ライフサイエンスコーパス: hindbrain

1 the inferior reticular nucleus (iRN, caudal hindbrain).

2 y and sensorimotor areas of the midbrain and hindbrain.

3 of neurons in optic tectum, cerebellum, and hindbrain.

4 o the correct dorsoventral column within the hindbrain.

5 vglut3 is restricted to the hypothalamus and hindbrain.

6 Galphat-S-ir reticular neurons in the caudal hindbrain.

7 strocaudal repositioning of these neurons in hindbrain.

8 ory dynamics of a neuronal population in the hindbrain.

9 FP, is crucial for CA guidance in the mouse hindbrain.

10 ogic deposition is largely restricted to the hindbrain.

11 , and sciatic nerve, as well as in embryonic hindbrain.

12 of facial branchiomotor (FBM) neurons in the hindbrain.

13 junctional complexes (AJCs) in the zebrafish hindbrain.

14 mere boundaries and proper patterning of the hindbrain.

15 s primarily residing in the hypothalamus and hindbrain.

16 een the sensory ganglion cell bodies and the hindbrain.

17 d abundant commissural axons crossing in the hindbrain.

18 c protease-activated receptors (PAR1) in the hindbrain.

19 te regulators of cell differentiation in the hindbrain.

20 edial and dorsal octavolateral nuclei in the hindbrain.

21 located at the junction of the midbrain and hindbrain.

22 edial longitudinal fasciculus (nMLF) and the hindbrain.

23 project to a specific nucleus in the dorsal hindbrain.

24 urons and extra-cerebellar nuclei in ventral hindbrain.

25 orts the major evolutionary stability of the hindbrain.

26 ystem, but are most common in the paediatric hindbrain.

27 to the laterodorsal tegmental nucleus in the hindbrain.

28 encephalon, diencephalon, mesencephalon, and hindbrain.

29 een paid to the expression of polySia in the hindbrain.

30 bolism, largely by actions on regions of the hindbrain.

31 tworks spanning the forebrain, midbrain, and hindbrain.

32 tion of orosensory, gustatory centers in the hindbrain.

33 semicircularis and periaqueductal gray, and hindbrain.

34 radoxically increased activity in the caudal hindbrain.

35 anges induced by administration of exogenous hindbrain 5-HT are dependent on central GLP-1 receptor s

36 In the electrosensory hindbrain, a corollary discharge that signals the timing

37 pression is higher in the forebrain than the hindbrain across the life span of the Tg mice, suggestin

38 otes (within the hypothalamus, midbrain, and hindbrain), although the A15 population was entirely abs

39 e fourth segment, or rhombomere (r4), of the hindbrain and a group of caudal efferent neurons (CENs)

40 t originating at the mid-line of the rostral hindbrain and are usually the result of that event propa

41 disorder characterized by a distinctive mid-hindbrain and cerebellar malformation, oculomotor apraxi

42 l-like receptor (TLR) 2 up-regulation in the hindbrain and cerebellum as a response to dying oligoden

43 forebrain, they were increased 40 -: 75% in hindbrain and cerebellum, and these patterns paralleled

44 s (150-450 Hz) were locally processed in the hindbrain and elicited motor behaviors.

45 border, and tension at boundaries within the hindbrain and forebrain.

46 in a district dorsomedial position along the hindbrain and gives rise to the inferior olive nuclei, d

47 cular thalamic nucleus (PVT) neurons receive hindbrain and hypothalamic inputs, and project to forebr

48 ocular mesenchyme led to defects in mid- and hindbrain and in craniofacial development, but was insuf

49 by attenuating stress-induced activation of hindbrain and limbic forebrain neurons.

50 focused on feeding circuits residing in the hindbrain and midbrain that govern homeostatic or hedoni

51 rs are ancient paralogues, which diverged in hindbrain and NC activities.

52 segmental reporter expression in the lamprey hindbrain and require the same transcriptional inputs (f

53 ow HS2ST interacts with other signals in the hindbrain and show that fibroblast growth factor (FGF) s

54 ects in the formation of the fore-, mid- and hindbrain and somites of E- embryos at 24 hpf.

55 and its border, and subsequently in the mid/hindbrain and somites.

56 At the head-trunk transition, hindbrain and spinal cord alignment to occipital and ver

57 ed features of Robo3 expression in midbrain, hindbrain and spinal cord commissural circuits, which to

58 lectively expressed in p1 progenitors of the hindbrain and spinal cord in the frog embryo, and that a

59 re we show that Nos1(PVH) neurons project to hindbrain and spinal cord regions important for food int

60 ammals, midline crossing at the level of the hindbrain and spinal cord requires the Robo3 receptor wh

61 ortex connect through descending pathways to hindbrain and spinal cord to activate muscle and generat

62 tributing to the control of infection of the hindbrain and spinal cord, possibly by regulating cell d

63 d that motor neurons shifted dorsally in the hindbrain and spinal cord, suggesting that Netrin-1/DCC

64 V2a interneurons are located in the hindbrain and spinal cord, where they provide rhythmic i

65 led that these neurons project mostly to the hindbrain and spinal cord.

66 pacemaker neurons on one side of the tadpole hindbrain and spinal cord.

67 ain how equivalent p3 progenitors within the hindbrain and the spinal cord produce functionally disti

68 o the middle reticular nucleus (mRN, rostral hindbrain) and the inferior reticular nucleus (iRN, caud

69 not activate GLP-1-producing neurons in the hindbrain, and liraglutide-dependent body weight reducti

70 ing the ventricles from the forebrain to the hindbrain, and observed more abundant staining in gravid

71 expression of zswim6 and onset of midbrain, hindbrain, and retinal expression at 48 hpf.

72 h larvae, which project to the hypothalamus, hindbrain, and spinal cord, including regions that expre

73 preoptic area (POA), hypothalamus, midbrain, hindbrain, and spinal cord.

74 ucleus, the torus semicircularis, the medial hindbrain, and the thalamus, and the flow of information

75 he gene mutated in Rett syndrome, within the hindbrain are critical for control of normal breathing.

76 ex may depend on the action of leptin in the hindbrain areas involved in the respiratory control such

77 Here we identify the mouse embryo hindbrain as a powerful model to study embryonic neuroge

78 in gastric motility are dependent on intact hindbrain astrocytes.

79 ssion appears in the most caudal part of the hindbrain at fetal stages, where it is maintained until

80 entary expression of CNP was detected in the hindbrain at the entry zones of sensory afferents.

81 single cell RNA sequencing of the zebrafish hindbrain at three different stages of patterning.

82 ble 5-HT-ir innervation, as did thalamic and hindbrain auditory and lateral line areas and vocal-acou

83 a common source for innervation of both the hindbrain auditory efferent nucleus and saccule, the mai

84 DA fibers innervating the inner ear and the hindbrain auditory efferent nucleus in the plainfin mids

85 is associated with a lengthening of incoming hindbrain axons to form delay lines, allowing for fine t

86 bsequently delaminate and migrate toward the hindbrain before completing differentiation.

87 nd CENs then migrate posteriorly through the hindbrain between 18 and 48 hrs postfertilization, along

88 siently during embryogenesis at the midbrain-hindbrain border, an area that gives rise to a variety o

89 genes homologous to those directing midbrain-hindbrain boundary (MHB) formation in vertebrates and th

90 e formation of the highly conserved midbrain-hindbrain boundary (MHB).

91 eepest constriction of the MHB, the midbrain-hindbrain boundary constriction (MHBC), and are critical

92 factor 10 (sox10) expression in the midbrain-hindbrain boundary, spinal cord and dorsal root ganglia.

93 y the choroid plexus (ChP) of the developing hindbrain, but not the telencephalon, in both mouse and

94 hemorrhages throughout the fore-, mid-, and hindbrain by E11.5.

95 We develop methods for stable hindbrain calcium imaging in free-moving mice, which sho

96 Hindbrain catecholamine (CA) neurons are essential for e

97 Hindbrain catecholamine neurons distribute glycemia-rela

98 ycemia-related sensory information activates hindbrain catecholaminergic neurons in a rate-dependent

99 r signals underlying early forebrain and mid/hindbrain cell differentiation from human embryonic stem

100 ion commands, a transformation achieved by a hindbrain cell group termed the velocity-to-position neu

101 ree that bifurcates between cortical and mid/hindbrain cell types.

102 e laterodorsal tegmental nucleus (LDTg) is a hindbrain cholinergic cell group thought to be involved

103 ular heterogeneity between telencephalic and hindbrain choroid plexi contributes to region-specific,

104 cle (telencephalic) versus fourth ventricle (hindbrain) choroid plexus.

105 Finally, laser ablations of hindbrain circuitry confirmed that visual and mechanosen

106 cuitry to include two non-overlapping PVH to hindbrain circuits.

107 lear afferents to the cochlear nuclei in the hindbrain, consistent with known expression of the Wnt r

108 Hindbrain cranial motor neurons are organized into discr

109 In contrast, in the hindbrain, DA terminals form traditional synaptic contac

110 er dysfunction results from rostral, and not hindbrain damage, indicating that rostral brain inputs a

111 that, before neural crest cell exit from the hindbrain, DAN is expressed in the mesoderm, and then it

112 e1-morphant zebrafish displayed midbrain and hindbrain degeneration, modeling PCH-like structural def

113 vealed correlations between the midbrain and hindbrain, demonstrating the utility of measuring intrin

114 MB via MYCN overexpression in primary human hindbrain-derived neuroepithelial stem (hbNES) cells or

115 ulloblastoma tumor suppressor that regulates hindbrain development and restricts the competence of ce

116 Here we show that during zebrafish hindbrain development dicer expression levels are contro

117 e 4 (egr4) as a novel regulator of posterior hindbrain development in Xenopus.

118 ryonic phenotypes including delayed mid- and hindbrain development, disruptions in motor behaviors th

119 for genes involved in anthropometric traits, hindbrain development, vascular and neurodegenerative di

120 can arise from cellular malfunctions during hindbrain development.

121 gliogenic progenitors involved in embryonal hindbrain development.

122 esent totally unrelated pathways involved in hindbrain development: neural migration and DNA transles

123 The vertebrate hindbrain develops as a series of well-defined neuroepit

124 that the mechanisms OENs use to navigate the hindbrain differ significantly from those employed by FB

125 zebrafish led to macrocephaly and posterior hindbrain displacement reminiscent of CM1.

126 Hindbrain dorsal interneurons (HDIs) are implicated in r

127 dely distributed throughout the midbrain and hindbrain early during larval development but very weakl

128 signalling, with defects extending into the hindbrain (en2 and egr2 expression reduced).

129 Motor centers in the hindbrain encode motor variables that are precisely tune

130 In characterizing a lamprey hoxalpha2 NC/hindbrain enhancer, we identify essential Meis, Pbx, and

131 CpG island methylator phenotype-positive hindbrain ependymomas are responsive to clinical drugs t

132 ocal copy number aberrations, poor-prognosis hindbrain ependymomas exhibit a CpG island methylator ph

133 hole-genome and whole-exome sequencing of 47 hindbrain ependymomas reveals an extremely low mutation

134 glutamate-dependent and required descending hindbrain excitation, similar to but preceding swimming,

135 In addition, exposure of cultured hindbrain explants to ectopic Netrin1 caused attractive

136 er analysis of data from the whole brain and hindbrain faithfully reflected the evolutionary history

137 red to attract pre-crossing axons toward the hindbrain floor plate, but is active in post-crossing gu

138 opulation at the extreme lateral edge of the hindbrain for which we were not able to identify axons.

139 r, our data support the view that excitatory hindbrain-forebrain projections are necessary for cispla

140 in rats and reveal the necessity of specific hindbrain-forebrain projections for cisplatin-induced an

141 d corticosterone (0.5 mug) directly into the hindbrain fourth ventricle.

142 t goldfish, we hypothesize that a permanent "hindbrain framework" may be a general property that is r

143 Hindbrains genetically defective in SVP formation owing

144 ular vascular plexus (SVP) extends through a hindbrain germinal zone populated by NPCs whose peak mit

145 Since hindbrain glucagon-like peptide-1 (GLP-1) neurons and no

146 Together, this data demonstrates a role for hindbrain GR signalling in regulating stress-related beh

147 novel mouse model that specifically ablates hindbrain GR to ascertain its role in behaviour, HPA-axi

148 enal syndrome, is critical for Shh-dependent hindbrain growth and development.

149 a premature decline in both NPC activity and hindbrain growth downstream of precocious cell cycle exi

150 forebrain specification and restricting mid-hindbrain growth.

151 ed by Fos immunoreactivity) in subregions of hindbrain gustatory nuclei were restored if the posterio

152 s with ROC thresholds for both forebrain and hindbrain had high negative and positive predictive valu

153 the forebrain, however in recent years, the hindbrain has become a region of interest for research i

154 neurons located in the raphe nucleus of the hindbrain have crucial roles in regulating brain functio

155 MRP in embryonic development of the auditory hindbrain have not been identified.

156 Hindbrain herniation measured on brain magnetic resonanc

157 This encompasses hindbrain herniation, ventriculomegaly, posterior fossa

158 pathways, and those expressed in the retina, hindbrain, hypothalamus, and pituitary.

159 We define network connectivity in the hindbrain important for the lateralized escape behavior

160 ) ependymomas are lethal malignancies of the hindbrain in infants and toddlers.

161 s throughout the diencephalon, midbrain, and hindbrain, in addition to their modulatory inputs from t

162 s widely expressed from the forebrain to the hindbrain, including the hippocampus, the mediobasal hyp

163 otor nucleus, then in caudal portions of the hindbrain, including the locus coeruleus, and much later

164 S-ir cells were observed in the midbrain and hindbrain, increasing the known populations.

165 In contrast, hindbrain infection resulted in localised disease, limit

166 Activity in a hindbrain inhibitory microcircuit is sufficient to modul

167 Hindbrain injection of a PKA inhibitor, KT5720, signific

168 ns during the partitioning of the developing hindbrain into lineage-restricted units called rhombomer

169 ions suggest that proper partitioning of the hindbrain into transient, genetically-defined segments c

170 rest (VNC) cells that emerge from the caudal hindbrain, invade the foregut and populate the gastroint

171 directly impact gastric control because the hindbrain is also the location of the vago-vagal reflex

172 f Hox gene expression to segmentation of the hindbrain is an ancient trait with origin at the base of

173 Segmentation of the vertebrate hindbrain is an evolutionarily conserved process.

174 neuron that leads FBMN migration through the hindbrain is an r4-derived FBMN/REN or an r5-derived CEN

175 scending input it receives from the mid- and hindbrain, its widespread projections to the forebrain a

176 m the so-called isthmic organizer at the mid/hindbrain junction; however, the underlying mechanism is

177 In NHP and human midbrain and hindbrain, Klb + cells are quite rare, as is expression

178 sed in cells of the hypothalamus and in some hindbrain lateral reticular neurons, and PSST5 in cells

179 occur via downstream orexin signaling to the hindbrain laterodorsal tegmental nucleus, thereby highli

180 Segmentation of the vertebrate hindbrain leads to the formation of rhombomeres, each wi

181 ial epigenetic mechanism balancing forebrain-hindbrain lineages and self-renewal-differentiation choi

182 ciliopathy characterized by a pathognomonic hindbrain malformation.

183 neous and characterized by a distinctive mid-hindbrain malformation.

184 ssed in two parallel somatotopically ordered hindbrain maps of the electrosensory lateral line lobe (

185 port suggested that astrocytes in the dorsal hindbrain may be the principal detectors of glucoprivic

186 gest that reduction of food intake following hindbrain MC4R activation is mediated by central vagal a

187 the nucleus tractus solitarius (NTS) form a hindbrain micro-circuit with preganglionic parasympathet

188 erebellar development, synapse assembly, and hindbrain morphogenesis, which substantially extended ou

189 tudies of sound producing fishes delineate a hindbrain network comprised of anatomically distinct com

190 ntal and functional ground plan for building hindbrain networks.

191 ulomotor system contains a simple example, a hindbrain neural circuit that takes velocity signals as

192 t streams emerge from the interaction of the hindbrain neural crest and the neighboring epibranchial

193 ression of sensory inputs onto an identified hindbrain neuron and provide evidence that Hip14 palmito

194 These findings reveal a novel hypothalamic-hindbrain neuronal circuit for sleep/wake control.

195 ng is a vital rhythmic behavior generated by hindbrain neuronal circuitry, including the preBotzinger

196 approach to stimulate select populations of hindbrain neurons and characterize how they modulate fre

197 PMV is therefore required for activation of hindbrain neurons and the full CRR during slow-onset hyp

198 role of glutamatergic GGC peptide-secreting hindbrain neurons in regulating metabolic homeostasis ha

199 ocks the ability of acute stress to activate hindbrain neurons that are immunoreactive for either pro

200 ntified groups of commissural reticulospinal hindbrain neurons.

201 ing or silencing Phox2b- or Atoh1-expressing hindbrain neurons.

202 Hindbrain NMDA receptors play important roles in reflexi

203 Using tegmental hindbrain nuclei neurons in zebrafish embryos together w

204 ance towards forebrain targets and away from hindbrain nuclei.

205 smatic nucleus (SCh) and select midbrain and hindbrain nuclei.

206 s in the nucleus tractus solitarius (NTS), a hindbrain nucleus critical for energy balance control.

207 s in the nucleus tractus solitarius (NTS), a hindbrain nucleus that projects monosynaptically to the

208 As pharmacological studies highlight the hindbrain nucleus tractus solitarius (NTS) as a brain re

209 Here, we show that the hindbrain nucleus tractus solitarius (NTS) is essential

210 significant (P < .05) neuropathology in the hindbrain of 24-week-old mice was quantifiable.

211 toneuronal segmental patterns persist in the hindbrain of adult goldfish, we hypothesize that a perma

212 tal imprint in the reticular and motor basal hindbrain of adult goldfish.

213 r, viral entry increased specifically in the hindbrain of IFNAR-deficient mice, suggesting that IFNAR

214 authner cells are the largest neurons in the hindbrain of teleost fish and most amphibians.

215 aging of whole neural network throughout the hindbrain of the larval zebrafish at a matured stage whe

216 the midbrain and hindbrain or remain in the hindbrain only, and the type of loop determines the dura

217 t can either encompass both the midbrain and hindbrain or remain in the hindbrain only, and the type

218 ls, yet was independent of vascular roles in hindbrain oxygenation.

219 We evaluated the contributions of the hindbrain parabrachial nucleus (PBN) to systemic Ex4-ind

220 ighlighting a novel hippocampus-hypothalamus-hindbrain pathway regulating meal size control.

221 +/-:Raldh2+/-; axon phenotypes are seen when hindbrain patterning and CN V gross morphology is altere

222 Here, we show that Ddx3x regulates hindbrain patterning and development by controlling Hox

223 in register with altered anterior-posterior hindbrain patterning and gross morphological disruption

224 s a noisy gradient during critical stages of hindbrain patterning and that cells use distinct intrace

225 RA activity in the neuroepithelium regulates hindbrain patterning directly and territory size specifi

226 These studies suggest that disruption of hindbrain patterning genes can alter monoamine system de

227 identify Ddx3x as essential for hindbrain patterning, cell fate determination, and as a

228 with the well-established function of RA in hindbrain patterning.

229 Hoxd3), normally required for early rostral hindbrain patterning.

230 Key to this process is a self-oscillating hindbrain population (HBO) that acts as a pacemaker for

231 so in contrast with spinal cord, analysis of hindbrain post-crossing axons in Robo1/2 mutant embryos

232 -Robo repulsive signaling, remains active in hindbrain post-crossing commissural axons to guide longi

233 ce the CSF flows along the apical surface of hindbrain progenitors not expressing Wnt5a, we examined

234 mate signaling in the amygdala, arising from hindbrain projections, is required for the full expressi

235 hypothalamic circuitry that engages distinct hindbrain regions and is innervated by discrete upstream

236 nflammation and was not seen in midbrain and hindbrain regions associated with bladder filling and vo

237 To study the selective innervation of hindbrain regions by sensory afferents in the zebrafish

238 c spatiotemporal expression in embryonic mid-hindbrain regions where monoamine neurons emerge.

239 ion, though the role of GR signalling in the hindbrain remains poorly characterised.

240 he feeding circuitry in the hypothalamus and hindbrain remains unclear.

241 study highlights that ablation of GR in the hindbrain results in excessive barbering, obsessive comp

242 Barrington's nucleus, Kolliker-Fuse nucleus, hindbrain reticular formation, and rostral NTS.

243 osition along the anterior-posterior axis of hindbrain rhombomere 8 determines expression of hox5 gen

244 the classical model, oculomotor circuits in hindbrain rhombomeres 5-6 develop and function independe

245 se measures by comparison of intravenous and hindbrain routes of Mycobacterium marinum infection, whi

246 map VPNI neural activity in zebrafish onto a hindbrain scaffold consisting of alternating excitatory

247 Examination of rat hindbrain sections revealed that neuronal processes expr

248 rved gene regulatory network that integrates hindbrain segmentation with segmentally restricted domai

249 ssion has not been coupled to the process of hindbrain segmentation.

250 expression in lamprey or its relationship to hindbrain segmentation.

251 We focus on the formation of hindbrain segments (rhombomeres) in the developing zebra

252 dulation of this noise affects patterning of hindbrain segments (rhombomeres) in the zebrafish embryo

253 a reveal known and novel markers of distinct hindbrain segments, of cell types along the dorsoventral

254 Peptide 1 (GLP-1)-expressing neurons in the hindbrain send robust projections to the paraventricular

255 the developing mouse and chick neural tube, hindbrain serotonergic neurons and spinal glutamatergic

256 Our results in the hindbrain show that the major midline attractant, Netrin

257 h the loss of RA, including the reduction in hindbrain size and the loss of posterior rhombomeres.

258 with xVGlut1 than xVIAAT, especially in the hindbrain, spinal cord, and cranial nerves.

259 nd subpallium), diencephalon, mesencephalon, hindbrain, spinal cord, and retina.

260 anoids resembling the cerebral cortex or the hindbrain/spinal cord and assemble them with human skele

261 ortical development, inferring a neocortical-hindbrain split in early progenitor cells and the key ge

262 ep via the serotonergic raphe nuclei (RN), a hindbrain structure that is critical for sleep in both d

263 The cerebellum is a large hindbrain structure that is increasingly recognized for

264 ne expression indicate a functional role for hindbrain structures during bower building.

265 es, including the ventral tegmental area and hindbrain structures such as the locus coeruleus and par

266 enotype (Tor1a(DeltaE/+)) selectively within hindbrain structures will produce an overtly dystonic an

267 els did not differ between the forebrain and hindbrain, suggesting that other factors modulating the

268 Importantly, RA specifies and patterns the hindbrain territory by antagonizing the activity of the

269 ion relative to mesodermal structures of the hindbrain territory.

270 the isoform 1 transgene was more abundant in hindbrain than forebrain and midbrain.

271 on of central GLP-1 receptors rostral to the hindbrain that are involved in the LiCl-mediated suppres

272 ts from, among others, areas of the mid- and hindbrain that are known to be involved in wake-sleep co

273 g regions of the thalamus, hypothalamus, and hindbrain that are much more extensive than those from t

274 tify three neuronal clusters in the anterior hindbrain that are well suited to execute the underlying

275 bellum is a prominent part of the vertebrate hindbrain that is critically involved in the regulation

276 epithelium to the first central site in the hindbrain, the nucleus of the lateral descending trigemi

277 The cardiac neural crest arises in the hindbrain, then migrates to the heart and contributes to

278 m that does not use hingepoints to shape its hindbrain, thereby enabling a direct assessment of the r

279 ne and neuropeptide, acts principally in the hindbrain to decrease food intake and has recently been

280 ates Mafb and Krox20 expression in the mouse hindbrain to specify r5, we show that in Xenopus this pr

281 New interneurons are added in the hindbrain to support more complex movements as young zeb

282 udy we investigated the projections from the hindbrain to the midbrain in more detail, using tracer i

283 was malformed and compressed into the lower hindbrain together with the hypothalamus midbrain and po

284 e growth of medulloblastoma, a Shh-dependent hindbrain tumor.

285 ined the development of spinal pathways from hindbrain V2a neurons and the role of these pathways in

286 Using a zebrafish hindbrain ventricle infection model, we demonstrate that

287 ne knockdown of zebrafish samhd1, we observe hindbrain ventricular swelling and brain hemorrhage.

288 etected by taste buds and transmitted to the hindbrain via sensory afferent neurons.

289 ferentiated larynx at the periphery, and the hindbrain vocal central pattern generator (CPG) centrall

290 The frog hindbrain vocal circuit contains a previously unexplored

291 hants replicated the human phenotype showing hindbrain volume loss.

292 We find that hindbrain volume varies significantly between species th

293 the role of glucocorticoid signalling in the hindbrain we have developed a novel mouse model that spe

294 Within the hindbrain, we have described how intrinsic membrane prop

295 throughout the brain, including the mid- and hindbrain, where they activate premotor circuits involve

296 The cerebellum is a crucial structure of hindbrain which helps in maintaining motor tone, posture

297 induction of boundary cells in the zebrafish hindbrain, which we find both require kinase-dependent s

298 many nuclei from the olfactory bulbs to the hindbrain, while vglut3 is restricted to the hypothalamu

299 ptic tectum) and motor areas (cerebellum and hindbrain), with similar visual area retinotopic maps of

300 and miR-9 biogenesis across the ventricular hindbrain zone, resulting in an increase of both prolife