ライフサイエンスコーパス: hindlimb

1 the trunk body wall not associated with the hindlimb.

2 on and three-dimensional articulation of the hindlimb.

3 ting acute and chronic regeneration in a rat hindlimb.

4 are differentially modified in forelimb and hindlimb.

5 entivectors in the skeletal muscle of murine hindlimb.

6 nd neovascularization in the murine ischemic hindlimb.

7 echanisms that change proportions within the hindlimb.

8 the recovered proprioceptive function of the hindlimb.

9 k as monitored by five muscle nerves in each hindlimb.

10 flow, and functional recovery in an ischemic hindlimb.

11 Euparkeria implies, however, a more abducted hindlimb.

12 ced a variety of functional movements in the hindlimb.

13 d for FMD and arteriogenesis in the ischemic hindlimb.

14 sing spinal MU input in the rat forelimb and hindlimb.

15 absence of a variable distal portion of the hindlimbs.

16 nic proteins in ischemic AMPKalpha2(DeltaMC) hindlimbs.

17 of the flight muscles and enlargement of the hindlimbs.

18 , followed by stepping-like movements in the hindlimbs.

19 is manifested, leading to a hopping gait in hindlimbs.

20 abnormal involuntary muscle contractions on hindlimbs.

21 pharyngeal mesoderm, peripheral neurons, and hindlimbs.

22 elopment of a fusiform body and reduction of hindlimbs [8-11], but the rarity of Oligocene whale skel

23 Mice underwent hindlimb Achilles' tendon transection and dorsal burn in

24 rons that project to L5 selectively disrupts hindlimb alternation allowing a continuum of walking to

25 These data illustrate that hindlimb alternation can be manipulated independently fr

26 Both hindlimb and dorsal muscles were studied at 7, 14 and 21

27 e consistently observed a protrusion between hindlimb and forelimb representation, which in rats corr

28 athway regulates skeletal development of the hindlimb and lower jaw through discrete populations of c

29 net total uptake of amino acids by the fetal hindlimb and lower skeletal muscle protein synthesis rat

30 The new specimen includes a partial right hindlimb and remiges from an adult or subadult bird.

31 EB, an enhancer of Tbx4, produces defects in hindlimbs and genitalia, establishing the importance of

32 fluorescent L4 DRG neurons, innervating the hindlimbs and lower back, were not significantly dimorph

33 d functional survival of AD-MSCs in ischemic hindlimbs and provoked a synergetic effect with AD-MSCs

34 echanism when the animal was standing on its hindlimbs and which was partially dependent on the endog

35 ifferentially expressed between forelimb and hindlimb, and across different stages.

36 l and derived osteological characters in the hindlimb, and might suggest a moderately adducted postur

37 rated (predictable) sensory signals from the hindlimbs are likely to occur.

38 ctions of RFA, CFA, and the caudally located hindlimb area (CHA), which is a part of M1, were determi

39 described, besides few reports of a rostral hindlimb area.

40 me3) analyses on its developing forelimb and hindlimb autopods at sequential embryonic stages to deci

41 otein synthesis rates that match the reduced hindlimb blood flow and oxygen consumption rates in IUGR

42 muscle protein synthesis rates match reduced hindlimb blood flow and oxygen consumption rates in the

43 Simultaneous bilateral EDL force and hindlimb blood flow measurements were made during electr

44 These adaptations resulted in hindlimb blood flow rates in IUGR that were similar to c

45 Absolute hindlimb blood flow was reduced in IUGR (IUGR: 32.9 +/-

46 gen and nutrient supply to the fetus affects hindlimb blood flow, substrate uptake and protein accret

47 a synergetic effect with AD-MSCs to restore hindlimb blood perfusion and limb functions.

48 in pythons, and HOXD gene expression in the hindlimb buds progresses to the distal phase, forming an

49 Python hindlimb buds then develop transitory pre-chondrogenic c

50 ranscription is weak and transient in python hindlimb buds, leading to early termination of a genetic

51 the ancestral enhancer drives expression in hindlimbs but not forelimbs, in locations that have been

52 by bilateral mechanical hypersensitivity of hindlimbs, but corpus callosotomy eliminated the analges

53 impaired blood flow recovery, foot movement, hindlimb capillary density, vessel diameter, and vascula

54 noticeable tremor, and most animals showed a hindlimb clasping phenotype.

55 Hindlimb CMAP and MUNE demonstrated strong correlations

56 cally transformed mice and the disruption of hindlimb coordination following ablation of descending L

57 We explored this in the somatosensory hindlimb cortex at an age when animals begin to use thei

58 forepaw sensory cortex into the deafferented hindlimb cortex, associated with sprouting of corticospi

59 erwent massive changes after injury and that hindlimb cortical areas were recruited to control the fo

60 Fore-hindlimb coupling favored a more stable diagonal couplin

61 deletion of the SAM domain induces a complex hindlimb defect associated with down-regulation of Trp63

62 Here we report that hindlimb development arrests in python embryos as a resu

63 nheritance; implicating 13 genes involved in hindlimb development in bilateral cases and 11 in unilat

64 d SMARCA4, known to play a role in embryonic hindlimb development.

65 acceptance of secondary skin grafts from the hindlimb donor strain and rejection of skin grafts from

66 during spontaneous feeding and the impaired hindlimb during locomotion were both significantly great

67 the right forelimb during feeding and right hindlimb during locomotion.

68 raising the possibility that re-emergence of hindlimbs during snake evolution did not require de novo

69 ence for a successive loss of forelimbs then hindlimbs during snake evolution.

70 limb enhancer played a role in reduction of hindlimbs during snake evolution.

71 unctuated evolution of allometric scaling of hindlimb elements during the radiation of Dipodoidea.

72 demonstrate that the HLEB of snakes has lost hindlimb enhancer function while retaining genital activ

73 ng scratching and behave like neurons in the hindlimb enlargement.

74 contralateral L5, which contains many of the hindlimb extensor MNs activated by the LVST.

75 nd tracing the pathway transynaptically from hindlimb extensor muscles using rabies virus (RV).

76 ats, these results show that the encoding of hindlimb features in motor cortex dynamics is comparable

77 g gastrulation, the forelimb, interlimb, and hindlimb fields are progressively generated and concomit

78 ter-connect these CPGs are thought to secure hindlimb-forelimb coordination, ensuring that diagonal l

79 therapy have the clear potential to protect hindlimb function from such adverse influence.

80 different aspects of recovered forelimb and hindlimb functions (i.e., stability, strength, coordinat

81 Hindlimb glucose uptake and lactate output rates were si

82 Net hindlimb glucose uptake and lactate output rates were si

83 ambs with IUGR at birth have higher rates of hindlimb glucose uptake, which may compensate for myocyt

84 placental insufficiency that develop to slow hindlimb growth and muscle protein accretion.

85 Metabolic adaptations to slow hindlimb growth are probably hormonally-mediated by mech

86 In IUGR and IUGR-AR lambs, hindlimb GURs were greater but fractional glucose oxidat

87 imb area (CFA), rostral forelimb area (RFA), hindlimb (HL) cortex (based on intracranial microstimula

88 The loss of V2b INs results in hindlimb hyperextension and a delay in the transition fr

89 ation of limb morphology and forelimb versus hindlimb identity.

90 improves skeletal muscle recovery following hindlimb immobilization.

91 ty electrical stimulation of the ipsilateral hindlimb in awake rats evoked field potentials in the C1

92 of blood vasculature in the mouse brain and hindlimb in the NIR-IIb window with short exposure time

93 for organogenesis of the lungs, pelvis, and hindlimbs in humans.

94 cing muscle damage and recovery in the lower hindlimbs in mice.

95 The development of hindlimbs in tetrapod species relies specifically on the

96 oral and pelvic fins in fishes and fore- and hindlimbs in tetrapods.

97 q palmitoylation in ischemic and nonischemic hindlimbs in vivo In summary, we demonstrate that NAC ac

98 Ischemia of the hindlimb induced expansion of clusters C5 and C8.

99 ythmic pattern is highly correlated with the hindlimb ipsilateral flexor activities.

100 capitulate this injury with a mouse model of hindlimb IR injury which leads to skeletal muscle fibros

101 animal irradiator to administer fractionated hindlimb irradiation to juvenile mice bearing implanted

102 In a mouse hindlimb ischaemia model, we examine the effects of hMSC

103 ike CD31(+)/c-Kit(+) cells in mice following hindlimb ischaemia.

104 In mice with unilateral hindlimb ischemia (40%-50% reduction in flow), ultrasoun

105 the susceptibility of the peripheral limb to hindlimb ischemia (HLI).

106 uscles of C57Bl/6J mice following unilateral hindlimb ischemia +/- the alpha-sialidase NA (neuraminid

107 These animals developed advanced hindlimb ischemia and digital autoamputation, secondary

108 njection, and iii) vascular flow recovery to hindlimb ischemia as indicated by laser Doppler and alph

109 These events can reverse hindlimb ischemia in mice for >24 hours and increase mus

110 essential for arteriogenesis in response to hindlimb ischemia in mice.

111 Here we found that hindlimb ischemia increases levels of resolvin D1 (RvD1)

112 When hindlimb ischemia is induced in diabetic mice and QKI-7

113 d flow recovery and neovessel formation in a hindlimb ischemia model compared with nondiabetic mice.

114 In a murine hindlimb ischemia model injection of ASCs with downregul

115 cell dose (5 x 10(4) cells) in a preclinical hindlimb ischemia model showing accelerated formation of

116 In a murine hindlimb ischemia model, TUDCA-treated MSC transplantati

117 Using a surgical murine hindlimb ischemia model, we first discovered a significa

118 ManN also promotes angiogenesis in a mouse hindlimb ischemia model, with accelerated limb blood flo

119 blood vessel perfusion function in a murine hindlimb ischemia model.

120 In this study, In vivo experiments with hindlimb ischemia models revealed that XBP1 deficiency i

121 ge numbers of newly formed arterioles in the hindlimb ischemia mouse model.

122 Hindlimb ischemia, NA, and hindlimb ischemia plus NA increased the magnitude of (99

123 reperfusion after occlusion was removed) or hindlimb ischemia reperfusion injury (left leg tournique

124 implantation in mouse and porcine models of hindlimb ischemia rescues severely damaged tissues by th

125 Unilateral hindlimb ischemia was induced by ligating femoral artery

126 se 1) in mice impaired recovery from chronic hindlimb ischemia, a model of peripheral artery disease.

127 We utilized the murine model of hindlimb ischemia, and in vivo Matrigel plug assay toget

128 In a murine model of hindlimb ischemia, daily oral fenofibrate treatment rest

129 Hindlimb ischemia, NA, and hindlimb ischemia plus NA inc

130 idening of the arterial lumen in response to hindlimb ischemia, potentially via functional interactio

131 mps in diabetes using a novel in vivo murine hindlimb ischemia-amputation model.

132 was prepared from C57BL/6-mice subjected to hindlimb ischemia.

133 d in vivo angiogenic properties in mice with hindlimb ischemia.

134 the murine femoral artery ligation model of hindlimb ischemia.

135 d miR-155 as a downregulated microRNA during hindlimb ischemia.

136 ndent vasodilator pathways, and in mice with hindlimb ischemia.

137 od flow recovery and capillary density after hindlimb ischemia.

138 ecovery and decreases angiogenesis following hindlimb ischemia.

139 y potentiates blood flow recovery from mouse hindlimb ischemia.

140 d pigs were subjected to RIPC (4x5/5 minutes hindlimb ischemia/reperfusion) or placebo (PLA) before 6

141 Rats dosed with CK-2066260 showed increased hindlimb isometric and isokinetic force in response to s

142 hat control antagonistic muscles at a single hindlimb joint.

143 on responses in the hindlimb motor cortex to hindlimb kinematics and hindlimb muscle synergies across

144 cy, associated with altered EMG patterns and hindlimb kinematics during gait.

145 Hindlimb kinematics, SO fascicle and muscle-tendon unit

146 ous degrees of spontaneous motor recovery of hindlimb/leg function.

147 Morphological comparison showed that lower hindlimb length in the introduced populations tended to

148 Slower hindlimb linear growth and muscle protein synthesis rate

149 We demonstrate slower hindlimb linear growth and muscle protein synthesis rate

150 nical coupling, to partly recover unassisted hindlimb locomotion after complete spinal cord injury.

151 Four adult cats that recovered stable hindlimb locomotion after spinal transection were implan

152 Hindlimb locomotion was restored by reestablished integr

153 mals, including mice, rats and cats, recover hindlimb locomotion with treadmill training.

154 cats and primates, cortical contribution to hindlimb locomotor movements is not critical in rats.

155 ence that, despite its broad contribution to hindlimb mesenchyme and facial epithelium, the Isl1-beta

156 s that give rise to Shh-expressing posterior hindlimb mesenchyme and Fgf8-expressing mandibular epith

157 ls differentially to contralateral trunk and hindlimb MNs in the mammalian spinal cord.

158 ndothelial cells and neovessels de novo in a hindlimb model of ischaemia and resulted in a 50% increa

159 emia reperfusion and injured leg muscle in a hindlimb model of ischemia reperfusion.

160 onized with the spatiotemporal activation of hindlimb motoneurons.

161 in rats suggests that cortical engagement in hindlimb motor control may depend on the behavioral cont

162 temporally scaled activity occurs in the rat hindlimb motor cortex in the absence of motor output and

163 targeted deletion of FGFR1 and FGFR2 in the hindlimb motor cortex limits the formation of new synaps

164 whole-body kinematics, muscle synergies, and hindlimb motor cortex modulation in freely moving rats p

165 We found that the activation of hindlimb motor cortex preceded gait initiation.

166 ped the neuronal population responses in the hindlimb motor cortex to hindlimb kinematics and hindlim

167 ubsets of lumbar motor neurons: HGF supports hindlimb motor neurons through c-Met; CNTF supports subs

168 saphenous nerve block before, but not after, hindlimb movement blocked movement-induced BTP.

169 ays to record neural activity during passive hindlimb movement in 7 anesthetized cats.

170 ouse (lamb1t) exhibits intermittent dystonic hindlimb movements and postures when awake, and hyperext

171 ted axons in the implants and improvement in hindlimb movements.

172 We further show that, in ischaemic rabbit hindlimbs, MRTF-A as well as Tss4 induce functional neov

173 ion were implanted with electrodes to record hindlimb muscle activity chronically and to stimulate th

174 logical analyses revealed that Stac3-deleted hindlimb muscle contained more slow type-like fibers tha

175 o frequent electrostimulation, Stac3-deleted hindlimb muscle contracted but the maximal tension gener

176 nal discharges reliably evoke contra-lateral hindlimb muscle contractions.

177 sponses to electrically-induced intermittent hindlimb muscle contractions.

178 Rodents dosed with CK-2066260 show increased hindlimb muscle force and power in response to submaxima

179 ted against muscle fatigue and increased mdx hindlimb muscle force by 40%, a value comparable to curr

180 le-derived stem cells under the epimysium of hindlimb muscle in mice leads to the de novo formation o

181 antibody (GSK577548) significantly improves hindlimb muscle innervation at 90 days, a late symptomat

182 ty using sarcolemmal membranes isolated from hindlimb muscle of control (CON, n = 11-12) and IUGR (n

183 thetic fibres, as well as cardiac and either hindlimb muscle or lumbar sympathetic nerves.

184 ng pairs of sympathetic nerves: forelimb and hindlimb muscle sympathetic fibres, as well as cardiac a

185 limb motor cortex to hindlimb kinematics and hindlimb muscle synergies across a spectrum of natural l

186 adherin/CD31+/CD45-) isolated from uninjured hindlimb muscle tissue undergo in vivo EndMT when transp

187 ite, both functionally similar (forelimb and hindlimb muscle) and functionally dissimilar (lumbar and

188 unctions in skeletal muscle, at least in the hindlimb muscle.

189 etermined the contractility of Stac3-deleted hindlimb muscle.

190 ct4-deficient perivascular cells in ischemic hindlimb muscle.

191 0 in fibres of the entire posterior group of hindlimb muscles (gastrocnemius, soleus, and plantaris)

192 Likewise, the hindlimb muscles of rats with ligated femoral arteries s

193 In EphA7(-/-) mice, hindlimb muscles possess fewer myofibers at birth, and t

194 Primary myoblasts were isolated from hindlimb muscles, and after 3 days in growth media (20%

195 rological impairment, delayed denervation of hindlimb muscles, and prolonged survival of spinal motor

196 nsory evoked potentials; brain, spinal cord, hindlimb muscles, bladder and rectum histology and/or im

197 s area control movements involving trunk and hindlimb muscles, those in the intermediate part control

198 tivity deriving from hindpaw inflammation or hindlimb nerve injury.

199 e revealed the emergence within the ischemic hindlimb of a small subset of YFP(+) CD144(+) CD11b(-) f

200 n- and cartilage-like tissues from the lower hindlimb of Early Cretaceous Confuciusornis.

201 scular injection of alpha-syn fibrils in the hindlimb of M83(+/-) mice leads to progressive alpha-syn

202 Unilateral exposure of the proximal hindlimb of mice (with or without ischemia produced by i

203 The hindlimb of theropod dinosaurs changed appreciably in th

204 egration of both the heads and the fore- and hindlimbs of abnormal cyclopic trisomy 18 and anencephal

205 of praying mantids (Mantidae), the elongated hindlimbs of grasshoppers (Orthoptera: Caelifera), and t

206 into the anterior tibial compartment of the hindlimbs of NOD-Rag1(null) IL2rgamma(null) immunodefici

207 mechanical hypersensitivity develops in the hindlimbs of rats in parallel with a reduction in all co

208 left-right asymmetry in the forelimbs and/or hindlimbs of the abnormal cyclopic trisomy 18 and anence

209 encephalomyelitis mouse model with complete hindlimb paralysis and death by 30 d after induction of

210 symptomatic dy(2j)/dy(2j) mice with apparent hindlimb paralysis and muscle fibrosis.

211 06 in mice results in progressive ataxia and hindlimb paralysis associated with motor neuron degenera

212 their ablation using PLP-CreERT resulted in hindlimb paralysis with immobility at approximately 30 d

213 nn cells displayed tremor that progressed to hindlimb paralysis, which correlated with diminished num

214 extended median survival by 50% and delayed hindlimb paralysis, with animals remaining ambulatory un

215 disease severity, resulting in recovery from hindlimb paralysis.

216 e decline of motor function and the onset of hindlimb paralysis.

217 ection from ischemia-induced neuron loss and hindlimb paralysis.

218 e driver line led to motor impairment due to hindlimb paresis.

219 reaching/grasping, spontaneous limb use, and hindlimb placement during walking.

220 g, by damage in voxels encompassing CFA/RFA; hindlimb placement, by damage in HL; and spontaneous for

221 ing, damage medial to HL reduced recovery of hindlimb placing, and damage lateral to CFA reduced reco

222 Confuciusornis may indicate a more crouched hindlimb posture.

223 of a hinge-like ankle joint and a more erect hindlimb posture.

224 fusion, between-limb proportions, and within-hindlimb proportions all evolved independently of one an

225 ed the crocodylian-like ankle morphology and hindlimb proportions of stem archosaurs and early pseudo

226 In contrast, the hindlimbs provide relatively weak thrust in all simulati

227 ug of GsMTx4 into the arterial supply of the hindlimb reduced the peak pressor (control: 24 +/- 2, Gs

228 ug of GsMTx4 into the arterial supply of the hindlimb reduced the peak pressor (control: 24 +/- 5, Gs

229 Low-intensity ES at hindlimb regions drives the vagal-adrenal axis, producin

230 elopmental mechanisms, and elongation of the hindlimb relative to the forelimb is not simply due to g

231 Furthermore, hindlimb remote ischemic preconditioning induced MCUb ex

232 6 mice were subjected to S/R with or without hindlimb RIC.

233 viable molecular mechanisms for diversity in hindlimb scale and feather distribution.

234 To address this issue, we use turtle hindlimb scratching as a model for fine motor control, s

235 ed in the left hindlimb, such that the right hindlimb serves as an internal control.

236 Contrast ultrasound perfusion imaging of hindlimb skeletal muscle and femoral artery diameter mea

237 rotein expression significantly increased in hindlimb skeletal muscle in the mSOD1(G93A) mouse model

238 We also conducted a GWAS on hindlimb skeletal muscle mass of 1,867 mice from an adva

239 reduced, and ex vivo ATP levels are lower in hindlimb skeletal muscle of the IUGR fetus.

240 ction (topical glibenclamide superfused onto hindlimb skeletal muscle) resolved a decreased blood flo

241 ke acutely influences signal transduction in hindlimb skeletal muscle.

242 well as reduced parasite burden in heart and hindlimb skeletal muscle.

243 mal membranes isolated from control and IUGR hindlimb skeletal muscle.

244 The hindlimb skeletal muscles were exposed to ultrasound fro

245 igin and therefore evoked by transmission in hindlimb SOCPs.

246 , loss of brain neurons; lumbar CSF leakage, hindlimb somatosensory-motor deficit with absence of mot

247 s sequentially from visual to barrel then to hindlimb somatosensory; the second principle is correlat

248 regulatory changes in the genes encoding the hindlimb-specific transcription factor Pitx1 and forelim

249 Here, we show that hindlimb standing and locomotion recover after spinal tr

250 Rats showed significant improvement in hindlimb stepping ability, quadrupedal weight support, a

251 surface of the spinal cord at L3-L7 induced hindlimb stepping-like movements on a moving treadmill b

252 plit-belt treadmill, with the left and right hindlimbs stepping at different speeds.

253 he sensory signal, some animals received the hindlimb stimulation only during phase 2.

254 h injury is specifically induced in the left hindlimb, such that the right hindlimb serves as an inte

255 flight and a publicly available data set for hindlimb suspension, a claimed surrogate model of microg

256 Hindlimb-targeted irradiation (3 x 8.2 Gy) of 4-week-old

257 models of ischemia in the heart, brain, and hindlimb that only in the brain does NADPH oxidase 4 (NO

258 bited shifts in investment from forelimbs to hindlimbs that were qualitatively similar to anatomical

259 Moreover, in the hindlimb there are only two muscle absence/presence diff

260 f altered regulation of CaV2.1 channels, the hindlimb tibialis anterior muscle in IM-AA mice exhibite

261 ETHODS AND When injected into mouse ischemic hindlimb tissue, CD34Exo, but not the CD34Exo-depleted c

262 trated progressive motor unit decline in the hindlimb to a greater extent than the forelimb.

263 ns result from a partial transformation from hindlimb to forelimb identity mediated by cis-regulatory

264 d to randomly occlude capillaries in the rat hindlimb to mimic the capillary rarefaction observed in

265 both a sciatic nerve cut-and-repair and rat hindlimb transplant model.

266 Preclinical swine hindlimb transplantation models have an important role i

267 of nerve regeneration after nerve injury and hindlimb transplantation.

268 Syngeneic and allogeneic orthotopic hindlimb transplantations were performed using male rats

269 Similarly, rats undergoing allogeneic hindlimb transplants treated with local injection of MSC

270 LEW) (n = 4) and allogeneic (BN-LEW) (n = 4) hindlimb transplants were performed and assessed for neu

271 ts received full-mismatched Brown Norway rat hindlimb transplants.

272 oved nerve regeneration following allogeneic hindlimb transplants.

273 is rat recipients of mismatched Brown Norway hindlimb transplants.

274 from the skin component of heterotopic swine hindlimb transplants.

275 from the skin component of heterotopic swine hindlimb transplants.

276 he tibialis anterior muscle and the onset of hindlimb tremor.

277 obot impedance control at the pelvis allowed hindlimb, trunk, and forelimb mechanical interactions.

278 selectively differentiate into forelimb- or hindlimb-type mesenchymes, depending on a concentration

279 r-Brown Norway rats at 10 months of age were hindlimb unloaded for a period of 2 weeks.

280 rmine if absence of MAT reduced bone loss in hindlimb-unloaded (HU) mice.

281 response of wild type (WT) and ob/ob mice to hindlimb unloading (HU).

282 chitectural and mechanical properties due to hindlimb unloading alone and simulated spaceflight.

283 e in UBR5 after recovery of muscle mass from hindlimb unloading in both adult and aged rats, as well

284 ated spaceflight, combining microgravity (by hindlimb unloading) and radiation exposure.

285 IR-II) fluorescence imaging, to image murine hindlimb vasculature and blood flow in an experimental m

286 In a rat hindlimb VCA model, local administration of this Treg-in

287 rapamycin (RPM) induced long-term orthotopic hindlimb VCA survival (BALB/c->C57BL/6), as did CTLA4Ig/

288 dies, which have emphasized the influence of hindlimb vs. forelimb lengths on sauropodomorph stance.

289 The soleus of the immobilized-reambulated hindlimb was found to have a greater amount of muscle da

290 The joint mobility of the hindlimb was quantified in 3D to address previous qualit

291 These mice display hindlimb weakness and impaired axonal conduction in scia

292 spinalized adult rats can recover unassisted hindlimb weight support and locomotion without explicit

293 bot rehabilitation that promotes recovery of hindlimb weight support functions on trunk motor cortex

294 1) ; P < 0.005), although flow normalized to hindlimb weight was similar between groups.

295 Hindlimb weight, linear growth rate, muscle protein accr

296 , P < 0.0001) was negatively associated with hindlimb weight.

297 orepinephrine was negatively associated with hindlimb weight.

298 restingly, CD34Exo, when treated to ischemic hindlimbs, were most efficiently internalized by endothe

299 menter-induced movement of the tumor-bearing hindlimb with a context produces conditioned place avoid

300 ion (T10), cats recovered stepping with both hindlimbs within 3 weeks.