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1 T9-L5 vertebrae and the ilium portion of the hip bones.
2 r without low BMD underwent lumbar spine and hip bone densitometry and a complete periodontal examina
3                     Lateral lumbar spine and hip bone density remained stable or improved in 65% and
4                                              Hip bone density was 1.06 percent higher in the calcium
5 ding influences of age, body mass index, and hip bone density were taken into account.
6 ed in a small but significant improvement in hip bone density, did not significantly reduce hip fract
7  AIDs or mild anemia and question the use of hip bone-derived cells as healthy experimental controls.
8                    Living inhabitants of the hip bone (e.g. osteocytes) are visible in their local ex
9  variation in bone mineral density (BMD) and hip bone geometry are associated with fracture risk.
10 iation between dietary patterns, measures of hip bone geometry, and subsequent fracture risk are scar
11 TATE was observed in the T9-L5 vertebrae and hip bones in all 17 patients compared with activity conc
12 men, PAD was associated with higher rates of hip bone loss and increased risk of nonspine fractures.
13 one mineral density at the hip and the heel, hip bone loss over 2 years, and fractures during 3.5 yea
14                               Total-body and hip bone mineral content (BMC) and bone mineral density
15  intake during childhood and adolescence and hip bone mineral content and bone mineral density (P < 0
16 scence was associated with a 3% reduction in hip bone mineral content and bone mineral density (P < 0
17 oints were changes in lumbar spine and total hip bone mineral densities (BMDs); secondary endpoints w
18 3.72%, 95% CI 1.54 to 5.89; p=0.26), nor did hip bone mineral density (2.09%, 95% CI -1.45 to 5.63 vs
19 yunsaturated fatty acid and fish intakes and hip bone mineral density (BMD) at baseline (1988-1989; n
20 mary endpoint was percentage change in total hip bone mineral density (BMD) from baseline to week 48
21          A candidate locus for regulation of hip bone mineral density (BMD) has been identified on ch
22              The database includes spine and hip bone mineral density (BMD) in 1056 premenopausal or
23                       Lumbar spine and total hip bone mineral density (BMD) were assessed at baseline
24                The primary outcome was total hip bone mineral density (BMD), with femoral neck BMD, l
25        The primary outcome measure was total hip bone mineral density (BMD); secondary measures were
26 ty, followed up prospectively for changes in hip bone mineral density and fractures.
27 percentage changes in lumbar spine and total hip bone mineral density at week 48, assessed by dual en
28 y for a treat-to-target approach, with total hip bone mineral density being the best specific target.
29 howed a smaller decrease in lumbar spine and hip bone mineral density but greater accumulation of lim
30    Participants were stratified by spine and hip bone mineral density categories.
31                         Adjustment for total-hip bone mineral density eliminated the elevated risk.
32 ne mineral density secondary outcomes, total hip bone mineral density increased more in the teriparat
33                                              Hip bone mineral density increases were greater with TAF
34                                        Total hip bone mineral density loss was similarly greater at w
35  measured by peripheral quantitative CT, and hip bone mineral density measured by dual-energy X-ray a
36 /d and was not associated with total body or hip bone mineral density measurements.
37  years or more, with a femoral neck or total hip bone mineral density T-score between -2.5 and -4.0 i
38 2]; p<0.0001), and mean percentage change in hip bone mineral density was 1.33% (2.20) in the elviteg
39 c effect of weight change on change in total hip bone mineral density was evaluated over 4 years (199
40                                              Hip bone mineral density was measured with dual x-ray ab
41 th differences in percentage change in total hip bone mineral density were 0.79 percentage point (95%
42 change from baseline to week 48 in spine and hip bone mineral density with a null hypothesis of zero
43 n (serum type I collagen C-telopeptide), low hip bone mineral density, absence of urticaria pigmentos
44         We assessed ankle-brachial index and hip bone mineral density, followed up prospectively for
45 els adjusted for age, body mass index (BMI), hip bone mineral density, knee surgery or pain, and phys
46 cs sex, serum type I collagen C-telopeptide, hip bone mineral density, urticaria pigmentosa, and alco
47 022 Gy/GBq for the mean of all vertebrae and hip bones, respectively.
48 0.069 +/- 0.033 Gy/GBq for the vertebrae and hip bones, respectively.
49                                  THA-derived hip bones serve as a major source of healthy hematopoiet
50 e in areal BMD of the lumbar spine and total hip, bone turnover markers C-terminal telopeptide cross-