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1 gions (periventricular white matter and both hippocampi).
2  suppression of spontaneous seizures in both hippocampi.
3 al-like discharges induced in vitro in mouse hippocampi.
4 ask in epileptic compared with non-epileptic hippocampi.
5 oven to depend on the presence of functional hippocampi.
6 57BL/6J mice and increased IL-1beta in their hippocampi.
7 re hypothalamic specific, with no changes in hippocampi.
8 al set of tissue samples from prenatal human hippocampi.
9 ronal loss, inflammation, and gliosis in the hippocampi.
10 ary hippocampal neuron cultures and in mouse hippocampi.
11 ctions of HFOs rates were found in epileptic hippocampi.
12 d methylation of the histone H3 in E17 fetal hippocampi.
13 ion were also increased in astrocytes in NBD hippocampi.
14 e) glycohydrolase mRNA were decreased in NBD hippocampi.
15 es in spared ipsi- and contralateral ventral hippocampi.
16 oligomers also increased in PS2APP;Trem2(ko) hippocampi.
17 in synaptic plasticity in eed and Mll mutant hippocampi.
18 lish or depress recurrent seizures in 70% of hippocampi.
19 RNA and protein levels were increased in NBD hippocampi.
20 nto SGs and away from polyribosomes, in both hippocampi.
21 were compared in epileptic and non-epileptic hippocampi.
22 uced by 44% in area CA1 of MTLE vs. non-MTLE hippocampi.
23 ficantly down-regulated in the neurons of AD hippocampi.
24 rd deviation in the Sommer sector of the MTS hippocampi.
25 ell as NFT formation, was observed in the AD hippocampi.
26 was associated with systemic hypoxia in both hippocampi.
27 lex families, had significantly smaller left hippocampi.
28 he brain, enhances neuronal survival in both hippocampi.
29  associated with increased activation of the hippocampi.
30 ulate white matter (WM), and the thalami and hippocampi.
31 tionalize the function of the left and right hippocampi.
32 188C>T) in postmortem AD olfactory bulbs and hippocampi.
33       Voxels were tailored to the individual hippocampi.
34 ere differences in T2 between right and left hippocampi.
35 ced by a mean of 42.7% compared with control hippocampi.
36  regions of interest within the head of both hippocampi.
37 r granule cell between control and sclerotic hippocampi.
38 eductions were not observed in non-epileptic hippocampi.
39 ong-term depression in USP6 transgenic mouse hippocampi.
40 ers reflect the extent of sclerosis in human hippocampi.
41  in brain metabolites characterize epileptic hippocampi.
42 quences predominantly involving the pons and hippocampi.
43 fferent spiking trains among eight different hippocampi.
44 e selectively reduced in the TFEB-transduced hippocampi.
45 o date comparing epileptic with normal human hippocampi.
46 es as well as in the thalami, amygdalae, and hippocampi.
47  cannot be used alone to distinguish between hippocampi.
48 lvement of the brainstem, basal ganglia, and hippocampi.
49 he largest effects seen in the amygdalae and hippocampi.
50 12 of 15 had abnormally low NAA in sclerotic hippocampi; 3 of these 12 also had abnormally low NAA co
51 iation (LTP) could not be induced in injured hippocampi; (3) GFAP and inducible NO synthase (iNOS) im
52                                              Hippocampi (5/group/time point) were harvested immediate
53 synchrony of neural oscillations between the hippocampi, a measure of brain function that indexed cog
54 First, metabolite changes occurring in brain hippocampi after application of 3 h of DBS to the animal
55 hermore, impaired growth of normal-appearing hippocampi after FSE suggests subtle injury even in the
56 ater than 50% seizure reduction in bilateral hippocampi after treatment.
57 rom this study of monkeys suggest that small hippocampi also reflect an inherited characteristic of t
58 G allele predicted lower volume of bilateral hippocampi among cannabis users relative to controls (bo
59 identified differentially expressed genes in hippocampi and amygdalae of the endogenous depression an
60 w that FNDC5/irisin levels are reduced in AD hippocampi and cerebrospinal fluid, and in experimental
61 n gray matter (GM) volume in their posterior hippocampi and concomitant changes to their memory profi
62 injured but also the contralateral uninjured hippocampi and correlated with the lesion induced atroph
63 (DHA) to arachidonic acid, were lower in the hippocampi and cortices and plasma of injured mice.
64 decreased in DS fibroblasts, and in both the hippocampi and cortices of young (age 15-40 years old) a
65  its cleavage products was also increased in hippocampi and cultured hippocampal neurons lacking Fbxo
66   Risk adjustment correlated with DOC in the hippocampi and deliberation time with DOC in the medial
67                      We performed RNA-seq in hippocampi and dorsolateral prefrontal cortices (DLPFCs)
68 refrontal cortex, retroesplenial cortex, and hippocampi and elevated connectivity between anterior an
69 and a pattern of atrophy including bilateral hippocampi and entorhinal cortex, right inferior parieta
70 n a set of 110 manually segmented atlases of hippocampi and find the manifold learning technique and
71  steady-state levels are reduced in human AD hippocampi and in the brain of an AD mouse model versus
72 en and women had significantly smaller right hippocampi and larger cerebrospinal fluid volumes than h
73 ated caspase 3 protein were increased in NBD hippocampi and localized to nuclei, mossy fibers, and de
74                                          His hippocampi and medial frontal cortex were significantly
75  levels of BDNF were associated with smaller hippocampi and poorer memory, even when controlling for
76 with bumetanide abolished seizures in 70% of hippocampi and significantly reduced the frequency, dura
77 dine 10 mg/kg/day) for 5 days, and cortices, hippocampi and spinal cords were collected for immunoblo
78 RNA miR-124 was increased in demyelinated MS hippocampi and targets mRNAs encoding 26 neuronal protei
79 exhibited greater activity in both posterior hippocampi and the right fusiform gyrus during smooth pu
80 Cs of both human and pilocarpine-treated rat hippocampi and those in the control rat DGCs.
81           Findings suggest that smaller left hippocampi and verbal memory deficits are an expression
82 ility to schizophrenia includes smaller left hippocampi and verbal memory deficits.
83 ysis included both the MTS and contralateral hippocampi, and covariance for changes in brain parenchy
84 response in the posterior cerebellar cortex, hippocampi, and lenticular nuclei bilaterally and the ri
85 hydrocephalus to cystic formations, abnormal hippocampi, and polymicrogyria.
86 ed significantly lower NAA in right and left hippocampi, and significantly higher Glu and Glu/NAA in
87 rtex and cortical lobes, basal ganglia (BG), hippocampi, and thalami.
88 ansgene spontaneously develop RIDNs in their hippocampi, and the formation of RIDNs correlates with t
89  lifetime of seizures on both left and right hippocampi, and the presence of any co-existing malforma
90 tionality of remaining tissue in the damaged hippocampi, and to appreciate the neural basis of a dist
91  induces tPA activity and cell death in both hippocampi, and unilateral treatment of rats with neuros
92  plasma, young mouse plasma, and young mouse hippocampi, appears in the brain after systemic administ
93  in triple-transgenic AD mice, CAR levels in hippocampi are low and further reduced after systemic in
94                        Consequently, Lis1+/- hippocampi are prone to interictal electrographic seizur
95 ed significant neuronal damage in KA-treated hippocampi at 16 h post-injection in both maternal and v
96 on microscopic analyses confirm that PS-cDKO hippocampi at 2 months postnatal do not yet exhibit syna
97 re used to measure structural differences in hippocampi between genotype groups.
98 or densities significantly decreased in both hippocampi (binding potential: controls 1.62 +/- 0.07; A
99       HS-1 degrees Rel had bilaterally small hippocampi, but no focal white matter atrophy was detect
100 were significantly decreased in demyelinated hippocampi, but not in demyelinated motor cortices from
101 vulnerable regions of the brain, notably the hippocampi, but was not sufficient to result in the more
102 AE by performing in vivo recordings from the hippocampi (CA1 and CA3) of moderate PAE and control adu
103 46), temporal cortices (Brodmann's area 22), hippocampi, caudate nuclei, and cerebella of schizophren
104 les, but volume changes in dorsal or ventral hippocampi, caudate-putamen, or thalamus were not detect
105 0 mRNA levels were elevated in tetanized rat hippocampi compared with those of sham controls that rec
106 ipitation assays in NG108-15 cells and mouse hippocampi confirmed specific Egr1 binding to the Ca(V)3
107 vitro slice preparation containing bilateral hippocampi connected by the VHC.
108 /B, was selectively increased in 4E-BP2(-/-) hippocampi, consistent with unaltered I-V relation of EP
109      LTP induced in chronic nicotine-treated hippocampi contained a component that is immune to rever
110                                    Sclerotic hippocampi contained fewer granule cells, fewer PV inter
111 better spatial learning abilities and larger hippocampi containing more and larger neurons compared t
112 uggests that radiation-induced injury to the hippocampi could play an important role in this cognitiv
113 s, and subcortical regions such as bilateral hippocampi depended predominantly on the perceptual diff
114 Inhibition of the MAPK/ERK cascade in dorsal hippocampi did not impair acquisition, but blocked the f
115 nome miRNA sequencing in surgically resected hippocampi did not reveal obvious differences in express
116                                    Sclerotic hippocampi displayed proportional losses of syt2-immunor
117                             Notably, the two hippocampi diverged in their responses to remoteness.
118 re is no T2 relaxation time abnormality, the hippocampi do not meet the criteria for MTS.
119 zure reduction greater than 98% in bilateral hippocampi during stimulation and greater than 50% seizu
120 e changes were attenuated in parkin-injected hippocampi, even in the presence of Tau pathology, sugge
121 n in an interface chamber were compared with hippocampi fixed by perfusion or by immersion of the who
122                    Using surgically acquired hippocampi from 129 TLE patients, we identify a gene-reg
123 s homolog NMNAT1, but not NMNAT3, in rTg4510 hippocampi from 6 weeks of age using recombinant adeno-a
124 62 were observed in cytoplasmic fractions of hippocampi from chronically stressed rats, and immunoflu
125                                              Hippocampi from control- and KD-fed rats were also compa
126                                              Hippocampi from hippocampal sclerosis patients (n = 28)
127           Using RNA-sequencing data from 100 hippocampi from mice with epilepsy (pilocarpine-temporal
128                                              Hippocampi from MMP9 null mice showed higher levels of l
129  interneuronal synapse formation, we studied hippocampi from mutant mice that completely lack the z+
130                                  Analysis of hippocampi from patients with intractable epilepsy revea
131 this pathway to experimental seizures and in hippocampi from patients with intractable temporal lobe
132 NA profiles from myelinated and demyelinated hippocampi from postmortem MS brains and performed valid
133 cular changes in myelinated and demyelinated hippocampi from postmortem MS brains.
134 gical changes in myelinated and demyelinated hippocampi from postmortem MS brains.
135                             Unexpectedly, in hippocampi from subsets of mice, abnormally low levels o
136            We measured changes in individual hippocampi from the mast cell mutant mouse strain, C57BL
137 c human epilepsy were validated by examining hippocampi from the pilocarpine model of chronic TLE.
138 itu hybridization and immunocytochemistry in hippocampi from three groups: TLE with hippocampal scler
139  activities of various apoptotic pathways in hippocampi from type 1 diabetic BB/Wor rats (hyperglycem
140                            Golgi analysis of hippocampi from weanling rats confirmed that development
141 ding field and whole-cell patch recording in hippocampi from wild-type and transgenic mice, we show t
142 foundly upregulated KCNQ2/3 transcription in hippocampi from wild-type, but not AKAP150(-/-), mice af
143                                              Hippocampi from WNV-NS5-E218A-recovered mice with poor s
144 ynapses or mtHSP70 was seen in myelinated MS hippocampi, further pointing toward a link between the c
145      Our results indicate that although both hippocampi generate HFOs with similar features that prob
146                                           AT hippocampi had lower levels of phosphorylated cAMP respo
147                                 Demyelinated hippocampi had minimal neuronal loss but significant dec
148 ing, intact planning in animals with damaged hippocampi has been repeatedly observed.
149            Injury was markedly attenuated in hippocampi having ipsilateral communicating arteries >50
150                      In autopsy and most NHS hippocampi, HCN1 mRNA expression was substantial in pyra
151 he diagnostic potential of GSH estimation in hippocampi (HP) and frontal cortices (FC) as a biomarker
152 d in the head and body of the right and left hippocampi in 123 subjects: 62 patients with Alzheimer d
153 ntiation between epileptic and non-epileptic hippocampi in humans.
154 crostructural integrity was impaired in both hippocampi in patients.
155  all samples, we found significantly smaller hippocampi in subjects with current PTSD compared with t
156 led activation of both TrkB and PLCgamma1 in hippocampi in the pilocarpine and kindling models in wil
157 uma exposure, we found evidence that smaller hippocampi indeed constitute a risk factor for the devel
158 t to left hippocampal volume ratios, smaller hippocampi initially, and reduced hippocampal growth.
159  tissue lactic acid and FFA were made in the hippocampi, ipsilateral cortex, contralateral cortex, an
160 erlying disease process in the contralateral hippocampi is different from MTS.
161 anscriptome profiling by RNA-seq analysis of hippocampi isolated from neonatal pups prenatally expose
162        PLCgamma1 activation was decreased in hippocampi isolated from trkB(PLC/PLC) compared with con
163 s +/- 0.31; right, 1.70 years +/- 0.37), and hippocampi (left, 1.93 years +/- 0.34; right, 1.78 years
164 matched cannabis users had smaller bilateral hippocampi (left, p=0.002; right, p=0.001) and left amyg
165  increased in myelinated and demyelinated MS hippocampi, mainly in the CA3/2 and CA1 subfields, which
166 rimary neuron-glia co-cultures from P0 mouse hippocampi, male neurons have more complex dendritic arb
167               Several regions, including the hippocampi, medial and lateral parietal cortex, exhibite
168 s (HS; n = 16), non-HS (n = 10), and autopsy hippocampi (n = 9) were studied for NMDAR1 (NR1) and NR2
169                        Compared with autopsy hippocampi, non-HS and HS patients showed increased NR2A
170 er cell of 29,201 from both fresh and frozen hippocampi, observing little difference in accessibility
171 lmodulin-dependent protein kinase II mRNA in hippocampi obtained during surgical resections for intra
172 n Illumina sequencing reads derived from the hippocampi of 10 colonial tuco-tucos housed in captivity
173 unit neuronal recordings were taken from the hippocampi of 10 male, New Zealand white rabbits during
174  Abeta and plaque loads were observed in the hippocampi of 11-month old Tg2576 mice born to mothers f
175 presynaptic and postsynaptic structures from hippocampi of 13 individuals aged 4 months to 71 years.
176 c imaging measurements were performed in the hippocampi of 14 control subjects and nine patients with
177                                  Whole human hippocampi of 14 subjects with bipolar disorder and 18 h
178  dentate granule cells acutely isolated from hippocampi of 28- to 35-day-old rats suggests that recep
179     Granule cells were acutely isolated from hippocampi of 7- to 14- and 45- to 52-day-old rats, and
180                                Sections from hippocampi of AD brains, brains with Alzheimer neurofibr
181 that UCH-L1 mRNA levels are decreased in the hippocampi of AD brains.
182  ADAM10/AP2 association was increased in the hippocampi of AD patients compared with healthy controls
183 y of these deficits were also present in the hippocampi of adult Df(16)A(+/-) and Zdhhc8-deficient mi
184 ss altered global transcription in the whole hippocampi of adult male and female rats.
185                                           In hippocampi of aged (21-28 months) mice, LTP was relayed
186 lular signal-regulated kinase (ERK2) MAPK in hippocampi of aged animals.
187                    Findings were compared to hippocampi of Alzheimer disease (AD) and non-neurologica
188 rmation and fewer dendritic polyribosomes in hippocampi of AMPKalpha2 cKO mice.
189               Conversely, microglia from the hippocampi of animals that had exercised were able to ac
190 l, FGF2 gene is delivered bilaterally to the hippocampi of APP+presenilin-1 bigenic mice via an adeno
191 sited amyloid in the vessels of cortices and hippocampi of APP/PS1DeltaE9/apoA-I(KO) mice, measured b
192 it and local field potential recordings from hippocampi of behaving rats with and without chronic epi
193 table metabolites can be quantified from the hippocampi of cognitively impaired individuals, and that
194 troscopy (MRS) studies of the left and right hippocampi of consenting Gulf War veterans (N=15; 10 wit
195 eurons and interneurons in the neocortex and hippocampi of Dravet adult post-mortem cases.
196 f the cannabinoid type 1 (CB(1)) receptor in hippocampi of epileptic rats following pilocarpine-induc
197 pared with NW piglets at 12 days of age, the hippocampi of EW piglets showed decreased gene expressio
198        In accordance with these conclusions, hippocampi of FABP5-null mice display excess accumulatio
199                          Recordings from the hippocampi of freely-moving epileptic rats revealed high
200 observed elevated Ephexin5 expression in the hippocampi of hAPP mice.
201 , synaptic pathology was not detected in the hippocampi of HMW oligomer-injected mice.
202 hexin5 expression was highly elevated in the hippocampi of human AD patients, indicating its potentia
203 ether HCN isoform expression was modified in hippocampi of individuals with TLE.
204 he differential expression of SNAP-25 in the hippocampi of infected neonates indicates a variable deg
205 rminal beta-amyloid fragment into the dorsal hippocampi of intact mice.
206 et (KD) increases UCP levels and activity in hippocampi of juvenile mice.
207 ed mPT in acutely prepared mitochondria from hippocampi of Kcna1-null animals.
208                                              Hippocampi of ketamine-treated mice were analysed by met
209        Similarly, GABA concentrations in the hippocampi of mice with a DG knockout of the GRIN1 gene
210 021 prevented PSD95-positive synapse loss in hippocampi of mice with EAE but did not affect developme
211 1H MRS described a metabolite profile in the hippocampi of MRI-negative TLE patients that was differe
212  (MRS), we examined metabolic changes in the hippocampi of MS patients, compared the findings to perf
213              Using neurons cultured from the hippocampi of newborn rats (both male and female), we ob
214                               Strikingly, in hippocampi of patients at early stages of late-onset Alz
215 ) cannabinoid receptor protein levels in the hippocampi of patients with Alzheimer's disease remain u
216 e found significant volume reductions in the hippocampi of patients with schizophrenia compared with
217  found in both ipsilateral and contralateral hippocampi of patients.
218 n agreement, beta1AR levels are decreased in hippocampi of PIST-deficient mice.
219               Previously, we showed that the hippocampi of prenatally [embryonic days (E) 11-17] chol
220 how that promoting translational recovery in hippocampi of prion-infected mice is neuroprotective.
221                                The posterior hippocampi of taxi drivers were significantly larger rel
222 in neurons and increased in astroglia in the hippocampi of TDF-treated wt and gp120-tg mice.
223 ired IRS-1 signaling was also present in the hippocampi of Tg mice with a brain condition that models
224 ignificant increase of new mature neurons in hippocampi of TgCRND8 compared with WT mice, suggesting
225                 On postnatal days 17 and 27, hippocampi of the ChD animals had the highest AChE and C
226 primary cultures of neurons derived from the hippocampi of the embryonic brain, suggesting that the a
227                  It is not known whether the hippocampi of the female brain are more vulnerable to al
228 er (GM) atrophy in the temporal lobe and the hippocampi of the NC+ group.
229  of the veterans with syndrome 2 and to both hippocampi of the veterans with syndrome 3.
230 ed in the microsomal fractions prepared from hippocampi of trained rats.
231  presence of dystrophic neurites in both the hippocampi of transgenic mice overexpressing amyloid pre
232 ffect in vivo, lactate was perfused into the hippocampi of unanesthetized rats while recording the fi
233 ndritic spines of CA1 pyramidal cells in the hippocampi of water maze-trained rats vs. controls.
234 se VCAM1 expression in cultured BECs and the hippocampi of young mice.
235             Proteomic analysis comparing the hippocampi of Zc3h14+/+ and Zc3h14Deltaex13/Deltaex13 mi
236          Among the proteins increased in the hippocampi of Zc3h14Deltaex13/Deltaex13 mice compared to
237                         Connectivity of both hippocampi predicted memory performance in patients.
238                We now report that, in intact hippocampi prepared from neonatal rats and transgenic mi
239 ated that GM density around the amygdala and hippocampi primarily drove the age estimation.
240 romoter heat shock elements by HSF1 in APP23 hippocampi, primary murine hippocampal neurons, and SH-S
241                  In vitro recordings in mice hippocampi produced similar speeds (0.10 +/- 0.03 m/s) a
242 n AQP4 levels in MTLE compared with non-MTLE hippocampi, quantitative ImmunoGold electron microscopy
243                                For bilateral hippocampi, quantitative markers of verticalization had
244                Therefore, the young and aged hippocampi react differently to estrogen replacement, wi
245 tive assessments of mRNA levels in epileptic hippocampi relative to autopsy controls were made by usi
246 ongation of T2 relaxation time identified in hippocampi remote from the seizure focus in all patient
247 of having 50% volume loss bilaterally in his hippocampi, retrieval in Jon was associated with increas
248   Magnetic resonance spectra measured in the hippocampi revealed a significantly lower glutamate conc
249 [3H]L-655,708 binding sites in rat and human hippocampi revealed a strong correlation with the affini
250 ed distant areas, including the neocortices, hippocampi, rostral migratory stream, and olfactory bulb
251                                Contralateral hippocampi show different HDM-LD changes, suggesting tha
252                          Furthermore, non-HS hippocampi showed increased NR1 and NR2B mRNA levels per
253 cant volume loss (P < 0.0001), contralateral hippocampi showed no significant volume loss.
254 rying with brain parenchymal volume, the MTS hippocampi showed significant volume loss (P < 0.0001),
255  to autopsies both sclerosis and mass lesion hippocampi showed that, in the stratum granulosum, the g
256 l of TLE using 100 epileptic and 100 control hippocampi shows the proconvulsive module is preserved a
257 orded at rest in epileptic and non-epileptic hippocampi suggests that they cannot be used alone to di
258 cutely dissociated from slices prepared from hippocampi surgically removed for the treatment of tempo
259  APOE4 carriers, GM volume loss affected the hippocampi, temporal and parietal lobes, right caudate n
260 cannabis users display smaller amygdalae and hippocampi than controls, and genetic variation accounts
261 sequence, more new neurons remained in their hippocampi than in sex-matched controls.
262  unexposed members-had significantly smaller hippocampi than non-PTSD pairs.
263       The improvers had significantly larger hippocampi than those that declined (P = 0.02) and the s
264 ent of prolongation of T2 relaxation time in hippocampi that are not the primary epileptogenic focus,
265 epilepsy is associated with abnormalities in hippocampi that are not the primary seizure focus.
266                                     In their hippocampi, the concentration of GABA was significantly
267                         In the contralateral hippocampi, the inferior surface of the hippocampal body
268 solute concentrations of metabolites in both hippocampi, the sites of early Alzheimer's disease, in p
269 ation of a white matter tract connecting the hippocampi, the ventral hippocampal commissure (VHC), wi
270 r piriform cortex and the dorsal and ventral hippocampi underwent MS: electrical stimulation of the l
271 rentiate between epileptic and non-epileptic hippocampi using a simple odd-ball task.
272 ciated viral delivery of the channel to both hippocampi was also effective in a model of temporal lob
273 l gray matter, white matter, ventricles, and hippocampi was performed by using software.
274   The mean difference between left and right hippocampi was smallest at fast FLAIR imaging.
275                                     In mouse hippocampi, we detected relatively low levels of alpha,
276 lobe epilepsy model) and 100 healthy control hippocampi, we identified 256 RNA sites (overlapping wit
277 d with T2-lesion volumes, and right and left hippocampi were affected equally.
278 e imaging (MRI) studies of the amygdalas and hippocampi were conducted in 50 non-epileptic controls (
279                        In contrast to AD, MS hippocampi were consistently negative for the terminal c
280                                              Hippocampi were evaluated as a reference structure.
281 brains were frozen in situ, and cortices and hippocampi were excised at 0 degrees C.
282                                 Frozen right hippocampi were fixed, sectioned (50 mum), immunostained
283                                    Sclerotic hippocampi were identified by the loss of Nissl-stained
284                                              Hippocampi were obtained from control and chronic DA sea
285                 Following retention testing, hippocampi were removed and protein extracted from cytos
286                     After the challenge, the hippocampi were removed to assay phosphatase 2A (PP2A) a
287                            We found that the hippocampi were significantly active during the retrieva
288                                        Human hippocampi were stained with a specific polyclonal antib
289 tal and temporal lobe regions (including the hippocampi) were associated with the decline in PIQ.
290 s callosum, anterior commissure, and fimbria hippocampi, were investigated for structural and functio
291 as required to saturate LTD in primed mutant hippocampi, whereas multiple low-frequency stimuli were
292 articularly in the left hemisphere; (2) that hippocampi will be smaller in multiplex relatives as com
293  will be positively correlated; and (4) that hippocampi will be smaller in patients with schizophreni
294 ression of inhibition when superfusing mouse hippocampi with amyloid-beta.
295 g early posthatching development had smaller hippocampi with fewer neurons and performed worse in a c
296               The mean metabolite content of hippocampi with less than 30% of neurons remaining was t
297 ure of rat hippocampal neurons and the mouse hippocampi with morphine or fentanyl for 3 days, seven m
298 ype II focal cortical dysplasia (IIa and b), hippocampi with or without hippocampal sclerosis (HS), a
299                          RNA-seq analysis of hippocampi with upregulation of Rbfox1 in conjunction wi
300 ippocampal sclerosis (HS; n = 17), epileptic hippocampi without HS, or non-HS (NHS; n = 10), and auto

 
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