ライフサイエンスコーパス: histiocyte

1 rple for CD163-positive (CD163(+)) monocytes/histiocytes.

2 this may be a general attribute of abnormal histiocytes.

3 e detected in intratumoral blood vessels and histiocytes.

4 as hsp27 is absent in all lymphoid cells and histiocytes.

5 epithelium, supports differentiation toward histiocytes.

6 anulomas were composed solely of epithelioid histiocytes (75.8%).

7 xtensive inflammation, composed primarily of histiocytes admixed with lymphocytes and occasional neut

8 All lesions were composed predominantly of histiocytes and a variable number of foreign body-type g

9 eosinophils, giant cells, epithelioid cell, histiocytes and calcified Schistosoma eggs with terminal

10 pha promotes the terminal differentiation of histiocytes and dendritic cells, we hypothesized that th

11 dy reaction characterized by the presence of histiocytes and multinucleated giant cells (MNGCs).

12 ing of the crosstalk between lymphocytes and histiocytes and their regulatoty mechanisms.

13 ough the blood to become tissue macrophages, histiocytes, and dendritic cells.

14 rivascular spaces, consisted of lymphocytes, histiocytes, and scattered eosinophils.

15 Histiocytes are white blood cells of the monocytic linea

16 ar, monomorphic appearance without lipidized histiocytes but sometimes more spindled or epithelioid m

17 vated in Langerhans cell histiocytosis (LCH) histiocytes, but only 60% of cases carry somatic activat

18 us and noninfectious inflammatory disorders, histiocytes can engulf nonapoptotic leukocytes and nonse

19 ockets of different cell types, in this case histiocytes, can show intermediate cancer/healthy lipid

20 e surface of canine MDCK epithelial and DH82 histiocyte cells and murine C8-D1A astrocytes.

21 nulomas with many multinucleated epithelioid histiocytes consistent with the diagnosis of sarcoidosis

22 LCH) results from the accumulation of tissue histiocytes derived from the same progenitor cells as mo

23 for retinal pigment epithelium and CD163 for histiocytes, each tagged with different chromogens, yell

24 prone to develop splenic rupture, as splenic histiocytes engage in more robust erythrophagocytosis, l

25 istent with this observation, hemophagocytic histiocytes from patients with macrophage activation syn

26 e patient with wild-type BRAF alleles in his histiocytes had compound mutations in the kinase domain

27 most cases, biopsy specimens infiltrated by histiocytes have somatic mutations in genes activating t

28 pirate smears and biopsies may show reactive histiocytes, hemophagocytosis, or myelofibrosis but neve

29 ubulopathy (LCPT) in 80% and in interstitial histiocytes in 36%.

30 Histiocytes in Rosai-Dorfman disease and hemophagocytic

31 i and were intermixed with the CD68-positive histiocytes in the absence of CD20 B lymphocytes.

32 of placenta, Kupper cells in the liver, and histiocytes infiltrating near prostate cancers.

33 debris, as well as extensive lymphocyte and histiocyte infiltration.

34 several biopsy specimens within adventitial histiocytes-macrophages, but these results did not corre

35 mine silver stain procedures showed numerous histiocytes, multinucleated giant cells, and numerous gl

36 cluded complex hyperplasia (n = 1), abnormal histiocytes (n = 1), simple hyperplasia (n = 2), polyps

37 mphocytes (n = 9), plasma cells (n = 6), and histiocytes (n = 6).

38 CD biopsies, we detected expression in foamy histiocytes of the phosphorylated forms of mTOR and of i

39 licular cell proliferation, large numbers of histiocytes, polymorphonuclear leukocytes, and multinucl

40 pression of inflammatory genes in human U937 histiocytes (resident macrophages).

41 ed features of histologically defined T-cell/histiocyte-rich B-cell lymphoma, including fewer genetic

42 6, 64%), significantly more when compared to histiocyte-rich inflammatory lesions (0/11, 0%, P<.001)

43 xpressed in <10% of lesional cells in all 11 histiocyte-rich inflammatory lesions (P<.001) and all 10

44 Eleven histiocyte-rich inflammatory lesions and 10 xanthelasma

45 T-cell/histiocyte-rich large B-cell lymphoma (TCRLBCL) is an ag

46 ogically, 3 patterns were identified: T-cell/histiocyte-rich large B-cell lymphoma-like (n = 36), gra

47 s), angiocentric lymphoma (3 of 3 patients), histiocyte-rich tumors (4 of 5 patients), and unspecifie

48 hologic features of HLH, as set forth in the Histiocyte Society diagnostic guidelines: fever, cytopen

49 ne kinase expression was localized to CD163+ histiocytes; tumors with CLTC::SYK fusions also demonstr

50 Authentic histiocytes were also found in most cases, with a mature

51 were negative for PD-1 ligands, but PD-L1(+) histiocytes were found within the T cell-rich zone of th

52 Neoplastic histiocytes were positive for macrophage markers and oft

53 oiesis in the spleen and abnormally enlarged histiocytes with ingested red blood cells (RBCs) in bone

54 infiltration of tissues by foamy CD68+CD1a- histiocytes, with 1500 known cases since 1930.

55 r characterized by the accumulation of foamy histiocytes within organs (in particular, frequent retro