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1 d crizotinib, respectively, in patients with histiocytosis.
2 he self-limiting, cutaneous form of human LC histiocytosis.
3 d in this respect CCH differed from human LC histiocytosis.
4 iopsy may reveal hemophagocytosis and marrow histiocytosis.
5 orfman disease is a rare non-Langerhans cell histiocytosis.
6 ns in 14 of 18 (78%) patients with suspected histiocytosis.
7 survival in 156 adults with Langerhans cell histiocytosis.
8 for clinical trials of targeted therapies in histiocytosis.
9 em for the fields of ophthalmic oncology and histiocytosis.
10 lichiosis-induced hemophagocytic lymphocytic histiocytosis.
11 leiomyomatosis and pulmonary Langerhans cell histiocytosis.
12 Erdheim-Chester disease and Langerhans'-cell histiocytosis.
13 ster disease (ECD) is a rare, non-Langerhans histiocytosis.
14 ystemic viral infections and Langerhans cell histiocytosis.
15 ytic human diseases, such as Langerhans cell histiocytosis.
16 Erdheim-Chester disease and Langerhans cell histiocytosis.
17 gated in patients with malignant BRAF(V600E) histiocytosis.
18 taneous and progressive macrophage-dominated histiocytosis.
19 blastoma, some sarcomas, and Langerhans cell histiocytosis.
20 plant (UCBT) for refractory Langerhan's cell histiocytosis.
21 rapy to the entire spectrum of patients with histiocytosis.
22 g pneumonia), and pulmonary Langerhans' cell histiocytosis.
23 ine staging and diagnosis of Langerhans cell histiocytosis.
24 , Rosai-Dorfman 21, xanthogranuloma 7, mixed histiocytosis 11, Langerhans cell histiocytosis 15, and
26 lar xanthogranuloma (22%), 7 Langerhans cell histiocytosis (19%), 5 Rosai-Dorfman disease (14%), 5 xa
27 CSF transgenic mice developed a disseminated histiocytosis and had increased circulating IL-17 produc
29 anoma, conjunctival amyloidosis, and primary histiocytosis and in 1 case of ocular surface squamous n
30 , MEK inhibitor therapy led to resolution of histiocytosis and inflammation in a patient with H syndr
31 , we reveal the molecular pathway leading to histiocytosis and inflammation in this genetic disorder.
34 presence of distinct kinase mutations in the histiocytosis and myeloid neoplasm resulted in discordan
35 erature outlining the recent advancements in histiocytosis and ocular oncology was reviewed and combi
37 ogic and molecular landscape of ALK-positive histiocytosis and provides guidance for the clinical man
38 se, characterized by short stature, familial histiocytosis and sinus histiocytosis with massive lymph
39 ed with Erdheim-Chester disease (ECD) (mixed histiocytosis) and discuss the significance of this asso
42 LC] proximal tubulopathy, LC crystal-storing histiocytosis, and LC crystalline podocytopathy) and les
43 ted with tuberous sclerosis, Langerhans cell histiocytosis, and lymphocytic interstitial pneumonia.
44 leiomyomatosis and pulmonary Langerhans cell histiocytosis are perhaps more frequently seen in the cl
45 cysts of Rathke's pouch and Langerhans cell histiocytosis, as well as with genetic neurodevelopmenta
46 e have developed a model for Langerhans cell histiocytosis comprising a CD1a-positive subcutaneous xe
50 s of 91 patients (46 males, 45 females) with histiocytosis (Erdheim-Chester 35, Rosai-Dorfman 21, xan
51 iocytic disorders, including Langerhans cell histiocytosis, Erdheim-Chester disease, and Rosai-Dorfma
53 functional evidence of the cell of origin of histiocytosis from actual patient materials has long bee
56 Furthermore, the majority of patients with histiocytosis had increased SFCT compared with age- and
58 n SLC29A3 cause H syndrome, characterized by histiocytosis, hyperinflammation, and immunodeficiency.
61 r and molecular basis for the development of histiocytosis in several human syndromes associated with
62 igotes were not observed in organs but sinus histiocytosis in the spleen and lymphoid hyperplasia wer
63 or CD34(+) cells to drive the development of histiocytosis in xenotransplantation assays in vivo.
70 al of adults with pulmonary Langerhans'-cell histiocytosis is shorter than that in the general popula
71 observed in 57% of cases of Langerhans cell histiocytosis (LCH) and 54% of cases of Erdheim-Chester
72 RAF mutation in a child with Langerhans cell histiocytosis (LCH) and demonstrate that the encoded pro
75 he most common histiocytoses-Langerhans cell histiocytosis (LCH) and Erdheim-Chester disease (ECD)-wh
76 ved in half of patients with Langerhans cell histiocytosis (LCH) and in 50% to 100% of patients with
79 wo girls were diagnosed with Langerhans cell histiocytosis (LCH) at the age of 16 and 7 months and de
82 ling pathway is activated in Langerhans cell histiocytosis (LCH) histiocytes, but only 60% of cases c
106 inal phenotype: for example, Langerhans cell histiocytosis (LCH) shares CD207(+) antigen with physiol
108 sis of patients with proven Langerhans' cell histiocytosis (LCH) was undertaken with the aim of evalu
109 olvement at presentation of Langerhans' cell histiocytosis (LCH) with subsequent disease course, surv
110 om skeletal collections with Langerhans Cell Histiocytosis (LCH), a benign osteolytic tumor-like diso
111 nterstitial pneumonia (DIP), Langerhans cell histiocytosis (LCH), and acute eosinophilic pneumonia (A
115 on a group of patients with Langerhans cell histiocytosis (LCH), which historically has poor disease
120 ents were identified: 7 with Langerhans cell histiocytosis (LCH); 15 with Erdheim-Chester disease (EC
122 eoside transporter, mutations in which cause histiocytosis-lymphadenopathy plus syndrome, a group of
124 ngerhans cell histiocytosis (so-called mixed histiocytosis [MH]), we identified an unexpected and her
127 een identified previously in solid tumor and histiocytosis patients, caused hyperactivated MAP kinase
128 ed <18 years enrolled in the French National Histiocytosis Registry, 331 developed HI (median age at
129 who received transplants for Langerhans cell histiocytosis relative to other indications for transpla
130 ologic and molecular spectra of ALK-positive histiocytosis remain, however, poorly characterized.
132 ECD and ECD overlapping with Langerhans cell histiocytosis (so-called mixed histiocytosis [MH]), we i
133 ons) coexisting with those characteristic of histiocytosis (such as BRAFV600E and MAP2K1 mutations).
134 disease (ECD) is a rare non-Langerhans cell histiocytosis that most commonly affects adults and is d
136 Erdheim-Chester disease or Langerhans'-cell histiocytosis, the response rate was 43% (95% CI, 18 to
137 gically confirmed pulmonary Langerhans'-cell histiocytosis to ascertain their vital status and whethe
138 e describe the largest study of ALK-positive histiocytosis to date, with detailed clinicopathologic d
139 disease (ECD) is a rare non-Langerhans cell histiocytosis, to whose pathogenesis neoplastic and immu
140 o include neurodegeneration with bone marrow histiocytosis, visceromegaly, brown pancreas, and linkag
141 eneous, the phenotype of patients with mixed histiocytosis was closer to that of isolated ECD than to
143 the diagnosis of pulmonary Langerhans'-cell histiocytosis were analyzed with the Cox proportional-ha
144 risk factors, patients with Langerhans cell histiocytosis were at significantly increased risk for P
145 s from Langerhans cell sarcoma and malignant histiocytosis, which are hyperproliferative lesions typi
146 CCH is a localized form of self-limiting LC histiocytosis, which predominantly expresses an epiderma
150 rt stature, familial histiocytosis and sinus histiocytosis with massive lymphadenopathy (SHML), chara
153 ell histiocytosis (LCH), previously known as histiocytosis X, is a reactive proliferative disease of