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1  their magnitude correlated with severity of histological damage.
2 essed them for the presence of ricin and for histological damage.
3 incided with reduced lung, liver, and kidney histological damage.
4 egulation of proinflammatory genes, and less histological damage.
5 strated by reduction of disease severity and histological damage.
6 rrelates with the loss of renal function and histological damage.
7 generation and progression to patchy chronic histological damage.
8 ntly improves neurological score and reduces histological damage.
9 often noted in adult cartilage lacking overt histological damage.
10 ssical clinical presentation and more severe histological damage.
11 cal parameters and a progressive increase in histological damage.
12                                  Analysis of histological damage 72h after resuscitation revealed a c
13 age on clinical presentation and severity of histological damage (after adjusting for age and sex, p
14 usion, in the treated hearts, there was less histological damage and less apoptosis of cardiac muscle
15 ion was noted between the overall intestinal histological damage and lethality in mice.
16 which was ultimately associated with reduced histological damage and more pronounced apoptosis.
17 he key mediators in the subsequent cycles of histological damage and repair, and clinical relapse and
18           The compound significantly reduced histological damage and serum amyloid A (SAA) levels in
19                                      Chronic histological damage and specific diseases had additive a
20 activity index (DAI), restoration of colonic histological damage and suppression of abnormal inflamma
21           In addition, periodontitis-related histological damages and apoptotic activity were also si
22 1 significantly alleviated colonic symptoms, histological damages and reshaped the gut microbiota.
23 ductions in clinical colitis scores, colonic histological damage, and colonic inflammation compared w
24           The ensuing cellular infiltration, histological damage, and decline in lung function were q
25  a bioluminescence study, protection against histological damage, and decrease of inflammatory cytoki
26 B-269970 resulted in lower clinical disease, histological damage, and proinflammatory cytokine levels
27 d loss, weight loss, colon shortening, colon histological damage, and splenomegaly than did wild-type
28 neurobehavioral deficits, E/I imbalance, and histological damage are all ameliorated by treatment wit
29                     There was no evidence of histological damage caused by Hemopure.
30  stem cells are of great interest to recover histological damage caused by neuro-degenerative disease
31      This dose did not induce any additional histological damage compared to controls or after a subs
32 ischemia (30 and 60 min) induced significant histological damages, delayed cortical and medullary Gad
33 i.e. NF-kappaB, Ikappa-Balpha, COX-2, iNOS), histological damage, disease activity index (DAI) and SC
34                                 Behavior and histological damage evaluation was performed for adult f
35        The incidence and severity of chronic histological damage have decreased substantially over th
36                        However, there was no histological damage in the brains of Tg and Wt mice as a
37        DiNP exposure significantly increased histological damage in the colon compared to control.
38 scores for clinical disease activity and for histological damage in the joints were both significantl
39           Notably, with increasing levels of histological damage, miR-21-5p was upregulated progressi
40 vere and/or sustained, can result in chronic histological damage of which interstitial fibrosis and t
41 teinuria in comparison to littermates, while histological damage, podocyte ultrastructure in STED ima
42 logical diagnoses versus nonspecific chronic histological damage remains unclear.
43 eficit scores, overall performance category, histological damage scores (viable neuron counts in CA1
44 ance, final neurological deficit scores, and histological damage scores.
45  sacrifice of experimental animals to assess histological damage, thus counteracting the principles o
46 r) or B2F1 Stm(r) developed hematuria and/or histological damage to glomeruli or thrombocytopenia, co
47 m sulfate (DSS)-induced clinical disease and histological damage to the colonic mucosa were significa
48 toxin in the loops, and reduced STEC-induced histological damage (villus blunting).
49   Logistic regression analysis revealed that histological damage was (P = 0.0017) independently assoc
50 linical chemistry results normalized, and no histological damage was apparent 3 weeks after autograft
51 pients no progressive clinically significant histological damage was apparent in follow-up protocol b
52                                              Histological damage was assessed at 24 hr.
53  a single 6-min bilateral carotid occlusion, histological damage was evident in the CA1 region of hip
54                                           No histological damage was observed at the most effective p
55                                        Minor histological damage was observed.
56                  DSS-induced weight loss and histological damage were ameliorated in oxymatrine-treat
57  fluid secretion, mannitol permeability, and histological damage were measured in ileal loops in vivo
58           In addition, clinical symptoms and histological damage were more severe in AKAP12 KO mice t
59  fibrosis, inflammatory gene expression, and histological damage were not significantly different fro
60 in the IC group and had moderately increased histological damage when compared to the Na-NP group.
61 etween the level of anosmia and the score of histological damage within the olfactory epithelium.