戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 f CNP/NPR-C signaling in preserving vascular homoeostasis.
2 ammalian retina, a process required for disc homoeostasis.
3 atory resolution that re-establish pulmonary homoeostasis.
4 inflammatory responses and improve metabolic homoeostasis.
5 rtant role for regulation of telomere length homoeostasis.
6 role in the regulation of arousal and stress homoeostasis.
7  their complex functions and effects on body homoeostasis.
8 ion, a key cytokine in normal colonic tissue homoeostasis.
9 essary for maintaining normal blood pressure homoeostasis.
10 ion and a modulator of intestinal epithelial homoeostasis.
11 s body fat redistribution and alters glucose homoeostasis.
12 lf-digestion machinery for cell survival and homoeostasis.
13 ion history of a tissue SC during its normal homoeostasis.
14 ions and mutations can interfere with tissue homoeostasis.
15 eted hormone that plays a key part in energy homoeostasis.
16  bone turnover, and abnormalities in calcium homoeostasis.
17 nd support teeth in situ and preserve tissue homoeostasis.
18 e to a meal, is essential for normal glucose homoeostasis.
19 ) could be important determinants of glucose homoeostasis.
20 ticular, is implicated in mammary epithelium homoeostasis.
21 on of iron absorption and of whole-body iron homoeostasis.
22  highlight a role of melanocortins in weight homoeostasis.
23 atment, which by itself could affect glucose homoeostasis.
24 usceptible to perturbations in mitochondrial homoeostasis.
25 teotoxic stress response to maintain protein homoeostasis.
26 cessary for embryonic development and tissue homoeostasis.
27 t is unclear how FMRP supports mitochondrial homoeostasis.
28 ls microbiota composition and gut epithelial homoeostasis.
29 ed roles in cellular signalling and membrane homoeostasis.
30 nd ETS1 and other key regulators of vascular homoeostasis.
31 ective response plays a key role in cellular homoeostasis.
32 ns for life such as cellular respiration and homoeostasis.
33 barrier epithelia is essential to organismal homoeostasis.
34 and a controller of immune system and tissue homoeostasis.
35 oteins in the cell is critical for achieving homoeostasis.
36 aintaining cancer cell metabolic and protein homoeostasis.
37 obiota that interact dynamically to preserve homoeostasis.
38 sis, increased haemolysis, and deranged iron homoeostasis.
39 uced batokine to coordinate systemic glucose homoeostasis.
40 organelles without cell division to maintain homoeostasis.
41 gical transitions in organogenesis and adult homoeostasis.
42 PAR-delta), which is implicated in bile acid homoeostasis.
43 reatic cancer cells and was essential for ER homoeostasis.
44 des and is known to influence systemic lipid homoeostasis.
45 BR), a bacteriostatic agent, merit metabolic homoeostasis.
46 y pathway is a major determinant of cellular homoeostasis.
47  cell biology, embryo patterning, and tissue homoeostasis.
48 ce of STAT5 tetramers in maintaining NK cell homoeostasis.
49 proliferate is critical to tissue repair and homoeostasis.
50 educed fat mass and improved glucose/insulin homoeostasis.
51 catenin signalling is crucial for intestinal homoeostasis.
52 ain development requires a fine-tuned copper homoeostasis.
53 iability is an integral component of glucose homoeostasis.
54 trols food consumption to maintain metabolic homoeostasis.
55 s necessary to dampen activation and restore homoeostasis.
56 ransduction involved in regulating cell wall homoeostasis.
57 of energy balance, thermogenesis and glucose homoeostasis.
58 erentiation and in the maintenance of tissue homoeostasis.
59 P1 and WT1) in connective tissue maintenance/homoeostasis.
60 nges during embryonic development and tissue homoeostasis.
61 olic process critical for cell viability and homoeostasis.
62 replenished by stem cells to maintain tissue homoeostasis.
63 p1), a GTPase is essential for mitochondrial homoeostasis.
64  a role for HTT on dynamin 1 function and ER homoeostasis.
65 state of vulnerability to poor resolution of homoeostasis after a stressor event and is a consequence
66 hythmicity and sleep need, rather than sleep homoeostasis alone.
67 ial part played by thyroid hormone in energy homoeostasis and adaptation to cold.
68 mportant hormone regulator of cardiovascular homoeostasis and an important biomarker for heart failur
69 en implicated in germ cell specification and homoeostasis and are essential to fertility-related proc
70  been shown to ameliorate deranged bile acid homoeostasis and attenuate raised concentrations of live
71 nce of deregulated WNT signalling in gastric homoeostasis and cancer is still unclear.
72 rates and on its role in tissue development, homoeostasis and cancer.
73 ngs link O-GlcNAcylation to muscular protein homoeostasis and contractility and reveal a mechanism of
74              Hepcidin has a key role in iron homoeostasis and could be a future diagnostic and therap
75 onsistent with improved neuronal cholesterol homoeostasis and decreased neuronal pathology.
76     This may be required to maintain protein homoeostasis and deliver metabolite intermediates for bi
77 veral new factors that promote mitochondrial homoeostasis and demonstrate that the UPR(mt), as curren
78 he activation of genes associated with blood homoeostasis and erythropoiesis, with a particular empha
79 tress response pathway, which regulates heme homoeostasis and expression of antioxidant enzymes, was
80 eletal system is crucial for bone formation, homoeostasis and fracture repair, yet the diversity and
81 highlight the importance of PORB for Pchlide homoeostasis and greening in Arabidopsis.
82 ation of macromolecules to maintain cellular homoeostasis and health.
83 ying pathways include alterations of protein homoeostasis and immune and inflammatory function.
84 ading to favourable alterations in acid-base homoeostasis and in the gut microbiome.
85 ) of wild-type and Batf3-/- neonatal mice at homoeostasis and investigated their role during infectio
86 ribes how Breg cells are critical in humoral homoeostasis and may have implications for the regulatio
87 -environment interactions to influence human homoeostasis and metabolic networks(1-4).
88 process together with preservation of tissue homoeostasis and microbiota growth.
89 nown as programmed cell death, is central to homoeostasis and normal development and physiology in al
90  RNA sequestration in P-bodies during tissue homoeostasis and oncogenesis.
91 ferentiation is crucial for postnatal tissue homoeostasis and organogenesis.
92   Our findings thus provide insights on Breg homoeostasis and present possible targets for Breg-relat
93 ediated SUMOylation of SHP in maintaining BA homoeostasis and protecting from the BA hepatotoxicity.
94 1 could be important determinants of glucose homoeostasis and provide further evidence for the possib
95 ography studies are needed to validate sleep homoeostasis and respiratory variability during sleep as
96 tes IgA production to maintain gut microbial homoeostasis and restrain IL-1alpha-dependent colitis an
97 l antibody against FGF23, improves phosphate homoeostasis and rickets in children aged 5-12 years wit
98 ein as a regulator of cellular immunological homoeostasis and suggest cellular prion protein as a nov
99  show that LTA is needed for divalent cation homoeostasis and that its absence has severe effects on
100 ramifications for normal tissue development, homoeostasis and the physiological functions of various
101 ll surface scavenger receptors contribute to homoeostasis and the response to pathogens and products
102  activity by regulating neuronal cholesterol homoeostasis and the TCA cycle.
103 lling pathways in the maintenance of glucose homoeostasis and their contribution to disease are unkno
104                                   Melanocyte homoeostasis and their response to ultraviolet radiation
105  Therefore, the NPs can help maintain immune homoeostasis and vascular function, two key factors in t
106 ated pathways and their impact on organismal homoeostasis, and aid the generation of clinically meani
107    It is important for embryogenesis, tissue homoeostasis, and cancer treatment.
108 n coordinating nutrient availability, energy homoeostasis, and cell growth.
109  importance of the microbiota for intestinal homoeostasis, and discuss the similarity between inflamm
110  (RBP4:TTR) related to lipid biology, energy homoeostasis, and endocrine signalling.
111 related to energy metabolism, cardiovascular homoeostasis, and haemostasis.
112 racellular proteolytic activity in capillary homoeostasis, and identify ADAMTS1 as a marker of activa
113 gh metabolic load on a depleted capacity for homoeostasis, and in women increases the risk of childbi
114 Hippo pathway plays a central role in tissue homoeostasis, and its dysregulation contributes to tumor
115 utamate are necessary to maintain epithelial homoeostasis, and provision of glutamate drives disrupti
116 ic development, cell differentiation, tissue homoeostasis, and removal of damaged and harmful cells f
117 ttention to aspects of vascular development, homoeostasis, and response to environmental effects.
118 toprotective response is central to cellular homoeostasis, and there is increasing interest in develo
119 elial cell layer is crucial to maintain lung homoeostasis, and this depends on intercellular adhesion
120 se cytokines function to maintain intestinal homoeostasis, and under what circumstances they contribu
121 se insulin action, and fully correct glucose homoeostasis are a distant vision.
122 ic mechanisms that maintain nuclear envelope homoeostasis are bulwarks to ageing and disease.
123 s in membrane trafficking and altered Ca(2+) homoeostasis are common features in many lysosomal stora
124 ple, variation in genes involved in synaptic homoeostasis are implicated in autism spectrum disorder
125 ions driving mammary gland morphogenesis and homoeostasis are poorly understood.
126 ne plays an essential role in cellular redox homoeostasis as a key constituent of the tripeptide glut
127  auxin oxidation is more important for auxin homoeostasis at lower hormone concentrations, whereas au
128 ay deregulation dramatically affects stomach homoeostasis at very short latencies.
129 ardiac development (MYOZ1, SYNPO2L), protein homoeostasis (BAG3), and cellular senescence (CDKN1A).
130 alance is vital for cell survival and tissue homoeostasis because imbalanced production of reactive o
131 stitutive process required for proper tissue homoeostasis but can be rapidly regulated by a variety o
132 tion of bile acids affects intestinal immune homoeostasis but its impact on inflammatory pathologies
133 hought to return the affected tissue back to homoeostasis but recent evidence supports a non-linear m
134  is driven by disruptions in host-microbiota homoeostasis, but current treatments exclusively target
135  reticulum (ER) is essential for cholesterol homoeostasis, but the mechanism and regulation of this t
136           By restoring microbial and mucosal homoeostasis, butyrate-releasing micelles may function a
137 n important function in maintaining cellular homoeostasis by assisting the folding of many proteins,
138 icroarchitecture features and measured sleep homoeostasis by calculating overnight change in slow wav
139 factor ETS-related gene (ERG) promotes liver homoeostasis by controlling canonical TGFbeta-SMAD signa
140 sphatases in mammalian cells, maintains cell homoeostasis by counteracting most of the kinase-driven
141 totoxic T lymphocytes, CTLs) maintain immune homoeostasis by eliminating virus-infected and cancerous
142               Lung lymphatics maintain fluid homoeostasis by providing a drainage system that returns
143 /GH3.6 and DFL2/GH3.10, which regulate auxin homoeostasis, by binding directly to the TA box in each
144                                Abnormal iron homoeostasis can induce cellular damage through hydroxyl
145 s, which is critically important to cellular homoeostasis, can be achieved at the level of gene expre
146 city, monoaminergic signalling, inflammatory homoeostasis, cellular metabolic pathways, and mitochond
147 s as a source of Wnt in bone development and homoeostasis, complementing their known function as targ
148           Our results reveal that PtdIns(4)P homoeostasis, coordinated by PIPKIgamma and INPP5E at th
149 n various behavioral aspects including sleep homoeostasis, decision-making, spatial working memory an
150 s crucial for normal brain function, and its homoeostasis depends on carrier-mediated transport.
151                                     Vascular homoeostasis, development and disease critically depend
152  is critical for the maintenance of cellular homoeostasis during adaptation to internal and environme
153 egulate cardiac myocyte hypertrophy, calcium homoeostasis, energetics, and cell survival, and process
154 a pivotal role for WNT signalling in gastric homoeostasis, FGP formation and adenomagenesis.
155 itches the mechanism for maintaining protein homoeostasis from a chaperone-based approach to an appro
156 rence that represses fission yeast phosphate homoeostasis gene pho1 provides a sensitive readout of g
157 ry distinct parts in modulation of metabolic homoeostasis, haemopoiesis, and osteogenesis.
158            Its key role in governing protein homoeostasis has made VCP/p97 an appealing anticancer dr
159         Most of these responses relate to ER homoeostasis; however, here we show that the PERK branch
160 ny to generate myonuclei for skeletal muscle homoeostasis, hypertrophy and repair.
161  inflammatory molecules, disrupted glutamate homoeostasis, impaired action of antipsychotics, and dev
162 , we find that EGF signalling alters protein homoeostasis in adults by increasing UPS activity and po
163 pports sodium/potassium pump-dependent ionic homoeostasis in areas of central nervous system injury.
164 his by revealing the molecular basis of iron homoeostasis in Bacillus subtilis.
165                        The disruption of ion homoeostasis in cancer cells can thus synergize with tar
166 that are vital for the maintenance of axonal homoeostasis in HSP.
167 l insights into the complex role of dopamine homoeostasis in human disease, and understanding of the
168 lays a crucial role in regulation of glucose homoeostasis in mammalian cells.
169 trasound stimulation (pFUS) improves glucose homoeostasis in mice and rats with insulin-resistant dia
170 brain, could explain the perturbed glutamate homoeostasis in MTLE.
171 inal fat relates to abnormalities in glucose homoeostasis in obese adolescents with prediabetes.
172  biosynthesis and maintaining cellular redox homoeostasis in rapidly proliferating cells.
173 n of PE may contribute to the improved lipid homoeostasis in rats on diets high in cholesterol and li
174 ic phenotype switching strategy for adaptive homoeostasis in the adult vasculature.
175 ation from HSCs by maintaining immunological homoeostasis in the bone marrow microenvironment, both i
176 s a dynamic Cl(-) reservoir regulating Cl(-) homoeostasis in the CNS.
177         This study identifies impaired sleep homoeostasis in the form of altered SWA progression duri
178 on receptor (AhR), a key regulator of immune homoeostasis in the gut(1,2).
179 icrobial interaction that maintains protease homoeostasis in the gut.
180 d by the host, thus helping maintain mucosal homoeostasis in the intestine.
181 logic therapy for the restoration of glucose homoeostasis in type-2 diabetes and other metabolic dise
182  metabolic genes and thereby controls energy homoeostasis in vivo.
183 poparathyroidism results in impaired mineral homoeostasis, including hypocalcaemia and hyperphosphata
184 to assess the phases of HSCT: pre-transplant homoeostasis, induced aplasia, early settling and engraf
185 )physiological processes, including cellular homoeostasis, infection propagation, cancer development
186 llular pathways including apoptosis, calcium homoeostasis, inflammation and immunity.
187 rt-term and long-term departures from immune homoeostasis, inhibition of appropriate pathogen recogni
188 cell (SC) proliferation is central to tissue homoeostasis, injury repair, and cancer development.
189 h to explore how circadian rhythms and sleep homoeostasis interact with behavioural and environmental
190 expression of factors crucial for colorectal homoeostasis is affected by physiologic differences in S
191  human metabolism and dysregulation of their homoeostasis is associated with numerous disorders.
192                                      Oxalate homoeostasis is controlled, in part, by the intestinal b
193 regulated gene transcription in adult tissue homoeostasis is currently unknown.
194 rgy metabolism and other aspects of cellular homoeostasis, is hindered by an incomplete understanding
195 ophagy to the maintenance of normal cellular homoeostasis, its changes in neurodegenerative disorders
196  adipose tissue dysfunction, insulin-glucose homoeostasis, lipid disturbances, and cardiovascular dis
197 r, and that interfering with telomere length homoeostasis may be one of the mechanism(s) by which oxi
198 otein; metabolic syndrome score; and glucose homoeostasis measures of fasting insulin, glucose, insul
199 e impact of immune checkpoints on intestinal homoeostasis, mice are challenged with anti-CTLA4 and an
200 failure of dopamine receptor and transporter homoeostasis might underlie the pathophysiology of this
201 p with 15-year changes in bodyweight and the homoeostasis model (HOMA) for insulin resistance.
202  tolerance status, missing data for baseline homoeostasis model assessment (HOMA) indexes, missing da
203 ctories of fasting and 2-h postload glucose, homoeostasis model assessment (HOMA) insulin sensitivity
204 ed fasting blood samples (for calculation of homoeostasis model assessment of insulin resistance [HOM
205 hways involved in nociception, regulation of homoeostasis, modulation of vasodilation, and reward cir
206 all macroalgae and that is important for ion homoeostasis, nutrient uptake and O2/CO2 exchange throug
207 the effects of VCP mutations on the cellular homoeostasis of human induced pluripotent stem cell-deri
208 ed below-ground allocation, and the apparent homoeostasis of radial growth, as ca increases today.
209 e channel that has a key role in maintaining homoeostasis of the airway surface liquid layer in the l
210  Astrocytes, also called astroglia, maintain homoeostasis of the brain by providing trophic and metab
211                            It contributes to homoeostasis of the microenvironment of the central nerv
212 e cells (PSCs) can perturb the biomechanical homoeostasis of the tumour microenvironment to favour ca
213 switching between functional states tuned to homoeostasis or regeneration.
214               Intestinal fungi regulate host homoeostasis, pathophysiological and physiological proce
215 t checkpoint for maintaining oligodendrocyte homoeostasis, pointing to a previously uncharacterized E
216 ion, and indicates that DAXX regulates redox homoeostasis, providing a mechanistic insight into the p
217 ase and Parkinson's disease, changes in iron homoeostasis result in altered cellular iron distributio
218 thways include dysfunction in global protein homoeostasis resulting from abnormal protein aggregation
219 o chronic cell stress and imbalance of ionic homoeostasis, resulting in axonal and neuronal death.
220 lutamatergic, potassium, and neuromodulatory homoeostasis, resulting in dysregulated synaptic transmi
221 s other danger signals that perturb cellular homoeostasis, resulting in host defence responses in the
222 st cells (MCs) play a central role in tissue homoeostasis, sensing the local environment through nume
223 tissues beyond those involved in mineral ion homoeostasis should remain an important focus of researc
224  extrinsic abnormal change affecting calcium homoeostasis stimulating production of parathyroid hormo
225 sms responsible for abnormalities in calcium homoeostasis, the differential diagnosis of hypercalcaem
226 chemical process that is critical for tissue homoeostasis, these results improve our fundamental unde
227 e marrow adipose tissue modulates whole-body homoeostasis through actions on bone cells, haematopoiet
228 ibute to the maintenance of cellular protein homoeostasis through assisting de novo protein folding a
229 ) regulatory T cells (Tregs) maintain immune homoeostasis through mechanisms that remain incompletely
230 uggests that BAT activation improves glucose homoeostasis through several mechanisms, which could poi
231  of a nucleoporin, RanBP2, in maintaining BA homoeostasis through SUMOylation of SHP.
232 p is critical in maintaining telomere length homoeostasis through telomere guanine damage repair, and
233 tion and IL-18 then help maintain epithelial homoeostasis to mediate protection against pre-neoplasti
234 eage-tracing from development, through adult homoeostasis, to fibrosis, to define morphologically and
235 ndocrine function of BAT controlling glucose homoeostasis under this thermoregulatory challenge.
236 biquitin-proteasome system maintains protein homoeostasis, underpins the cell cycle, and is dysregula
237 values, an indicator of neuronal cholesterol homoeostasis, were significantly higher than post-saline
238 gy homeostasis, which acts to restore energy homoeostasis whenever cellular energy charge is depleted
239 e in maintaining whole-body calcium (Ca(2+)) homoeostasis, which is primarily mediated by altering th
240 microbiome maintains a state of basal immune homoeostasis, which modulates immune responses to microb
241  complex regulatory networks, which maintain homoeostasis while accurately distinguishing pathogenic
242 noglycoside sensitivity and phosphoinositide homoeostasis, with important implications for signalling
243 iption at puberty, alters luminal epithelial homoeostasis, yet remains deficient in homologous recomb

 
Page Top