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1 s (Assessment of Telavancin for Treatment of Hospital-Acquired Pneumonia).
2 entilator-associated pneumonia or ventilated hospital-acquired pneumonia).
3 ce of ventilator-associated pneumonia or ICU-hospital-acquired pneumonia.
4 One patient died 13 days after TMVR from hospital-acquired pneumonia.
5 unity-acquired pneumonia and a rare cause of hospital-acquired pneumonia.
6 on use was associated with increased odds of hospital-acquired pneumonia.
7 an important cause of community-acquired and hospital-acquired pneumonia.
8 se was associated with 30% increased odds of hospital-acquired pneumonia.
9 ated pneumonia and 9,747 were at risk of ICU-hospital-acquired pneumonia.
10 mon pathogens associated with fatal cases of hospital-acquired pneumonia.
11 pportunistic pathogen and a leading cause of hospital-acquired pneumonia.
12 ormance for detecting community-acquired and hospital-acquired pneumonia.
13 divided into community-acquired pneumonia or hospital-acquired pneumonia.
14 an improved prognosis, particularly for ICU-hospital-acquired pneumonia.
15 compared to those of their controls; 81% had hospital-acquired pneumonia, 13% had bloodstream infecti
16 monas aeruginosa is the most common cause of hospital-acquired pneumonia and a killer of immunocompro
17 ophylaxis may, however, increase the risk of hospital-acquired pneumonia and Clostridia difficile inf
19 gionnaires' disease is an important cause of hospital-acquired pneumonia and is caused by infection w
20 Staphylococcus aureus is a major cause of hospital-acquired pneumonia and is emerging as an import
21 icular, this bacterium is a leading cause of hospital-acquired pneumonia and is responsible for infec
22 e cohort of patients, we found that both ICU-hospital-acquired pneumonia and ventilator-associated pn
23 e is an important pathogen in the context of hospital-acquired pneumonia, and a better understanding
24 Pseudomonas aeruginosa is a leading cause of hospital-acquired pneumonia, and approximately 80% of pa
25 nal and urinary tract infections, as well as hospital-acquired pneumonia, and for gram-negative infec
26 ition, standardization of antibiotherapy for hospital-acquired pneumonia, and systematic approach to
27 um Pseudomonas aeruginosa, a common cause of hospital-acquired pneumonia, as a model for investigatin
29 ase 3 studies of telavancin for treatment of hospital-acquired pneumonia, cure rates for those with b
32 le data and unanswered questions surrounding hospital-acquired pneumonia, discuss alterations of the
33 a frequent cause of antimicrobial-resistant hospital-acquired pneumonia, especially in critically il
34 ppear to be at increased risk for death from hospital-acquired pneumonia, even after controlling for
36 e existing definitions of pneumonia based on hospital acquired pneumonia (HAP) classifications may be
37 sorders associated with an increased risk of hospital-acquired pneumonia (HAP) and acute respiratory
38 fessionals who care for patients at risk for hospital-acquired pneumonia (HAP) and ventilator-associa
39 an audit on the adherence to guidelines for hospital-acquired pneumonia (HAP) can improve the outcom
42 ening >=4 NIHSS points) before thrombectomy, hospital-acquired pneumonia (HAP) during hospitalization
43 otyped S. aureus isolates from patients with hospital-acquired pneumonia (HAP) enrolled in two regist
44 an audit on the adherence to guidelines for hospital-acquired pneumonia (HAP) for can improve the ou
45 ute respiratory distress syndrome (ARDS) and hospital-acquired pneumonia (HAP) in critically ill pati
48 o characterize the systemic host response in hospital-acquired pneumonia (HAP) when compared with com
50 8 (1.24-1.52); p < 0.0001) and even more ICU-hospital-acquired pneumonia (hazard ratio, 1.82 [1.35-2.
52 ed 150 patients (79.3% male) with sepsis and hospital-acquired pneumonia in 71.7% as the primary indi
53 emphasized the importance of preventing ICU-hospital-acquired pneumonia in nonventilated patients.
54 tion between acid-suppressive medication and hospital-acquired pneumonia in patients with acute strok
56 able logistic regression, the adjusted OR of hospital-acquired pneumonia in the group exposed to acid
57 t Pseudomonas aeruginosa, a leading cause of hospital-acquired pneumonia, interferes with the ability
59 p in all patients with community-acquired or hospital-acquired pneumonias is a crucial measure for ma
60 s (eg, individuals with ventilator-requiring hospital-acquired pneumonia), it is imperative to kill >
61 for ventilator-associated pneumonia and ICU-hospital-acquired pneumonia mortality due to P. aerugino
62 cally significant gastrointestinal bleeding, hospital-acquired pneumonia, mortality, and the use of e
63 ality rates for patients with non-ventilator hospital-acquired pneumonia (NV-HAP) are among the highe
66 ication was ordered in 52% of admissions and hospital-acquired pneumonia occurred in 2219 admissions
67 ve medication was ordered in 1,340 (80%) and hospital-acquired pneumonia occurred in 289 (17.2%).
68 ceptor blockers did not increase the risk of hospital-acquired pneumonia (odds ratio 1.53; 95% confid
69 e included if they had community-acquired or hospital-acquired pneumonia or ventilator-associated pne
72 nsy, and significant decreases were seen for hospital-acquired pneumonia, scarlet fever, pyelonephrit
73 of P. aeruginosa isolates from patients with hospital-acquired pneumonia secreted type III proteins,
74 To determine the strategies of prevention of hospital-acquired pneumonia that reduce mortality in int
78 es of ventilator-associated pneumonia or ICU-hospital-acquired pneumonia were handled as time-depende