ライフサイエンスコーパス: house dust

1 uring Der p 1 weight/weight concentration in house dust.

2 ious studies could detect peanut allergen in house dust.

3 dous to the airways because of inhalation of house dust.

4 products and leaches from the products into house dust.

5 d paint) was the major Pb species present in house dusts.

6 re routes of OHCs, ingestion of OHCs (i) via house dust and (ii) via cat food.

7 of children (n = 17), PBDEs were measured in house dust and child handwipes sampled during a home vis

8 We conclude that exposure to soil, house dust and decaying plant material enhances gut micr

9 We collected house dust and indoor air samples in 49 California homes

10 ilability (RBA) measurements for a subset of house dust and residential soils collected in the AHHS,

11 ) was developed to estimate Pb exposure from house dust and soil, in addition to other potential hous

12 House dust and soils can be major sources of lead (Pb) e

13 were measured in food or water, 67 in air or house dust, and 52 in biospecimens.

14 Paired samples of cat serum, house dust, and cat food were analyzed for brominated fl

15 tably, peanut proteins have been detected in house dust, and their levels correlate with peanut aller

16 ds (PFAAs) that have been widely detected in house dust, aquatic biota, surface water, and wastewater

17 Also, chemical mixtures present in house dust at environmentally relevant levels can activa

18 Absolute soil and house dust bioavailability averaged 33% (SD +/- 4%) and

19 whether hen's egg allergen is detectable in house dust collected from different household areas and

20 Higher house dust concentrations of cockroach, mouse, and cat a

21 House dust contains many organic contaminants that can c

22 House dust contains mite allergens as well as bacterial

23 the first report of in vivo Pb RBA for U.S. house dust contaminated in situ with paint Pb and corrob

24 al synergistic associations of coexposure to house dust endotoxin and ambient air pollutants with ast

25 , the first report of an association between house dust endotoxin and leukocyte count in a national s

26 Coexposure to elevated concentrations of house dust endotoxin and PM(2.5) (CMAQ) was synergistica

27 sion models to test for associations between house dust endotoxin and sensitization to specific foods

28 We investigated the relationship between house dust endotoxin concentration and peripheral leukoc

29 (HDM group) was superior to non-standardized house dust extract (HD group) for subcutaneous immunothe

30 th environmental adjuvants, including common house dust extract (HDE), to test their role in maintain

31 act was more effective than non-standardized house dust extract for subcutaneous immunotherapy; howev

32 Rush immunotherapy with house dust extract is safe and effective for Japanese ch

33 BA were subjected to rush immunotherapy with house dust extract.

34 immunotherapy, rather than non-standardized house dust extract.

35 o evaluate the adipogenic activity of indoor house dust extracts and a suite of semivolatile organic

36 to an innocuous protein using TLR ligands or house dust extracts as adjuvants developed mixed eosinop

37 Ten of 11 house dust extracts exhibited significant triglyceride a

38 Chemical measurements of the FAs in house dust extracts revealed a positive and significant

39 House dust from 17 Swedish homes and serum from the part

40 Bacterial exposure from house dust has been associated with asthma and atopy in

41 Increased pesticide concentrations in house dust in agricultural areas have been attributed to

42 potency for contaminant mixtures as found in house dust, maternal serum, and infant serum; to study w

43 median concentrations for these chemicals in house dust, maternal serum, or infant serum from Nordic

44 HDM allergens with common components of the house dust matrix, through either their binding to aller

45 Microbial composition of house dust may influence allergic outcomes in adults.

46 ouse models of allergic airway inflammation (house dust mice and Alternaria alternata) and OVA-induce

47 House dust microbiome analysis using 16S ribosomal RNA s

48 sion, antenatal environmental tobacco smoke, house dust microbiome, and allergen content (all P < 0.0

49 Here we show by modeling differences in house dust microbiota composition between farm and non-f

50 We sought to examine associations of house dust microbiota with adult asthma, atopy, and hay

51 ere sensitized and challenged with high-dose house dust mite (>10 mug) or with low-dose house dust mi

52 e house dust mite (>10 mug) or with low-dose house dust mite (<3 mug).

53 The prevalence of sensitization to house dust mite (-4.3%; 95% CI, -6.0% to -2.6%) and cat

54 tradermal SIT in children/adults allergic to house dust mite (10 trials), grass pollen or other inhal

55 Balb/c mice were challenged with PBS or house dust mite (HDM) (Days 0, 7, 14-18) and exposed to

56 House dust mite (HDM) acts on the airway epithelium to i

57 The house dust mite (HDM) allergen Der p 13 could be a lipid

58 in crystallographic studies suggest that the house dust mite (HDM) allergen Der p 5 potentially inter

59 a natural field study, sublingual tablets of house dust mite (HDM) allergen extracts (STG320) were ef

60 how endogenous CD4(+) T cells specific for a house dust mite (HDM) allergen form and function.

61 determined early immunological responses to house dust mite (HDM) allergen in offspring challenged f

62 t the safety and efficacy of challenges with house dust mite (HDM) allergen in the Fraunhofer allerge

63 events preceding sensitization to the common house dust mite (HDM) allergen remains to be elucidated.

64 House dust mite (HDM) allergens are a common cause of al

65 Upon inhalation, house dust mite (HDM) allergens are deposited at the nas

66 House dust mite (HDM) allergens are responsible for the

67 Mice were sensitized with house dust mite (HDM) allergens from days 3, 15, or 60 a

68 alyses revealed that IgE from crustaceans or House dust mite (HDM) allergic patients showed cross-rea

69 rly controlled atopic asthma associated with house dust mite (HDM) allergy, relative to non-asthmatic

70 immunotherapy (AIT) lack recommendations for house dust mite (HDM) allergy.

71 kout and wild-type mice were challenged with house dust mite (HDM) and infected with RV1B to determin

72 -mediated allergic CRS mouse model, based on house dust mite (HDM) and Staphylococcus aureus enteroto

73 or WT mice were challenged over 3 weeks with house dust mite (HDM) antigen.

74 emonstrate that Streptococcus pneumoniae and house dust mite (HDM) bear similar phosphorylcholine (PC

75 mice alleviates airway remodeling following house dust mite (HDM) challenge with decreases in mucus

76 istosoma egg antigen (SEA) immunization, and house dust mite (HDM) challenge without affecting cytoto

77 S. mansoni IgG antibodies in extracts of the house dust mite (HDM) Dermatophagoides farinae, the Aust

78 In the house dust mite (HDM) Dermatophagoides pteronyssinus, De

79 inhalation of airborne allergens such as the house dust mite (HDM) effectively activates both innate

80 immunized through the intranasal route with house dust mite (HDM) extract derived from Dermatophagoi

81 House dust mite (HDM) extract is a common trigger of ast

82 We used alphaT-catenin knockout mice and a house dust mite (HDM) extract model of atopic asthma, wi

83 examined constitutive and TNFalpha/IL-1beta, house dust mite (HDM) extract or lipopolysaccharide (LPS

84 ntrol mice were intranasally challenged with house dust mite (HDM) extract or PBS five days per week

85 House dust mite (HDM) extract was used to induce allergi

86 ession in ECs challenged with IL-4/IL-13 and house dust mite (HDM) extract.

87 in mice with prolonged i.n exposure to crude house dust mite (HDM) extract.

88 ced by means of intranasal administration of house dust mite (HDM) extract.

89 on induced by ovalbumin (OVA) in mice and by house dust mite (HDM) in guinea pigs, as well as investi

90 (ECP)] induced by bronchial instillation of house dust mite (HDM) in patients with asthma on mainten

91 A damage and DNA damage responses induced by house dust mite (HDM) in vivo and in vitro.

92 The house dust mite (HDM) is a major perennial allergen sour

93 Sensitization to house dust mite (HDM) is a risk factor for the developme

94 Using a house dust mite (HDM) model of allergic asthma and parab

95 -/-) ) mice were used in ovalbumin (OVA) and house dust mite (HDM) models of AAI.

96 ole of Sema3E in airway angiogenesis using a house dust mite (HDM) murine model of allergic asthma.

97 ty were evaluated in the ovalbumin (OVA) and house dust mite (HDM) murine models.

98 siologically relevant aeroallergens, such as house dust mite (HDM) or Alternaria alternata, to induce

99 Mice were sensitized to house dust mite (HDM) or cockroach at day 0, treated wit

100 ) mice were challenged with saline, DEPs, or house dust mite (HDM) or DEP+HDM.

101 d profiles during type 2 immune responses to house dust mite (HDM) or helminth infection and to ident

102 cells (PBECs) in response to RSV, poly(I:C), house dust mite (HDM) or IL-33 using RT-qPCR, Luminex an

103 As house dust mite (HDM) sensitization is dependent on TLR4

104 airway inflammation was induced following a house dust mite (HDM) sensitization protocol.

105 acy, safety and immunological response of SQ house dust mite (HDM) SLIT-tablet treatment in relation

106 nd during the last 8 weeks of treatment in 2 house dust mite (HDM) SLIT-tablet trials (n = 1768).

107 acebo-controlled, randomized clinical trial, house dust mite (HDM) sublingual AIT was found to be eff

108 The house dust mite (HDM) sublingual allergen immunotherapy

109 The house dust mite (HDM) sublingual immunotherapy (SLIT) ta

110 Mice were exposed to house dust mite (HDM) to provoke Th2-mediated immune res

111 ent mice, challenged with ovalbumin (OVA) or house dust mite (HDM), and accessed for TH2 inflammation

112 l exposure to common human allergens such as house dust mite (HDM), in the absence of additional adju

113 Exposure to environmental antigens, such as house dust mite (HDM), often leads to T helper 2 (Th2) c

114 Exposure to allergens, such as house dust mite (HDM), through the skin often precedes a

115 ng 24 (Trim24) was predicted to be active in house dust mite (HDM)- and helminth-elicited Il4(gfp+)al

116 r D. farinae were assessed in sera from 1302 house dust mite (HDM)-allergic patients living in variou

117 aim was to analyze the role of B cells in a house dust mite (HDM)-based murine asthma model.

118 House dust mite (HDM)-challenged Apoe(-/-) mice display

119 tch signaling induced by DCs is critical for house dust mite (HDM)-driven allergic airway inflammatio

120 igate the therapeutic efficacy of SAHM1 in a house dust mite (HDM)-driven asthma model.

121 ing the Cd11c promotor) to acute and chronic house dust mite (HDM)-driven asthma models.

122 e sought to address the role of B cells in a house dust mite (HDM)-driven TH2-high asthma mouse model

123 e) control mice were evaluated in a model of house dust mite (HDM)-induced AAI.

124 expansion and cytokine production to prevent house dust mite (HDM)-induced airway inflammation and re

125 s adaptive immune responses in patients with house dust mite (HDM)-induced airways disease.

126 sure to DEPs and in response to DEP-enhanced house dust mite (HDM)-induced allergic airway inflammati

127 in patients with AR and in a mouse model of house dust mite (HDM)-induced allergic asthma and whethe

128 evaluating several dosages in patients with house dust mite (HDM)-induced allergic rhinoconjunctivit

129 role of PAG1 in a preclinical mouse model of house dust mite (HDM)-induced allergic sensitization and

130 TJs in the nasal epithelium of patients with house dust mite (HDM)-induced AR and in an HDM-induced m

131 leukocyte infiltration, protecting mice from house dust mite (HDM)-induced asthma or Leishmania major

132 s, an induced psoriasis-like inflammation, a house dust mite (HDM)-induced asthma-like allergic lung

133 ptotic cells by lung phagocytes might dampen house dust mite (HDM)-induced lung inflammation has not

134 current study, we employed a mouse model of house dust mite (HDM)-induced lung inflammation to explo

135 OVA- and house dust mite (HDM)-induced murine asthma models were

136 s and the stimulatory effects of IL-1beta on house dust mite (HDM)-induced release of thymic stromal

137 et (ALK) has been developed for treatment of house dust mite (HDM)-induced respiratory allergic disea

138 e sought to investigate the role of TPL-2 in house dust mite (HDM)-mediated allergic airway inflammat

139 helminth Schistosoma mansoni or the allergen house dust mite (HDM).

140 ans of repeated intranasal administration of house dust mite (HDM).

141 -specific B-cell responses during 2 years of house dust mite AIT were compared between responder and

142 ild-type mice with Aspergillus fumigatus and house dust mite allergen and compared the effects on air

143 NC/Nga mice were sensitized with house dust mite allergen and treated topically with HOCl

144 In addition, we discuss of house dust mite allergen extracts as a prototypical comp

145 Diverse factors contribute to house dust mite allergenicity through the activation of

146 levels of IgE specific for staple foods and house dust mite allergens in DOCK8-deficient patients an

147 junction downregulation in a mouse model of house dust mite allergic airway inflammation.

148 House dust mite allergics showed IgE reactivity only to

149 Since 2015, for patients with house dust mite allergies, we used a standardized house

150 Specifically, for patients with house dust mite allergies, which are often underestimate

151 th tablets containing carbamylated monomeric house dust mite allergoids was to determine the most eff

152 Chronic exposure to house dust mite and A alternata were compared in a neona

153 Age-specific prevalence of sensitization to house dust mite and cat did not differ between year-of-b

154 lower levels of sensitization, especially to house dust mite and cat, after the age of 20 years.

155 sthma and rhinitis, focusing on responses to house dust mite and grass.

156 ysteine protease and major allergen from the house dust mite and is associated with allergic rhinitis

157 After exposure to house dust mite antigen, Zbtb46-negative CD64(+) inflamm

158 oup of mice was also intranasally exposed to house dust mite antigen.

159 House dust mite concentrations varied across cohorts.

160 zing intranasal doses of an extract from the house dust mite Dermatophagoides farinae (Df) sharply in

161 one of the most important allergens from the house dust mite Dermatophagoides pteronyssinus Identific

162 fungal driven and proinflammatory, the other house dust mite driven and chemokine dominant, with the

163 velopment and TH2 polarization, as seen in a house dust mite exposure model.

164 hyperoxia promoted allergic TH responses to house dust mite exposure.

165 This study hypothesizes that standardized house dust mite extract (HDM group) was superior to non-

166 ing dimaprit in both the ovalbumin (OVA) and house dust mite extract (HDM) murine models of respirato

167 s over 3-weeks to either saline, DEP, and/or house dust mite extract (HDM).

168 lveolar lavage fluid of mice challenged with house dust mite extract after oral administration (50 mg

169 Mice were chronically exposed to house dust mite extract and then infected with influenza

170 dust mite allergies, we used a standardized house dust mite extract for subcutaneous immunotherapy,

171 patients), the initial dose of standardized house dust mite extract was 1 JAU or less.

172 Standardized house dust mite extract was more effective than non-stan

173 model was exposed to allergens (ovalbumin or house dust mite extract) to decipher in vivo the implica

174 House dust mite extract, Alternaria alternata extract, o

175 In rSCIT using standardized house dust mite extract, lowering the target dose at the

176 ation in mice following acute challenge with house dust mite extract.

177 a decrease in skin prick test reactivity to house dust mite from 7.0 +/- 1.3 to 2.7 +/- 0.5 mm (P =

178 to any allergen was present in 17.2% and to house dust mite in 8.7%.

179 life and as allergen levels of dog, cat, and house dust mite in bed dust samples at 1 year.

180 Using prolonged exposure to house dust mite in mice, we developed a mouse model of p

181 When challenged with house dust mite in vivo, Gimap5-deficient mice displayed

182 ical features of allergic asthma provoked by house dust mite in vivo.

183 Our data indicate that exposure to house dust mite markedly reduces Sema3E expression in mo

184 he spatiotemporal dynamics of Tr1 cells in a house dust mite model of allergic airway inflammation.

185 In the house dust mite model, Tfr cells repress the production

186 es to allergic airway disease using a murine house dust mite model.

187 We used ovalbumin and house dust mite models of asthma.

188 us to interrogate the role of T(FR) cells in house dust mite models.

189 Asthma was induced in mice using intranasal house dust mite or aerosol ova-albumin challenge, and ch

190 emale BALB/c mice were repeatedly exposed to house dust mite or Alternaria alternata three times a we

191 ice by repeated intranasal administration of house dust mite or the fungal allergen Alternaria altern

192 Group 21 and 5 allergens are homologous house dust mite proteins known as mid-tier allergens.

193 A and EAACI Asthma Guidelines (separated for house dust mite SCIT, SLIT tablets and SLIT drops; patie

194 neutrophilic asthma via different methods of house dust mite sensitization and challenge.

195 We used a model of house dust mite sensitization to challenge wild-type, Bc

196 l and maternal history of atopy, eczema, and house dust mite sensitization.

197 man atopic dermatitis skin lesions with high house dust mite sensitization.

198 nd difficult to diagnose, the efficacy of SQ house dust mite sublingual immunotherapy tablets has bee

199 Treatment with SQ (standardised quality) house dust mite sublingual tablet for 1 year resulted in

200 acerbation day (from 11% [placebo] to 5% [SQ house dust mite sublingual tablet]) and an increased pro

201 a mild AR day (from 16% [placebo] to 34% [SQ house dust mite sublingual tablet]).

202 ecent publications were identified by using "house dust mite" as a key search term to evaluate the cu

203 models of experimental asthma (ovalbumin and house dust mite); miRNAs deregulated in both models were

204 y to the age of 7 with asthma, atopy (grass, house dust mite, and cat skin prick test) and atopic vs.

205 Alternaria alternata, Aspergillus fumigatus, house dust mite, and ovalbumin) for 4 wk.

206 acute challenge with Alternaria alternata or house dust mite, and secretion of IL-33 and activation o

207 serum specific IgE to common aeroallergens (house dust mite, cat, and grass) and total IgE levels we

208 articipants underwent skin prick testing for house dust mite, cat, grasses and moulds.

209 he airways upon intranasal immunization with house dust mite, confirming the ability of IL-9-producin

210 at least one of the allergens: birch, grass, house dust mite, or cat.

211 allergens from Aspergillus fumigatus and the house dust mite, resulting in an asthma-like pathology c

212 otein D (rfhSP-D) has been shown to suppress house dust mite- and Aspergillus fumigatus-induced aller

213 We use experimental models of house dust mite- or ovalbumin-induced airway inflammatio

214 ybridoma technology using human B cells from house dust mite-allergic patients was used to identify f

215 ll tolerated and reduces the CPT reaction in house dust mite-allergic patients.

216 nd IgA2 contribute to the clinical status of house dust mite-allergic patients.

217 ing and IL-3-secreting Th cells were high in house dust mite-allergic patients.

218 eriostin were also decreased in the lungs of house dust mite-challenged ERp57-deleted mice.

219 duced by 6.2-fold in pulmonary epithelium of house dust mite-challenged mice.

220 -specific Notch deficiency in mice prevented house dust mite-driven eosinophilic airway inflammation

221 To evaluate the use of house dust mite-impermeable bedding and its impact on se

222 Here we used a murine model of house dust mite-induced (HDM-induced) allergic inflammat

223 Experimental acute canine house dust mite-induced AD lesions exhibit an activation

224 During house dust mite-induced airway allergy, rEos features re

225 House dust mite-induced airway inflammation was assessed

226 TRPC1 intensifies house dust mite-induced airway remodeling by facilitatin

227 hat both DNA-HSP65 and CpG/CFP downregulated house dust mite-induced allergic airway inflammation via

228 contrast, in Th2-polarized settings such as house dust mite-induced allergic airway inflammation, th

229 ted the role of hBD2 in a steroid-sensitive, house dust mite-induced allergic airways disease (AAD) m

230 ed the impact of PKM2 on the pathogenesis of house dust mite-induced allergic airways disease in C57B

231 rresponsiveness, and mucus production during house dust mite-induced allergic asthma.

232 ltration into the airways of mice undergoing house dust mite-induced allergic response.

233 rolled dose-finding study, 131 patients with house dust mite-induced allergic rhinoconjunctivitis wer

234 y delivery of NP-CpG could prevent and treat house dust mite-induced allergy by modulating immunity d

235 ing primary cell assays and a mouse model of house dust mite-induced asthma, we compared IL-4 vs IL-1

236 ation was assessed in a mouse model of acute house dust mite-induced asthma.

237 aptive immune responses were dispensable for house dust mite-induced endoplasmic reticulum stress and

238 nce recovered, subjected to an ovalbumin- or house dust mite-induced experimental asthma protocol.

239 Blockade of Runx2 inhibited the house dust mite-induced goblet cell differentiation with

240 and MCTR3 inhibited lung eosinophilia after house dust mite-induced inflammation.

241 determined in the setting of ovalbumin- and house dust mite-induced lung inflammation.

242 ema3e(-/-) mice into WT recipients increases house dust mite-induced Th2/Th17 inflammation in the air

243 ting epithelial cells and dendritic cells of house dust mite-sensitized mice to dampen IFN-beta expre

244 cross all populations and at different ages, house dust mite-specific IgG/IgE ratios (but not IgG4/Ig

245 at the baseline and after sensitization with house dust mite.

246 asthma via regulation of immune response to house dust mite.

247 t of a commonly studied airway allergen, the house dust mite.

248 ivate GPCRs such as Alternaria alternata and house dust mite.

249 and respiratory challenge with an extract of house dust mite.

250 and asymptomatic sensitization to pollen and house dust mite.

251 House dust mite/HDM atopy patch test/APT elicits positiv

252 response is dominated by a single allergen (house dust mite; HDM).

253 nes were confirmed in reporter assays and in house-dust-mite (HDM) induced AAI and primary human bron

254 ceptors, substantially reduced ovalbumin- or house-dust-mite-induced airway inflammation and bronchia

255 In vivo, loss of T(FR) cells increased house-dust-mite-specific IgE and lung inflammation.

256 the genome, transcriptome and microbiome of house dust mites (HDM) has shown that Staphylococcus aur

257 House dust mites (HDM) may serve as carriers of bacteria

258 ithelial cells, BEAS-2B, directly exposed to house dust mites (HDM) resulted in enhanced DNA damage,

259 The most frequent allergens are house dust mites (HDM), which act in vivo on the bronchi

260 (AIT) with an allergoid in the treatment of house dust mites (HDM)-induced allergic rhinitis and/or

261 irome in medically important mites including house dust mites (HDM).

262 Twenty-three adults allergic to house dust mites (HDMs) (M+) and 15 nonsensitive, nonall

263 House dust mites (HDMs) are among the most important all

264 House dust mites (HDMs) are sources of an extensive repe

265 Allergic rhinitis induced by house dust mites (HDMs) is a highly prevalent but often

266 veloped countries demonstrate sensitivity to house dust mites (HDMs).

267 uted allergens, including those derived from house dust mites (HDMs).

268 al allergens including insect allergens from house dust mites and cockroaches contribute to allergic

269 nalyses for a reduction in SPT reactivity to house dust mites and perennial allergens.

270 xtracellular traps potentiated the uptake of house dust mites by CD11b(+)Ly-6C(+) dendritic cells and

271 House dust mites have been implicated in the etiology an

272 Some allergens such as derived from house dust mites have proteolytic activity which can ind

273 eous application of Dermatophagoides farinae house dust mites to sensitized atopic dogs.

274 Allergic sensitization to cat, dog, and house dust mites was diagnosed longitudinally using skin

275 redominately caused by sensitization against house dust mites with a nearly complete penetrance of th

276 tory allergens (ie, grass, olive/ash pollen, house dust mites), specific IgE did not show marked diff

277 rse, (2) timothy grass/birch, (3) molds, (4) house dust mites, (5) peanut/wheat flour/mugwort, (6) pe

278 plicate a high exposure to indoor allergens (house dust mites, pets, molds, etc), tobacco smoke, and

279 and allergic airway inflammation induced by house dust mites, pulmonary function and cytokine profil

280 irected against staple food antigens but not house dust mites.

281 eatment for local allergic rhinitis (LAR) to house dust mites.

282 with allergic rhinitis and sensitization to house dust mites.

283 otective high dose of LPS before exposure to house dust mites.

284 allergic airway inflammation in response to house dust mites.

285 volved include seasonal or perennial such as house dusts mites, pollens, animal epithelia, moulds (al

286 We show here that sensitization of mice with house-dust mites (HDMs) in the presence of low-dose lipo

287 The adipogenic activity in house dust occurred at concentrations below EPA estimate

288 It has been detected in house dust of several European countries according to re

289 esults suggest that many SVOCs ubiquitous in house dust, or their metabolites, are possible PPARgamma

290 ng a better understanding of determinants of house dust Pb bioavailability.

291 al, and residential use exposure pathways to house dust pesticide concentrations: meta-regression of

292 t correlations between paired hand wipes and house dust samples were observed for PFOS, PFOA, and 6:2

293 most prevalent PFAS in both fire station and house dust samples, with medians of approximately 100 ng

294 nd Technology (NIST) organic contaminants in house dust standard reference material (SRM).

295 lytes were also detected and quantified in a house dust standard reference material, SRM 2585, demons

296 sh, whereas median endotoxin levels in Amish house dust was 6.8 times as high.

297 e abundance of a number of bacterial taxa in house dust was associated with increased or decreased as

298 verall diversity of bacterial communities in house dust was similar by asthma status but was lower (P

299 al community structure and concentrations in house dust were determined by using next-generation DNA

300 When compared to paired hand wipes and house dust, wristbands were found to have similar or gre