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1 etabolites of microbial communities from the human colon.
2 f this important family of bacteria from the human colon.
3 em to be involved in the excretory system of human colon.
4 thylation patterns of cells sampled from the human colon.
5 the complex multicellular environment of the human colon.
6 istently identified within mouse and healthy human colon.
7 ommon anatomical alterations detected in the human colon.
8 ernal surfaces, and tortuous 3D shape of the human colon.
9 lar, cellular, and morphologic properties of human colon.
10 sion assays and ex vivo using recellularized human colon.
11 and peptide YY (PYY) in vitro from mouse and human colons.
12 feration, migration and viability assays and human colon adenocarcinoma (Caco-2, ATCC, Rockville, MD)
14 hat eIF3i expression was upregulated in both human colon adenocarcinoma and adenoma polyps as well as
19 eening a kinase/phosphatase shRNA library in human colon adenocarcinoma HT-29 cells, we found that kn
20 and in vivo, in a cisplatin resistant HT-29 human colon adenocarcinoma model, with and without the p
21 ion was used to analyze levels of miR-627 in human colon adenocarcinoma samples and nontumor colon mu
29 ression were inversely correlated in primary human colon and breast cancers, and in intestinal adenom
33 terleukin 6 (IL6) and its receptor (IL6R) in human colon and lung cancer cell lines by flow cytometry
35 croarray, and quantitative RT-PCR studies of human colon and pancreatic tumors demonstrating signific
37 y expressed on the surface epithelium of the human colon and was observed to colocalize with TcdB in
42 sed interleukin 23 (IL-23) protein levels in human colon biopsy specimens positive for C. difficile t
43 at FOXA1 is robustly expressed in the normal human colon but significantly downregulated in colon ade
44 roduction from (13) C-labelled fibres in the human colon by measurement of (13) C-labelled SCFA conce
45 study, we investigated EHEC adherence to the human colon by using in vitro organ culture (IVOC) of co
46 A), was implemented in different cell lines: human colon cancer (Caco-2), human hepatocarcinoma (HepG
48 acrophages; its genotoxicity was assessed in human colon cancer (HCT116) and primary murine colon epi
49 s (P1 and P2), we generated Tet-On-inducible human colon cancer (HCT116) cell lines that express eith
51 ntly, treatment of mouse xenograft models of human colon cancer and multiple myeloma resulted in tumo
53 and visualized in a xenograft mouse model of human colon cancer by PET, which demonstrates that this
56 s and their glutathione conjugates, prevents human colon cancer cell growth in culture as well as in
57 d between the theory and experiments using a human colon cancer cell line (COLO205) as the capture ta
58 brane was tested by confocal microscopy in a human colon cancer cell line exclusively expressing Nox1
68 ng, and exerted antiproliferative effects in human colon cancer cell lines and CRC cells isolated by
69 cytoplasm and nuclei of several established human colon cancer cell lines and this localization patt
76 nhanced RIP140 expression strongly repressed human colon cancer cell proliferation in vitro and after
78 l, with an IC(50) of 24.43 microM in HCT-116 human colon cancer cells and an IC(50) of 25.82 microM i
79 d in the lamellipodial leading edges of HT29 human colon cancer cells and are colocalized with aquapo
80 effect of anthocyanin-rich plant extracts on human colon cancer cells and determine their mechanism o
82 ation of THAP11-dependent gene expression in human colon cancer cells and suggest that the THAP11-HCF
83 SAHA increase phosphocholine (PC) levels in human colon cancer cells and tumor xenografts as observe
85 of excess R2 subunits protects p53-deficient human colon cancer cells from cisplatin-induced DNA dama
88 Here, we reveal that silencing GUCY2C in human colon cancer cells increases Akt-dependent TGF-bet
89 rane-facing glutamate with glutamine renders human colon cancer cells largely resistant to ceramide-i
93 lopment of acquired cross-drug resistance of human colon cancer cells that harbor different genetic b
95 pounds on growth and antioxidative status in human colon cancer cells was investigated in this study.
96 rowth of primary tumors formed by murine and human colon cancer cells was reduced in mice by genetic
98 y to cause cytotoxicity to HCT-116 and HT-29 human colon cancer cells with ED50 values of 134.4 and 2
99 ng strand cleavage and growth suppression in human colon cancer cells with G12D or G12V mutations, th
100 mab synergistically reduced the viability of human colon cancer cells with wild-type but not mutant K
102 ivation of beta-catenin and proliferation of human colon cancer cells, and increased tumor growth in
103 mRNA and protein abundance twofold in HCT116 human colon cancer cells, and induction was further enha
104 the nucleus of primary colorectal tumors and human colon cancer cells, and oncogenic activation of Ra
105 aused cytotoxicity to Caco-2, HT-29, HCT-116 human colon cancer cells, and reduced inflammatory respo
107 nly enhanced cancer cell killing in cultured human colon cancer cells, but also improved antitumor ac
109 best in a luciferase knockdown study in HT29 human colon cancer cells, which were found to be more di
124 py, here we find that cohesin depletion in a human colon cancer derived cell line results in endomito
125 formation of mouse epidermal cells JB6 Cl41, human colon cancer DLD-1, breast cancer T-47D, and melan
126 ory microenvironment and studied circulating human colon cancer HCT116 cells in response to a variety
127 owth of mouse xenograft tumours derived from human colon cancer HCT116 cells that contain wild type p
130 findings establish a new role for LIN28B in human colon cancer pathogenesis, and suggest LIN28B post
133 ohistochemical analysis of approximately 450 human colon cancer specimens (Stage III) reveals that P1
134 plementary experimental approaches and using human colon cancer specimens, human and murine colon can
137 notypic heterogeneity between two iso-clonal human colon cancer sublines HCT116 and HCT116b on their
138 oth human colon tumor cell lines and primary human colon cancer tissue xenografted into immunodeficie
142 destruction-replenishment) was performed in human colon cancer xenografts (n = 38) by using a clinic
143 e form of matriptase in vivo was measured in human colon cancer xenografts and in a patient-derived x
144 ation on antiangiogenic treatment effects in human colon cancer xenografts compared with ex vivo refe
145 nd reproducible to assess tumor perfusion in human colon cancer xenografts in mice and allows for ass
147 Materials and Methods Twenty-three mice with human colon cancer xenografts were randomized to receive
148 ectal cancer often differ significantly from human colon cancer, being largely restricted to the smal
161 multiple monkey kidney fibroblasts (MKF) and human colon cancerous (HCT-8) cells in close proximity,
162 otein, and is stabilized in both APC-mutated human colon cancers and Apc(min/+) intestinal polyps.
164 oli (APC) gene is mutated in the majority of human colon cancers and often occurs simultaneously with
165 tro, and that gene-specific 5-hmC changes in human colon cancers are directly correlated with changes
166 deletion mutation in beta-catenin present in human colon cancers both weaken tumor intercellular Ecad
168 and RIP140 protein levels were decreased in human colon cancers compared with those in normal mucosa
169 Consistent with these findings, analyses of human colon cancers demonstrated increased Dsg2 protein
172 dependency signature" is enriched in primary human colon cancers with mutations in both APC and KRAS,
173 thylated in Cancer 1 (HIC1), often occurs in human colon cancers, among others, via CpG island hyperm
174 n was significantly increased in BRAF(V600E) human colon cancers, and patients whose tumors had high
175 of the PC secretory pathway was observed in human colon cancers, only furin showed highly diffuse ex
176 , we investigated the expression of eIF3i in human colon cancers, tested its contribution to colon on
185 CAM accumulated on the lateral interfaces of human colon carcinoma and normal intestinal epithelial c
186 rrelation between MET and TNS4 expression in human colon carcinoma and ovarian carcinoma suggests TNS
190 olyploid cells resulting from the culture of human colon carcinoma cell lines or primary mouse epithe
191 inked glycosylation profiles of the isogenic human colon carcinoma cell lines SW480 (primary tumor) a
192 ay high, structure-dependent toxicity to the human colon carcinoma cell-line HCT116 p53(++), causing
193 ar basis of intracellular Ca(2+) handling in human colon carcinoma cells (HT29) versus normal human m
194 in effective sensitization of the metastatic human colon carcinoma cells to FasL-induced apoptosis.
195 city and potent activity in sensitization of human colon carcinoma cells to FasL-induced apoptosis.
197 ingle-strand breaks, activated DNA repair in human colon carcinoma cells, and effectively suppressed
207 eration, we tested TOP1mt KO fibroblasts and human colon carcinoma HCT116 cells and measured mtDNA af
208 investigate global DNA methylation changes, human colon carcinoma HCT116 cells, which were hypomorph
209 tion in two human embryonic stem cell lines, human colon carcinoma line HCT116, and mouse embryonic s
211 thermore, berberine suppresses the growth of human colon carcinoma xenograft in nude mice in an RXRal
213 ainst MCF-7 (human breast carcinoma), HT-29 (human colon carcinoma), A2780 (human ovarian carcinoma),
215 l models: mouse hepatocellular carcinoma and human colon carcinoma, whereby the metabolism has been p
217 L17A, IL22, and interferon-gamma by cultured human colon CD3-negative, IL7-receptor-positive cells.
218 oxicity as measured using an in vitro normal human colon cell (NCM460) assay, compared to chlorinatio
219 ion to IBD map to HNF4A binding sites in the human colon cell line CaCo2, suggesting impaired HNF4A b
220 Compounds 5c and 5j, tested in vitro on the human colon cell line HT-29, blocked the growth of cells
222 t GH suppresses p53 and reduces apoptosis in human colon cell lines as well as in induced human pluri
226 Mitochondrial dysfunction was induced in human colon-derived epithelial cell lines or colonic bio
227 es in expression of alpha-1 adrenoceptors in human colon emphasize the translational relevance of our
229 RRalpha are not detectable in nontransformed human colon epithelial cells, and depletion of the AMPKg
231 RT1 was also detected in the goblet cells of human colon epithelial tissue and primary culture of col
232 rons projecting to the guinea pig rectum and human colon expressed alpha-synuclein in their axons.
235 three-stage continuous-culture model of the human colon (gut model), which represents different anat
236 t strains of Escherichia coli present in the human colon have been linked to the initiation of inflam
237 uate the activities against proliferation of human colon HCT116 and mammary adenocarcinoma MCF-7 canc
238 jected intravenously into nude mice carrying human colon HCT116 tumors efficiently suppressed tumor g
240 tempt at modelling microbial dynamics in the human colon incorporating both uncertainty and adaptatio
242 c device with the ability of detecting a few human colon invasive cancer cells (SW48) in a mixed cell
246 gh there are many factors that determine the human colon microbiota composition, diet is an important
248 biofilm-positive, but not biofilm-negative, human colon mucosal homogenates induced colon tumor form
250 orthotopic organoid transplantation model of human colon organoids engineered to harbor different CRC
251 technology to delete key DNA repair genes in human colon organoids, followed by delayed subcloning an
257 2'-deoxycytidine, was quantified in diseased human colon samples and found to be present at levels si
258 f phosphatidylcholines in mouse brain and in human colon samples with the desorption laser spot size
259 -5 mm) were stitched into 1 freshly resected human colon specimen and followed by an ex vivo molecula
263 normal and inflammatory bowel disease (IBD) human colon tissue as well as hyperplasia human tonsils.
264 three surrogate peptides in trypsin-digested human colon tissue homogenates; and a small-molecule dru
265 lial electrical resistance (TEER) in primary human colon tissues and Caco-2 cells in vitro through up
268 es in mice and single-cell RNA sequencing in human colon to investigate how sympathetic postganglioni
270 Wnt inhibitors are specifically cytotoxic to human colon tumor biopsy cultures as well as colon cance
271 ll growth, invasion, and metastasis, of both human colon tumor cell lines and primary human colon can
272 or B4 (EPHB4) is aberrantly overexpressed in human colon tumor cell lines and selectively required fo
274 PXR bound to the FGF19 promoter in both human colon tumor cells and "normal" intestinal crypt ce
275 CD133-positive (CD133(+)) subpopulations of human colon tumor cells that exhibited more potent tumor
276 ly correlated with H2O2-stress signatures in human colon tumor cohorts, but positively correlated wit
277 verexpression decreased the proliferation of human colon tumor epithelial cells and blunted H(2)O(2)-
278 els of NTR1, miR-21, and miR-155 increase in human colon tumor samples and correlate with tumor stage
279 R-21, and miR-155 increased significantly in human colon tumor samples, compared with normal tissues,
282 d expression of KIT in cultured cells and in human colon tumor xenografts and this contributed to the
283 ed sphere formation and viability of primary human colon tumor-initiating cells harboring mutant KRAS
284 undifferentiated colonosphere cultures from human colon tumors and used them to generate stably diff
288 vival, while elevated expression of TIGAR in human colon tumors suggested that deregulated TIGAR supp
290 issue microarrays from 151 paraffin-embedded human colon tumors, with adjacent normal and adenoma tis
296 systematically more than 70 pairs of primary human colon tumours by applying next-generation sequenci
299 al success in habitats such as the rumen and human colon where nitrogen is rarely limiting for growth
300 olonizes and forms stable A/E lesions on the human colon, which are likely to contribute to intestina
301 ingle immune cells across the healthy, adult human colon, with paired characterization of immune cell