ライフサイエンスコーパス: hunger

1 s sense the fed or fasted state and regulate hunger.

2 tivated, they rapidly (within minutes) drive hunger.

3 egulated in an opposite fashion and decrease hunger.

4 to drive food consumption during periods of hunger.

5 olutions in mice, independently of thirst or hunger.

6 ed food cues drive feeding in the absence of hunger.

7 sing neurons in the arcuate nucleus regulate hunger.

8 amic sites to drive eating in the absence of hunger.

9 the impacts of the price increases on global hunger.

10 nctional exponent of ARC(AgRP) neuron-driven hunger.

11 ting that circuit selection is determined by hunger.

12 re downstream of the metabolic regulation of hunger.

13 , buy their medications, and alleviate their hunger.

14 la melanogaster to identify neurons encoding hunger.

15 etite and food consumption in the absence of hunger.

16 likely play a more general role in encoding hunger.

17 sitive to shifts in sucrose concentration or hunger.

18 dictive cues elicit eating in the absence of hunger.

19 mplicit bargain to ensure each other against hunger.

20 e hypothalamus are crucial to the control of hunger.

21 hich can be modified by internal states like hunger.

22 regulating hormones to transiently suppress hunger.

23 e global health and socio-economic impact of hunger.

24 hen activates AgRP neurons to fully increase hunger.

25 ppreciated step in homeostatic regulation of hunger.

26 l craving, similar to the way fasting causes hunger.

27 general discomfort (18% vs. 42%; P = 0.010), hunger (44% vs. 67%; P = 0.024), and weakness (22% vs. 4

28 ts with high RMRs showed increased levels of hunger across the day (P < 0.0001) and greater food inta

29 these afferent neurons capable of triggering hunger advances understanding of how this intense motiva

30 Hunger affects the behavioral choices of all animals, an

31 al ingestion, and the return of appetite and hunger after fasting.

32 cantly increased gastric volumes and reduced hunger (all P < 0.001).

33 2030 for a baseline scenario that would meet hunger alleviation targets for over 9 billion people.

34 Hunger also modulates behaviors related to food seeking

35 tode Caenorhabditis elegans to elucidate how hunger alters valence.

36 s of cognitive restraint, disinhibition, and hunger and 1-y changes in these eating behaviors predict

37 HGI postexercise meal suppresses feelings of hunger and augments postprandial fullness sensations mor

38 Hunger and chronic undernourishment impact over 800 mill

39 procedures successfully increase subjective hunger and decrease blood glucose, we do not find signif

40 ings, fructose versus glucose led to greater hunger and desire for food and a greater willingness to

41 After each block, participants rated hunger and desire for food.

42 palatable food across varying conditions of hunger and food palatability in male rats.

43 d to monitor gastrointestinal complaints and hunger and fullness scores.

44 Appetite-related sensations (i.e., hunger and fullness) were recorded by visual analog scal

45 s (bitter, sweet, and umami) on food intake, hunger and fullness, gastrointestinal symptoms, and gast

46 analog scales were used to assess ratings of hunger and fullness.

47 intake, but the high-egg group reported less hunger and greater satiety postbreakfast.

48 Lower hunger and higher fullness ratings were seen premeal and

49 Moreover, DB decreases hunger and increases satiety ratings after a meal and sh

50 tinal physiology that contribute to reducing hunger and increasing sensations of satiation.

51 food can stimulate feeding in the absence of hunger and lead to maladaptive overeating behavior.

52 ederal legislation and programs to alleviate hunger and malnutrition, improve consumers' nutrition kn

53 r 1a (GHSR1a), in the hypothalamus to signal hunger and promote food intake.

54 Hunger and prospective consumption dose-dependently decr

55 ion selection to promote food seeking during hunger and safety during satiety.

56 nvestigated brain response to rewards during hunger and satiated states to examine whether diminished

57 No differences were observed in hunger and satiation curves between morbidly obese and n

58 the metabolic transitions that occur during hunger and satiation, and provides a platform to study t

59 loss, serum obesity-related hormone levels, hunger and satiety assessments, and quality of life (QOL

60 nize and respond appropriately to children's hunger and satiety cues can lead to "normal" weight gain

61 l Rather than Emotional Reasons, Reliance on Hunger and Satiety Cues, and Unconditional Permission to

62 Instead, hunger and satiety homeostatically modulate the microstr

63 ges that occur during the transition between hunger and satiety in Drosophila melanogaster.

64 gnitive neurocircuitry did not differ during hunger and satiety in the RAN group.

65 , motilin and ghrelin plasma concentrations, hunger and satiety ratings, and food intake in healthy v

66 Accordingly, hunger and satiety regulate adult neurogenesis by modula

67 imilar energy intake, the SD group had lower hunger and satiety scores compared with the bvFTD group.

68 In contrast, hunger and satiety scores did not differ between the bvF

69 Neuronal circuits regulating hunger and satiety synthesize information encoding the e

70 Key factors driving eating behavior are hunger and satiety, which are controlled by a complex in

71 pha-MSH, provides insight into regulation of hunger and satiety.

72 All participants completed surveys on hunger and satiety.

73 timing did not affect rhythms of subjective hunger and sleepiness, master clock markers (plasma mela

74 Hunger and snack food intake were reduced only after a l

75 lly stimulated, they potently induce intense hunger and subsequent food intake.

76 The patients had a sustainable reduction in hunger and substantial weight loss (51.0 kg after 42 wee

77 vagal tone (heart rate variability (HRV)) on hunger and the postprandial response to GL.

78 e that STROBE-driven behavior is modified by hunger and the presence of taste ligands, and find that

79 leviate the negative valence associated with hunger and the rewarding properties of food discovery ev

80 ompared with the sham group, VAS ratings for hunger and the urge to eat declined significantly more (

81 Hunger and thirst are ancient homeostatic drives for foo

82 Hunger and thirst have distinct goals but control simila

83 tly being uncovered, less is known about how hunger and thirst interact.

84 PeriLC(VGLUT2) neurons are a hub between hunger and thirst that specifically controls motivation

85 oals should be pursued (e.g., when balancing hunger and thirst) and how to combine these signals with

86 ar abundance signals and oppositely regulate hunger and thirst.

87 during two physiological deficiency states: hunger and thirst.

88 events following motivational shifts between hunger and thirst.

89 exture of food in the mouth independently of hunger and thus of reward value and pleasantness.

90 rupted, it is often by routine sensations of hunger and urge.

91 satiety, while the ghrelin receptor promotes hunger and weight gain.

92 eptions of dietary restraint, disinhibition, hunger, and control of eating.

93 ed forebrain areas known to mediate feeding, hunger, and satiation while minimally affecting brainste

94 stry of Social Development and Fight Against Hunger, and the Information System for the Public Budget

95 ement in people's food pleasantness ratings, hunger, and weight.

96 king energy state to the motivational drive, hunger, and, finally, limbic and cognitive processes tha

97 Hunger/appetite scores decreased in the first 2 weeks af

98 sible mechanism for how various responses to hunger are coordinated.

99 The neural mechanisms underlying hunger are poorly understood.

100 mines how the energy deficits that result in hunger are represented in the brain and promote feeding

101 The motivational states of thirst and hunger are represented in the brain by shared and unique

102 ttenuates the rewarding effects of food- and hunger-associated brain stimulation reward.

103 an independent role of oleoylethanolamide in hunger-associated interoceptive signaling.

104 orted greater decreases in disinhibition and hunger at 1- and 10-y follow-ups (all P < 0.001 in both

105 significant relation between eating rate and hunger at the end of the meal or up to 3.5 h later.

106 inated impulsive action in DRL engendered by hunger, at a dose (1 mug) that significantly affected ne

107 8 min, respectively; P < 0.05), although the hunger AUC was not statistically different.

108 wer levels of 6-mo and 1-y disinhibition and hunger (beta = 0.13-0.29, P < 0.01 in men; beta = 0.11-0

109 obesity agents by lowering the set-point for hunger between meals.

110 nts, intake that is not only associated with hunger but also the incentive value of food.

111 BG was related to hunger but only in low disinhibited eaters.

112 combination of tastants inhibits feelings of hunger, but only the latter also reduces food intake.

113 Both solid and higher viscous food reduce hunger by - 4.97 mm (95% confidence interval (CI) - 8.13

114 Ending all forms of hunger by 2030, as set forward in the UN-Sustainable Dev

115 Hypoglycemia, which also stimulates hunger by activating CNS neurons, albeit independently o

116 asingly making efforts to reduce poverty and hunger by pursuing sustainable energy and agricultural p

117 food preference driven by nutrient-specific hunger can be essential for survival, yet little is know

118 In addition, hunger can promote the expression of food-associated mem

119 regulated behavior that integrates taste and hunger cues to balance food intake with metabolic needs.

120 tion in mice with caloric-deficiency-induced hunger decreases feeding.

121 The neuronal mechanisms underlying hunger-dependent behavioral plasticity are not fully cha

122 tain a cellular-level understanding of these hunger-dependent cortical response biases, we performed

123 mushroom-body-innervating DANs to facilitate hunger-dependent sugar memory expression.

124 ing attributes of acetic acid by virtue of a hunger-dependent switch in their behavioral response to

125 ly activated by starvation and evoke intense hunger-display electrical and biochemical hyperactivity

126 but the mechanisms underlying this sustained hunger drive remain unknown (Chen et al., 2016).

127 sting these cells may supply the fundamental hunger drive that motivates feeding.

128 Hunger, driven by negative energy balance, elicits the s

129 A microanalysis of hunger-driven and palatability-driven feeding was carrie

130 ssion of SLC5A11 is sufficient for promoting hunger-driven behaviors and enhancing the excitability o

131 ssion in SLC5A11-expressing neurons produces hunger-driven behaviors even in fed flies, mimicking the

132 neurons is sufficient to promote feeding and hunger-driven behaviors; silencing these neurons has the

133 another OA receptor previously implicated in hunger-driven exuberant sugar intake.

134 Malnutrition or hidden hunger due to micronutrient deficiencies affects about o

135 height and weight, eating in the absence of hunger (EAH) at 5 y, and inhibitory control (a measure o

136 Hunger enhances sensitivity to reward, yet individuals w

137 is is true for 2 forms of low leptin-induced hunger, fasting and poorly controlled type 1 diabetes.

138 an be modulated by behavioral states such as hunger, fear, stress, or a change in environmental conte

139 ulating eating behavior as well as decreased hunger feelings and diminished incentive value of food.

140 olved in this association between subjective hunger feelings and gastrointestinal motility during the

141 tilin-induced phase III contractions induced hunger feelings through a cholinergic pathway.

142 in China has been associated with a growing hunger for energy consumption and steadily-increasing CO

143 shares common neural mechanisms with that of hunger for food.

144 ically cause hypertension, but it promotes a hunger for salt and a transition from salt resistance to

145 sential prerequisites for ending poverty and hunger, for improving institutions and participation in

146 The Healthy, Hunger-Free Kids Act of 2010 (prepolicy period: 2007-201

147 ss-sectional study of students, the Healthy, Hunger-Free Kids Act of 2010 was associated with better

148 The Healthy, Hunger-Free Kids Act of 2010, implemented nationwide in

149 After implementation of the Healthy Hunger-Free Kids Act, change was associated with signifi

150 5 months after implementation of the Healthy Hunger-Free Kids Act.

151 s in change for BMI, household wealth index, hunger, general health perception score, or adherence to

152 In contrast, during hunger, gustatory inputs enter the hypothalamus and driv

153 Hunger had a similar opposite effect on insula-to-ventra

154 in the ventral caudal putamen indicated that hunger had opposite effects on tastant response in the c

155 y, circadian misalignment also increased the hunger hormone ghrelin by ~8% during wake periods in fem

156 Since ghrelin was discovered as a pro-hunger hormone, many additional roles for it have been i

157 manipulating central receptors for the gut "hunger" hormone ghrelin.

158 ir similarity for the meal-induced change in hunger (ICC: 0.41; P = 0.03), fullness (ICC: 0.39; P = 0

159 Behavioral tests demonstrated that perceived hunger, importance of eating, eating frequencies, and li

160 ial neural insensitivity to these effects of hunger in anorexia nervosa.

161 pamine circuit that encodes protein-specific hunger in Drosophila The activity of these neurons incre

162 s reveal a unique role for NPY in sustaining hunger in the interval between food discovery and consum

163 ional regulation of key neurons that control hunger in vivo.

164 forms of obesity characterized by excessive hunger, including melanocortin-4 receptor deficiency, th

165 cerbated if male quality declines and female hunger increases in declining populations.

166 Although hunger-induced behavioral changes are well documented, t

167 (1 mug) that significantly affected neither hunger-induced PR enhancement nor hyperactivity.

168 show that a metabolic challenge by cold and hunger induces diurnality in otherwise nocturnal mice.

169 study was to elucidate how the hypothalamic hunger-inducing hormone acyl-ghrelin (AG), which is also

170 y chemical 'obesogens' that drive adiposity, hunger, inflammation and suppress metabolism.

171 We hypothesized that increasing hunger intensity would curb expression of social behavio

172 Hunger is a complex motivational state that drives multi

173 Hunger is a hard-wired motivational state essential for

174 Hunger is a powerful drive that stimulates food intake.

175 The sensation of hunger is caused by an imbalance between energy intake a

176 Hunger is controlled by specialized neural circuits that

177 One immediate response to hunger is increased food consumption.

178 value in that they respond to food only when hunger is present and in that activations correlate line

179 Although progress is being made, how hunger is represented in the brain and how it coordinate

180 With regard to hunger, it is thought that leptin-sensing neurons work e

181 o have two core components-experience (e.g., hunger, joy) and agency (e.g., planning, self-control).

182 , these pathways are recruited at increasing hunger levels, such that low-risk changes (higher sugar

183 de (AgRP)) bypasses these signals to restore hunger-like response patterns in insular cortex.

184 tization of species for aquaculture to fight hunger, malnutrition and micronutrient deficiency; ultim

185 ectors, could play a big role in eradicating hunger, malnutrition and nutrient-deprivation globally.

186 OMC) neurons are extremely slow in affecting hunger (many hours).

187 sed sensitivity to the motivational drive of hunger may explain the ability of individuals with AN to

188 bic and cognitive processes that bring about hunger-mediated increases in reward value and perception

189 e molecular and cellular mechanisms by which hunger modulates neural circuit function to generate cha

190 ty of the sGPNs in the AMMC, suggesting that hunger modulates the responsiveness of the secondary swe

191 teracts with visual processing, by examining hunger modulation of food-associated visual responses in

192 These findings reveal that hunger neurons drive feeding by transmitting a positive

193 triggers broad dysregulation of hypothalamic hunger neurons that is incompletely reversed by weight l

194 ex, chemogenetic activation of hypothalamic 'hunger neurons' (expressing agouti-related peptide (AgRP

195 al N = 795), we examine the effects of acute hunger on prosociality in a wide variety of non-interdep

196 oss all tasks reveals a very small effect of hunger on prosociality in non-interdependent tasks (d =

197 ucose, we do not find significant effects of hunger on prosociality.

198 Hunger or appetite decreased for 4 weeks after embolizat

199 weight on quality of life (IWQOL-Lite), and hunger or appetite using a visual assessment scale.

200 ce and was not explained simply by increased hunger or lower satiety.

201 ting that this system is not involved in the hunger or satiety mechanisms that govern normal feeding.

202 ays that modulate the physiological state of hunger or satiety.

203 Accordingly, while artificial induction of hunger or thirst in sated mice via activation of specifi

204 During artificial hunger or thirst, food or water cues further shifted act

205 During natural hunger or thirst, food or water cues rapidly and transie

206 Selection of hunger- or thirst-appropriate memory emerges from compet

207 Hunger (P = 0.001) and desire to eat (P = 0.001) ratings

208 intragastric administration of DB decreased hunger (P = 0.008) and increased satiety ratings (P = 0.

209 ot differ significantly, yet fluctuations in hunger (P = 0.019) and desire to eat (P = 0.026) over th

210 ANOVA confirmed that subjectively rated hunger (P = 0.569; SED: 3.8), fullness (P = 0.404; SED:

211 signals originating inside the body, such as hunger, pain and heart rate.

212 the frequency and length of their strides if hunger pangs compete with their desire to see cherry blo

213 ions were also significantly associated with hunger peaks (P < 0.05), and this association involved a

214 phase III contractions on the occurrence of hunger peaks and the involvement of a cholinergic pathwa

215 e sought to 1) investigate the occurrence of hunger peaks and their relation to phase III contraction

216 The occurrence of hunger peaks and their significant association with phas

217 An algorithm was developed to detect hunger peaks.

218 Thirst, but not hunger, potentiated the phasic dopamine response to the

219 Here we describe the in vivo dynamics of hunger-promoting AgRP neurons during the development of

220 trient infusion rapidly and durably inhibits hunger-promoting AgRP neurons in awake, behaving mice.

221 her cell types, potently and durably inhibit hunger-promoting AgRP neurons in the hypothalamus.

222 gnaling hormones (e.g., higher levels of the hunger-promoting hormone ghrelin) or hedonic brain respo

223 e from the satiety hormone leptin toward the hunger-promoting hormone ghrelin.

224 The strongest observations were in men for hunger (r = 0.69, P = 0.002) and desire to eat (r = 0.61

225 sting GLP-1, fasting PYY, PYY responses, and hunger-rating responses to the test drink (P < 0.05).

226 ch to the duodenum (P = 0.001) and decreased hunger ratings (P = 0.04).

227 Despite similar hunger ratings due to fasting in both conditions, partic

228 istration decreases both antral motility and hunger ratings during the fasting state, possibly becaus

229 In the morning and evening, hunger ratings increased (both P = 0.02), whereas rating

230 ect on fasting gastrointestinal motility and hunger ratings, motilin and ghrelin plasma concentration

231 ence concurrent energy intake and subjective hunger ratings.

232 nt, we analyzed the hierarchical position of hunger related to rival drive states.

233 In states of hunger relative to satiety, the rewarding value of food

234 Instead, stimulation of hunger requires an intervening endocrine step, namely ac

235 The relation between hunger, satiation, and integrated gastrointestinal motil

236 assess the effects of sleeve gastrectomy on hunger, satiation, gastric and gallbladder motility, and

237 Fasting and postprandial hunger, satiation, hormone concentrations, and gastric a

238 Although physiological state (e.g., hunger, satiety) and associated hormones are known to af

239 Interactions of Group (RAN, CW) x Visit (hunger, satiety) revealed that, for the CW group, hunger

240 mg pure capsaicin) in healthy volunteers on hunger, satiety, and gastrointestinal symptoms and the r

241 ual analog scale scores were used to measure hunger, satiety, and gastrointestinal symptoms.

242 re not observed to have different effects on hunger, satiety, lipid profiles, or other inflammatory a

243 sive behavioral testing assessing changes in hunger/satiety regulation were performed during the firs

244 Internal physiological factors, such as hunger, scale signals encoded by dopaminergic neurons an

245 combination of tastants and umami decreased hunger scores compared with placebo.

246 strate oxidation, core temperature, cold and hunger scores, and plasma parameters were repeated every

247 strate oxidation, core temperature, cold and hunger scores, or plasma parameters were found, nor was

248 nt goals (SDGs): "no poverty" (SDG 1), "zero hunger" (SDG 2), and "health and well being" (SDG 3).

249 l LCN2 serum levels correlate inversely with hunger sensation in challenged subjects.

250 than after the LGI meal (P < 0.001), whereas hunger sensations were approximately 9% lower after the

251 utcome) and alter body temperature, cold and hunger sensations, plasma metabolic parameters, and ener

252 Hunger shifts the behavioral response from aversion to a

253 Ghrelin is the only known hunger signal derived from the peripheral tissues.

254 were excited by food deprivation and the gut hunger signal ghrelin.

255 e III contractions have been identified as a hunger signal.

256 ) pro-opiomelanocortin (POMC)-expressing and hunger signaling (orexigenic) agouti-related peptide (Ag

257 cused on the pathways underlying satiety and hunger signaling, while other studies have concentrated

258 Ghrelin, a hunger signalling peptide derived from the peripheral ti

259 between leucokinin and other neuromodulatory hunger signals at the level of the DANs.

260 orexigenic peptide ghrelin (Ghr) stimulates hunger signals in the hypothalamus via growth hormone se

261 r, satiety) revealed that, for the CW group, hunger significantly increased activation in reward sali

262 battery of behavioral assays, we found that hunger significantly increases its capacity to suppress

263 ally precise ingestion that was regulated by hunger state and sucrose concentration.

264 stronger food cue biases and sensitivity to hunger state and trial history.

265 We found that as hunger state escalated, resident animal social interacti

266 afish behavior and expose many ways in which hunger state influences their action selection to promot

267 circuit that integrates gustatory input and hunger state to modulate food ingestion.

268 ed by the fish as a function of its internal hunger state, behavioral history, and the locations and

269 y for common confounders, like, for example, hunger state, menstrual phase, and BMI, as well as how t

270 ssing dKCNQ channels, thereby conferring the hunger state.

271 active or repulsive depending on an animal's hunger state.

272 lating chemosensory valence as a function of hunger state.

273 sm by which these neurons rapidly coordinate hunger states with glucose homeostasis.

274 Furthermore, intense hunger states, especially those evoked via AgRP photoact

275 es of foods promote and guide consumption in hunger states, whereas satiation should dampen the senso

276 n the insular cortex of behaving mice across hunger states.

277 eption by tracking and predicting thirst and hunger states.

278 Why choose suicide bombing rather than a hunger strike or self-immolation?

279 r arms had equal access to an Action Against Hunger-supported programme.

280 ing lunch and in an eating in the absence of hunger task.

281 calories from snack foods in the absence of hunger than did children in a control group.

282 ief stimulation of AgRP neurons can generate hunger that persists for tens of minutes, but the mechan

283 duce motivational drives, such as thirst and hunger, that regulate behaviors essential to survival.

284 Eradicating world hunger-the aim of Sustainable Development Goal 2 (SDG2)-

285 ostatic and physiological variables, such as hunger, thirst, and effort levels, by orchestrating sens

286 ogical processes and ingestive drives (i.e., hunger, thirst, and salt appetite).

287 inhibitor that not only could satisfy 'drug hunger' through its slow-onset long-lasting DAT inhibito

288 d weight loss (control, daily self-weighing, hunger training, diet/exercise app, brief support).

289 However, the molecular details of how hunger triggers changes in the activity of neural circui

290 ndings, people hold strong lay theories that hunger undermines prosociality.

291 ty in the ventral striatum when curiosity or hunger was elicited, which extends into the dorsal stria

292 Premeal hunger was lower before dinner with the LCBF than with t

293 Ratings of hunger were decreased during nightshift days despite dec

294 Blood glucose (BG) and hunger were measured 30 and 150 min after consumption of

295 haviors (i.e., eating even in the absence of hunger), which were predictive of having a high body mas

296 -/perinatal famine exposure during the Dutch Hunger Winter of 1944-1945 and mortality through age 63

297 eptional exposure to famine during the Dutch Hunger Winter.

298 improved gastrointestinal symptoms and less hunger with GMP-MFs.

299 including alertness, social recognition, and hunger, yet, their mechanism of action is poorly underst

300 l's flagship social protection program, Zero Hunger (ZH), which aims to reduce food insecurity and po