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1 trations of hCG 6 months after evacuation of hydatidiform mole.
2  concentrations 6 months after evacuation of hydatidiform mole.
3 orms are derived from the precursor lesions, hydatidiform moles.
4 hat these tumors are not related to complete hydatidiform moles.
5 nal-triploidy and the development of partial hydatidiform moles.
6 d to investigate the involvement of LIGHT in hydatidiform moles.
7 ostic methods for accurate classification of hydatidiform moles.
8 eference-standard DNA mixtures of homozygote hydatidiform mole and heterozygote blood DNA at varying
9 , NLRP7 and NLRP2, cause familial biparental hydatidiform mole and multilocus imprinting disturbance,
10 e genome-wide restriction maps of a complete hydatidiform mole and three lymphoblast-derived cell lin
11 iform moles (purely androgenetic in complete hydatidiform moles and diandric triploid in partial hyda
12 egnancy-related disorders, such as recurrent hydatidiform moles and fetal growth restriction, and thu
13 emalignant disorders of complete and partial hydatidiform mole, and the malignant disorders of invasi
14                                              Hydatidiform moles are intriguing pathologic entities re
15                                              Hydatidiform moles are known to pose an extremely high r
16 haliana, Drosophila melanogaster and a human hydatidiform mole cell line (CHM1) from SMRT sequencing.
17 read technologies, but it was derived from a hydatidiform mole cell line with a nearly homozygous gen
18 sed 77 twin pregnancies, comprising complete hydatidiform mole (CHM) and healthy co-twin, to ascertai
19 is based on the use of a homozygous complete hydatidiform mole (CHM) as the reference.
20  for alpha satellite containing reads in the hydatidiform mole (CHM1, haploid-like) genome.
21 used genomic DNA from an essentially haploid hydatidiform mole, CHM1.
22 ong-read nanopore sequencing of the complete hydatidiform mole CHM13 genome, combined with complement
23 g centromeric satellite arrays in a complete hydatidiform mole (CHM13).
24                                     Complete hydatidiform moles (CHMs) are diploid tumors that result
25 diagnosis, routine morphologic assessment of hydatidiform moles continues to suffer from interobserve
26                     Rare familial biparental hydatidiform moles (due to NLRP7 or KHDC3L mutations) sh
27 rations 6 months after uterine evacuation of hydatidiform mole, even when values are falling.
28                          Familial biparental hydatidiform mole (FBHM) is the only known pure maternal
29        We used data generated from a haploid hydatidiform mole genome (CHM1) and a diploid human geno
30             76 (<1%) of 13,960 patients with hydatidiform moles had persistently high hCG concentrati
31 nd sFlt-1 by ELISA in 17 women with complete hydatidiform mole (HM) and 20 gestational-age-matched no
32                                              Hydatidiform mole (HM) is an abnormal gestation characte
33 entify novel genes responsible for recurrent hydatidiform moles (HMs), we performed exome sequencing
34 development of the conceptus into a complete hydatidiform mole in which extraembryonic trophoblastic
35 istinguishable from an androgenetic complete hydatidiform mole, in which abnormal extra-embryonic tis
36 eotide polymorphism markers in most complete hydatidiform moles indicating that these tumors are not
37 t elevated LIGHT in the trophoblast cells of hydatidiform mole induces sFlt-1, which might underlie t
38           The most frequent type of complete hydatidiform mole is a 46, XX homozygote formed by the f
39 iform moles and diandric triploid in partial hydatidiform moles) is a fundamental genetic event leadi
40      The homozygous nature of these complete hydatidiform moles makes them unique resources for human
41 niparental tissues of germline origin, i.e., hydatidiform moles (paternal origin) and complete ovaria
42                                      Partial hydatidiform moles (PMs) rarely require chemotherapy and
43 sentation of the paternal genome in sporadic hydatidiform moles (purely androgenetic in complete hyda
44                        We leverage a haploid hydatidiform mole to identify highly identical sequences
45 esent evidence of homozygosity of a complete hydatidiform mole using 20 diallelic markers distributed
46             These diseases vary from partial hydatidiform mole, which rarely metastasizes and infrequ