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1 structed, thereby protecting the kidney from hydronephrosis.
2 ereas non-surviving null mice exhibit marked hydronephrosis.
3  fluid-filled due to the severe polyuria and hydronephrosis.
4  obstruction of urine outflow and death from hydronephrosis.
5 e cystic kidney disease, renal fibrosis, and hydronephrosis.
6 ued ectopic Ptch2 expression and obstructive hydronephrosis.
7 ther during gestation or postnatally, caused hydronephrosis.
8 tion, bladder enlargement, gut dilation, and hydronephrosis.
9 ower likelihood of spontaneous resolution of hydronephrosis.
10 , and variably expressive phenotypes such as hydronephrosis.
11 age to enteral feeding solutions to treating hydronephrosis.
12 ascular invasion, micropapillary disease, or hydronephrosis.
13 strate the predominant role of cPLA2alpha in hydronephrosis.
14 ching or in distal ureter maturation, and no hydronephrosis.
15  with associated hydroureter, megaureter and hydronephrosis.
16 n between the expression of these miRNAs and hydronephrosis.
17  genes in mutant ureters before the onset of hydronephrosis.
18 indicated in infants with isolated antenatal hydronephrosis.
19 he ureter and this is followed by congenital hydronephrosis.
20 ary tract was affected by diverticulosis and hydronephrosis.
21 logy increases with the severity of prenatal hydronephrosis.
22 s of alkaline phosphatase, urolithiasis, and hydronephrosis.
23 ation of the child with prenatally diagnosed hydronephrosis.
24 of bladder, and the development of end-stage hydronephrosis.
25 h wild-type mice, despite similar degrees of hydronephrosis.
26 elvic junction obstruction, a common form of hydronephrosis.
27 been most often applied to the evaluation of hydronephrosis.
28 ations may contribute to hereditary forms of hydronephrosis.
29 onal knockout mice have renal hypoplasia and hydronephrosis.
30 ruction, poor renal function and significant hydronephrosis.
31 tomotic stenosis (6.8% vs. 0.4%, P=0.02) and hydronephrosis (12.9% vs. 5.3%, P=0.02) in the SKT cohor
32  stage (755 [90%] vs 255 [90%]), presence of hydronephrosis (97 [12%] vs 27 [10%]), and receipt of ne
33 e from the kidney to the bladder can lead to hydronephrosis, a common birth defect associated with ob
34 cess can result in malpositioned ureters and hydronephrosis, a common cause of renal disease in child
35 ctile function may underlie diseases such as hydronephrosis, a deleterious condition that can cause s
36 e Pax3-Cre transgene) resulted in congenital hydronephrosis accompanied by reduced branching, abnorma
37 ry tumor smaller than 7 cm, no or unilateral hydronephrosis, adequate bladder function, and lack of m
38 h stricture of the transplant ureter causing hydronephrosis and allograft dysfunction, whose ureteral
39 uplex and multiplex kidneys, renal agenesis, hydronephrosis and cystic kidney disease.
40 Analysis of Ret mutant mice at birth reveals hydronephrosis and defective ureter maturation, abnormal
41 esoderm-derived renal progenitors results in hydronephrosis and failure to develop a patent pelvic-ur
42 gressive renal hemosiderosis concurrent with hydronephrosis and glomerulonephritis.
43 can of the chest, abdomen, and pelvis showed hydronephrosis and hydroureter with marked cortical thin
44 ult of obstruction without manifestations of hydronephrosis and it can be confused with leakage from
45 occur in humans, including renal hypoplasia, hydronephrosis and mega-ureter, abnormalities also seen
46 aneous recessive mutation that causes severe hydronephrosis and obstructive nephropathy in affected m
47                      Three patients had mild hydronephrosis and one had distal hydroureter; these con
48 efective pacemaker cell differentiation with hydronephrosis and provide a cellular basis for one of t
49 s most commonly applied to the evaluation of hydronephrosis and provides valuable insight into a wide
50  variant histology, lymphovascular invasion, hydronephrosis and/or high-grade upper tract disease(3-5
51 nically relevant abnormality (eg, unilateral hydronephrosis and/or urolithiasis in patients with an o
52 ficient mice exhibit a dilated renal pelvis (hydronephrosis) and a small papilla.
53 nine of urinary tract complaints (three with hydronephrosis), and five of toxic shock syndrome after
54 rifices resulting in the back flow of urine, hydronephrosis, and altered renal function indicators.
55 ular peritoneal thickening, lymphadenopathy, hydronephrosis, and bowel obstruction were less common a
56 s sharply reduced, apparently as a result of hydronephrosis, and fractional excretion of electrolytes
57 sents early with intestinal hypoperistalsis, hydronephrosis, and hydroureters.
58 -fold increased sensitivity to cleft palate, hydronephrosis, and lethality.
59  truncation, vertebral segmentation defects, hydronephrosis, and limb hypoplasia, resembling humans w
60 mass, retroperitoneal lymphadenopathy, right hydronephrosis, and mesenteric tumor deposits ( Fig 1A )
61 ary tumours less than 7 cm, no or unilateral hydronephrosis, and no extensive or multifocal carcinoma
62 ing late gestation expansion of the bladder, hydronephrosis, and rapid demise after parturition.
63 sent with enuresis, urinary-tract infection, hydronephrosis, and voiding dysfunctions as a result of
64 nd disrupted in conditions like diabetes and hydronephrosis, appears to facilitate synchronized respo
65 treating children with a history of prenatal hydronephrosis are included in this review.
66          The underlying causes of functional hydronephrosis are not well understood.
67 aluation of the CT studies by using proximal hydronephrosis as a criterion for minimal invasion impro
68 sensitive to dioxin-induced cleft palate and hydronephrosis as compared with embryos from an Ahr(+/+)
69 genesis and function causing hydroureter and hydronephrosis at birth.
70 at, when abnormal, can cause obstruction and hydronephrosis at birth; whether ND defects underlie sim
71        Loss of Rab35 leads to nonobstructive hydronephrosis because of loss of ureter epithelium.
72  caused bilateral UPJ obstruction and severe hydronephrosis beginning at embryonic day 17.5.
73 arge tumor suppressor, results in congenital hydronephrosis characterized by urinary tract abnormalit
74         miR-143/145-deficient mice developed hydronephrosis, characterized by severe papillary atroph
75                       Congenital progressive hydronephrosis (cph) is a spontaneous recessive mutation
76 onounced glomerulosclerosis was evident, and hydronephrosis developed with low penetrance.
77 o lack of mature germ cells, and 50% develop hydronephrosis due to ureteral hyperplasia.
78 rvals to detect complications like calculus, hydronephrosis, etc.
79 did not enable discrimination of obstructive hydronephrosis from unobstructive hydronephrosis in chil
80 more commonplace, the management of neonatal hydronephrosis has become one of the most interesting an
81  cell-mediated ureteritis, leading to kidney hydronephrosis (hereafter called acetate-induced renal d
82                                    Antenatal hydronephrosis (HN) impacts up to 5% of pregnancies and
83                       Dlg1-null mice exhibit hydronephrosis, hydroureter, and occasionally hypoplasti
84 om bladder epithelial cells causing prenatal hydronephrosis, hydroureter, and vesicoureteric reflux.
85 CBA) dams led to cleft palate in only 8% and hydronephrosis in 69% of embryos.
86  examination revealed the presence of severe hydronephrosis in almost all animals, affecting lean as
87                                       Kidney hydronephrosis in C2RD was caused by ureteral obstructio
88 bstructive hydronephrosis from unobstructive hydronephrosis in children.
89  influence the incidence of cleft palate and hydronephrosis in developing mice exposed to the polluta
90 ound examination revealed a hilar mass, with hydronephrosis in five and stenosis of renal vessels in
91 ing of the molecular basis of nonobstructive hydronephrosis in humans.
92 rodibenzo-p-dioxin) induces cleft palate and hydronephrosis in mice, when exposed in utero; these eff
93 urinary concentrating ability, polyuria, and hydronephrosis in mice.
94 s in supernumerary kidneys, megaureters, and hydronephrosis in mice.
95 benzo-p-dioxin (TCDD) induces nonobstructive hydronephrosis in mouse neonates through upregulation of
96 yonic day 9 (E9) led to palatal clefting and hydronephrosis in nearly 100% of embryos by E17.
97 or UOC when there is no evidence of relevant hydronephrosis in the ultrasonography.
98              SFU classification was used for hydronephrosis in ultrasound reports, and UTD classifica
99 ticularly TE, with occult (=without relevant hydronephrosis in US) UOC and renal outcomes.
100 areful observation of patients with moderate hydronephrosis in well functioning kidneys.
101                    The diagnosis of prenatal hydronephrosis, indications for and timing of prenatal i
102                                              Hydronephrosis is a common disease characterized by dila
103                                     Prenatal hydronephrosis is diagnosed with an incidence of 1:100 t
104              The natural history of prenatal hydronephrosis is difficult to determine, and therefore
105 nts with a history of any degree of prenatal hydronephrosis is routine, but ascertaining which patien
106                                        Fetal hydronephrosis is the most common anomaly detected on an
107                   The presence and degree of hydronephrosis is very important in the management of ma
108 hibited severe polyuria (10 ml/day), extreme hydronephrosis, low plasma potassium, high blood pH, hyp
109        Loss of Rab35 leads to nonobstructive hydronephrosis, making the Rab35 mutant mouse a novel ma
110 ction must be treated, it is also clear that hydronephrosis may well exist without significant obstru
111 de VUR (n = 2), renal dysplasia (n = 2), and hydronephrosis (n = 1).
112 dings were cholelithiasis (n=3), obstructive hydronephrosis (n=1), small-bowel dilatation (n=1), and
113 , high plasma potassium, metabolic acidosis, hydronephrosis of varying severity, and high plasma reni
114 as defined as stable serum creatinine and no hydronephrosis on follow-up.
115 ts between kidneys with increasing degree of hydronephrosis (P > .5).
116 l reflux (P=0.02) and four of four developed hydronephrosis (P=0.002).
117       The most common findings detected were hydronephrosis, pelviectasis, and dilated ureter.
118                CT findings (tissue rim sign, hydronephrosis, perinephric fat stranding, perinephric f
119 culus increases the risk of hematuria and/or hydronephrosis, presenting with colicky pain as in the p
120 sitional cell carcinoma of the renal pelvis, hydronephrosis proximal to the tumor may cause overstagi
121         The postnatal management of prenatal hydronephrosis remains controversial.
122 knockouts experienced vesicoureteral reflux, hydronephrosis, renal dysfunction, and, in the offspring
123 nary outflow tract obstruction, hydroureter, hydronephrosis, renal failure and reduced bladder capaci
124 esence of a tissue rim sign and the grade of hydronephrosis, renal fascial thickening, and renal pare
125 ings represented 8.7% (131/1509) among which hydronephrosis represented 23% (30/131).
126 cedure-related major complications occurred (hydronephrosis requiring nephrostomy due to gross hematu
127 gnostic challenge in discriminating COU from hydronephrosis secondary to vesicoureteral reflux, parti
128 machinery in the mutants before the onset of hydronephrosis suggest that the congenital obstructive n
129                         One patient had mild hydronephrosis that resolved.
130 have no renin mRNA expression in the kidney, hydronephrosis, thickening of renal arterial walls, and
131 , were hypertension (13 [7%] vs eight [4%]), hydronephrosis (three [2%] vs seven [4%]), back pain (fi
132 reening has changed from simple detection of hydronephrosis to selection for specific diagnosis-based
133 ation of the pathway leading to the onset of hydronephrosis using the TCDD-exposed mouse model will d
134  indomethacin-rescued -/- adults except that hydronephrosis was mild.
135            The incidence of cleft palate and hydronephrosis was not significantly different in fetuse
136                                     Proximal hydronephrosis was present in 80% of overstaged cases (e
137           The incidences of cleft palate and hydronephrosis were assessed and genomic DNA from embryo
138                        Narrowed ureters with hydronephrosis were found only in the Tl1a transgenic mi
139 dred twenty-six children suspected of having hydronephrosis were hydrated prior to undergoing both co
140  whose first ultrasound scans for congenital hydronephrosis were performed between 2008 and 2011.
141 The impaired bladder function and subsequent hydronephrosis were secondary to involvement of the cent
142                       Young null mutants had hydronephrosis, were severely dehydrated, and approximat
143 l morphology with no evidence of significant hydronephrosis, whereas non-surviving null mice exhibit
144 ry tract abnormalities, including congenital hydronephrosis, which is the leading cause of renal fail
145 renal/ureteral duplication, hydroureter, and hydronephrosis, which were visible prenatally.
146 n of all children with a history of prenatal hydronephrosis with a voiding cystourethrogram may reduc
147 ren with a history of any degree of prenatal hydronephrosis with an ultrasound and voiding cystoureth
148 red with degree of obstruction and degree of hydronephrosis with analysis of variance.
149 canning 5 weeks posttransplant revealed mild hydronephrosis with several parenchymal cystic areas mea
150 ent date, age, carcinoma in situ status, and hydronephrosis) with propensity scores to patients who u
151 of Six1 in the ureter led to hydroureter and hydronephrosis without anatomical obstruction when kidne
152 ction is the most common cause of congenital hydronephrosis, yet the underlying pathogenesis is undef

 
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