ライフサイエンスコーパス: hydroperoxide

1 anganese in the presence of TBHP (tert-butyl hydroperoxide).

2 dation products (conjugated dienes and lipid hydroperoxides).

3 ed cyclization cascade of a chiral bis(epoxy)hydroperoxide.

4 ies suggested to be either a superoxide or a hydroperoxide.

5 from oxidative stress induced by tert-butyl hydroperoxide.

6 ificantly increased resistance to tert-butyl hydroperoxide.

7 f Ohr in bacterial responses toward distinct hydroperoxides.

8 ility of OhrR to sense intracellular organic hydroperoxides.

9 enocysteine, leading to accumulation of PUFA hydroperoxides.

10 which oxidize polyunsaturated fatty acids to hydroperoxides.

11 alize olefins to afford a new class of alkyl hydroperoxides.

12 (Gpx4) is a major scavenger of phospholipid hydroperoxides.

13 hydroperoxide lyase (HPL) action on linoleyl hydroperoxides.

14 by the iron-dependent accumulation of lipid hydroperoxides.

15 1 failed to give metabolites with any of the hydroperoxides.

16 e peroxides, peroxynitrite, and phospholipid hydroperoxides.

17 olecule scavengers of amino acid and protein hydroperoxides.

18 inhibiting the formation of TBARS and lipid hydroperoxides.

19 the reduction of hydrogen peroxide and lipid hydroperoxides.

20 iary benzylic carbons are oxygenated to give hydroperoxides.

21 that protects from the accumulation of lipid hydroperoxides.

22 ssays for quantification of formaldehyde and hydroperoxides.

23 actions of the Criegee intermediates to form hydroperoxides.

24 t of Pseudomonas aeruginosa toward different hydroperoxides.

25 hydroxides was clearly favoured over that of hydroperoxides.

26 xidases were equally sensitive to fatty acid hydroperoxides.

27 ow involves oxidation of complex 2 by Pt(IV) hydroperoxide 4.

28 duced from oxidation of isoprene 4-hydroxy-3-hydroperoxide (4,3-ISOPOOH) under low-NO conditions, wer

29 ilic dehydration products of the cholesterol hydroperoxides, 4-, 6-, and 7-ketocholesterol.

30 cholesterol (chol) autoxidation gives chol 7-hydroperoxide (7-OOH) as the sole primary product is sho

31 tom substituted cyclic alkenes by tert-butyl hydroperoxide (70% TBHP in water) using catalytic dirhod

32 the ChOOH 3beta-hydroxycholest-5-ene-7alpha-hydroperoxide (7alpha-OOH) in liposomes, stimulated cell

33 d, octanoic acid, nonanoic acid), along with hydroperoxides (9- and 13-hydroperoxy-octadecadienoylgly

34 Cells treated with tert-butyl hydroperoxide, a known ROS stimulus, were also evaluated

35 The total hydroperoxides accumulated along the 10 days of fermenta

36 e almost comparable to derivatives of flavin hydroperoxide acting as oxidizing agents in monooxygenas

37 The tryptophan-derived hydroperoxide acts in vivo as a signalling molecule, ind

38 water and a high (>1,000 mmol O(2)/kg lipid hydroperoxides after 4 weeks) in gels with > 2.4% water

39 id oxidation was low (~30 mmol O(2)/kg lipid hydroperoxides after 6 weeks) in gels with < 0.23% water

40 -dicyano-p-benzoquinone) or TBHP (tert-butyl hydroperoxide), along with FeCl24 H2O (10 mol %).

41 rmination of the differing relative rates of hydroperoxide and acid formation with changes in hydroca

42 hemistry, generated by the presence of cumyl hydroperoxide and Cu(2+) in solution, when supplemented

43 mation were identified, the iminium ion, the hydroperoxide and dimer of THIQ, and a new ring opened i

44 We replaced hydrogen peroxide with t-butyl hydroperoxide and found that, although the palladium eno

45 Prx1 to hyperoxidation caused by both urate hydroperoxide and hydrogen peroxide.

46 amino acids after incubation with tert-butyl hydroperoxide and hypochlorous acid in vitro, we identif

47 Reaction of an azulichlorin with tert-butyl hydroperoxide and KOH in dichloromethane/methanol gave a

48 duction and evolution reactions, and lithium hydroperoxide and lithium hydroxide are identified as pr

49 oxidants, such as hydrogen peroxide, cumene hydroperoxide and menadione, compared to the OS strains.

50 and methylene produced negative effects, and hydroperoxide and nitrate groups produced the smallest a

51 The lipid hydroperoxide and p-Anisidine values of emulsions genera

52 d derivative failed to react with tert-butyl hydroperoxide and potassium hydroxide, demonstrating tha

53 Furthermore, fatty acid hydroperoxide and SIN-1 both induced Ohr expression in t

54 Based on hydroperoxide and TBARS analysis, sinapic acid and rutin

55 to extend the lag phase to 20 days for lipid hydroperoxide and to 14 days for hexanal production.

56 ROS reflect a relative abundance of organic hydroperoxides and alcohols contained in SOA.

57 markers of oxidative stress including lipid hydroperoxides and alkenals were significantly higher in

58 eactions of a series of gold(III) peroxides, hydroperoxides and alkylperoxides.

59 ticles (50-80 um) was stored for 82 days and hydroperoxides and anisidine value measured in the total

60 ir inhibition against the formation of lipid hydroperoxides and carbonyl compounds in a stripped sunf

61 as demonstrated by the automated analysis of hydroperoxides and carboxylic acids (by microplate reade

62 Hydroperoxides and carboxylic acids are key primary prod

63 r production of some lipid mediators such as hydroperoxides and EPA-derived prostaglandins, such as 1

64 h the interaction of carbonyl compounds with hydroperoxides and H2O2 in acidic media, as such reactio

65 E/ml emulsion delayed the formation of lipid hydroperoxides and headspace hexanal in the 5.0%(wt) cor

66 sonance (EPR) spectroscopy and compared with hydroperoxides and hexanal in complex starch-protein-lip

67 re much stronger oxidizing agents than alkyl hydroperoxides and in some cases are almost comparable t

68 -ohrR) displayed limited or no resistance to hydroperoxides and INH.

69 emulsion), by slowing down the formation of hydroperoxides and malondialdehyde (MDA) compounds.

70 role in modulating the levels of fatty acid hydroperoxides and peroxynitrite, both of which are invo

71 ulated duodenal digests of the porridges had hydroperoxides and therefore caused LDL oxidation.

72 storage of an O/W emulsion was tested by the hydroperoxides and thiobarbituric acid reactive species

73 Lipid hydroperoxides and thiobarbituric acid reactive substanc

74 eroxides (tyrosyl, tryptophan, and histidine hydroperoxides) and CBA, and these values (7-23 M(-1) s(

75 mation of (1) an ozonide intermediate, (2) a hydroperoxide, and (3) cis,cis-muconic acid.

76 oxify ROS such as hydrogen peroxide, organic hydroperoxide, and peroxynitrite.

77 Cyclic peroxides, hydroperoxides, and epoxy alcohols are major products fo

78 tus, oxidative stress index, levels of lipid hydroperoxides, and the activities of paraoxonase, aryle

79 ously determine both types of products using hydroperoxide- and acid-sensitive moieties conjugated to

80 e dioxirane intermediate via addition of the hydroperoxide anion to the ketone and revealed that this

81 y the concurrent activation of persulfate by hydroperoxide anion, which is generated by the base cata

82 ation of a carbanion intermediate and that a hydroperoxide (anion) is involved.

83 nols, various allylic alcohols, and an alkyl hydroperoxide are viable coupling partners in this proce

84 pimers of each of the regioisomeric 4- and 6-hydroperoxides are formed as is the 5alpha-hydroperoxide

85 Hydroperoxides are the primary oxidation products from u

86 particles suggests that alpha-pinene-derived hydroperoxides are thermally labile; thus, analysis of p

87 Amino acid hydroperoxides are unstable intermediates that can furth

88 Ti(IV), 160 mM cyclohexene, 24 mM tert-butyl hydroperoxide) are 9 +/- 2 M(-2) s(-1); whereas within c

89 a secondary rather than tertiary surfactant hydroperoxide, arguing for an orthogonal alkene on water

90 -artemisinin metabolites (dihydroartemisinic hydroperoxide, artemisinic acid, dihydroartemisinic acid

91 lated Cu-catalyzed reaction using tert-butyl hydroperoxide as oxidant and how to overcome this limita

92 with unprecedented selectivity to give alkyl hydroperoxides as the major products.

93 Primary, secondary, and tertiary hydroperoxides as well as silyl, benzyl, and acyl peroxi

94 n the regioselectivity of protonation of the hydroperoxide, as suggested in the enzymatic systems.

95 Cis,trans-conjugated dienes associated with hydroperoxides, as well as monoepoxides, cis,trans-2,4-a

96 Hydroperoxide-based kinetic parameters (IP, induction pe

97 of 1 equiv of strong acid to 2 afforded the hydroperoxide-bridged dicopper(II) species [Cu(II)(2)(XY

98 ate method was not suitable to measure lipid hydroperoxides by the both in auto- and photo-oxidation

99 This is in contrast to CHA, where the hydroperoxide channel is the dominant oxidation pathway,

100 ated trafficking of redox-active cholesterol hydroperoxides (ChOOHs) can result in site-specific Mito

101 wild-type strain (MSWt) induced with cumene hydroperoxide (CHP) and t-butyl hydroperoxide (t-BHP).

102 hereas organic hydroperoxides such as cumene hydroperoxide (CHP) deactivate AphB and OhrR.

103 een the base-catalyzed hydrolysis of Co(III)-hydroperoxide [Co(III)-OOH](-) to release H(2)O(2) and t

104 oxanes, isoprostanes, resolvins, hydroxides, hydroperoxides, coming from eicosapentaenoic (EPA), arac

105 ned by analysing protein-bound peroxides and hydroperoxide compounds in water-methanol and chloroform

106 Hydroperoxide content of the samples was also measured.

107 as significant in terms of the inhibition of hydroperoxide content, while formation of secondary lipi

108 eriod which indicates the formation of lipid hydroperoxides could be detected via this method.

109 the effects of the DNA-damaging agent cumene-hydroperoxide (cum-OOH) and a chemopreventive cranberry

110 l, left ventricle myocardial levels of lipid hydroperoxides, cytochrome-c, and mitochondrial aconitas

111 ine (1a) into a catalytic chain-breaking and hydroperoxide-decomposing antioxidant by replacing the 2

112 ere asthma (r = -0.55; P = 0.03) and t-butyl-hydroperoxide decreased LXA4 and 15-epi-LXA4 biosynthesi

113 er-soluble compounds (e.g., strong acids and hydroperoxides) deposit with low surface resistance wher

114 ane may account for the formation of the bis-hydroperoxide derivative.

115 icated that fatty acid (but not cholesterol) hydroperoxides docked well into the active site of Ohr f

116 se two key steps: (i) the formation of ethyl hydroperoxide (EHP) accompanying with the hydrogen trans

117 Downstream, the LOX products 13-fatty acid hydroperoxides esterified to galactolipids and phospholi

118 ; (ii) substrate radical rebound to a Cu(II) hydroperoxide favors the proximal, nonprotonated oxygen;

119 try, observing SO(3) and identifying organic hydroperoxide formation from reaction with SO(2) and for

120 s a new mechanism for amino acid and protein hydroperoxide formation in biological systems.

121 At 37 degrees C, the rate of lipid hydroperoxide formation increased with decreasing drople

122 lamb, pork and chicken to investigate their hydroperoxide formation potential.

123 Hydroperoxide formation was examined at different pH val

124 ations-monitored either by O2 consumption or hydroperoxide formation-are the most reliable way to obt

125 synthesis of peroxy radical addition-induced hydroperoxide formation.

126 rogeneous reaction of aldehydes with organic hydroperoxides forming peroxyhemiacetals can lead to a l

127 um tuberculosis (MtAhpE-SH) reacts fast with hydroperoxides, forming a stable sulfenic acid (MtAhpE-S

128 mmetrical perepoxide transition state in the hydroperoxide-forming step.

129 Here we also show formation of tryptophan hydroperoxide from tryptophan exposed to co-generated fl

130 Our results demonstrate that ROS and lipid hydroperoxides function as not-yet-recognized unconventi

131 R-1, glutathione-S-transferase, phospholipid hydroperoxide glutathione peroxidase and peroxidase were

132 the following order: hydroxyl >> carboxyl > hydroperoxide > nitrate >> methylene (where nitrate and

133 rapidly and specifically with linoleate 10S-hydroperoxide (>2,500 turnovers/s) with a hydroperoxide

134 mitochondrial superoxide and cellular lipid hydroperoxides, had reduced activities of superoxide dis

135 discovery that varying the structure of the hydroperoxide has a significant impact on the enantiosel

136 idation reaction of olefins with the help of hydroperoxides has also been explored.

137 fluorometric method for the determination of hydroperoxides (HP) in edible oils and fats using the re

138 to unequivocally verify the presence of the hydroperoxide, hydroxide, and ketone oxidation products

139 Several species of OxCE, such as keto, hydroperoxide, hydroxy, and epoxy cholesteryl ester deri

140 rious kinds of oxidation products comprising hydroperoxides, hydroxy-dienes and other alcohols, epoxi

141 e, Prx1 and Prx2 are likely targets of urate hydroperoxide in cells.

142 stimulated cells took up substantially more hydroperoxide in Mito than controls, with a resulting lo

143 gainst oxidative damage caused by tert-butyl hydroperoxide in the human hepatocellular HepG2 cell lin

144 6-hydroperoxides are formed as is the 5alpha-hydroperoxide in the presence of a good H-atom donor.

145 s been accomplished in minutes using t-butyl hydroperoxide in the presence of tetrabutylammonium iodi

146 monium iodide as the catalyst and tert-butyl hydroperoxide in water (T-Hydro) as the oxidant affords

147 This approach was used to detect protein hydroperoxides in cell lysates obtained from macrophages

148 quantification of both carboxylic acids and hydroperoxides in hydrocarbon samples.

149 nes by its unique activity to reduce complex hydroperoxides in membrane bilayers and lipoprotein part

150 In contrast, the ability to inactivate lipid hydroperoxides in oxidized low-density lipoprotein was c

151 intact ohrR gene could be induced by organic hydroperoxides in vitro and in the intracellular environ

152 gen and fumitremorgin A relied not only on a hydroperoxide/indole hemiaminal cyclization, but also on

153 The 20xi-hydroperoxides induced spectral shifts in CYP27A1 and CY

154 nds protected HepG2 cells against tert-butyl hydroperoxide-induced oxidative cytotoxicity.

155 glutathionylation correlated with tert-butyl hydroperoxide-induced ROS generation.

156 om a semi-hydroquinone species and a Co(III)-hydroperoxide intermediate.

157 d to the peroxide ligand to yield a kappa(1)-hydroperoxide intermediate.

158 echanism involving decomposition of hydroxyl hydroperoxide intermediates followed by hemiacetal ring

159 X4) prevents ferroptosis by converting lipid hydroperoxides into non-toxic lipid alcohols(3,4).

160 Urate hydroperoxide is a product of the oxidation of uric acid

161 Notably, cumene hydroperoxide is produced in a higher yield and at milde

162 onor-Acceptor (D-A) cyclopropanes with alkyl hydroperoxides is reported to furnish various peroxycarb

163 e SOA components, including isoprene hydroxy hydroperoxide (ISOPOOH), isoprene-derived epoxydiols (IE

164 by the two main isomers of isoprene hydroxyl hydroperoxide (ISOPOOH), the primary low-NO(x) isoprene

165 a Fenton-like reaction between linoleic acid hydroperoxide (LAHP) tethered on iron oxide nanoparticle

166 te the transient imbalance between the lipid hydroperoxide level and antioxidant status related to a

167 is but attenuated protein carbonyl and lipid hydroperoxide levels in 3T3-L1 cells.

168 d lower plasma troponin and myocardial lipid hydroperoxides levels (vs. controls, both p<0.05, analys

169 es acting as pro-oxidants as measured by the hydroperoxides levels.

170 ers like TBA value, carbonyl value and lipid hydroperoxides (LHPODs) exhibited significant negative c

171 action between linoleic acid (LH) and its 13-hydroperoxide (LOOH) catalysed by lipoxygenase.

172 60 degrees C and the kinetic curves of lipid hydroperoxides (LOOH) accumulation were drawn.

173 meters and rate constants representing lipid hydroperoxides (LOOH) formation and decomposition during

174 hydroperoxide production (H(2)O(2) and lipid hydroperoxides (LOOHs)).

175 lso examined serum lipid peroxidation (lipid hydroperoxides, LPH and 4-hydroxy-2-nonenal, 4-HNE), in

176 er concentration of hexanal, probably due to hydroperoxide lyase (HPL) action on linoleyl hydroperoxi

177 pathway, the allene oxide synthase (AOS) and hydroperoxide lyase (HPL) branches, which are responsibl

178 0S-hydroperoxide (>2,500 turnovers/s) with a hydroperoxide lyase activity specific for the 10S-hydrop

179 ical characteristics, lipoxygenase activity, hydroperoxide lyase activity; linoleic acid and linoleni

180 se and the newly identified catalase-related hydroperoxide lyase and given the role of aldehydes in p

181 e signal, in ceh1 (constitutively expressing hydroperoxide lyase1)-mutant plants leads to large-scale

182 rate, is known only in plant cytochrome P450 hydroperoxide lyases.

183 ress cake remarkably reduced levels of lipid hydroperoxides, malondialdehyde (MDA) and 4-hydroxy-tran

184 straction through a cyclic network of peroxy-hydroperoxide-mediated free-radical chain reactions.

185 Oxidation of Prxs by urate hydroperoxide might affect cell function and be partiall

186 The formation of urate hydroperoxide might be a key event in vascular inflammat

187 ly oxidized RO2 radicals containing a single hydroperoxide moiety.

188 and spectroscopic characteristics of lithium hydroperoxide monohydrate are scrutinized both experimen

189 s including hydrazines, methylamine, t-butyl hydroperoxide, N-hydroxylamine, alpha-chloroacetaldehyde

190 suggest that the oxidation of Prx2 by urate hydroperoxide occurs by a three-step mechanism, where th

191 studies on this reaction suggest that the C2 hydroperoxide of dibenzothiophene sulfone reacts with fl

192 Hydroperoxides of amino acid and amino acid residues (ty

193 activity with restricted specificity toward hydroperoxides of unsaturated fatty acid.

194 tions including the decomposition of organic hydroperoxides, (*)OH oxidation of primary or secondary

195 derived thermodynamics lead to a ferric heme hydroperoxide OO-H BDFE determination, that Fe(III)-OOH

196 Select samples were exposed to t-butyl-hydroperoxide or sEH inhibitor (sEHI) before activation.

197 Urate hydroperoxide oxidized Prx2 from intact erythrocytes to

198 Urate hydroperoxide oxidizes glutathione and sulfur-containing

199 olic 2-Cys Prx1 and Prx2 revealed that urate hydroperoxide oxidizes these enzymes at rates comparable

200 d V materials activate H2O2 to form pools of hydroperoxide, peroxide, and superoxide intermediates.

201 HNO3), hydrogen cyanide (HCN), hydroxymethyl hydroperoxide, peroxyacetic acid, organic hydroxy nitrat

202 The "ene" hydroperoxide photoproducts are not toxic on their own,

203 s, they are among the most important organic hydroperoxides present in clouds.

204 f Lto1 function renders cells susceptible to hydroperoxide pro-oxidants, though this type of sensitiv

205 a small amount of an initially formed metal hydroperoxide proceeds to generate a metal-oxyl species

206 hat denervation induces muscle mitochondrial hydroperoxide production (H(2)O(2) and lipid hydroperoxi

207 cholesterol yields a more complex mixture of hydroperoxide products than previously appreciated.

208 rols and the subsequent decomposition of the hydroperoxide products to cytotoxic derivatives are disc

209 sed on the formation of polar compounds than hydroperoxides provided more reliable results to evaluat

210 e fillets were free fatty acids (FFA), lipid hydroperoxides (PV) and thiobarbituric acid reactive sub

211 Fatty acid profiles, formation of hydroperoxides (PV), thiobarbituric acid reactive substa

212 ht to be controlled only by the phospholipid hydroperoxide-reducing enzyme glutathione peroxidase 4 (

213 rimarily regulates the expression of organic hydroperoxide reductase (Ohr) in bacteria.

214 scriptional regulator for the enzyme organic hydroperoxide reductase (Ohr).

215 The effector proteins, alkyl hydroperoxide reductase and acetyl-CoA acetyltransferase

216 The one-cysteine peroxiredoxin alkyl hydroperoxide reductase E from Mycobacterium tuberculosi

217 oxidative stress, suggesting that the alkyl hydroperoxide reductase is an important regulator of red

218 Organic hydroperoxide reductase regulator (OhrR) in bacteria is

219 , a dedicated peroxiredoxin reductase, alkyl hydroperoxide reductase subunit F (AhpF), catalyzes the

220 Finally, the alkyl hydroperoxide reductase, a primary scavenger of endogeno

221 I aneuploidy, wherein these proteins support hydroperoxide removal with the reducing equivalents gene

222 Organic hydroperoxide resistance (Ohr) enzymes are unique Cys-ba

223 used directly with our previously described hydroperoxide-sensitive coumarin-conjugated phosphine pr

224 roxide-shunt reaction is proposed, where the hydroperoxides serve as both donor for reduced oxygen an

225 A P450 hydroperoxide-shunt reaction is proposed, where the hydr

226 Copper(II) hydroperoxide species are significant intermediates in p

227 e, BDFE = 66.5 kcal/mol in THF), forming the hydroperoxide species HP and TEMPO radical.

228 O-H bond dissociation free energy (BDFE) of hydroperoxide species HP was calculated to be 73.5 kcal/

229 inium triflate, yielding the low-spin ferric hydroperoxide species, [(F(8))Fe(III)-(OOH)] (HP).

230 by a concerted process involving a manganese hydroperoxide species.

231 r (OhrR) in bacteria is a sensor for organic hydroperoxide stress and a transcriptional regulator for

232 The organic hydroperoxide stress resistance regulator (OhrR) is a Ma

233 o in vitro but also to intracellular organic hydroperoxide stress.

234 o sense and respond to intracellular organic hydroperoxide stress.

235 bon chain within the conjugated diene of the hydroperoxide substrate, is known only in plant cytochro

236 and repress its expression, whereas organic hydroperoxides such as cumene hydroperoxide (CHP) deacti

237 When exposed to tert-butyl hydroperoxide, survival of the lon-1 mutant was impaired

238 In addition, when exposed to tert-Butyl hydroperoxide, survival of the lon-2 mutant was impaired

239 e protection as well as decreased tert-butyl hydroperoxide (t-BHP) induced oxidative cytotoxicity in

240 ffect against oxidative stress in tert-butyl hydroperoxide (t-BHP)-challenged RAW264.7 macrophages.

241 with cumene hydroperoxide (CHP) and t-butyl hydroperoxide (t-BHP).

242 osen for efficient co-delivery of tert-butyl hydroperoxide (TBHP) and iron pentacarbonyl (Fe(CO)(5)).

243 an organophotoredox-catalyst and tert-butyl hydroperoxide (TBHP) as an oxidant at ambient temperatur

244 trazoles have been achieved using tert-butyl hydroperoxide (TBHP) as the methyl source, alkyl diacyl

245 tions of N,N-dialkylanilines with tert-butyl hydroperoxide (TBHP) as the oxidant.

246 in dioxane as solvent and aqueous tert-butyl hydroperoxide (TBHP) as the terminal oxidant.

247 uring oxidative stress induced by tert-butyl hydroperoxide (TBHP) in HEK293T cells and C. elegans.

248 s, proteins were pre-treated with tert-butyl hydroperoxide (TBHP), a bulky peroxide, to oxidise only

249 ed significant protection against tert-butyl hydroperoxide (tBHP), a damaging organic peroxide.

250 nd cytoprotective effects against tert-butyl hydroperoxide (TBHP)-induced cytotoxicity of Caco-2 cell

251 ffects of sheep whey protein from tert-butyl hydroperoxide (tBHP)-induced oxidative stress in muscle

252 noxalinol salen catalyst and with tert-butyl hydroperoxide (TBHP).

253 oceeds cleanly in the presence of tert-butyl hydroperoxide (TBHP, 2) using low loadings of VO(O(i)Pr)

254 avage reactions of arylalkenes by tert-butyl hydroperoxide that occur by free radical processes provi

255 s through formation of an amino acid-derived hydroperoxide that regulates vascular tone and blood pre

256 ethylnon-7-ene-1 sulfonate) leading to "ene" hydroperoxides that in the dark inactivate planktonic Es

257 epoxidation reaction of olefins using alkyl hydroperoxides, that the molybdenum-oxo moiety is an act

258 epimers of the major product, cholesterol 7-hydroperoxide, the epimers of each of the regioisomeric

259 n priming (SOP), the singlet oxygen leads to hydroperoxides then to peroxyl radicals, tetraoxides, an

260 ysteine in Gpx4 prevents elimination of PUFA hydroperoxides; these findings suggest new strategies fo

261 eing among the most abundant multifunctional hydroperoxides, they are among the most important organi

262 Hydroperoxide, thiobarbituric acid reactive substances (

263 sting that OhrR senses intracellular organic hydroperoxides through Cys residue.

264 mation of oxygen-containing defects by lipid hydroperoxides through photo-oxidation.

265 n to mediate the reduction of cholesterol 25-hydroperoxide to 25-hydroxycholesterol, a role of potent

266 pidly oxidize while reducing the toxic lipid hydroperoxide to a nonreactive lipid hydroxide, whereas

267 ), the selective decomposition of cyclohexyl hydroperoxide to cyclohexanone, and the asymmetric amina

268 Addition of cholesterol 25-hydroperoxide to the enzymes CYP27A1 and CYP11A1 induced

269 Heating of crystals of lophine hydroperoxide to ~115 degrees C results in detectable em

270 BA) probe reacts with amino acid and protein hydroperoxides to form the corresponding fluorescent pro

271 by the iron-dependent accumulation of lipid hydroperoxides to lethal levels.

272 tosis, converting unstable ferroptotic lipid hydroperoxides to nontoxic lipid alcohols in a tissue-sp

273 tated rapid decomposition of preformed lipid hydroperoxides to secondary lipid oxidation products.

274 s of oxygen transfer from these heterocyclic hydroperoxides to thioanisole, obtained by calculations

275 ctive cleavage of the resulting alpha-alkoxy hydroperoxide, trapping of the generated alkyl radical w

276 onstants for the reaction between amino acid hydroperoxides (tyrosyl, tryptophan, and histidine hydro

277 al-time monitoring of amino acid and protein hydroperoxides using the CBA-based assay.

278 Oxidation markers (hydroperoxide value and head space n-hexanal) increased

279 milar in all emulsions as concluded from the hydroperoxide value.

280 ive oxidation of L-tryptophan to a tricyclic hydroperoxide via a previously unrecognized oxidative ac

281 ing storage time, a progressive formation of hydroperoxide was found as evidenced by the increase in

282 ted hemoprotein reactivity toward fatty acid hydroperoxides, we detected a novel candidate in the cya

283 For first time total hydroperoxides were determined by FOX-1 assay to follow

284 es which can use as substrate the fatty acid hydroperoxides were differentially regulated in bacteriz

285 n the presence of superoxide, high yields of hydroperoxides were formed by LPO and urate.

286 Phenols and hydroperoxides were identified as being the most sensiti

287 constants between Ohr and several fatty acid hydroperoxides were in the 10(7)-10(8) M(-1)s(-1) range

288 cardial lactate) and oxidative stress (lipid hydroperoxides) were measured by enzyme-linked immunosor

289 Primary oxidation products (lipid hydroperoxides) were measured with a ferrous oxidation-x

290 ing multifunctional organosulfates (OSs) and hydroperoxides, were chemically characterized in both la

291 aturated oleyl chain is oxidized to an allyl hydroperoxide, which surprisingly is immune to further o

292 owever, oxidation by multifunctional organic hydroperoxides, which are expected to have higher water

293 kenes to form endoperoxides, diooxetanes, or hydroperoxides, which are not observed in our system.

294 Fatty acid hydroperoxides, which are readily solubilized by bacteri

295 ten-3-ol formed in reaction of linoleate 10S-hydroperoxide with hematin or ferrous ions.

296 Intriguingly, the reaction of lithium hydroperoxide with triiodide exhibits a faster kinetics,

297 actions of primary and secondary propargylic hydroperoxides with a variety of nucleophiles including

298 approach, the rate constants for amino acid hydroperoxides with ebselen, a glutathione peroxidase mi

299 s show that these reactions form substituted hydroperoxides with no energy barrier.

300 products of cholesterol, namely 25- and 20xi-hydroperoxides, with the four principal cholesterol-meta