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1 ressive KC; and 9 of 116 eyes (8%) had acute hydrops.
2 d perilymph, a disorder termed endolymphatic hydrops.
3 elease from hair cells but not endolymphatic hydrops.
4 ot fixed but can be altered by endolymphatic hydrops.
5 onstrate the ability to detect endolymphatic hydrops.
6 es several hematological disorders and fetal hydrops.
7  placental hypoxia associated with Hb Bart's hydrops.
8 med to consistently elicit a typical corneal hydrops.
9 l was similar in eyes with and without prior hydrops.
10 iated with a high risk of developing corneal hydrops.
11    This may present prenatally as non-immune hydrops.
12 identified risk factors for developing acute hydrops.
13 al grafts after transplantation for resolved hydrops.
14  presented with moderate to severe anemia or hydrops.
15 largement, leading to low cardiac output and hydrops.
16 r insidiously, consistent with endolymphatic hydrops.
17 abyrinth with the formation of endolymphatic hydrops.
18 irth at 7 months, again with nonimmune fetal hydrops.
19 ressive KC; and 9 of 116 eyes (8%) had acute hydrops.
20                      Eleven cases of corneal hydrops (5.8%) occurred in our series during a mean foll
21                         Stage 5 demonstrates hydrops; 5a, acute onset: Descemet's membrane rupture an
22 vival rates compared with eyes without prior hydrops: 86.5% +/- 4.0% vs 86.5% +/- 2.6% at 1 year, 61.
23  either in the presence or in the absence of hydrops (95 percent confidence interval, 86 to 100 perce
24 e-free survival rates with and without prior hydrops: 98.6% +/- 1.3% vs 97.1% +/- 1.3% at 1 year, 97.
25 rs with experimentally-induced endolymphatic hydrops, a hallmark of Meniere's disease.
26          The third infant demonstrated acute hydrops after medically controlling IOP in 1 eye before
27                    Eleven fetuses had severe hydrops; all had polyhydramnios and a structurally abnor
28                                Endolymphatic hydrops also can occur after noise trauma and its presen
29 t the time of PK in 44.6% of eyes with prior hydrops and 7.6% without prior hydrops (P < .001).
30                 Thus, although endolymphatic hydrops and cochlear synaptopathy are both observed afte
31 es were corneal neovascularization following hydrops and complications following PK.
32 ignificant predictors of in utero death were hydrops and earlier diagnosis, and of postnatal death we
33 multivariable analysis of all cases included hydrops and endocardial fibroelastosis.
34 red with whites, who were at a lower risk of hydrops and endocardial fibroelastosis.
35 uterine transfusions in order to avoid fetal hydrops and fetal death.
36 ent compression of the right ventricle (RV); hydrops and low cardiac output are often associated.
37 dies of reliable biomarkers of endolymphatic hydrops and real-time imaging are warranted to improve u
38 of fetal CLM subgroups at increased risk for hydrops and respiratory compromise at delivery have not
39  clinical manifestations, ranging from fetal hydrops and symptomatic anemia requiring lifelong transf
40 s rare in KD without associated gall bladder hydrops and tends to occur in older patients.
41 neal features in eyes that developed corneal hydrops and those that did not develop this complication
42 between women who had fetuses with Hb Bart's hydrops and those with normal pregnancies or placenta-as
43  1.4 identifies fetuses at very low risk for hydrops, and a maximum CVR < 0.9 is associated with asym
44 risk of sudden raised IOP resulting in acute hydrops, and early treatment may help to prevent this ph
45  outcomes including ventricular dysfunction, hydrops, and fetal demise.
46 n the duration of corneal edema during acute hydrops, and have improved the survival of corneal graft
47 ged with maternal steroids in the setting of hydrops, and prenatal surgical intervention was uncommon
48            Fetal lung masses associated with hydrops are nearly 100% fatal.
49 ificantly correlated with a reduced risk for hydrops [area under the curve (AUC), 0.93; 95% confidenc
50 e, acute and chronic red cell aplasia, fetal hydrops, arthropathy, and other disorders.
51 g system may be easily used in endolymphatic hydrops assessment.
52        None of the 16 DMEK eyes manifested a hydrops at any time during or after surgery.
53                                       Severe hydrops at first intrauterine transfusion was present in
54                                    Four with hydrops at presentation died perinatally, despite initia
55 inner ear pathologies, such as endolymphatic hydrops, by changing the metabolite profiles in the inte
56           In patients with MD, endolymphatic hydrops can be studied on MRI using 3D-FLAIR delayed pos
57 with MD were observed: lack of endolymphatic hydrops (cases #1 and #7), various grades of cochlear hy
58 cases #1 and #7), various grades of cochlear hydrops (cases #2 and #3), various grades of vestibular
59 ses #2 and #3), various grades of vestibular hydrops (cases #4, #5, and #6), endolymphatic hydrops he
60 phatics and may present with nonimmune fetal hydrops, chylothorax, chylous ascites, or lymphedema.
61 nd posterior corneal pathology such as acute hydrops, Descematocele and pre-Descemet's dystrophies.
62     Attempts are being made to prevent fetal hydrops due to congenital heart defects, to recruit hypo
63  the leading cause of nonimmunological fetal hydrops during pregnancy.
64  can cause hematological disorders and fetal hydrops during pregnancy.
65 nctata, CHILD syndrome, lathosterolosis, and hydrops-ectopic calcification-moth-eaten skeletal dyspla
66 throderma and limb defects (CHILD syndrome), hydrops-ectopic calcification-moth-eaten skeletal dyspla
67 ven the association of HI with endolymphatic hydrops (EH), imaging-based techniques for quantificatio
68 normal inner ear fluid buildup-endolymphatic hydrops (EH)-with pressure rise and repetitive microtrau
69 rlier diagnosis, and of postnatal death were hydrops, endocardial fibroelastosis, and lower ventricul
70 r corneal perforation demonstrated a corneal hydrops, evident both intra- and postoperatively.
71 al effusions (2 of 2), ascites (6 of 8), and hydrops fetalis (5 of 8).
72 erited form of lymphatic-related (nonimmune) hydrops fetalis (LRHF).
73 etuses with incessant tachycardia and either hydrops fetalis (n=24) or ventricular dysfunction (n=2)
74                                   Non-immune hydrops fetalis (NIHF) is a complex condition with a hig
75                                    Nonimmune hydrops fetalis (NIHF), a fetal abnormality that is ofte
76 cted fetuses and children included nonimmune hydrops fetalis (NIHF), pleural and pericardial effusion
77 CD2) gene in multiple fetuses with nonimmune hydrops fetalis (NIHF).
78 ) Bart's disease is the most common cause of hydrops fetalis among Southeast Asians.
79 efects in the formation of the heart lead to hydrops fetalis and are likely the cause of embryonic le
80 /-) embryos die at midgestation with extreme hydrops fetalis and cardiovascular abnormalities, includ
81        The Calcrl-/- embryos exhibit extreme hydrops fetalis and cardiovascular defects, including th
82 m of GLD with a high incidence of non-immune hydrops fetalis and childhood onset of facial and four l
83 t with severe nonimmune hemolytic anemia and hydrops fetalis at birth.
84 a complicated by ventricular dysfunction and hydrops fetalis carries a significant risk of morbidity
85                              The majority of hydrops fetalis cases are nonimmune conditions that pres
86                                              Hydrops fetalis describes fluid accumulation in at least
87 vious sibling born with hemolytic anemia and hydrops fetalis died on the second day of life.
88 se control group or Group 4; n = 4); and (5) hydrops fetalis from non-Bart's causes (Group 5; n = 5).
89 s in 8 group A versus 0 group B (P < 0.007), hydrops fetalis in 8 group A versus 0 group B (P < 0.007
90 absence of Adm may be one cause of nonimmune hydrops fetalis in humans.
91             BHFS is the most common cause of hydrops fetalis in Southeast Asia; however, owing to glo
92 re the correction of alpha-thalassemia major hydrops fetalis in transgene-free iPS cells using zinc f
93             In utero infection may result in hydrops fetalis or congenital anemia.
94 icular tachycardia, even when accompanied by hydrops fetalis or ventricular dysfunction.
95                          We report a case of hydrops fetalis secondary to cCMV infection with minimal
96                            Hemoglobin Bart's hydrops fetalis syndrome (BHFS) represents the most seve
97                       Hemoglobin (Hb) Bart's hydrops fetalis syndrome (BHFS) resulting from alpha(0)-
98 nd neonatal deaths associated with nonimmune hydrops fetalis uncovered 2 heterozygous missense varian
99 oglobin [Hb] H disease) or lethal (Hb Bart's hydrops fetalis).
100 ted ova, 9 stillbirths, 1 termination due to hydrops fetalis, 1 cleft palate, and 2 threatened aborti
101 uding fifth disease, arthritis, myocarditis, hydrops fetalis, and aplastic crisis.
102 that is characterized by multiple anomalies, hydrops fetalis, and death within the first 8 wk of life
103 od cells, potentially causing severe anemia, hydrops fetalis, and fetal death.
104 pregnancies can be affected by fetal anemia, hydrops fetalis, and perinatal death.
105 haemoglobin H disease and haemoglobin Bart's hydrops fetalis, are an important public health concern
106                                 Early lethal hydrops fetalis, arthrogryposis, microcephaly, and polym
107  of diverse pathological outcomes, including hydrops fetalis, fetal myocarditis, meningoencephalitis,
108 lly relevant thalassemias (hemoglobin Bart's hydrops fetalis, hemoglobin H disease, beta-thalassemia
109 h homozygous mutants develop polyhydramnios, hydrops fetalis, spina bifida occulta and osteochondrody
110 oss of beta-glucuronidase activity can cause hydrops fetalis, with in utero or postnatal death of the
111  aplastic crisis, pure red cell aplasia, and hydrops fetalis.
112 unexplained cases of severe neonatal NSHA or hydrops fetalis.
113 linical and electrophysiologic predictors of hydrops fetalis; and 3) to describe the medium-term foll
114 oup 2; n = 4); (3) Hb Bart's fetuses without hydrops (Group 3; n = 7); (4) placental associated compl
115 d moderate anemia; and 31, including 12 with hydrops, had severe anemia.
116 d in vivo imaging of the cornea during acute hydrops has led to an enhanced understanding of the path
117 ydrops (cases #4, #5, and #6), endolymphatic hydrops herniation into the semi-circular canal (case #6
118        Effective management of acute corneal hydrops in keratoconus is based on recognizing and addre
119 coma surgery in 1 eye and demonstrated acute hydrops in the fellow eye within 1 week.
120                                Endolymphatic hydrops, increased endolymphatic fluid within the cochle
121  Although there is known to be endolymphatic hydrops involved in the pathological process, the pathog
122                            In fact, prenatal hydrops is a common manifestation of MPS VII because of
123                                Acute corneal hydrops is an incompletely understood complication of ke
124 V-IgG in fetuses with B19V-derived anemia or hydrops is most likely due to a limited materno-fetal tr
125                              Eyes with prior hydrops (n = 74) had lower endothelial rejection-free su
126                            No cases of fetal hydrops occurred, and 6 participants (46%) did not recei
127 ted hereditary stomatocytosis and non-immune hydrops of unknown aetiology.
128 evere childhood manifestations include fetal hydrops or sudden infant death syndrome.
129  odds of receiving keratoplasty were corneal hydrops (OR 4.87 [95% CI 4.07-5.82]), Leber congenital a
130 es with prior hydrops and 7.6% without prior hydrops (P < .001).
131 er gestation at presentation correlated with hydrops (p < 0.02, p < 0.05), but mechanism of tachycard
132 associated with perinatal outcome, including hydrops, respiratory distress at birth, need for supplem
133                           In fetuses without hydrops that are at risk because of maternal red-cell al
134 uctural changes that take place during acute hydrops, the factors that influence its duration, and se
135                   The presence or absence of hydrops was assessed by intra- and postoperative optical
136 examining 3D T2 sequences, and endolymphatic hydrops was identified on delayed post-contrast FLAIR se
137                                              Hydrops was present in 12 of 30 fetuses.
138 rization, a frequent complication of corneal hydrops, was associated with increased risk of endotheli
139 m the women with fetuses with Hb Bart's with hydrops were smaller than those from the women in the no
140 the complete removal of DM did not produce a hydrops, whereas a combined defect in DM and the posteri
141 l were significantly associated with corneal hydrops, whereas the presence of corneal scarring was a
142 phaly presenting with acute onset of corneal hydrops with prominent bulging and refractory steroid-in
143 86 to 100 percent for the 23 fetuses without hydrops), with a false positive rate of 12 percent.

 
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