ライフサイエンスコーパス: hydroquinone

1 een an alkene, alpha-chloroamide, and flavin hydroquinone.

2 m the oxidized quinone to the doubly reduced hydroquinone.

3 ne, 4-dimethylaminophenol, benzaldehyde, and hydroquinone.

4 erivatives such as catechol, resorcinol, and hydroquinone.

5 formation of two tautomeric forms of emodin hydroquinone.

6 f GS-trichloro-p-hydroquinone to trichloro-p-hydroquinone.

7 ore rigid semiquinone and to the much looser hydroquinone.

8 the inhibition of tube formation induced by hydroquinone.

9 uction, yielding a glutathiylated conjugated hydroquinone.

10 se effects induced by the benzene metabolite hydroquinone.

11 (red)), and (iii) two-electron fully reduced hydroquinone.

12 and the conversion of vitamin K to vitamin K hydroquinone.

13 hesizes tricholignan A, a redox-active ortho-hydroquinone.

14 n to be a key step in formation of the ortho-hydroquinone.

15 oth an unstable neutral blue semiquinone and hydroquinone.

16 ster electron transfer affords the colorless hydroquinone.

17 none with a more negative potential than the hydroquinone.

18 o(salophen)-catalyzed aerobic oxidation of p-hydroquinone.

19 intermediate oxidizes a second equivalent of hydroquinone.

20 gher than those observed in solutions of 1,4-hydroquinone.

21 f magnitude greater than in solutions of 1,4-hydroquinone.

22 in solutions of simple quinones such as 1,4-hydroquinone.

23 athionyl-hydroquinones (GS-hydroquinones) to hydroquinones.

24 ition, and GS-HQRs convert the conjugates to hydroquinones.

25 clusively upon reaction with monosubstituted hydroquinones.

26 sence of GS-HQRs suggests they use common GS-hydroquinones.

27 ydroquinones and then enzymatic reduction to hydroquinones.

28 ed the GS-hydroquinones to the corresponding hydroquinones.

29 se in the oxidizability of these chlorinated hydroquinones.

30 duction of quinones and quinoid compounds to hydroquinones.

31 etic mechanism, generating the corresponding hydroquinones.

32 PcpA (2,6-dichloro-p-hydroquinone 1,2-dioxygenase) from Sphingobium chlorophe

33 ts, is a member of the recently discovered p-hydroquinone 1,2-dioxygenases.

34 e propose a general reaction mechanism for p-hydroquinone 1,2-dioxygenases.

35 xposure to the benzene metabolites catechol, hydroquinone, 1,2,4-benzenetriol, and p-benzoquinone.

36 hich is chemically reduced to 2,5-dichloro-p-hydroquinone (2,5-DiCHQ).

37 This study focused on 2,6-dimethoxy hydroquinone (2,6-DMHQ), an analogue to a common fungal

38 treatment with Thiamidol (79%) compared with hydroquinone (61%).

39 ns to generate tert-butanol and 1-acetyl-1,4-hydroquinone, 8, apparently by an SN1 mechanism.

40 oxygeldanamycin (17AAG) to the corresponding hydroquinone, a more potent 90-kDa heat shock protein (H

41 an overall 2 e-, 2 H+ reduction to make the hydroquinone, a much better description of the overall r

42 A tight hydrophobic pocket provides p-hydroquinones access to the Fe(II) centre.

43 The resultant alpha-FAD hydroquinone (alpha-FADH(-)) transfers one electron furt

44 Hydroquinone also caused an increase in Ten Eleven Trans

45 ulation by reducing vitamin K epoxide to the hydroquinone, an essential cofactor for the gamma-glutam

46 tested in the voltammetric determination of hydroquinone and catechol in solutions of increasing com

47 en appears to be first-order each in ionized hydroquinone and dioxygen, yielding hydrogen peroxide st

48 proton across the hydrogen bond between the hydroquinone and His154 at the cytochrome b site.

49 atechol), their 1,4-dihydroxyphenyl isomers (hydroquinone and homogentisic acid) are able to modify a

50 ansforms p-nitrophenol stoichiometrically to hydroquinone and hydroxyquinol.

51 e capable of cleaving the aromatic ring of p-hydroquinone and its substituted variants, is a member o

52 edel-Crafts reaction of a highly substituted hydroquinone and N-fumaryl ketimine generated from the c

53 A novel hydroquinone and NADH oxidase with protein disulfide-thi

54 otential of approximately 110 mV between the hydroquinone and semiquinone state.

55 ct, and a hydrogen-bonded adduct between the hydroquinone and the Co(III)-O2 species.

56 nterface by the reduction of benzoquinone to hydroquinone and the resulting interfacial pH change is

57 e that could accommodate various substituted hydroquinones and a hydrogen network of three Tyr residu

58 Our findings suggest that hydroquinones and benzoquinones, which are interchangeab

59 y laccase-catalyzed domino reactions between hydroquinones and cyclic 1,3-dicarbonyls using aerial ox

60 This study showed that reactions between hydroquinones and iron oxides could produce favorable co

61 l roles: channeling GS-hydroquinones back to hydroquinones and reducing benzoquinones via spontaneous

62 d the structural aspects of stabilization of hydroquinones and their ability to generate reactive oxy

63 enzoquinones via spontaneous formation of GS-hydroquinones and then enzymatic reduction to hydroquino

64 phenols and solvent water adducts (catechol, hydroquinone) and ammonium ion.

65 FMN N5, the anionic ionization state of the hydroquinone, and for a change in the hybridization stat

66 Hydroquinone, and its mercapturic acid pathway metabolit

67 erity Index scores significantly better than hydroquinone, and more subjects improved following treat

68 ate derivative 5,6-isopropylidine ascorbate, hydroquinone, and the hydroxylamine TEMPOH all rapidly a

69 he electronic structure of the fully reduced hydroquinone anionic state, FlH(-), however, has been th

70 ner, demonstrating increased efficacy of the hydroquinone ansamycin relative to its parent quinone.

71 uinone ansamycins are metabolized by NQO1 to hydroquinone ansamycins and that Hsp90-mediated trans-ci

72 hat both trans- and cis-amide isomers of the hydroquinone ansamycins exhibited increased binding affi

73 QO1, was used, and HPLC analysis showed that hydroquinone ansamycins were formed by the MDA468/NQ16 c

74 ard a selective desymmetrization of the meso-hydroquinone are also reported.

75 Hydroquinones are important mediators of electron transf

76 being that the ortho adducts (o-aminophenol, hydroquinone) are formed in a higher ratio in comparison

77 e reactant and product, 1,4-benzoquinone and hydroquinone, are separated during the assay by differen

78 reen by P. veronii and the identification of hydroquinone as a metabolite in the degradation pathway.

79 spray solvent flow rate and the addition of hydroquinone as a redox buffer to the spray solvent were

80 istic experiments support the role of flavin hydroquinone as a single electron reductant, flavin semi

81 ith a scan rate of 100 mV in the presence of hydroquinone as electron mediator and 0.1M phosphate buf

82 These conditions employing biorenewable hydroquinone as reagent were developed from initial expe

83 nto the electrode surface in the presence of hydroquinone as the redox mediator.

84 With unsubstituted hydroquinone as the substrate 2,3-disubstituted p-benzoq

85 hiol-hydroquinones, for example, S-cysteinyl-hydroquinone, as substrates.

86 In turn, benzoquinone is electroreduced into hydroquinone at the electrode.

87 oxygen consumption rate data, indicative of hydroquinone auto-oxidation.

88 potential physiological roles: channeling GS-hydroquinones back to hydroquinones and reducing benzoqu

89 c measurements confirm the formation of beta-hydroquinone (beta-HQ) clathrate with molecular hydrogen

90 FLEEL and vitamin K hydroquinone both stabilized the propeptide-carboxylase

91 ne with recombinant UBIAD1 revealed that the hydroquinone, but not the quinone form of menadione, was

92 a quinone with a thiol to form a substituted hydroquinone by reductive 1,4-Michael addition.

93 din, which is then converted to chrysophanol hydroquinone by the reductase ClaC and the dehydratase C

94 Selective reduction of quinones to hydroquinones by addition of small molar excesses of dit

95 inert but can be converted into photolabile hydroquinones by mild reduction in situ.

96 ive yield along with well-defined, colorless hydroquinone byproducts.

97 GS-hydroquinones can be spontaneously formed from benzoquin

98 iciency site (with 2,6-dimethoxyphenol and p-hydroquinone catalytic efficiencies of approximately 70

99 Hydroquinone caused reactivation of a methylated reporte

100 s treated with the Nrf2 activator tert-butyl hydroquinone, chromatin immunoprecipitation with Nrf2 an

101 A 2-coordinate Pd(0)-hydroquinone complex (4-H) was prepared using a one-pot

102 role in PCP degradation and converted the GS-hydroquinone conjugates back to the intermediates of the

103 te, likely due to the accumulation of the GS-hydroquinone conjugates inside the cell.

104 The fate and effect of GS-hydroquinone conjugates were unknown.

105 QDS, a widely used analogue for quinone- and hydroquinone-containing molecules in NOM) was immobilize

106 d electron-transfer behavior of quinone- and hydroquinone-containing molecules in particulate NOM.

107 esulting low diffusivity of its quinone- and hydroquinone-containing molecules.

108 he elevated potential of the FMN semiquinone/hydroquinone couple (-172 mV) is also an adaptation that

109 semiquinone couple = +70 mV, FMN semiquinone/hydroquinone couple = -180 mV, and heme = -180 mV) indic

110 The potentials of the semiquinone/hydroquinone couple of both FMN and FAD are altered to a

111 ive carbon electrodes, combining the quinone/hydroquinone couple with the Br2/Br(-) redox couple, yie

112 is much higher than that of the semiquinone/hydroquinone couple.

113 idized/semiquinone (-301 mV) and semiquinone/hydroquinone couples (-464 mV).

114 he first synthesis of the marine benzoxepane hydroquinone cyclosiphonodictyol A and its bis(sulfato)

115 The pK(a) of parent hydroquinone decreased by 1 unit as the degree of chlori

116 iron sulfur protein and an integral membrane hydroquinone dehydrogenase, respectively.

117 -type cytochrome that functions as the major hydroquinone dehydrogenase.

118 The hydroquinone detected in vitro is a dead-end product mos

119 -hydroquinone (TriCH) and then to dichloro-p-hydroquinone (DiCH) in the PCP degradation pathway.

120 esults provide mechanistic insights into the hydroquinone dioxygenases.

121 ctrochemical detection was carried out using hydroquinone diphosphate (HQDP) as enzymatic substrate.

122 chol monophosphate, ascorbic 2-phosphate and hydroquinone diphosphate) that could previously not be u

123 5-hydroxymethylcytosine formation following hydroquinone exposure as well as the induction of glutam

124 This work demonstrates that hydroquinone exposure leads to active and functional DNA

125 tage of cells accumulating in G2+M following hydroquinone exposure, indicating that it may have a rol

126 tation the anionic semiquinone (FAD(*-)) and hydroquinone (FADH(-)) have longer lifetimes that are co

127 ur ET dynamics dictate that only the anionic hydroquinone flavin can be the functional state in photo

128 he target of warfarin and provides vitamin K hydroquinone for the carboxylation of select glutamic ac

129 potential of the flavin mononucleotide (FMN) hydroquinones for one-electron reduction in the Desulfov

130 (nonenzymatic) reduction of emodin to emodin hydroquinone, for example with sodium dithionite, is obl

131 did not use GS-benzoquinones or other thiol-hydroquinones, for example, S-cysteinyl-hydroquinone, as

132 ted pentad molecules, AQ is converted to its hydroquinone form (AQH2) via reversible intramolecular e

133 using the crystal structure of the relevant hydroquinone form and compared to the results of the Clo

134 odeling supported increased stability of the hydroquinone form of 19-phenyl-DMAG in the active site o

135 onalized surface is toggled from the reduced hydroquinone form to the oxidized benzoquinone form by t

136 is non-emissive, while upon reduction to the hydroquinone form, B-VKQH2, BODIPY fluorescence is resto

137 the monoanion, HQ(-), the kinetically active hydroquinone form, reducing Cu(II) with an intrinsic rat

138 thought to convert the alkoxide-epoxide to a hydroquinone form.

139 current associated with the oxidation of the hydroquinone formed.

140 mical shifts of the BPP34C10, its internal p-hydroquinone forms pi-pi-stacking interactions with only

141 ir of redox-active substituents, quinone and hydroquinone groups, which allow the enantiomerization t

142 ne (GSH)-dependent reduction of glutathionyl-hydroquinones (GS-hydroquinones) to hydroquinones.

143 this end, a liquid battery is designed using hydroquinone (H2BQ) aqueous solution as catholyte and gr

144 es, the amperometric signal using the system hydroquinone/H2O2 was related to the levels of p53-autoa

145 (I) by O2 and the reduction of Cu(II) by 1,4-hydroquinone (H2Q) in the presence of O2 in 0.7 M NaCl s

146 cular cobalt complex, Co(salophen), and para-hydroquinone (H2Q) serve as effective cocatalysts for th

147 orresponding (two-electron) reduction to the hydroquinone (H2Q) via the Pauling bond-length/bond-orde

148 e, HAT= 1,4,5,8,9,12-hexaazatriphenylene) by hydroquinone (H2Q), N-acetyl-tyrosine (N-Ac-Tyr) or guan

149 article contact, the presence of the aqueous hydroquinone (H2Q)/benzoquinone (Q) couple in a flowing

150 Benzene and its metabolite hydroquinone have been shown to lead to decreased levels

151 rs sought to determine whether cytokines and hydroquinone (HQ) affect mCRP expression in cultured hum

152 hiafulvalene (BPTTF) and weaker aryl donors, hydroquinone (HQ) and 1,5-dioxynaphthalene (DNP), respec

153 Phenolic compounds, such as hydroquinone (HQ) and 4-aminophenol (4-AP), were detecte

154 as significantly enhanced in the presence of hydroquinone (HQ) and hydrogen peroxide (H(2)O(2)).

155 es is studied using model phenolic compounds hydroquinone (HQ) and its oxidized counterpart benzoquin

156 do-reference electrode) upon the addition of hydroquinone (HQ) as electron transfer mediator and H(2)

157 the addition of H(2)O(2) in the presence of hydroquinone (HQ) as mediator was used as transduction s

158 upon the addition of H2O2 in the presence of hydroquinone (HQ) as mediator were used to monitor the e

159 rent arising upon addition of H2O2 and using hydroquinone (HQ) as redox mediator in solution.

160 upon the addition of H2O2 in the presence of hydroquinone (HQ) as redox mediator was used as the tran

161 abeled with horseradish peroxidase (HRP) and hydroquinone (HQ) as the redox mediator.

162 change measured in the presence of H(2)O(2)/hydroquinone (HQ) at screen-printed carbon electrodes (S

163 Hydroquinone (HQ) is one of the most frequently used and

164 the effect of nonlethal oxidant injury with hydroquinone (HQ) on MMP-2 activity.

165 Transient kinetics of hydroquinone (HQ) oxidation by wt-TcDyP showed that conv

166 ction was realized by spiking 10 microL of a hydroquinone (HQ) solution into 40 microL of buffer solu

167 amperometric transduction with the H(2)O(2)/hydroquinone (HQ) system at disposable electrodes.

168 tor and amperometric detection with the H2O2/hydroquinone (HQ) system at dual screen-printed carbon e

169 ion at -0.20V was carried out using the H2O2/hydroquinone (HQ) system.

170 ectrodes (SPCEs) in the presence of H(2)O(2)/hydroquinone (HQ) upon magnetic capture of the modified

171 We previously reported that hydroquinone (HQ), a major pro-oxidant in cigarette smok

172 ared from the reaction of bisphenol A (BPA), hydroquinone (HQ), and resorcinol (RS) in different solv

173 The gold standard for treatment is hydroquinone (HQ), which reduces pigmentation through it

174 none (sq), or the two-electron fully reduced hydroquinone (hq).

175 tored by amperometric transduction using the hydroquinone (HQ)/H(2)O(2) system upon capturing the mod

176 ectrode for amperometric detection using the hydroquinone (HQ)/H2O2 system.

177 e tyrosinase inhibitor Thiamidol compared to hydroquinone in women with mild to moderate melasma.

178 es to obtain the corresponding catechols and hydroquinones in good to excellent yields.

179 adical and fully reduced neutral and anionic hydroquinones in solution.

180 convergent access to highly substituted para-hydroquinones in unprotected form via a one-pot Diels-Al

181 itions, Aer cannot be further reduced to the hydroquinone, in contrast to the proposed Aer signaling

182 nones, and neutral and anionic fully reduced hydroquinones--in solution and in inert protein environm

183 tetramers consisting of four monomer units (hydroquinone, indolequinone, and its two tautomers), in

184 ent with anti-ChM-I siRNA markedly abrogated hydroquinone-induced inhibition of tube formation in TrH

185 ChM-I in TrHB-MECs and protected cells from hydroquinone-induced inhibition of tube formation.

186 d tube formation in TrHBMECs; NQO1 inhibited hydroquinone-induced up-regulation of ChM-I in TrHB-MECs

187 Hydroquinone inhibited TrHBMEC tube formation at concent

188 entrin A and herbindole B from a common meso-hydroquinone intermediate prepared by a ruthenium-cataly

189 cleophilic attack in Michael-type additions, hydroquinone is not and acts as a passivating agent.

190 Hydroquinone is oxidized into benzoquinone by the HRP/H2

191 Since GS-trichloro-p-hydroquinone is uncommon in nature, the extensive presen

192 farin-resistant enzyme reducing vitamin K to hydroquinone (KH(2)) is probably not NQO1, (3) there app

193 ch depends upon the oxygenation of vitamin K hydroquinone (KH2).

194 ertiary butyl phenol and monobenzyl ether of hydroquinone, known triggers of vitiligo.

195 ising from the reaction of (L)Pd(OAc)(2) and hydroquinones (L = bathocuproine, 4,5-diazafluoren-9-one

196 istent with that expected of semiquinone and hydroquinone-like moieties respectively.

197 quinone group, such as 1,4-benzoquinone and hydroquinone, likely contributed to the transit toxicity

198 derivatives consisting of 1,4-disubstituted hydroquinones linked by methylene bridges in the 2,5-pos

199 chlorophenolicum made the mutant lose the GS-hydroquinone lyase activities in the cell extracts.

200 The GS-hydroquinone lyase reactions catalyzed by PcpF are rathe

201 Monobenzyl ether of hydroquinone (MBEH) is a Food and Drug Administration ap

202 ulture model to investigate the mechanism of hydroquinone-mediated changes in DNA methylation.

203 arbon counter electrodes, was facilitated by hydroquinone-mediated electron transfer.

204 e substrate (hydrogen peroxide) along with a hydroquinone mediator then allowed an enzymatic reaction

205 peaks developed using H(2)O(2) activator and hydroquinone mediator.

206 ic solutions and support the hypothesis that hydroquinone moieties can reduce Fe(III) in natural wate

207 on of covalent bonds by oxidizing unreactive hydroquinone moieties in LHA to reactive, electrophilic

208 ng electrons from electron-rich phenolic and hydroquinone moieties in the DOM, while O3 reacted via e

209 ne ligand was synthesized with a noninnocent hydroquinone moiety as the central arene (1-H).

210 tion rather than direct redox cycling of the hydroquinone moiety is a source of adaphostin-induced RO

211 s an inner sphere Pd-based process where the hydroquinone moiety only subsequently participates in th

212 teraction of a paraquat beta-proton with the hydroquinone moiety; this is the first time this interac

213 nthesized on controlled pore glass using the hydroquinone-O,O'-diacetic acid linker.

214 nts for the reduction of seven NACs by three hydroquinones of different protonation states.

215 of the cavitand by forming H-bonds with the hydroquinone OH groups.

216 ight into the content and impact of quinones/hydroquinones on the optical properties of HS and CDOM.

217 ely, the results show how Co(salophen) and p-hydroquinone operate synergistically to mediate O2 reduc

218 ted cell surface proteins that catalyze both hydroquinone or NADH oxidation and protein disulfide-thi

219 ants show little or no detectable effects on hydroquinone or quinone exchange and binding at the Q(o)

220 Using flavodoxin hydroquinone or reduced ferredoxin obtained by electron

221 two catalytically active domains, termed the hydroquinone oxidation (Q(o)) and quinone reduction (Q(i

222 osynthetic Rhodobacter capsulatus results in hydroquinone oxidation rates that are between 5 and 50-f

223 te between the arylamine N-hydroxylation and hydroquinone oxidation reactions.

224 by measuring the current required to reduce hydroquinone oxidized during the regeneration of the HRP

225 Quinone/hydroquinone PCET redox couples were used to produce a p

226 -catalyzed deoxyenation of the corresponding hydroquinone precursors is described.

227 pending on the degree of the chlorination of hydroquinone/quinone and the presence or absence of GSH.

228 The benzoquinone and hydroquinone redox couple was examined as a representati

229 Cyclic voltammetry (CV) of the quinone/hydroquinone redox couple was used to monitor the nucleo

230 Benzoquinone/hydroquinone redox interconversion by the reversible Os(

231 This slurry allows the 2e(-)/2H(+) quinone/hydroquinone redox reactions while suppressing proton re

232 anges in the stabilities of the oxidized and hydroquinone redox states of the FMN, none of the replac

233 ine, beta-naphthoflavone, and tertiary butyl hydroquinone reduced MIOX expression.

234 Our results show that 1,4-hydroquinone reduces Fe(III) in acidic conditions, gener

235 Glutathionyl-hydroquinone reductases (GS- HQRs) are a newly identifie

236 S-Glutathionyl-hydroquinone reductases (GS-HQRs) are a new class of glu

237 pigmented human cartilage tissue both showed hydroquinone-resembling NMR signals.

238 rmediates and by-products, including several hydroquinone, resorcinol and catechol derivatives, eithe

239 0 s(-1) mM(-1) for 2,6-dimethoxyphenol and p-hydroquinone, respectively) was localized at the main he

240 r reduction of benzoquinone and oxidation of hydroquinone, respectively.

241 eduction of the quinone to the corresponding hydroquinone results in a chemiluminescent signal.

242 ne steps, including the hydroxylation of the hydroquinone ring by CLK-1 and two O-methylation steps m

243 pillar[5]arene, a macrocycle containing five hydroquinone rings linked through their para positions b

244 ing compounds, including phenol, resorcinol, hydroquinone, serotonin, and ascorbic acid, had minimal

245 In contrast, monosubstituted hydroquinones show a limited amount of ring cleavage but

246 An excess of the hydroxylamine TEMPOH or of hydroquinone similarly reduces Fe(III)~CO(2), and TEMPO

247 , the semiquinone radicals generated in pure hydroquinone solution are rapidly oxidized by dioxygen,

248 proton-coupled electron transfer from a semi-hydroquinone species and a Co(III)-hydroperoxide interme

249 established HAT- and EA-based LFERs for six hydroquinone species.

250 tabilizing the vase form only in the reduced hydroquinone state of the cavitand by forming H-bonds wi

251 ction to either the ASQ or the fully reduced hydroquinone state produces the same conformational resp

252 ase form and are only present in the reduced hydroquinone state.

253 whereas the excited anionic semiquinone and hydroquinone states donate an electron to the adenine mo

254 eutral, and anionic semiquinone, and anionic hydroquinone states.

255 at a higher rate in both the semiquinone and hydroquinone states.

256 Two merotriterpenoid hydroquinone sulfates designated adociasulfate-13 (1) an

257 as developed for determination of tert-butyl-hydroquinone (TBHQ) in edible vegetable oils, based on C

258 ane fusion and virus infectivity, tert-butyl hydroquinone (TBHQ), shows that the inhibitor binds in a

259 utylated hydroxytoluene (BHT) and tert-butyl hydroquinone (TBHQ), were determined in different edible

260 One of the enzymes is tetrachloro-p-hydroquinone (TeCH) reductive dehalogenase (PcpC), which

261 K 2,3-epoxide to vitamin K and to vitamin K hydroquinone, the latter required by the enzyme gamma-ca

262 termined the effect of substrates: vitamin K hydroquinone, the pentapeptide FLEEL, and NaHCO(3), on t

263 Reduction of quinones to hydroquinones, the major redox active moieties in NOM, i

264 y of (L)Pd(II)(OAc)(2)-mediated oxidation of hydroquinones, the microscopic reverse of quinone-mediat

265 he O(2)-dependent, two-electron oxidation of hydroquinones, the protein was reprogrammed to catalyse

266 The asymmetric reduction of emodin hydroquinone to (R)-3,8,9,10-tetrahydroxy-6-methyl-3,4-d

267 eir corresponding radicals ArO(*) and TEMPO, hydroquinone to benzoquinone, and dihydroanthracene to a

268 pressure-corrected diffusion coefficient of hydroquinone to demonstrate the effect of this phenomeno

269 gulation of ChM-I may explain the ability of hydroquinone to inhibit TrHB-MEC tube formation.

270 ructural basis of electron transfer from FMN-hydroquinone to its partners, three deletion mutants in

271 e of formal potential on pH and oxidation of hydroquinone to quinone by O(2) at basic pH.

272 ation, cytochrome bc(1) efficiently oxidizes hydroquinone to quinone, but how it performs this reacti

273 epoxide reductase (VKOR) generates vitamin K hydroquinone to sustain gamma-carboxylation of many bloo

274 (GSH)-dependent reduction of GS-trichloro-p-hydroquinone to trichloro-p-hydroquinone.

275 n shown to catalyze the oxidation of various hydroquinones to benzoquinones in the presence of t-BuOO

276 e to air, consistent with the reoxidation of hydroquinones to quinones.

277 -HQRs from yeast and bacteria reduced the GS-hydroquinones to the corresponding hydroquinones.

278 reduction of glutathionyl-hydroquinones (GS-hydroquinones) to hydroquinones.

279 elasma Area and Severity Index scores on the hydroquinone-treated side.

280 proved on both the Thiamidol-treated and the hydroquinone-treated sides of the face.

281 In summary, hydroquinone treatment up-regulated ChM-I and inhibited

282 -dependent conversion of TeCH to trichloro-p-hydroquinone (TriCH) and then to dichloro-p-hydroquinone

283 onors such as NADH, pinacyanol chloride, and hydroquinone undergo the TAML-catalyzed oxidation by O2.

284 ch is composed of a crown ether containing a hydroquinone unit and a 1,5-diaminonaphthalene unit, int

285 intramolecular electron transfer between the hydroquinone unit and the oxidized metal macrocycle occu

286 le diprotonated derivative in which only the hydroquinone unit resides inside the cavity of the tetra

287 ol; 0.14 muM for pyrogallol and 0.21 muM for hydroquinone, using square-wave voltammetry (SWV).

288 es spontaneously reacted with GSH to form GS-hydroquinones via Michael addition, and four GS-HQRs fro

289 Hydroquinone was found to up-regulate chondromodulin-I (

290 0.2V vs. Ag|AgCl while a mixture of H2O2 and hydroquinone was injected into the microfluidic device.

291 lectrochemical oxidation of enzyme-generated hydroquinone was measured.

292 When 2,3-disubstituted hydroquinones were employed as starting materials the 2,

293 In addition, two chlorinated o-hydroquinones were prepared, 6-chloro-biphenyl-3,4-diol

294 s a reduction of a quinone intermediate to a hydroquinone, which goes hand-in-hand with a stabilizing

295 site is assumed to be a polycyclic aromatic hydroquinone whose oxidation to the corresponding conjug

296 nyl compounds using catalytic amounts of 1,4-hydroquinone with a copper nanoparticle electron transfe

297 Interactions of 1,4-hydroquinone with soluble iron species over a pH range o

298 ons led to the reduction of benzoquinones to hydroquinones with the concomitant oxidation of GSH to o

299 substrate specificity for 2,6-disubstituted hydroquinones, with halogens greatly preferred at those

300 demonstrate that direct excitation of flavin hydroquinone within "ene"-reductase active sites enables