ライフサイエンスコーパス: hydroxyindoleacetic acid

1 mpulse control, alcohol abuse, and low CSF 5-hydroxyindoleacetic acid.

2 e test and/or measurement of CSF levels of 5-hydroxyindoleacetic acid.

3 Interestingly, cerebrospinal fluid (CSF) 5-hydroxyindoleacetic acid (5-HIAA) and fenfluramine-induc

4 the platelet-derived serotonin metabolite 5-hydroxyindoleacetic acid (5-HIAA) as a GPR35 ligand.

5 he relationship between aggression and CSF 5-hydroxyindoleacetic acid (5-HIAA) concentration with tha

6 separation after birth; their behavior and 5-hydroxyindoleacetic acid (5-HIAA) concentrations in CSF

7 an increase in homovanillic acid (HVA) or 5-hydroxyindoleacetic acid (5-HIAA) concentrations in the

8 Low concentrations of CSF 5-hydroxyindoleacetic acid (5-HIAA) have been consistently

9 liability for antisocial behavior and CSF 5-hydroxyindoleacetic acid (5-HIAA) in newborns was explor

10 he present study investigated the level of 5-hydroxyindoleacetic acid (5-HIAA) in perforated and nonp

11 owed reduced levels of the 5-HT metabolite 5-hydroxyindoleacetic acid (5-HIAA) in several regions inc

12 (DA), serotonin (5-HT) and its metabolite 5-hydroxyindoleacetic acid (5-HIAA) in the PVN.

13 47 vs 122.0 +/- 50.7 pmol/mL; P<.01), and 5-hydroxyindoleacetic acid (5-HIAA) levels were significan

14 We aimed to determine the impact of 5-hydroxyindoleacetic acid (5-HIAA) on septic shock as a n

15 5-hydroxyindoleacetic acid (5-HIAA), a biomarker of pharma

16 scans and changes in the level of urinary 5-hydroxyindoleacetic acid (5-HIAA), a metabolite of serot

17 eir migration to the inflammatory mediator 5-hydroxyindoleacetic acid (5-HIAA), a serotonin metabolit

18 CSF homovanillic acid (HVA), 5-hydroxyindoleacetic acid (5-HIAA), and 3-methoxy-4-hydro

19 d duodenal mucosal concentrations of 5-HT, 5-hydroxyindoleacetic acid (5-HIAA), and kynurenic acid (K

20 s in tissue levels of 5-HT, its metabolite 5-hydroxyindoleacetic acid (5-HIAA), and norepinephrine in

21 tic acid (DOPAC), homovanillic acid (HVA), 5-hydroxyindoleacetic acid (5-HIAA), and serotonin (5-HT)

22 doses and the serotonin (5-HT) metabolite, 5-hydroxyindoleacetic acid (5-HIAA), at the highest dose.

23 f tryptophan and the serotonin catabolite, 5-hydroxyindoleacetic acid (5-HIAA), but not that of the n

24 in the brain content of serotonin (5-HT), 5-hydroxyindoleacetic acid (5-HIAA), dopamine (DA), norepi

25 by reduced brainstem serotonin (5-HT) and 5-hydroxyindoleacetic acid (5-HIAA), fewer 5-HT(1A) autore

26 fer from controls in CSF concentrations of 5-hydroxyindoleacetic acid (5-HIAA), homovanillic acid (HV

27 ptamine (5-HT) and its primary metabolite, 5-hydroxyindoleacetic acid (5-HIAA), in prefrontal cortex

28 Cerebrospinal fluid was assayed for 5-hydroxyindoleacetic acid (5-HIAA), norepinephrine, 3-met

29 Brain levels of 5-HT and its metabolite, 5-hydroxyindoleacetic acid (5-HIAA), were determined using

30 hromogranin A (CgA), serotonin, or urinary 5-hydroxyindoleacetic acid (5-HIAA), while topographic loc

31 cells respond to the serotonin metabolite 5-hydroxyindoleacetic acid (5-HIAA).

32 y serotonin and the metabolite interferant 5-hydroxyindoleacetic acid (5-HIAA).

33 analyze levels of 5-HT and its metabolite, 5-hydroxyindoleacetic acid (5-HIAA); levels of TPH2; and 5

34 els of the major metabolites of serotonin (5-hydroxyindoleacetic acid [5-HIAA]), dopamine (homovanill

35 Her 5-hydroxyindoleacetic acid and chromogranin A levels were

36 Thyroid function was normal, as were 5-hydroxyindoleacetic acid and chromogranin A levels.

37 The CSF 5-hydroxyindoleacetic acid and homovanillic acid levels di

38 We used CSF (AVP), CSF 5-hydroxyindoleacetic acid, and the prolactin response to

39 Tumor markers (chromogranin A and 5-hydroxyindoleacetic acid) before and after treatment wer

40 oholism and lower mean cerebrospinal fluid 5-hydroxyindoleacetic acid concentration than late-onset a

41 f alcoholism and lower cerebrospinal fluid 5-hydroxyindoleacetic acid concentration.

42 ly predictive of lower cerebrospinal fluid 5-hydroxyindoleacetic acid concentrations in controls but

43 5-HT and 5-hydroxyindoleacetic acid content of limbic nuclei were d

44 rebral glucose utilization, while 5-HT and 5-hydroxyindoleacetic acid content were quantified in limb

45 dopamine, and norepinephrine metabolites (5-hydroxyindoleacetic acid, homovanillic acid, and 3-metho

46 particularly low mean cerebrospinal fluid 5-hydroxyindoleacetic acid, homovanillic acid, and tryptop

47 t changes were observed in CSF measures of 5-hydroxyindoleacetic acid, homovanillic acid, dehydroepia

48 had raised ratios of homovanillic acid to 5-hydroxyindoleacetic acid in cerebrospinal fluid, of rang

49 Tail pinch had no effect on 5-hydroxyindoleacetic acid in either brain region.

50 nding of low cerebrospinal fluid levels of 5-hydroxyindoleacetic acid in higher-lethality suicide att

51 concentrations of the serotonin metabolite 5-hydroxyindoleacetic acid in the CSF of suicide cases ver

52 noradrenaline and the serotonin metabolite 5-hydroxyindoleacetic acid in the hippocampus.

53 s on extracellular 5-hydroxytryptamine and 5-hydroxyindoleacetic acid in the striatum and hippocampus

54 ve determination of tryptophan, serotonin, 5-hydroxyindoleacetic acid, kynurenine, and kynurenic acid

55 lamine increased, and serotonin metabolite 5-hydroxyindoleacetic acid levels decreased, to a much gre

56 ancreatic polypeptide, and 24-hour urinary 5-hydroxyindoleacetic acid levels in all patients.

57 angiographic findings, chromogranin A and 5-hydroxyindoleacetic acid levels were measured and were 7

58 tered significantly by forced swimming but 5-hydroxyindoleacetic acid levels were reduced to 60% of b

59 nockout mice had normal levels of 5-HT and 5-hydroxyindoleacetic acid, possibly because of an up-regu

60 ge, region of origin, and urinary level of 5-hydroxyindoleacetic acid predicted survival by univariat

61 a mediating phenotype, cerebrospinal fluid 5-hydroxyindoleacetic acid, provides preliminary evidence

62 Concentrations of dopamine and 5-hydroxyindoleacetic acid, the main metabolite of seroton

63 mine to 90% above basal levels and reduced 5-hydroxyindoleacetic acid to 45% of basal levels in the s

64 amino acid-derived metabolites, including 5-hydroxyindoleacetic acid (total dietary fiber - compleme

65 resected was 33 cm, and mean 24-hour urine 5-hydroxyindoleacetic acid was 120 mg.

66 tly increased (2-fold), but its metabolite 5-hydroxyindoleacetic acid was not.

67 d DA and higher levels of MAO A metabolite 5-hydroxyindoleacetic acid were found in the forebrain reg

68 yphenylacetic acid, homovanillic acid, and 5-hydroxyindoleacetic acid were measured in the brainstem,

69 ith PRL[d-FEN] responses (but not with CSF 5-hydroxyindoleacetic acid), which in turn was correlated