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1 ulation of intermediates of this pathway and hydroxyl radical.
2 ldiacylglycerol from singlet oxygen and from hydroxyl radical.
3 oxidative cycles normally controlled by the hydroxyl radical.
4 t wetland) that generated singlet oxygen and hydroxyl radical.
5 ecies or decay and, in doing so, produce the hydroxyl radical.
6 higher particle concentrations produced more hydroxyl radical.
7 were allowed to decay over time induced less hydroxyl radical.
8 ioxide, which forms when nitrite reacts with hydroxyl radical.
9 OH oxidation and is stable towards attack by hydroxyl radicals.
10 ss iron can lead to the formation of harmful hydroxyl radicals.
11 ts with GSH, and react with H2 O2 generating hydroxyl radicals.
12 cesses and changes the budget of atmospheric hydroxyl radicals.
13 o its critical role as a nighttime source of hydroxyl radicals.
14 tended metal release and production of toxic hydroxyl radicals.
15 photochemical reactor where it reacted with hydroxyl radicals.
16 akage inhibition induced by both peroxyl and hydroxyl radicals.
17 r where it is an important source of daytime hydroxyl radicals.
18 more efficiently by sulfate radicals than by hydroxyl radicals.
19 romatic rings by substitution of hydrogen by hydroxyl radicals.
20 of processes whose photochemistry generates hydroxyl radicals.
21 onstants for reactions of the compounds with hydroxyl radicals.
22 y binds to nucleosomes and protects DNA from hydroxyl radicals.
23 ue to its catalytic role in the formation of hydroxyl radicals.
24 sibly, cell expansion via H(2) O(2) -derived hydroxyl radicals.
25 of solvent-exposed amino acid side chains by hydroxyl radicals.
26 binding protein that protects chromatin from hydroxyl radicals.
27 ration of superoxide, hydrogen peroxide, and hydroxyl radicals.
28 r hydrogen peroxide treatment, that generate hydroxyl radicals.
29 leosomes and protecting chromosomal DNA from hydroxyl radicals.
32 ) is produced by the formal dehydration of a hydroxyl radical adduct of dG as well as by deprotonatio
33 s activated water molecules to form covalent hydroxyl radical adducts at nearby residues, which were
34 ochlorous acid and hypochlorite) to generate hydroxyl radical, along with ozone and a suite of haloge
38 undergoes O-O homolytic cleavage to yield a hydroxyl radical and Cu(II)OH rather than heterolytic cl
39 ous acid (HONO) is a photochemical source of hydroxyl radical and nitric oxide in the atmosphere that
40 ities involved in these reactions are likely hydroxyl radical and singlet oxygen based on the use of
43 le which couples the oxidation of ammonia by hydroxyl radical and the reaction of nitric acid with am
45 to TiO2 to produce three- and four-fold more hydroxyl radicals and hydrogen peroxide, respectively, t
46 e of the catalytic decomposition of ozone to hydroxyl radicals and increase of the hydrophilicity of
48 drogen peroxide (H2O2) with the formation of hydroxyl radicals and the consequent oxidation of the pe
49 r the parameters affecting the production of hydroxyl radicals and their spin trapping with DMPO were
50 Apo-transferrin increased the formation of hydroxyl radicals and this related with a faster degrada
51 brils can destroy H2O2 and generate damaging hydroxyl radicals and, so, are not necessarily inert end
52 processes such as photolysis, reactions with hydroxyl radicals, and aerosol uptake were found to be i
53 xamined as hydrogen donors to DPPH, ABTS and hydroxyl radicals, and as electron donors in the FRAP as
54 four ROS studied (hydrogen peroxide, ozone, hydroxyl radicals, and superoxide anions), only H2O2 and
58 te anions or bisulfite anions) with holes or hydroxyl radicals are the active species for MO photodeg
59 Our mechanistic investigations reveal that hydroxyl radicals are unreactive toward HFPO-DA, while e
60 footprinting approach inside cells in which hydroxyl radicals are used to oxidatively modify protein
61 cavengers of the ROS superoxide (O2(*-)) and hydroxyl radical, are preferentially internalized by T l
63 lease from Hb and the subsequent reaction of hydroxyl radicals, as strong oxidizing agents, with CDs
64 contents and scavenging of ABTS(+), DPPH and hydroxyl radicals, as well as metal chelation of the sol
65 ct at an equally fast rate with atrazine (k (hydroxyl radical + atrazine) = 3 x 10(9) M(-1) s(-1)).
66 xidation and enters the cytoplasm inflicting hydroxyl radical attack on intracellular proteins and DN
68 ere we describe footprinting of Abeta1-42 by hydroxyl radical-based fast photochemical oxidation of p
69 ence is presented for in vitro generation of hydroxyl radicals because of redox cycling of environmen
71 ation and steady-state concentrations of the hydroxyl radical by cloud chemistry models and for organ
72 ic pollutants with oxidants such as ozone or hydroxyl radicals by compound-specific stable isotope an
75 Further research on processes related to the hydroxyl radical chemistry in the environmental compartm
79 idth, propeller twist, roll, helix twist and hydroxyl radical cleavage predictions for the entire gen
82 tial distribution of radicals, we found that hydroxyl radical concentration was strongly dependent on
87 nd DOM isolates revealed that reactions with hydroxyl radicals dominated the transformation of tested
88 en UV spectrophotometric response, effective hydroxyl radical dose delivered, and peptide and protein
89 o compare HRPF data from sample to sample, a hydroxyl radical dosimeter is needed that can measure th
90 ne (epsilon(C) = -3.6 to -4.6 per mille) and hydroxyl radicals (epsilon(C) = 0.0 to -1.2 per mille).
91 d on activity of DPPH radical, ABTS radical, hydroxyl radical, Fe(2+) chelating ability and reducing
98 f small angle X-ray scattering (SAXS), X-ray hydroxyl radical footprinting, circular dichroism, and H
100 radicals were regionally more important than hydroxyl radicals for alkane oxidation and were also imp
102 orbic acid depletion (denoted as OP(AA)) and hydroxyl radical formation (denoted as OP(*OH)) from bot
103 complex which might yield Fe(IV) instead of hydroxyl radical formation as suggested in literature.
104 ammatory reactions since E. coli can inhibit hydroxyl radical formation by eliminating substrates of
106 ticles (Fe3O4 MNPs) to the medium to produce hydroxyl radicals from H2O2, owing to the peroxidase-lik
107 se results demonstrate that plasma-generated hydroxyl radicals from water can be used to map protein
108 omolecules), that generates micros bursts of hydroxyl radicals from water, to measure changes in prot
110 (RP-MS), first introduced in 1999, utilizes hydroxyl radicals generated directly within aqueous solu
111 degraded in aqueous systems in presence of a hydroxyl radical generating system such as ascorbic/iron
113 in, after accounting for both differences in hydroxyl radical generation and nonanalyte radical consu
114 reas ovotransferrin completely inhibited the hydroxyl radical generation by a system containing ascor
115 , W(18)@Hf(12)-DBB-Ir significantly enhances hydroxyl radical generation from Hf(12) SBUs, singlet ox
118 showed that Ag NPs were not able to catalyse hydroxyl radical generation, but that they directly oxid
119 NPs under SSL exposure could be explained by hydroxyl radical generation, the enhanced toxicity of Ag
120 gen abstraction reactions from isobutanol by hydroxyl radical have been calculated using multi-path v
121 species - superoxide, hydrogen peroxide and hydroxyl radicals - have long been suspected of constrai
122 so suggested that SO4(*-) was transformed to hydroxyl radical (HO(*)) and carbonate radical (CO3(*-))
126 nned a range of reactivity with UV light and hydroxyl radical (HO(*)) in three different types of sou
127 ersulfate into sulfate radical (SO4(*-)) and hydroxyl radical (HO(*)) over time scales of several wee
128 o acids (FAA) against Fenton system-mediated hydroxyl radical (HO(*)) production in aqueous solution,
133 dation process that produces highly reactive hydroxyl radicals (HO(*)) and chlorine radicals (Cl(*))
136 advanced oxidation processes (AOPs) produce hydroxyl radicals (HO*) which can completely oxidize ele
137 ased mixing ratio of hydroperoxyl radical to hydroxyl radical ([HO2]/[OH]) and increased [NO2]/[NO] w
138 10(3) L mgC(-1) s(-1) (mgC = mg carbon); k (hydroxyl radical + humic acids) = 1.4 x 10(4) L mgC(-1)
139 ment of the contributions of singlet oxygen, hydroxyl radical, hydrogen peroxide, and triplet dissolv
141 photogeneration of equimolar superoxide and hydroxyl radical in desiccated and aqueous soils, respec
142 We provide an extensive view on the role of hydroxyl radical in different environmental compartments
143 d retinal pigmented epithelial cells against hydroxyl radicals in a dose-dependent manner by maintain
145 alytic system with the most effective use of hydroxyl radicals in oxidation treatment scenarios.
146 NIA (FER), is required for the production of hydroxyl radicals in the female gametophyte, which cause
148 hophorothioated DNA reacted to both H2O2 and hydroxyl radicals in vivo, and protected genomic DNA as
152 lysis of voltammograms demonstrated that the hydroxyl radical is a principal contributor to the volta
154 trary to frequent reports in the literature, hydroxyl radical is not a key species participating in e
155 her concentrations of oxides of nitrogen and hydroxyl radicals, is more efficient in terms of O(3) an
156 (kO3) (<0.1-7.9 x 10(3) M(-1) s(-1)) or with hydroxyl radicals (k(*)OH) (0.9 x 10(9) - 6.5 x 10(9) M(
160 Using time-resolved X-ray-mediated in situ hydroxyl radical labeling, we probed real-time solvent a
162 de dismutase), hydrogen peroxide (catalase), hydroxyl radicals (mannitol) and singlet oxygen (sodium
163 ferrin and ovotransferrin; could prevent the hydroxyl radical mediated degradation of beta-glucan.
164 of archaeal RNase P, we used a site-specific hydroxyl radical-mediated footprinting strategy to pinpo
165 copies that bound the RPR and site-specific hydroxyl radical-mediated footprinting to localize the K
166 ficantly increases the signal quality of the hydroxyl radical modification products and the dose-resp
167 ess (e.g., hydrogen peroxide, superoxide and hydroxyl radicals, nitric oxide, ascorbic acid, and glut
169 for an autoxidation mechanism, initiated by hydroxyl radical (OH) addition to C=C bonds and propagat
170 el air through a PAM reactor over integrated hydroxyl radical (OH) exposures ranging from approximate
175 nic aerosol (SOA) via aqueous reactions with hydroxyl radical (OH), singlet oxygen ((1)O2*), and exci
177 pproximately 60% to the primary formation of hydroxyl radical (OH), which is a key oxidant in the deg
179 of nitroblue tetrazolium into formazan, and hydroxyl radicals (OH( *)) were detected by the hydroxyl
181 is work, we have elucidated the mechanism of hydroxyl radicals (OH(*)) generation and its life time m
183 atmospheric photochemical oxidants, such as hydroxyl radicals (OH(*)), nitrogen oxides (NOx), and oz
186 be competitive with multiphase oxidation by hydroxyl radicals (OH) and ozonolysis of gaseous alpha-t
187 ir-water interface from two potent oxidizers hydroxyl radicals (OH) and singlet delta oxygen (SDO).
191 sphere is initiated primarily by addition of hydroxyl radicals (OH) to C4 or C1 in a ratio 0.57 +/- 0
192 phase oxidation of cis-pinonic acid (CPA) by hydroxyl radicals (OH) was studied using a relative rate
193 es of BCA and LA due to reactions with O(3), hydroxyl radicals (OH), and due to photolysis were calcu
194 photolysis of nitrous acid (HONO) generates hydroxyl radicals (OH), and since OH is fast reacting, i
198 um yields, contradictory evidence exists for hydroxyl radical ((*)OH) and hydroxylating species.
199 ap, 5,5-dimethyl-1-pyrroline-N-oxide (DMPO), hydroxyl radical ((*)OH) and sulfate radical anion (SO4(
202 n rates, illustrating a possible role of the hydroxyl radical ((*)OH) in the anodic formation of sulf
205 nsitizers from hydroxylating species such as hydroxyl radical ((*)OH) present in dissolved organic ma
206 the UV/H2O2 AOP via UV direct photolysis and hydroxyl radical ((*)OH) reaction, so that DBPs most lik
207 dicals are recognized to form as products of hydroxyl radical ((*)OH) scavenging by halides, their co
209 rocess (AOP) frequently employed to generate hydroxyl radical ((*)OH) to treat reverse osmosis permea
210 els, forming: (1) nitrogen dioxide (NO2) and hydroxyl radical ((*)OH), (2) nitrite (NO2(-)) and oxyge
211 ived reactive intermediates (RIs), including hydroxyl radical ((*)OH), singlet oxygen ((1)O2) and sup
214 generation of reactive oxygen species (ROS) [hydroxyl radicals ((*)OH) and hydrogen peroxide (H2O2)]
215 ter ((3)DOM*), singlet oxygen ((1)O(2)), and hydroxyl radicals ((*)OH), for surface waters collected
216 orus chemistry, and its association with the hydroxyl radical ((.) OH) to yield metaphosphorous acid
218 dence of the photochemical production of the hydroxyl radical (*OH) from dissolved organic matter (DO
219 reaction for production of highly efficient hydroxyl radicals (*OH) and consequently suppressing the
220 ctivity of the impregnated solids to produce hydroxyl radical (.OH) from H2O2 decomposition was evalu
221 the well understood mechanism through which hydroxyl radical (.OH) produced by nitrate and nitrite p
222 s evaluated by (DPPH.), superoxide (O2(.-)), hydroxyl radicals (.OH) and hypochlorous acid (HOCl) ass
223 (GAC) (O(3)/GAC) to promote the formation of hydroxyl radicals (.OH) was evaluated at 1.0 mg O(3)/mg
225 side, the impact of the reactivity of indoor hydroxyl radicals on health and well-being is another em
227 found able to scavenge superoxide anion and hydroxyl radicals, organic nitro-radicals (ABTS, DPPH) a
228 l precursor/product mix formed after aqueous hydroxyl radical oxidation and droplet evaporation under
229 nt contributor to RNA strand scission by the hydroxyl radical, particularly under anaerobic condition
230 iologically relevant radicals, including the hydroxyl radical, peroxyl radicals, the trioxidocarbonat
233 ties of platinum nanoparticles combined with hydroxyl radical probes produced at the particle surface
239 in doxorubicin uptake, temperature increase, hydroxyl radical production and nuclear membrane modific
240 ous-phase oxidation reaction: iron-catalyzed hydroxyl radical production from hydrogen peroxide (Fent
242 oeostasis can induce cellular damage through hydroxyl radical production, which can cause the oxidati
244 lows mass spectrometry-based high-resolution hydroxyl radical protein footprinting (HR-HRPF) measurem
247 hemical oxidation of proteins (IV-FPOP) is a hydroxyl radical protein footprinting method used to stu
248 r dissociation-based high spatial resolution hydroxyl radical protein footprinting to identify two se
249 r dissociation-based high spatial resolution hydroxyl radical protein footprinting, which shows great
250 Bimolecular reaction rate constants with hydroxyl radicals ranged from (2.04 +/- 0.37) x 10(9) to
252 unted for 17 +/- 19% of the regional kinetic hydroxyl radical reactivity of nonbiogenic VOCs suggesti
253 oxidation by active chlorine, as well as by hydroxyl radicals resulting from its reaction with iron
254 II)-concentrations, absence vs presence of a hydroxyl radical scavenger (dimethyl sulfoxide, DMSO), a
257 cies levels (nitric oxide, superoxide anion, hydroxyl radical scavenger capacity) and cellular antiox
259 bed sunlight was affected by the presence of hydroxyl radical scavengers, indicating the likely invol
260 trolysine yields decreased in the absence of hydroxyl radical scavengers, suggesting that future rese
261 ing ability (DRSA), reducing power (RP), and hydroxyl radical scavenging ability (HRSA) assays to tes
262 h in-vitro assays namely, DPPH(*), ABTS(*+), hydroxyl radical scavenging ability, reducing activity,
264 l as determined by ABTS and DPPH assays, and hydroxyl radical scavenging capacity, reducing power as
265 s requires high flux density to overcome the hydroxyl radical scavenging reactions produced by the bu
267 ous Ion-chelating Ability, 221.46 mug mL(-1) Hydroxyl radical scavenging, 279.02 mug mL(-1) Peroxyl r
268 phenolics, flavonoids, ABTS free radical and hydroxyl radicals scavenging and anti-inflammatory activ
269 comparable 1,1-diphenyl-2-picrylhydrazyl and hydroxyl radical-scavenging activities in most cases.
271 d similar antioxidant properties, except for hydroxyl radical-scavenging activity, higher on green te
272 ighest reducing power (absorbance 0.366) and hydroxyl-radical-scavenging activity (91%) at 15 mg/mL;
274 roduced higher amounts of singlet oxygen and hydroxyl radicals than free MB, possibly due to better d
277 bond homolysis, leading to the formation of hydroxyl radicals that give rise to alcohol/ketone (A/K)
278 duced hydrogen peroxide photolysis generates hydroxyl radicals that react with solvent-accessible sid
279 s contrasts with the behavior of ROS such as hydroxyl radicals that selectively abstract allylic and/
280 ltraviolet (UV) radiation (lambda=254nm) and hydroxyl radicals, the intensity of the emitted photolum
281 in particular have the potential to generate hydroxyl radicals, the most hazardous among all ROS.
283 mical oxidation of proteins (FPOP), utilizes hydroxyl radicals to oxidatively modify solvent accessib
285 en, superoxide anion, hydrogen peroxide, and hydroxyl radicals, to afford superb antitumor efficacy o
286 d reactive conformation and guides a derived hydroxyl radical toward formation of a copper-oxyl inter
287 f photochemically generated oxidants such as hydroxyl radicals, ultimately leading to an enhanced atm
288 of aromatic hydrocarbons promptly react with hydroxyl radicals undergoing oxidation to form phenols a
290 that the molecules investigated reacted with hydroxyl radical via both HAT and SPLET in the solvents
291 ated Fe(II) represent an important source of hydroxyl radical via the Fenton reaction in cloudwater.
296 f atmospheric oxidants such as ozone and the hydroxyl radical, which controls the self-cleansing capa
298 ly absorbed X-rays and efficiently generated hydroxyl radicals, which enhanced the radiotherapy effec
299 de superoxide, hydrogen peroxide (H2O2), and hydroxyl radicals, whose killing is amplified by iron vi