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1 ration of (E)-3-methylglutaconyl-CoA to (3S)-hydroxymethylglutaryl-CoA.
2 f acetyl-CoA, acetoacetyl-CoA, succinyl-CoA, hydroxymethylglutaryl-CoA, and malonyl-CoA confirmed tha
3    In this study, the comparative effects of hydroxymethylglutaryl-CoA (HMG-CoA) reductase inhibitors
4                                              Hydroxymethylglutaryl-CoA (HMG-CoA) reductase is the pri
5                                        Avian hydroxymethylglutaryl-CoA (HMG-CoA) synthase is inactiva
6  acetoacetyl-CoA, succinyl-CoA, malonyl-CoA, hydroxymethylglutaryl-CoA (HMG-CoA), and acetyl-CoA in I
7 dicate that HMGCLL1 is an extramitochondrial hydroxymethylglutaryl-CoA lyase.
8 iNOS expression, and NF-kappaB activation by hydroxymethylglutaryl-CoA, mevalonate, and farnesyl pyro
9                   The pleiotropic actions of hydroxymethylglutaryl CoA reductase inhibitors (statins)
10 d that "high-risk" patients are treated with hydroxymethylglutaryl CoA reductase inhibitors to reduce
11 nhibited by both lovastatin, an inhibitor of hydroxymethylglutaryl CoA reductase, and alendronate.
12 REBP binding to three specific target genes: hydroxymethylglutaryl-CoA reductase (Red), fatty acid sy
13 (AMPK), phosphorylates and regulates in vivo hydroxymethylglutaryl-CoA reductase and acetyl-CoA carbo
14 main, we made a fusion protein between yeast hydroxymethylglutaryl-CoA reductase and GFP.
15  protein pathway and increases expression of hydroxymethylglutaryl-CoA reductase and low density lipo
16 ed in the regulated degradation of wild-type hydroxymethylglutaryl-CoA reductase in the ER membrane.
17 cutely injured lungs from sepsis (SAILS) and hydroxymethylglutaryl-CoA reductase inhibition with simv
18 or the hepatic uptake of this liver-specific hydroxymethylglutaryl-CoA reductase inhibitor in rat and
19  hamster ovary cell lines with lovastatin (a hydroxymethylglutaryl-CoA reductase inhibitor) and meval
20 sulfate, and thyroid hormone, as well as the hydroxymethylglutaryl-CoA reductase inhibitor, pravastat
21 dence suggests that long term treatment with hydroxymethylglutaryl-CoA reductase inhibitors, or stati
22 ord "surgery" and the MeSH term for statins "hydroxymethylglutaryl-CoA reductase inhibitors." Studies
23 lated degradation consistent with the native hydroxymethylglutaryl-CoA reductase proteins.
24 ion (cytochrome c) or cholesterol synthesis (hydroxymethylglutaryl-CoA reductase).
25 carboxykinase, and type I deiodinase but not hydroxymethylglutaryl-CoA reductase, cytochrome c, and a
26 e stimulated the rapid inactivation of their hydroxymethylglutaryl-CoA reductase, presumably through
27 of gamma-tocotrienol, indicating the role of hydroxymethylglutaryl-CoA reductase.
28 -binding protein-2 and reduced expression of hydroxymethylglutaryl-CoA reductase.
29 ding protein cleavage-activating protein and hydroxymethylglutaryl-CoA reductase.
30 tein maturation and promoting degradation of hydroxymethylglutaryl-CoA reductase.
31 e, we evaluated mice lacking mitochondrial 3-hydroxymethylglutaryl CoA synthase (HMGCS2) to determine
32 his enzyme is unique in that it is the first hydroxymethylglutaryl CoA synthase that is insensitive t
33 e, the first characterization of an archaeal hydroxymethylglutaryl CoA synthase.
34 n regulatory factor 1 (IRF-1), mitochondrial hydroxymethylglutaryl-CoA synthase (HMG-CoA synthase), a
35 pecific beta-lactone inhibitor of eukaryotic hydroxymethylglutaryl-CoA synthase (HMGCS).
36  an effective competitive inhibitor of avian hydroxymethylglutaryl-CoA synthase (Ki = 28 microm).
37 talyses the first reaction in ketogenesis: 3-hydroxymethylglutaryl-CoA synthase 2 (HMGCS2).
38 nd catalase, and show that these, as well as hydroxymethylglutaryl-CoA synthase and sarcosine dehydro
39 nding cassette transporters ABCA1 and ABCG5, hydroxymethylglutaryl-CoA synthase and the LDL receptor)
40 odular polyketide synthase (PKS) proteins, a hydroxymethylglutaryl-CoA synthase cassette and three fl
41 -methyl and beta-ethyl branches catalyzed by hydroxymethylglutaryl-CoA synthase homologues that utili
42 ency resulted in up-regulation of intestinal hydroxymethylglutaryl-CoA synthase mRNA and an increase
43                                              Hydroxymethylglutaryl-CoA synthase-catalyzed condensatio
44 nzymes that enolize this analog, occurs with hydroxymethylglutaryl-CoA synthase.