戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 oxia) and increased inspired carbon dioxide (hypercarbia).
2 seline to 1.96 +/- 0.18 muL/mm(2)/min during hypercarbia.
3 nt improvement in oxygenation while limiting hypercarbia.
4 nchanged during hypoxia and increased during hypercarbia.
5  a reduction in tidal volume with subsequent hypercarbia.
6 and permits identification of the causes for hypercarbia.
7 ion for exacerbation of COPD associated with hypercarbia.
8 uch as increased intraabdominal pressure and hypercarbia.
9 -REM sleep, with continuous desaturation and hypercarbia.
10 er trial comparing hypoxia (17% FIO(2)) with hypercarbia (2.7% FICO(2)).
11 urons in the BF impairs cortical arousals to hypercarbia [4], but which BF cell types mediate cortica
12 ally detrimental conditions (e.g. hypoxemia, hypercarbia, acidosis, hypothermia, hypervolemia, and in
13 ala as an important chemosensor that detects hypercarbia and acidosis and initiates behavioral respon
14 t was not receiving lorazepam in response to hypercarbia and failed to rise while the patient was rec
15 entilation, potentially aggravating systemic hypercarbia and hypoxemia.
16 ence points to impaired arousal responses to hypercarbia and hypoxia, which ultimately leads to asphy
17 f their jugular veins and aorta in room air, hypercarbia, and 100% O2.
18 equences include increased airway pressures, hypercarbia, and decreased pulmonary compliance.
19 early onset (min to hr) rise in temperature, hypercarbia, and muscular rigidity following exposure to
20 ng resting rCBF or the elevation elicited by hypercarbia (arterial CO2 pressure, Pa,CO2, approximatel
21 ccelerated by hyperoxemia, hyperglycemia and hypercarbia but inhibited by hypoxemia and hypoglycemia;
22 ncrease in ventilation due to the O2-induced hypercarbia [calculated as the production (delta VE/delt
23 did not change significantly relative to the hypercarbia condition but remained significantly elevate
24                                              Hypercarbia decreased SaO(2) (-2.6+/-0.6%, P=0.002) comp
25                             Both hypoxia and hypercarbia decreased the balance between pulmonary and
26  deadspace are sufficient to account for the hypercarbia developed by patients with acute exacerbatio
27 anges in PaCO2 and SaO2; and that O2-induced hypercarbia does not indicate a failure of respiratory c
28               Although both groups developed hypercarbia during oleic acid infusion, PaCO2, steadily
29 ventilation-perfusion ratios), adding to the hypercarbia from increased deadspace.
30 mia/hypoglycemia, hyperoxemia/hypoxemia, and hypercarbia/hypocarbia - conditions common in cancer pat
31 luded mucosal laceration in six patients and hypercarbia in one.
32 ole of respiratory control during O2-induced hypercarbia in patients with chronic obstructive pulmona
33 teric perfusion decreased intestinal mucosal hypercarbia, leading to improvement of intramucosal pH.
34  = .014) after adjustment for age, degree of hypercarbia, maximal respiratory rate, use of an arteria
35                         To determine whether hypercarbia occurs following the use of open suctioning
36  study compares the impact of hypoxia versus hypercarbia on oxygen delivery, under conditions of fixe
37 did not significantly underestimate arterial hypercarbia or acidosis.
38 e latency to arousal produced by exposure to hypercarbia or auditory stimuli.
39 pes mediate cortical arousals in response to hypercarbia or other sensory stimuli is unknown.
40 compared with cells reperfused with relative hypercarbia (PCO2 of 71 torr, pH 6.8) or hypocarbia (PCO
41 nsion, hypernatremia, acidosis, hypoxia, and hypercarbia predicts hospital mortality, surgical intens
42       Compared with normocarbic reperfusion, hypercarbia significantly reduced cell death from 54.8%
43  hagfish exposed acutely to severe sustained hypercarbia tolerated among the most severe blood acidos