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1 nticatabolic agents to reduce the effects of hypercortisolemia.
2 specially in the hippocampus, are related to hypercortisolemia.
3 bjected to common bile duct ligation develop hypercortisolemia.
4                                     They had hypercortisolemia (53% increase in area under the curve)
5 k between olfactory deficits and MDD lies in hypercortisolemia, a cardinal symptom of MDD.
6    ACTH-secreting pituitary adenomas lead to hypercortisolemia and cause significant morbidity and mo
7 ete corticotropin autonomously, resulting in hypercortisolemia and Cushing disease.
8 ortisol-metabolizing enzymes, contributed to hypercortisolemia and hence corticotropin suppression.
9 PA axis activity, characterized by overnight hypercortisolemia and increased activity in ultradian se
10 te that chronic stress, associated with mild hypercortisolemia and prolonged SNS activation, favors a
11 stress reliably induces ovarian dysfunction, hypercortisolemia, and excessive adrenergic activation i
12 s disease 2019 appears to be associated with hypercortisolemia, and increased synthesis of glucocorti
13    To address this, the authors have modeled hypercortisolemia by administering corticosterone in the
14 excess, due to primary aldosteronism (PA) or hypercortisolemia, causes hypertension and cardiovascula
15  a novel approach for treating patients with hypercortisolemia due to pituitary corticotroph adenomas
16 ught to better define the role of antecedent hypercortisolemia in generating HAAF, using two complime
17 ed in the overreaction to the stress and the hypercortisolemia in the development of depression--prob
18                              Amelioration of hypercortisolemia in the stressed state leads to an impr
19 icosterone levels; it also reversed signs of hypercortisolemia, including elevated glucose levels and
20 he disorders, a causal relationship in which hypercortisolemia induces diabetes or diabetic vascular
21 fering from major depressive disorder (MDD), hypercortisolemia is a common finding.
22                                      Chronic hypercortisolemia is a hallmark of neuroendocrine and ps
23 de strong evidence that hypoglycemia-induced hypercortisolemia is not primarily responsible for the g
24                                              Hypercortisolemia is usually present in psychotic depres
25 ies have suggested that hypoglycemia-induced hypercortisolemia may be responsible for blunting subseq
26             This study suggests that chronic hypercortisolemia may contribute to anxiety-related beha
27 motional neglect during childhood as well as hypercortisolemia may lead to heightened threat-related
28   Our results showed no effect of antecedent hypercortisolemia on epinephrine responses to subsequent
29 (ACTH)-secreting pituitary adenomas leads to hypercortisolemia predisposing to diabetes, hypertension
30 rs may counteract the deleterious effects of hypercortisolemia seen in bipolar disorder by upregulati
31                                  However, in hypercortisolemia, the role of DNA methylation of 11beta
32     Critical illness is often accompanied by hypercortisolemia, which has been attributed to stress-i
33 ion was regulated through DNA methylation in hypercortisolemia with cortisol-producing adenoma (CPA),