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1 t major architectural chromatin proteins are hyperdynamic and bind loosely to chromatin in ES cells.
2 uction did not prevent the occurrence of the hyperdynamic and hypercardiovascular response during the
3 ous, unrestrained rats prevented LPS-induced hyperdynamic and hypodynamic circulatory shock, hyperlac
4                                              Hyperdynamic binding is a property of pluripotent cells,
5                              We suggest that hyperdynamic binding of structural chromatin proteins is
6 ures enriched for active chromatin marks and hyperdynamic binding of structural chromatin proteins.
7                  Our hypotheses were that 1) hyperdynamic blood flow is absent in septic shock; 2) no
8 ction may exist to coordinately maintain the hyperdynamic cardiac contractile performance of the PLN-
9 ngendorff perfusion indicated that the basal hyperdynamic cardiac function of the knockout mouse was
10                           Intriguingly, this hyperdynamic cardiac function was maintained throughout
11 egardless of the resting EF, all players had hyperdynamic cardiac responses with exercise.
12                        All sheep developed a hyperdynamic cardiovascular response by 40 hrs that was
13                        The sheep developed a hyperdynamic cardiovascular response characterized by a
14                                          The hyperdynamic cardiovascular state (high cardiac index an
15                    According to this report, hyperdynamic chromatin is both specific and functionally
16 Lipoic acid prevented the development of the hyperdynamic circulation (cardiac index [CI]: 15.7 +/- 2
17            All but one of the patients had a hyperdynamic circulation (cardiac index, 4.2 to 7.3 lite
18                                 In patients, hyperdynamic circulation (p = 0.04), vascular dysfunctio
19 al hypertension develops differently, with a hyperdynamic circulation and angiogenic biomarker profil
20 l hypertensive (PHT) gastric mucosa leads to hyperdynamic circulation and increased susceptibility to
21 aused haemodynamic changes consistent with a hyperdynamic circulation and induced increases in muscle
22 ent hypermetabolic response characterized by hyperdynamic circulation and severe muscle catabolism an
23      Patients with subclinical PHT have less hyperdynamic circulation and significantly lower portal
24                       In models, severity of hyperdynamic circulation and specific fibrosis pattern (
25 dilatation (splanchnic and systemic) and the hyperdynamic circulation are hemodynamic abnormalities t
26         Additionally, vasodilatation and the hyperdynamic circulation are hemodynamic abnormalities t
27 Vasodilatation (splanchnic and systemic) and hyperdynamic circulation are hemodynamic abnormalities t
28 olely from portal hypertension, although the hyperdynamic circulation contributes to variceal growth
29        Peripheral vasodilation initiates the hyperdynamic circulation in cirrhosis.
30 s been implicated in the pathogenesis of the hyperdynamic circulation in portal hypertension.
31     We have previously demonstrated that the hyperdynamic circulation in the partial portal vein-liga
32  lipoic acid prevents the development of the hyperdynamic circulation in the rat model of biliary cir
33                                          The hyperdynamic circulation was dose dependently reversed b
34                                     Although hyperdynamic circulation was still maintained by day 7,
35 lysis, sepsis also enhances glycolysis under hyperdynamic circulation with adequate oxygen delivery.
36 NO production, blunts the development of the hyperdynamic circulation, and decreases portal pressure
37 ment of liver cirrhosis, is characterized by hyperdynamic circulation, angiogenesis, and portosystemi
38                          L-NMMA reversed the hyperdynamic circulation, causing an increase in arteria
39 revented the development of hyperlactatemia, hyperdynamic circulation, cellular myocardial depression
40                                      Next to hyperdynamic circulation, increased bile acids, VWF-Ag,
41           Caffeine decreased PP, ameliorated hyperdynamic circulation, PSS, mesenteric angiogenesis,
42 e control group showed a continuation of the hyperdynamic circulation.
43 iliary cirrhosis, portal hypertension, and a hyperdynamic circulation.
44  first 24 hrs of sepsis (n = 21) developed a hyperdynamic circulation.
45             The injury induced a hypotensive-hyperdynamic circulation; increases in pulmonary capilla
46 rtal vein ligation or sham operation until a hyperdynamic circulatory state developed.
47                                            A hyperdynamic circulatory state frequently is observed in
48  day 4, portal vein-ligated rats developed a hyperdynamic circulatory state with a normal central blo
49 receiving the bacterial infusion developed a hyperdynamic circulatory state with hypotension, decreas
50 fter 24 hrs of sepsis, all sheep developed a hyperdynamic circulatory state with increased cardiac in
51  of portal hypertension and the vasodilatory-hyperdynamic circulatory state, resulting in a progressi
52 is and ascites showed a typical pattern of a hyperdynamic circulatory state, when compared with their
53  Pseudomonas infusion, all sheep developed a hyperdynamic circulatory state, with increased cardiac i
54 hepatic fibrosis, portal hypertension, and a hyperdynamic circulatory state.
55 ues, and the work overload associated with a hyperdynamic circulatory state.
56 hunts, pulmonary arteriovenous shunts, and a hyperdynamic circulatory state.
57 ylene conjugate in septic sheep reverses the hyperdynamic circulatory state.
58                                          The hyperdynamic circulatory syndrome observed in chronic li
59 econd, experimental models used to study the hyperdynamic circulatory syndrome; and third, the vasodi
60 vity was correlated with a prevention of the hyperdynamic contractile response and enhanced myocardia
61 bserved in most infants (29/40; 72%); 17 had hyperdynamic contractility, and 24 had altered LV geomet
62 heart, but the earliest signs of disease are hyperdynamic contraction and impaired relaxation.
63                     These data indicate that hyperdynamic contraction is essential for HCM pathobiolo
64 aracterized by left ventricular hypertrophy, hyperdynamic contraction, and impaired relaxation of the
65  Myh6(R403Q/+) mice, R-carvedilol normalized hyperdynamic contraction, suppressed arrhythmia, and inc
66           R-enantiomer carvedilol attenuates hyperdynamic contraction, suppresses arrhythmia, and at
67  the hearts of older mutant mice also showed hyperdynamic contraction, with increased end-systolic ch
68                This finding was explained by hyperdynamic CSF movement, manifesting as higher caudal
69 alues were used to categorize LV function as hyperdynamic (EF>73%), normal (53%-73%), mildly-to-moder
70 ncreased wall thickness, small chamber size, hyperdynamic ejection fraction, and left ventricular con
71                       This typically induces hyperdynamic ejection, impaired relaxation, delayed earl
72 s characterized by diastolic dysfunction and hyperdynamic features.
73 ncy in mice did not alter cardiac structure, hyperdynamic function, or antifibrotic effects induced b
74 pport the increased metabolic demands of the hyperdynamic hearts.
75 months after thermal injury, ameliorates the hyperdynamic, hypermetabolic, hypercatabolic, and osteop
76 and prolonged QT interval) and functionally (hyperdynamic left ventricular [LV] contractility along w
77 s of normal (55-70%), depressed (< 55%), and hyperdynamic left ventricular ejection fraction (> 70%)
78 lar resistance was inversely associated with hyperdynamic left ventricular ejection fraction (odds ra
79                                Patients with hyperdynamic left ventricular ejection fraction had a hi
80                                              Hyperdynamic left ventricular ejection fraction was asso
81      After multivariate logistic regression, hyperdynamic left ventricular ejection fraction was inde
82 lar dimensions, left atrial enlargement, and hyperdynamic left ventricular systolic function.
83 ass 4 (21%; mostly high cardiac output, with hyperdynamic LV function).
84 data are consistent with the hypothesis that hyperdynamic microtubules impair axonal transport and ac
85                                              Hyperdynamic microvascular blood flow was not found.
86  describes a new trait of pluripotent cells: hyperdynamic or "breathing" chromatin.
87 icrobial sepsis is characterized by an early hyperdynamic phase (2-10 hrs after cecal ligation and pu
88         Sepsis is characterized by an early, hyperdynamic phase and a late, hypodynamic phase.
89 crobial sepsis is characterized by an early, hyperdynamic phase followed by a late, hypodynamic phase
90 gulated CYP4A3 is associated with the early, hyperdynamic phase of sepsis and the down-regulated CYP2
91 ays or even prevents the transition from the hyperdynamic phase to the hypodynamic phase of sepsis, a
92 ays or even prevents the transition from the hyperdynamic phase to the hypodynamic phase of sepsis, a
93 o be responsible for the transition from the hyperdynamic phase to the hypodynamic phase of sepsis.
94 sequently observed that produced a transient hyperdynamic phase; however, progressive RV distension d
95 to meet the increased ATP utilization in the hyperdynamic phospholamban-deficient hearts.
96 tate to meet the increased ATP demand in the hyperdynamic phospholamban-deficient hearts.
97     We show that in SCA mice, anemia-induced hyperdynamic physiology was gradually superimposed with
98                    Upon differentiation, the hyperdynamic proteins become immobilized on chromatin.
99              A Cushing reflex, followed by a hyperdynamic response and diabetes insipidus, occurred i
100                                   A systemic hyperdynamic response characterized by an increase in bo
101 GI2 synthesis prevents the occurrence of the hyperdynamic response during early sepsis.
102 dergoing exercise testing had an appropriate hyperdynamic response in cardiac function.
103                                 Although the hyperdynamic response observed in septic patients underg
104 rotects against I/R injury by preventing the hyperdynamic response of isolated perfused hearts during
105 appear to play a major role in producing the hyperdynamic response under such conditions.
106                      The Cushing phenomenon, hyperdynamic response, and diabetes insipidus were obser
107 eptide, plays a major role in initiating the hyperdynamic response.
108 lop a standardized and reproducible model of hyperdynamic sepsis after smoke inhalation in sheep.
109 ensin II and norepinephrine and induction of hyperdynamic sepsis by administration of live Escherichi
110                  During infusion of E. coli, hyperdynamic sepsis developed with hypotension, tachycar
111          This animal model closely resembles hyperdynamic sepsis in humans and may be of great value
112 nthase inhibitors in a large animal model of hyperdynamic sepsis in which acute kidney injury occurs
113 ures and maintains mean arterial pressure in hyperdynamic sepsis without reversal of sepsis-induced v
114                 In sheep with nonhypotensive hyperdynamic sepsis, creatinine clearance halved (32 to
115                                           In hyperdynamic sepsis, intrarenal infusion of a highly sel
116                              In experimental hyperdynamic sepsis, renal denervation was associated wi
117          In this ovine model of experimental hyperdynamic sepsis, renal excretory function decreases
118                   In a large animal model of hyperdynamic sepsis, renal hyperemia was associated with
119 lyoxyethylene conjugate in an ovine model of hyperdynamic sepsis.
120                             All patients had hyperdynamic septic shock and impaired renal function.
121 tion of TNF-alpha attenuated the severity of hyperdynamic shock induced by a subsequent infusion of e
122  5 hrs or 10 hrs after CLP (i.e., the early, hyperdynamic stage of sepsis), the thoracic aorta was is
123 taining endothelial cell function during the hyperdynamic stage of sepsis.
124 nitric oxide) is depressed during the early, hyperdynamic stage of sepsis.
125 itric oxide significantly contributes to the hyperdynamic state associated with sepsis.
126 re only normalized at T3 associated with the hyperdynamic state at this point.
127  All three doses of hemoglobin reversed this hyperdynamic state by increasing mean arterial pressure
128 ) plays a significant role in modulating the hyperdynamic state during early sepsis.
129 ponse syndrome of fever, leukocytosis, and a hyperdynamic state is common in trauma patients, especia
130 l replicates the mortality, hypotension, and hyperdynamic state seen in clinical sepsis.
131 The E. coli infusions were associated with a hyperdynamic state, pulmonary hypertension, systemic hyp
132 ndotoxemia induced an immediate hypotensive, hyperdynamic, tachycardic state with progressive lactic
133 1 (AMBP-1) prevented the transition from the hyperdynamic to the hypodynamic stage in the progression

 
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