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1 ve been linked to psychosis-related dopamine hyperfunction.
2 us liver disease and correlates with splenic hyperfunction.
3 ffects ranging from hypomorphic to silent to hyperfunctioning.
4 ompensate for damage both by hypertrophy and hyperfunction after renal injury or ablation, these chan
5 l photoreceptor development characterized by hyperfunction and excess of the minority S (short wavele
6  deletion in mice leads to ependymal ciliary hyperfunction and hydrocephalus accompanying ASD-related
7  the GluN3A reactivation corrected the NMDAR hyperfunction and prevented the full range of HD signs a
8         By inhibiting this pathway, dopamine hyperfunction and/or NMDA hypofunction abnormalities imp
9 igestive diseases, varicose veins, pituitary hyperfunction, and other peripheral nerve disorders.
10 y in the hippocampus and consequent dopamine hyperfunction; and fifth, deficits in corollary discharg
11 sphonia associated with acute illness, vocal hyperfunction, benign lesions, and muscle tension.
12 for behavioral aberrations associated with a hyperfunctioning dopamine system.
13 her characteristic findings of MAS including hyperfunctioning endocrinopathies, polyostotic fibrous d
14        Surgery is an effective treatment for hyperfunctioning glands and benign and malignant tumours
15 ing parathyroid resection indicates multiple hyperfunctioning glands and to determine the appropriate
16  human parathyroids are generally limited to hyperfunctioning glands owing to the difficulty in obtai
17 better; in 6 patients (9%), visualization of hyperfunctioning glands was best on late images; and in
18 ne-needle aspiration biopsy of the suspected hyperfunctioning glands, using histopathologic results a
19 ocalize and confirm complete excision of all hyperfunctioning glands, was compared with group II, who
20  be most valuable in the group with multiple hyperfunctioning glands, with sensitivity of 88%, wherea
21  approaches, it is necessary to localize all hyperfunctioning glands.
22 ity of these patients do not have additional hyperfunctioning glands.
23  and further exploration revealed additional hyperfunctioning glands.
24  be indicative of the presence of additional hyperfunctioning glands.
25 y 50% and BIJ PTH successfully localized the hyperfunctioning glands.
26  30% of these cases are caused by one or two hyperfunctioning glands.
27 omparator, four histologically characterized hyperfunctioning human parathyroids with varying oxyphil
28 reatic beta-cell to establish a template for hyperfunction in early life and beta-cell failure with a
29           Here, we approximated striatal D2R hyperfunction in mice via designer receptor-mediated act
30  whereas patients with OCD showed larger and hyperfunctioning insular-striatal regions that may be po
31                                        NMDAR hyperfunction might impair synaptic plasticity.
32 aphy is the first-line study for assessing a hyperfunctioning nodule.
33 d by toxic multinodular goitre, and solitary hyperfunctioning nodules.
34  an underlying cause and correcting hypo- or hyperfunction of an endocrine gland, however, can often
35  involved, and the earliest known deficit is hyperfunction of glutamate-type N-methyl-d-aspartate rec
36 ugs for the treatment of diseases related to hyperfunction of L-type channels, such as Torsades de Po
37                            We speculate that hyperfunction of pancreatic islets did not lead to obesi
38 hether this interaction is implicated in the hyperfunction of these receptors.
39 n a central hypernoradrenergic state and the hyperfunctioning of specific central CRH pathways that e
40                                 Importantly, hyperfunctioning of this sensory mnemonic system of thre
41 the impact of NMDA hypofunction and dopamine hyperfunction on OFC neurons, suggesting that these neur
42 nder the curve (AUC) for localization of the hyperfunctioning parathyroid gland or glands at sestamib
43 t could predict the likelihood of additional hyperfunctioning parathyroid glands and let the surgeon
44  500 mg, there is a 9% chance for additional hyperfunctioning parathyroid glands based on the WIN nom
45 mogram predicts the likelihood of additional hyperfunctioning parathyroid glands during parathyroidec
46                     Radioguided resection of hyperfunctioning parathyroid glands has been shown to be
47 tiglandular disease, Met-PET/CT identified 2 hyperfunctioning parathyroid glands in 1 patient, 1 glan
48 differences between early and late images of hyperfunctioning parathyroid glands in 44 patients (69%)
49 T/CT imaging in preoperative localization of hyperfunctioning parathyroid glands in a larger series o
50 (18)F]flurpiridaz PET/CT for localization of hyperfunctioning parathyroid glands in comparison with o
51     Although operative failure can be due to hyperfunctioning parathyroid glands in ectopic locations
52 ld become the preferred method for detecting hyperfunctioning parathyroid glands in patients with cli
53 choline PET/CT in identifying and localizing hyperfunctioning parathyroid glands in preoperative sett
54          Conclusion: In most patients (89%), hyperfunctioning parathyroid glands were adequately visu
55 (18)F]flurpiridaz PET/CT for localization of hyperfunctioning parathyroid glands were retrospectively
56 nalysis, differentiating single and multiple hyperfunctioning parathyroid glands, showed PET/CT to be
57 ately predicted the likelihood of additional hyperfunctioning parathyroid glands.
58  an endocrine disorder caused by one or more hyperfunctioning parathyroid glands.
59 or presurgical detection and localization of hyperfunctioning parathyroid tissue by (11)C-MET and (18
60 sitive predictive value for the detection of hyperfunctioning parathyroid tissue in patients after su
61 onine ((11)C-MET) PET/CT for localization of hyperfunctioning parathyroid tissue in patients with pHP
62                 Preoperative localization of hyperfunctioning parathyroid tissue in patients with pri
63 seases in being associated with target organ hyperfunction rather than organ damage.
64 o determine how constitutive NMDAR hypo- and hyperfunction, respectively, affect the glutamate recept
65        Early human NR2E3 disease with S cone hyperfunction showed thickened retinal layers within an
66 ntial treatments for diseases of parathyroid hyperfunction, such as primary and secondary hyperparath
67                             According to the hyperfunction theory, aging is a quasi-program favoring
68                  Among these concepts is the hyperfunction theory, which postulates that processes co
69 rophysiology as a correlate for the dopamine hyperfunction thought to underlie psychosis in patients.
70  receptor have been identified as a cause of hyperfunctioning thyroid adenomas, and germline mutation
71                                              Hyperfunctioning thyroid nodules (HTNs) were presumed to
72 ate localization and confirm excision of all hyperfunctioning tissue, the success rate of reoperative
73 ulated, such as genetic factors, masticatory hyperfunction, trauma, and continued growth, but the und
74 hat they exhibited hippocampal NMDA receptor hyperfunction, which likely drives the enhanced LTP.
75 em, together with dorsal and fronto-striatal hyperfunctioning, which may reflect poor affect reactivi
76 pressed subchondral bone loss and osteoclast hyperfunction while beta-agonist (isoproterenol) exacerb
77 mechanisms that account for the compensatory hyperfunction with insulin resistance (so-called beta-ce