ライフサイエンスコーパス: hyperfunctioning

1 ffects ranging from hypomorphic to silent to hyperfunctioning.

2 for behavioral aberrations associated with a hyperfunctioning dopamine system.

3 her characteristic findings of MAS including hyperfunctioning endocrinopathies, polyostotic fibrous d

4 Surgery is an effective treatment for hyperfunctioning glands and benign and malignant tumours

5 ing parathyroid resection indicates multiple hyperfunctioning glands and to determine the appropriate

6 human parathyroids are generally limited to hyperfunctioning glands owing to the difficulty in obtai

7 better; in 6 patients (9%), visualization of hyperfunctioning glands was best on late images; and in

8 ne-needle aspiration biopsy of the suspected hyperfunctioning glands, using histopathologic results a

9 ocalize and confirm complete excision of all hyperfunctioning glands, was compared with group II, who

10 be most valuable in the group with multiple hyperfunctioning glands, with sensitivity of 88%, wherea

11 approaches, it is necessary to localize all hyperfunctioning glands.

12 ity of these patients do not have additional hyperfunctioning glands.

13 and further exploration revealed additional hyperfunctioning glands.

14 be indicative of the presence of additional hyperfunctioning glands.

15 y 50% and BIJ PTH successfully localized the hyperfunctioning glands.

16 30% of these cases are caused by one or two hyperfunctioning glands.

17 omparator, four histologically characterized hyperfunctioning human parathyroids with varying oxyphil

18 whereas patients with OCD showed larger and hyperfunctioning insular-striatal regions that may be po

19 aphy is the first-line study for assessing a hyperfunctioning nodule.

20 d by toxic multinodular goitre, and solitary hyperfunctioning nodules.

21 n a central hypernoradrenergic state and the hyperfunctioning of specific central CRH pathways that e

22 Importantly, hyperfunctioning of this sensory mnemonic system of thre

23 nder the curve (AUC) for localization of the hyperfunctioning parathyroid gland or glands at sestamib

24 t could predict the likelihood of additional hyperfunctioning parathyroid glands and let the surgeon

25 500 mg, there is a 9% chance for additional hyperfunctioning parathyroid glands based on the WIN nom

26 mogram predicts the likelihood of additional hyperfunctioning parathyroid glands during parathyroidec

27 Radioguided resection of hyperfunctioning parathyroid glands has been shown to be

28 tiglandular disease, Met-PET/CT identified 2 hyperfunctioning parathyroid glands in 1 patient, 1 glan

29 differences between early and late images of hyperfunctioning parathyroid glands in 44 patients (69%)

30 T/CT imaging in preoperative localization of hyperfunctioning parathyroid glands in a larger series o

31 (18)F]flurpiridaz PET/CT for localization of hyperfunctioning parathyroid glands in comparison with o

32 Although operative failure can be due to hyperfunctioning parathyroid glands in ectopic locations

33 ld become the preferred method for detecting hyperfunctioning parathyroid glands in patients with cli

34 choline PET/CT in identifying and localizing hyperfunctioning parathyroid glands in preoperative sett

35 Conclusion: In most patients (89%), hyperfunctioning parathyroid glands were adequately visu

36 (18)F]flurpiridaz PET/CT for localization of hyperfunctioning parathyroid glands were retrospectively

37 nalysis, differentiating single and multiple hyperfunctioning parathyroid glands, showed PET/CT to be

38 ately predicted the likelihood of additional hyperfunctioning parathyroid glands.

39 an endocrine disorder caused by one or more hyperfunctioning parathyroid glands.

40 or presurgical detection and localization of hyperfunctioning parathyroid tissue by (11)C-MET and (18

41 sitive predictive value for the detection of hyperfunctioning parathyroid tissue in patients after su

42 onine ((11)C-MET) PET/CT for localization of hyperfunctioning parathyroid tissue in patients with pHP

43 Preoperative localization of hyperfunctioning parathyroid tissue in patients with pri

44 receptor have been identified as a cause of hyperfunctioning thyroid adenomas, and germline mutation

45 Hyperfunctioning thyroid nodules (HTNs) were presumed to

46 ate localization and confirm excision of all hyperfunctioning tissue, the success rate of reoperative

47 em, together with dorsal and fronto-striatal hyperfunctioning, which may reflect poor affect reactivi