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1 hyperinsulinemic-euglycemic clamp and a 2-h hyperglycemic clamp.
2 n littermates (ZCL rats) before and during a hyperglycemic clamp.
3 (pot) or AIR(max), but requires conduct of a hyperglycemic clamp.
4 ons, after a 30-g oral glucose, and during a hyperglycemic clamp.
5 s (1stIR and 2ndIR) were assessed by using a hyperglycemic clamp.
6 /min) during euglycemia and after 6 hours of hyperglycemic clamp.
7 ynthesis and glucose turnover rates during a hyperglycemic clamp.
8 on during glucose tolerance tests as well as hyperglycemic clamps.
9 oral glucose challenge and hyperinsulinemic-hyperglycemic clamps.
10 an oral glucose tolerance test and two-step hyperglycemic clamp (100 and 300 mg/dL) followed by intr
11 normal-weight AA versus C peers during a 2-h hyperglycemic clamp (12.5 mmol/L) on two occasions: 1) i
12 sensitivity and insulin clearance and a 2-h hyperglycemic clamp (12.5 mmol/l) to assess first- and s
15 8 micromol x kg(-1) x min(-1)), and during a hyperglycemic clamp, a failure to suppress endogenous gl
17 We measured insulin secretion during a 2-h hyperglycemic clamp and calculated the disposition index
20 beta-Cell function was determined with the hyperglycemic clamp and morphometric analysis of pancrea
21 d-phase insulin responses to a 2-h 13 mmol/l hyperglycemic clamp and the insulin response to a subseq
23 se-induced insulin secretion both in vivo in hyperglycemic clamps and ex vivo in isolated islets from
25 ndirect calorimetry), insulin secretion (2-h hyperglycemic clamp), and body composition (dual-energy
26 se in ex vivo perifusion experiments, during hyperglycemic clamps, and following i.p. glucose challen
27 in the overnight fasting state and during a hyperglycemic clamp ( approximately 150 mg/dl) in 10 rec
30 dogs underwent a hyperinsulinemic (4x basal) hyperglycemic clamp (arterial blood glucose 146 +/- 2 mg
31 ges, glucose tolerance tests, euglycemic and hyperglycemic clamps, as well as isolated islet and peri
33 insulin release in response to i.v. glucose (hyperglycemic clamps at 250 or 350 mg/dl plasma glucose)
34 -cell function was measured with a nine-step hyperglycemic clamp before and 48 h and 14 days after th
36 rst- and second-phase insulin secretion (2-h hyperglycemic clamp), body composition and abdominal adi
37 travenous glucose tolerance test (IVGTT) and hyperglycemic clamp characterized the insulinotropic eff
43 tration curve during the first 12 min of the hyperglycemic clamp (DeltaC-pep[AUC]0-12) was inversely
44 rements, and hyperinsulinemic-euglycemic and hyperglycemic clamp experiments were performed in RLIP76
45 n and two women, BMI 32.6 +/- 0.6) underwent hyperglycemic clamping for 24 h with hourly determinatio
48 went oral glucose tolerance tests (OGTT) and hyperglycemic clamp (HC) procedures, followed by arginin
50 travenous glucose tolerance test (IVGTT) and hyperglycemic clamp (HGC) in 17 nondiabetic subjects (14
51 1c [HbA1c] 7.8 +/- 1.1%) or hyperinsulinemic-hyperglycemic clamp (HH) (n = 10; 44 +/- 12 years, 8 fem
52 e reabsorption was measured with the stepped hyperglycemic clamp in 15 subjects with type 2 diabetes
53 glucose infusion rate/insulin (mg/kg/min) by hyperglycemic clamp in 50 adolescents with T1D (age 16.1
54 by applying mathematical modeling during the hyperglycemic clamp in 60 normal glucose tolerance (NGT)
55 ein for 150 min to create a hyperinsulinemic-hyperglycemic clamp in EX-Basal, EX-Sim, and sedentary d
57 e clearance versus insulin levels during the hyperglycemic clamp in the two small groups showed both
59 ensitivity and secretion were assessed using hyperglycemic clamps in adults and frequently sampled in
60 on GLP-1-stimulated insulin secretion during hyperglycemic clamps in nondiabetic Caucasian individual
62 sma C-peptide concentration curve during the hyperglycemic clamp increased by 22 +/- 4 and 23 +/- 4%
63 r change in plasma glucose levels during the hyperglycemic clamp, individuals with T1DM had significa
67 -1, we performed a five-step (1-h intervals) hyperglycemic clamp on seven heterozygous members (NM) a
68 Intravenous infusion of indinavir during hyperglycemic clamps on rats significantly suppressed th
69 intravenous glucose challenge followed by a hyperglycemic clamp protocol, during which the plasma-in
70 glucose turnover during fasting and during a hyperglycemic clamp.RESULTSExendin 9-39 increased fastin
71 he first (10-80 min) and second (90-160 min) hyperglycemic clamp steps increased by 3.8-fold and 1.9-
73 meostasis by hyperinsulinemic-euglycemic and hyperglycemic clamp studies and energy expenditure by in
75 Dawley rats (n = 28) were subjected to a 4-h hyperglycemic clamp study (approximately 11 mmol/l).
76 rglycemia on incretin receptor expression, a hyperglycemic clamp study was performed for 96 h with re
77 and free fatty acid (FFA) turnover using the hyperglycemic clamp technique in combination with isotop
78 ealthy subjects (HS) was conducted using the hyperglycemic clamp technique together with duodenal nut
83 active such as during fasting, diabetes, and hyperglycemic clamp, the concentration of GR mRNA increa
85 re challenged with the same hyperinsulinemic-hyperglycemic clamp to simulate identical postprandial-l
87 AM, n = 5; CHADN, n = 6), a hyperinsulinemic-hyperglycemic clamp was used to assess whether CHADN cou
91 ucose infusion rates during hyperinsulinemic-hyperglycemic clamps were increased with GLUT4 overexpre
93 for hepatic glycogen synthesis and turnover, hyperglycemic clamps were performed with somatostatin [0
94 ulin secretion rates (ISR) measured during a hyperglycemic clamp with either GLP-1 receptor blockade
95 e diet [P-HFF]) underwent a hyperinsulinemic-hyperglycemic clamp with intraportal glucose infusion.
96 a randomized order involving 2-h euglycemic-hyperglycemic clamps with coadministration of: 1) SU (gl
97 ut (Cx36(-/-)) mouse phenotype and performed hyperglycemic clamps with rapid sampling of insulin in C