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1 ail skin showed hyperplasia with evidence of hyperkeratosis.
2  with histological findings of epidermolytic hyperkeratosis.
3 fied as the molecular basis of epidermolytic hyperkeratosis.
4  PLS patients display both periodontitis and hyperkeratosis.
5 ate red patches and localized or generalized hyperkeratosis.
6 dermolysis bullosa simplex and epidermolytic hyperkeratosis.
7  and histologically by acanthosis and marked hyperkeratosis.
8 ng-Meara phenotype with severe palmo-plantar hyperkeratosis.
9 der characterized by generalized scaling and hyperkeratosis.
10 f nail dystrophy, alopecia, and palmoplantar hyperkeratosis.
11 itive layer of cells and was associated with hyperkeratosis.
12 ere dystrophy of the nails, and palmoplantar hyperkeratosis.
13 ents with oral leukokeratosis and follicular hyperkeratosis.
14 r hemorrhage, onychoschizia, and distal nail hyperkeratosis.
15 acterized by diffuse, yellowish palmoplantar hyperkeratosis.
16 erized by thickening of the skin with marked hyperkeratosis.
17 ienced premature tooth loss and palm plantar hyperkeratosis.
18 g skin and involves follicular occlusion and hyperkeratosis.
19 ysis of cases of pregnancy-associated nipple hyperkeratosis.
20 ong with other skin abnormalities, including hyperkeratosis.
21 ation, delayed differentiation and extensive hyperkeratosis.
22  cultures with IL1A was sufficient to induce hyperkeratosis.
23 mmation and associated epidermal hyperplasia/hyperkeratosis.
24  other keratins were upregulated, suggesting hyperkeratosis.
25 a, and by 2 weeks of age develop significant hyperkeratosis.
26 very kinetics in patients with epidermolytic hyperkeratosis.
27 celerated recovery kinetics in epidermolytic hyperkeratosis.
28 r layer, mild acanthosis, and orthokeratotic hyperkeratosis.
29  skin may be normal, except for palmoplantar hyperkeratosis.
30 agnosed with hyperpigmentation was 673; with hyperkeratosis, 243; and with skin cancer (Bowen's disea
31   The most common AEs were arthralgia (33%), hyperkeratosis (27%), and pyrexia (24%).
32 14 [38.9%], respectively [P = .67]), plantar hyperkeratosis (47 [39.5%] and 14 [38.9%], respectively
33  in the dabrafenib and trametinib group, and hyperkeratosis (70 patients, 33%) in the dabrafenib only
34 arly, wound-associated epidermal hyperplasia/hyperkeratosis, a hallmark of adult HK1.fos phenotypes,
35  phenotype mimicking a form of epidermolytic hyperkeratosis, a keratin gene disorder.
36 ed embryonic barrier formation and postnatal hyperkeratosis (abnormal accumulation of cornified cells
37 perplasia of the epithelium typified by mild hyperkeratosis, acanthosis, and elongation and isolated
38 er stratum granulosum cells in epidermolytic hyperkeratosis after barrier disruption.
39 kin in which affected infants have epidermal hyperkeratosis and a defective permeability barrier.
40 opy of nuclear-lamin-deficient skin revealed hyperkeratosis and a disordered stratum corneum with an
41 ssive congenital ichthyosis characterized by hyperkeratosis and a disruption in the epidermal permeab
42 ed multifocal hyperplasia without associated hyperkeratosis and aberrant expression of keratin 6.
43 ion, we show that murine epidermis developed hyperkeratosis and acanthosis 4 d after an adenoviral ve
44 ith overt hyperkeratosis of the forestomach, hyperkeratosis and acanthosis of the epidermis, and hypo
45 c nipple changes in pregnancy do not include hyperkeratosis and are expected to resolve or improve po
46                                              Hyperkeratosis and blister formation are relatively mild
47 teractive effect on lung cancer risk between hyperkeratosis and cigarette smoking was identified, whi
48  resolution, including epidermal thickening, hyperkeratosis and dermal fibroplasia/fibrosis, serve as
49  the Keratin 2e and Egfr mutations, in which hyperkeratosis and epidermal thickening precede epiderma
50 fication with overlapping features including hyperkeratosis and erythema.
51 hil-derived cys-LTs and the CysLT(2)R in the hyperkeratosis and fibrosis of allergic skin inflammatio
52 y associated with a correct diagnosis, while hyperkeratosis and fissures and ridges were independent
53 te apoptosis, which might contribute to skin hyperkeratosis and hypotrichosis, was also detected in s
54 miquimod-induced psoriasis model by reducing hyperkeratosis and inflammation.
55 nd cutaneous blistering at birth followed by hyperkeratosis and less frequent blistering later in lif
56 is of suprabasal keratinocytes and secondary hyperkeratosis and melanocytosis.
57 atinocytes resulted in epidermal hyperplasia/hyperkeratosis and novel 12-O-tetradecanoylphorbol-13-ac
58 ental anomalies, hypotrichosis, palmoplantar hyperkeratosis and onychodysplasia, syndactyly, and clef
59 zed by persistent plaque-like or generalized hyperkeratosis and transient red patches of variable siz
60 nthosis), thickening of the cornified layer (hyperkeratosis), and increased vascularity in the dermis
61 0 deficient mouse, a model for epidermolytic hyperkeratosis, and a mouse model for Bloom syndrome are
62 sing MCPyV T antigens developed hyperplasia, hyperkeratosis, and acanthosis of the skin with addition
63 e exhibit epidermal and adnexal hyperplasia, hyperkeratosis, and almost total alopecia.
64 epidermis resulted in epidermal hyperplasia, hyperkeratosis, and an increased labeling index that per
65 t at a normal rate and developed oily coats, hyperkeratosis, and apparent vision defects.
66 uding immune suppression, thymus involution, hyperkeratosis, and carcinogenesis.
67 ibited significant epidermal hyperplasia and hyperkeratosis, and developed spontaneous squamous cell
68 e developed severe epidermal hyperplasia and hyperkeratosis, and did not survive beyond 3 weeks of ag
69 th delayed cerebellar development, epidermal hyperkeratosis, and follicular dystrophy.
70 barrier disease characterized by acanthosis, hyperkeratosis, and immune cell accumulation.
71 ocyte hyperproliferation, skin inflammation, hyperkeratosis, and increased apoptosis.
72 , epidermal and sebaceous gland hyperplasia, hyperkeratosis, and increased epidermal thickness.
73  characterized by acanthosis, parakeratosis, hyperkeratosis, and inflammatory infiltrates in the derm
74 asia characterized by alopecia, palmoplantar hyperkeratosis, and nail dystrophy.
75 6A mutations and cutaneous cysts, follicular hyperkeratosis, and natal teeth with K17 mutations.
76  psoriasis, atopic dermatitis, epidermolytic hyperkeratosis, and Netherton's syndrome.
77 essing mice, developed progressive alopecia, hyperkeratosis, and skin lesions containing profuse acti
78 arcinoma, ten with precancerous lesions with hyperkeratosis, and ten subjects without laryngeal disea
79  of the barrier abnormality in epidermolytic hyperkeratosis are unknown, however.
80 cated keratin 10 gene exhibit acanthosis and hyperkeratosis as seen in the human disease.
81 sting of early-onset patches of erythema and hyperkeratosis, as well as SCA manifesting in the fourth
82 clude basal hyperplasia, lack of maturation, hyperkeratosis, atypical nuclei, perinuclear clearing, a
83 d by early-onset poikiloderma, pachyonychia, hyperkeratosis, bone anomalies and neutropenia, predispo
84 ability barrier abnormality in epidermolytic hyperkeratosis can be attributed to abnormal lamellar bo
85 ed atrophic and pigmentary lesions, striated hyperkeratosis, coarse lusterless hair and alopecia, cat
86                          Two individuals had hyperkeratosis confined to palms, soles, and anogenital
87 ) is an autosomal dominant disorder in which hyperkeratosis confined to the palms and soles is charac
88 tion, it did not protect from acanthosis and hyperkeratosis, demonstrating that neutrophils are dispe
89 at the histopathologic feature of follicular hyperkeratosis distinguished these 6 patients from previ
90 utosomal dominant skin disease epidermolytic hyperkeratosis (EHK) is an arginine to histidine substit
91 h clinical features similar to epidermolytic hyperkeratosis (EHK).
92 hich models the human disease, epidermolytic hyperkeratosis (EHK).
93 nherited blistering disorders, epidermolytic hyperkeratosis, epidermolysis bullosa simplex, epidermol
94 ologic and dermatoscopic findings of KP were hyperkeratosis, hypergranulosis, mild T helper cell type
95 trophils and T lymphocytes into the skin and hyperkeratosis/hyperplasia of the nonglandular stomach.
96 ed a markedly thinned epidermis and striking hyperkeratosis, hypoplastic sebaceous glands, and a fibr
97 ncing in four probands with undiagnosed skin hyperkeratosis/ichthyosis, we identified compound hetero
98 ty-five cases of pregnancy-associated nipple hyperkeratosis identified during a 5-year period (Januar
99 arkable occurrence of severe palmar--plantar hyperkeratosis in both patients suggests that the kerati
100 vement of hemichannels in the development of hyperkeratosis in KID syndrome.
101 aviolet A treatments reduced hyperplasia and hyperkeratosis in murine skin.
102 r compared with normal epidermis, and a mild hyperkeratosis in the cornified layer.
103 arked epidermal papillomatous acanthosis and hyperkeratosis in the skin, with a notable decrease in t
104 keratinocyte hyperproliferation, acanthosis, hyperkeratosis, intraepidermal neutrophil microabscesses
105 utosomal recessive disorder characterized by hyperkeratosis involving the palms, soles, elbows, and k
106                                Epidermolytic hyperkeratosis is a dominantly inherited ichthyosis, fre
107                                Epidermolytic hyperkeratosis is a hallmark feature of light and electr
108                                Epidermolytic hyperkeratosis is characterized by tonofilament clumping
109                                              Hyperkeratosis is significantly associated with an incre
110 ecture leading to a severe form of epidermal hyperkeratosis known as ichthyosis hystrix Curth-Macklin
111  these mice showed epidermal hyperplasia and hyperkeratosis, marked thickening of the dermis and hypo
112 nant skin blistering disorder, epidermolytic hyperkeratosis (MIM 113800), which is caused by mutation
113         Deletion of K2 caused acanthosis and hyperkeratosis of the ear and the tail epidermis, corneo
114   The mice present histologically with overt hyperkeratosis of the forestomach, hyperkeratosis and ac
115 s disorder allow differentiation from nevoid hyperkeratosis of the nipple and areola.
116 , after estrogen treatment, and/or in nevoid hyperkeratosis of the nipple and areola.
117                                              Hyperkeratosis of the nipple and/or areola can develop i
118                         Pregnancy-associated hyperkeratosis of the nipple can be symptomatic and pers
119 ologic entity be called pregnancy-associated hyperkeratosis of the nipple.
120 recessive condition manifested clinically by hyperkeratosis of the palms and soles and rapidly progre
121                   Slurp2(-/-) mice exhibited hyperkeratosis on the volar surface of the paws (i.e., p
122 ized by intraepidermal blistering, epidermal hyperkeratosis, or abnormalities in skin appendages, suc
123 o moderately dry, scaly skin with or without hyperkeratosis, palmoplantar keratoderma, and erythroder
124 resented with persistent skin rash featuring hyperkeratosis, parakeratosis and acanthosis, with infil
125 vere phenotype characterized by hyperplasia, hyperkeratosis, parakeratosis, and impaired epidermal ba
126 26.12 reduced symptom severity, for example, hyperkeratosis, parakeratosis, epidermal acanthosis, and
127 enodermatosis characterized by epidermolytic hyperkeratosis restricted to the palm and sole epidermis
128  Cx26-G45E mice displayed reduced viability, hyperkeratosis, scaling, skin folds, and hair loss.
129        The chronic epidermal hyperplasia and hyperkeratosis seen in these mice is similar to that see
130 ) mice showed mild epidermal hyperplasia and hyperkeratosis, severe hyperplasia of the sebaceous glan
131  recovery rates were faster in epidermolytic hyperkeratosis than in age-matched controls.
132  MdM experience pain in conjunction with the hyperkeratosis that has been attributed to fissures or m
133 hinning of stratum corneum, thereby reducing hyperkeratosis that likely underlies ectropion in patien
134 in phenotype is characterized by acanthosis, hyperkeratosis, the presence of a mixed inflammatory cel
135 g from mild psoriasiform scaly skin to overt hyperkeratosis, typically develops within the first thre
136 nt caused vaginal epithelial hyperplasia and hyperkeratosis, uterine inflammation, adenomyosis, and f
137                                              Hyperkeratosis was also evident.
138                             In two patients, hyperkeratosis was improved on the gentamicin side, as d
139 rted in GJB3, yet the extent and severity of hyperkeratosis was milder compared to the corresponding
140       Patients' self-reported improvement of hyperkeratosis was significantly greater on the gentamic
141                 Aggregate formation, but not hyperkeratosis, was suppressed by the deletion of both K
142 PL --> (157)NQSPLQPL) leads to epidermolytic hyperkeratosis, we tested and showed that the analogous
143  histological analysis, both hyperplasia and hyperkeratosis were evident.
144 hyperplasia, acanthosis, orthokeratosis, and hyperkeratosis were only observed in mice maintained in
145 dentified, which suggests that patients with hyperkeratosis who have been exposed to arsenic should c
146 oss groups, except for a higher incidence of hyperkeratosis with brensocatib.
147 amination revealed extensive areas of marked hyperkeratosis with focal parakeratosis, acanthosis, der
148 ted individuals later developed palmoplantar hyperkeratosis with patchy erythema and scale elsewhere
149 gic features were conspicuous orthokeratotic hyperkeratosis, with papillomatosis and acanthosis being
150 tagonist led to a dose-dependent decrease in hyperkeratosis without a reduction in non-polar lipids i
151 atosis type 1, Bloom syndrome, epidermolytic hyperkeratosis, X-linked ichthyosis, Netherton syndrome,

 
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