ライフサイエンスコーパス: hyperkeratotic

1 the epidermis was markedly hyperplastic and hyperkeratotic.

2 ary school-aged female patient with bands of hyperkeratotic-affected skin on the upper and lower extr

3 1, in which affected individuals have marked hyperkeratotic bands on the palms and soles.

4 We report that hyperkeratotic calluses arising in the glabrous skin of

5 sorder typified by dystrophic nails, painful hyperkeratotic calluses in glabrous skin, and lesions in

6 ntly causes oral lesions as well as PPK-like hyperkeratotic calluses on Krt16(-/-) front and hind paw

7 ated protein-1 (Slurp1), associated with the hyperkeratotic disorder mal de Meleda, is abundantly exp

8 and a potential target for treatment of skin hyperkeratotic disorders.

9 for the inherited ichthyoses, but also other hyperkeratotic disorders.

10 Unmodified XLI keratinocytes regenerated a hyperkeratotic epidermis lacking STS expression with def

11 is, a skin disease characterized by a thick, hyperkeratotic epidermis that recapitulates Steller's se

12 e produced that exhibited a hyperplastic and hyperkeratotic epithelium overlying a large dermal fibro

13 papulosquamous dermatosis, characterized by hyperkeratotic follicular papules and erythematous desqu

14 reeminent with UV-A-dependent phototoxicity, hyperkeratotic folliculitis, hand-foot skin reaction, ha

15 progresses to the subacute phase and papular-hyperkeratotic in the chronic phase.

16 -93) years underwent gentle curettage of the hyperkeratotic lesions followed by aminolevulinic acid p

17 lesions, whereas 0 of 17 normal tissues or 2 hyperkeratotic lesions had detectable telomerase activit

18 tate PPKs are characterized by circumscribed hyperkeratotic lesions on the palms and soles with consi

19 th bullous ichthyosis in early childhood and hyperkeratotic lichenified plaques in the flexural areas

20 cterized by bilaterally distributed pruritic hyperkeratotic nodules on extremities and trunk.

21 n Americans and features intensely pruritic, hyperkeratotic nodules on the extremities and trunk.

22 tory skin disease characterized by pruritic, hyperkeratotic nodules.

23 paired re-epithelialization characterized by hyperkeratotic nonmigratory wound epithelium is a hallma

24 is revealed that sweat ducts were blocked by hyperkeratotic or parakeratotic plugs.

25 rhesus LCV infection in these proliferative, hyperkeratotic, or parakeratotic epithelial cell lesions

26 Follicular hyperkeratotic papules and marked pruritus were also pro

27 Clinical manifestations include recurrent hyperkeratotic papules and plaques that occur mainly in

28 ed characteristic pink, violaceous, or brown hyperkeratotic papules and plaques throughout the body,

29 d indocyanine green (ICG) readily penetrated hyperkeratotic PC skin and normal callused regions compa

30 On day 4, ML.myc2 transgenic pups develop a hyperkeratotic phenotype, which progressively worsens un

31 rinatal lethality occurred due to the severe hyperkeratotic phenotype, which restricted movement.

32 focal and diffuse palmoplantar keratoderma, hyperkeratotic plaques on the trunk and limbs, varying d

33 diffuse, nonepidermolytic PPK and verrucous hyperkeratotic plaques over the joints and in flexures a

34 ad Greither disease associated with variable hyperkeratotic plaques.

35 atal phenotype that consists of skin that is hyperkeratotic, scaly, and essentially devoid of fur.

36 d a widespread, progressive, violaceous, and hyperkeratotic skin eruption.

37 isease characterized by intense pruritus and hyperkeratotic skin nodules.

38 c inflammatory skin disease characterized by hyperkeratotic skin nodules.

39 ll and at 4-5 days of age develop patches of hyperkeratotic skin where no hair grows, resulting in a

40 elop a syndrome characterized by dry, scaly, hyperkeratotic skin, growth failure, and death.

41 ormed dermal venule-like channels underlying hyperkeratotic skin.

42 Overall, lesions showed a scaly and hyperkeratotic surface (45 [33.6%]), yellowish keratin (