ライフサイエンスコーパス: hypermobility

1 alities, gait/balance disturbance, and joint hypermobility.

2 ective tissue abnormalities, including joint hypermobility.

3 , using a cutoff of >/=6 for the presence of hypermobility.

4 t have the orthopedic manifestation of joint hypermobility.

5 sess associations of specific variables with hypermobility.

6 e encompassed by the seemingly trivial term, hypermobility.

7 y account for some subgroups of benign joint hypermobility.

8 dition, a positive stress test, and urethral hypermobility.

9 sex, hand pain, chondrocalcinosis, and hand hypermobility.

10 s also evaluated to assess lesser degrees of hypermobility.

11 up of the hand in association with articular hypermobility.

12 rome, are characterized by generalized joint hypermobility.

13 Hypermobility, a largely unacknowledged though epidemiol

14 Hypermobility, also termed ligamentous laxity, may prese

15 Hypermobility among children in the cohort (mean age 13.

16 575 assistance dogs were scored for both hip hypermobility and 13 behaviour characteristics using pre

17 ificant negative association between present hypermobility and age was observed.

18 well-established relationship between joint hypermobility and anxiety in humans, that has not previo

19 o previous studies showing an association of hypermobility and CMC OA, in this cohort there was no ev

20 der larvae prolonged feeding, and suppressed hypermobility and cooperative burrowing behaviors.

21 The association of hypermobility and DIP OA was not evaluated, because evid

22 est a positive association between hip joint hypermobility and emotional arousal in domestic dogs, wh

23 e of these procedures in women with urethral hypermobility and genuine stress incontinence seems clea

24 hat there is an inverse relationship between hypermobility and hand and knee OA, and that hypermobili

25 Hypermobility and hypermobility syndrome are common cond

26 We assessed hypermobility and its relationship to radiographic hand

27 haracterized by an association between joint hypermobility and musculoskeletal pains, the latter occu

28 ng osteopenia, blue sclera, soft skin, joint hypermobility and neuromuscular junction dysfunction in

29 ts (OR 1.57) (P=0.037 for the interaction of hypermobility and obesity).

30 evaluated the association between articular hypermobility and primary OA.

31 The association of hypermobility and radiographic OA of the PIP, CMC, and m

32 have evaluated the association of articular hypermobility and radiographic osteoarthritis (OA) in hu

33 , chest wall deformity, dural ectasia, joint hypermobility, and acetabular protrusion.

34 clude feeding difficulties in infancy, joint hypermobility, and characteristic facial features such a

35 rs of older larvae, including food aversion, hypermobility, and cooperative burrowing.

36 is leading to premature loss of teeth, joint hypermobility, and mild skin findings.

37 A self-report questionnaire on joint hypermobility as well as data on age, height, weight, es

38 intrinsic sphincter deficiency and urethral hypermobility, assessing symptom severity and predicting

39 l and intersex differences, with UL mobility/hypermobility being the most consistently significant de

40 They may observe the hypermobility but fail to appreciate its significance in

41 ide hope that more common varieties of joint hypermobility can be understood and that effective thera

42 Recognition of hypermobility combined with a thorough patient evaluatio

43 tissue/myopathy overlap disorders with joint hypermobility, contractures, mild skeletal dysplasia and

44 levels and extensive joint radiographic and hypermobility data were also available for the GOGO coho

45 6,022 children evaluated, the prevalence of hypermobility (defined as a Beighton score of >/=4 [i.e.

46 cal symptoms including gastric reflux, joint hypermobility, dysautonomia, flushing and pruritus, and

47 TNX deficiency causes a phenotype similar to hypermobility Ehlers-Danlos syndrome involving joint hyp

48 we performed hand and knee examinations and hypermobility evaluations (Beighton criteria) and obtain

49 dy demonstrated a joint-protective effect of hypermobility for radiographic OA of PIP joints.

50 ubset of OI patients also present with joint hypermobility; however, the role of tendon dysfunction i

51 including progressive kyphoscoliosis, joint hypermobility, hypotonia, hyperelastic skin, hearing los

52 compelling new knowledge is the finding that hypermobility, if sought, is the most common finding amo

53 describe the point prevalence and pattern of hypermobility in 14-year-old children from a population-

54 as Morquio syndrome or achondroplasia, have hypermobility in a more limited distribution.

55 erum HA levels did not differ in relation to hypermobility in either cohort.

56 suggestion of a positive association between hypermobility in girls and variables including physical

57 ased; however, the mechanisms underlying Ty1 hypermobility in most rtt mutants are poorly characteriz

58 othesis that COMP levels are associated with hypermobility in patients with OA and individuals withou

59 ors have a substantial contribution to joint hypermobility in the adult female population.

60 or the past was based on recall of childhood hypermobility in the first 2 decades of life.

61 ither checkpoint pathway is required for Ty1 hypermobility in two rtt mutants that are competent for

62 We have shown that the prevalence of hypermobility in UK children is high, possibly suggestin

63 DMT induced hypermobility in wild-type mice but not in sigma-1 recep

64 criteria for generalized ligamentous laxity (hypermobility) in children are widely used, their validi

65 ie classic Ehlers-Danlos syndrome, and joint hypermobility is an important clinical manifestation.

66 hypermobility and hand and knee OA, and that hypermobility is associated with lower serum COMP levels

67 In rtt101delta mutants, hypermobility is stimulated through the DNA damage pathw

68 Joint hypermobility is widely prevalent in all communities yet

69 ive tissue, manifesting as early-onset joint hypermobility, joint contractures, muscle weakness and b

70 Extensive joint hypermobility, lower serum cartilage oligomeric matrix p

71 ogenesis and how a history of cervical spine hypermobility may be a needed predisposing physical char

72 In addition, hypermobility may be associated with collagen disorders

73 A clinical examination revealed hypermobility of the upper lip and absence of generalize

74 ence of slight vertical maxillary excess and hypermobility of the upper lip.

75 ssociated with vertical maxillary excess and hypermobility of the upper lip.

76 Heightened awareness of subtle hypermobility or symptomatic joint laxity on physical ex

77 st surgery, back trauma, heavy manual labor, hypermobility, or any other remarkable medical history.

78 an abnormal joint phenotype, similar to the hypermobility phenotype in classic Ehlers-Danlos syndrom

79 atologists to accept the challenges posed by hypermobility-related disorders, which have, in the past

80 Many conditions that cause joint hypermobility resolve with nonoperative management, but

81 ility Ehlers-Danlos syndrome involving joint hypermobility, skin hyperelasticity, pain and gastrointe

82 tive tissue disorders characterized by joint hypermobility, skin hyperelasticity, perivascular tissue

83 nherited form of EDS (characterized by joint hypermobility, skin hyperextensibility, and cardiac valv

84 tissue, with common features including joint hypermobility, soft and hyperextensible skin, abnormal w

85 ectal evacuation are common in patients with Hypermobility Spectrum Disorder (HSD) or Hypermobile Ehl

86 Joint hypermobility syndrome (JHS) is characterized by an asso

87 Hypermobility and hypermobility syndrome are common conditions with univer

88 Benign joint hypermobility syndrome, for example, shares similar auto

89 management of joint problems associated with hypermobility syndrome.

90 families, one of which diagnosed with joint hypermobility syndrome/Ehlers-Danlos syndrome hypermobil

91 In persons with hypermobility, the odds of OA in PIP joints was lower (f

92 ords of patients with Ehlers-Danlos Syndrome hypermobility type (HEDS), including demographic informa

93 ypermobility syndrome/Ehlers-Danlos syndrome hypermobility type (JHS/EDS-HT), characterized by idiopa

94 excluded, and for at least some cases of the hypermobility type of EDS, a condition marked by gross j

95 ch, language, and behavior; hypotonia; joint hypermobility; visual system defects; and other common c

96 The prevalence of hypermobility was 13% in the CARRIAGE family and 5% in t

97 The overall prevalence of hypermobility was 19.5% in MZ twins and 22.1% in DZ twin

98 GO subsets without radiographic OA, in which hypermobility was also associated with a significantly r

99 Tooth hypermobility was also present.

100 In the CARRIAGE family, hypermobility was associated with a significantly reduce

101 Hypermobility was defined as a Beighton score of >/=4, a

102 dy mass index-adjusted heritability of joint hypermobility was estimated to be 70% (95% confidence in

103 Whereas large-joint hypermobility was more common, lens dislocation and reti

104 Significantly greater concordance for joint hypermobility was observed in the MZ twins when compared

105 The Beighton criteria for assessment of hypermobility were recorded on a 0-9-point scale.

106 The lower odds of PIP OA with hypermobility were significant after adjusting for sex a

107 hanism of gene transfer to drive chromosomal hypermobility while self-transferring with additional vi

108 sion was used to examine the relationship of hypermobility with radiographic OA in each joint group,