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1 nic and, to an even greater degree, by acute hyperosmolarity.
2 elial cell function is adversely affected by hyperosmolarity.
3 ncrease (RVI) following cell shrinkage under hyperosmolarity.
4 DNMBP itself promotes P-body formation under hyperosmolarity.
5 there was normal inhibition by forskolin and hyperosmolarity.
6 ased intracellular Na(+) ([Na(+)](i)) and by hyperosmolarity.
7 APK is a stress-response kinase activated by hyperosmolarity.
8 each other and stimulated by dehydration and hyperosmolarity.
9 esulted from the sustained hyperglycemia and hyperosmolarity.
10 stress and was maintained through 360 min of hyperosmolarity.
11  mass spectrometry, increased in response to hyperosmolarity.
12 exposing confluent endothelial monolayers to hyperosmolarity.
13 d that the autophagic flux was unaffected by hyperosmolarity.
14 xpressing wild-type ASGP-Rs was inhibited by hyperosmolarity.
15  pathways mediate the responses to touch and hyperosmolarity.
16  NODM), comprising ketoacidosis (334, 8.1%), hyperosmolarity (131, 3.2%), renal complications (1,286,
17 otrophenol (11-fold), rotenone (7-fold), and hyperosmolarity (8-fold).
18                                              Hyperosmolarity (800 mOsm), achieved by either NaCl/urea
19 l conditions encountered in the lung cavity (hyperosmolarity, acidic pH, and low oxygen tension, amon
20                                              Hyperosmolarity activated both p38 and p42/44 mitogen-ac
21                                              Hyperosmolarity activates different components of severa
22                Exposure of yeast to external hyperosmolarity activates the Hog1 stress-activated prot
23                                              Hyperosmolarity also stimulated the uptake of myo-inosit
24 p38alpha phosphorylation that was induced by hyperosmolarity and anisomycin.
25 erate stresses, including serum deprivation, hyperosmolarity and ionizing radiation.
26                                              Hyperosmolarity and low O2 tension induced the invasive
27  in a greater apoptotic response of cells to hyperosmolarity and microtubule disruption.
28 h conditions found in human tissues, such as hyperosmolarity and presence of aminoglycoside antibioti
29 n kinase (ERK) was diminished in response to hyperosmolarity and serum factors in MEKK1(-/-) cells.
30     These experiments provide a link between hyperosmolarity and tear instability, suggesting that hy
31 ed in cells in response to oxidative stress, hyperosmolarity and treatment with serum.
32            These neurons respond to cooling, hyperosmolarity and wetness of the corneal surface.
33                                        Cold, hyperosmolarity, and abscisic acid (ABA) signaling induc
34 es, including exposure to hydrogen peroxide, hyperosmolarity, and carbon starvation.
35 ose biosynthesis, hydrophilins, responses to hyperosmolarity, and hypersalinity, reactive oxygen spec
36 e induced in response to lipopolysaccharide, hyperosmolarity, and interleukin 1.
37 ctivation, which can be induced by oxidants, hyperosmolarity, and proinflammatory cytokines, leading
38 ory neurons mediate responses to nose touch, hyperosmolarity, and volatile repellent chemicals.
39               Tear film instability and tear hyperosmolarity are considered core mechanisms in the de
40                  Sustained hypernatremia and hyperosmolarity are safely tolerated in pediatric patien
41 tivated by UV-C irradiation, heat shock, and hyperosmolarity as well as by tumor necrosis factor alph
42 hough BetT activity increased in response to hyperosmolarity, BetT mediated significant uptake under
43 utations in osm-10 eliminate the response to hyperosmolarity but have no effect on responses to nose
44 fically by methylmethane sulfonate (MMS) and hyperosmolarity but not by ultraviolet radiation, ionizi
45                                              Hyperosmolarity (but not hypo-osmolarity) resets clocks
46                                              Hyperosmolarity by added sucrose (50 and 100 mM) also in
47                                              Hyperosmolarity by added sucrose inhibited the spontaneo
48 ast, the activation of the stress kinases by hyperosmolarity, by the DNA-cross-linking agent diepoxyb
49 gene, was highly induced under physiological hyperosmolarity conditions.
50 guanylyl cyclase assays indicated that acute hyperosmolarity decreased NPR-B activity in a reversible
51                                         Tear hyperosmolarity, defined by a referent of 316 mOsmol/L,
52 udy was to investigate the role of TonEBP in hyperosmolarity-dependent autophagy in NP.
53 We conclude that in lung venular capillaries hyperosmolarity deteriorates barrier properties, possibl
54                                              Hyperosmolarity did not change the phosphorylation of UL
55        Unlike insulin, activation of NKCC by hyperosmolarity did not involve PI3-kinase but was suppr
56 ophagy in NP cells was not TonEBP-dependent; hyperosmolarity did not upregulate autophagy as previous
57                               Concomitantly, hyperosmolarity diminished total levels of protein synth
58                             We conclude that hyperosmolarity does not play a role in autophagy induct
59                                        Acute hyperosmolarity elevated intracellular calcium in a conc
60                                        Thus, hyperosmolarity enhanced action potential-evoked release
61                               Immunoblots of hyperosmolarity-exposed, cultured rat lung microvascular
62 nels and identified OSCA2.1, a member of the hyperosmolarity-gated calcium-permeable channel (OSCA) f
63 osmosensor involved in the regulation of the hyperosmolarity glycerol mitogen-activated protein kinas
64 orylated on Ser-38 and Ser-63 in response to hyperosmolarity, heat shock, and arsenite treatment but
65 n of neurotransmitter release was induced by hyperosmolarity, high potassium, or action potential fir
66    alpha-PAK is not activated in response to hyperosmolarity in 3T3-L1 cells.
67 tant role in cell resistance and adaption to hyperosmolarity in many tissues like kidney and liver.
68             Increased tear evaporation, tear hyperosmolarity, increased ocular surface staining, incr
69                                              Hyperosmolarity induced a significant retrieval of Ntcp
70 eptor potential (TRP) channels, mediates the hyperosmolarity induced Ca(2+) release.
71      In our laboratory, we are interested in hyperosmolarity-induced apoptosis in neuronal cells.
72 vergent pathway for both in vivo loading and hyperosmolarity-induced clock changes.
73 uroursodeoxycholate (TUDC) and cAMP reversed hyperosmolarity-induced Fyn activation and triggered re-
74 bing genomic loci, providing a mechanism for hyperosmolarity-induced global impairment of transcripti
75                      Muscle contraction- and hyperosmolarity-induced glucose transport may be regulat
76 ckade of Na(+)/H(+) exchangers prevented the hyperosmolarity-induced IEC inflammatory response.
77 ause p38 and p42/44 inhibition prevented the hyperosmolarity-induced increase in IL-8 production.
78 ficient mutant of focal adhesion kinase, the hyperosmolarity-induced increases in activity of focal a
79 monstrate that PS protects human cornea from hyperosmolarity-induced inflammation and oxidative stres
80 o hyperosmolar filtration (P < 0.01), and by hyperosmolarity-induced Lp increase (P < 0.01).
81 diated ocular surface disease, inhibited the hyperosmolarity-induced MMP production and JNK activatio
82                       TUDC also reversed the hyperosmolarity-induced retrieval of bile salt export pu
83           Kinetic analysis revealed that the hyperosmolarity-induced stimulation was associated with
84                                Additionally, hyperosmolarity inhibited mTORC1 activation and cell pro
85                                              Hyperosmolarity is a central mechanism causing ocular su
86 pal slices activates p38 and JNK and whether hyperosmolarity is a potential apoptotic stimulus in thi
87                                              Hyperosmolarity is believed to be the disease marker and
88                                    Medullary hyperosmolarity is protected from washout by countercurr
89                  Slow increases in tear film hyperosmolarity may cause the gradual increase in discom
90                    We hypothesized that this hyperosmolarity may contribute to colonic inflammation b
91 of 0 mm/5' in both eyes, accompanied by tear hyperosmolarity, mild meibomian gland dysfunction, reduc
92                                              Hyperosmolarity of the ocular surface triggers inflammat
93  drug formulation (polyoxyl 35 castor oil or hyperosmolarity of the SU5416 preparation).
94                 We determined the effects of hyperosmolarity on lung microvascular barrier properties
95         In the present study, the effects of hyperosmolarity on p38 activation and protein synthesis
96 However little is known about the effects of hyperosmolarity on short term regulation of the Na(+)-ta
97  and Caco-2 were used to study the effect of hyperosmolarity on the IEC inflammatory response.
98                             The influence of hyperosmolarity on the uptake of taurine, myo-inositol,
99 nd is activated by cellular stresses such as hyperosmolarity or DNA damage.
100 physiological derivatives (such as oxidants, hyperosmolarity, or glycation products) on tissues direc
101 r dysfunctions by multiple factors including hyperosmolarity, oxidant formation, and protein kinase C
102       gamma-PAK translocation in response to hyperosmolarity parallels Cdc42 translocation to the par
103 n culture study supported that extracellular hyperosmolarity plays no role in promoting autophagy in
104                     This initial response to hyperosmolarity precedes and temporally regulates the ac
105                                              Hyperosmolarity promotes translocation of gamma-PAK from
106  We conclude that a brief period of vascular hyperosmolarity protects against acid-induced ALI when t
107 hese studies provide the first evidence that hyperosmolarity regulates TauT activity and expression i
108 e to our knowledge that in lung capillaries, hyperosmolarity remodels the endothelial barrier and the
109 lishment of these bridges upon adaptation to hyperosmolarity represses the operon.
110  the combination of low O2 concentration and hyperosmolarity resulted in an approximate 10- to 15-fol
111 was designed to evaluate the effect of donor hyperosmolarity secondary to diabetes insipidus, an almo
112                                              Hyperosmolarity sensing is detected at the level of the
113 rophil induced either by stress stimuli (UV, hyperosmolarity, sphingosine) or by anti-Fas antibody or
114  factor, nuclear factor (NF)-kappaB, because hyperosmolarity stimulated both NF-kappaB DNA binding an
115 protein kinases, which effect contributed to hyperosmolarity-stimulated IL-8 production, because p38
116                                  In summary, hyperosmolarity stimulates IEC IL-8 production, which ef
117       No adverse effects of supraphysiologic hyperosmolarity such as renal failure, pulmonary edema,
118 instability produce transient shifts in tear hyperosmolarity that lead to chronic epithelial stress,
119                          Exposure of IECs to hyperosmolarity triggered expression of the proinflammat
120  studies of other cell types have shown that hyperosmolarity triggers autophagy.
121 nts with normal osmolarity (<312 mOsm/L) and hyperosmolarity values (>/=312 mOsm/L) had respective OS
122 vated levels can also activate p38 kinase by hyperosmolarity via a PKC-independent pathway.
123 fied that preferentially binds Nedd4-2 under hyperosmolarity was Dynamin Binding Protein (DNMBP)/Tuba
124  In addition, the proinflammatory effects of hyperosmolarity were, in a large part, mediated by activ
125 463) led to elevated p38-MAPK activity under hyperosmolarity, which was mediated by WNK463-dependent

 
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