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1 lastogenesis contributes at least in part to hyperostosis.
2 xyvitamin D, soft tissue calcifications, and hyperostosis.
3 l lead to conditions such as osteoporosis or hyperostosis.
4 ion, and the combined outcome is generalized hyperostosis.
5  with high skull binding were diagnosed with hyperostosis.
6 lies that features generalized craniotubular hyperostosis.
7 last activity in diffuse idiopathic skeletal hyperostosis and bone loss in diabetics.
8  in a patient with craniosynostosis, cranial hyperostosis, and long bone fragility.
9 r families affected by macrocephaly, cranial hyperostosis, and vertebral endplate sclerosis.
10                           Infantile cortical hyperostosis (Caffey disease) is characterized by sponta
11 ng results in a CMF phenotype of progressive hyperostosis combined with architecturally abnormal, poo
12 e, osteoporosis, diffuse idiopathic skeletal hyperostosis, crystalline arthropathy, neuropathic arthr
13 ly affected with diffuse idiopathic skeletal hyperostosis (DISH) and/or chondrocalcinosis, were ident
14 Conclusion Early diffuse idiopathic skeletal hyperostosis (DISH) criteria had high sensitivity and sp
15       Background Diffuse idiopathic skeletal hyperostosis (DISH) is a condition characterized by the
16                  Diffuse idiopathic skeletal hyperostosis (DISH) is a condition where adjacent verteb
17 uchem (VB) disease, two generalized skeletal hyperostosis disorders that result from hyperactive Wnt
18                We have encountered a case of hyperostosis frontalis interna in which the uptake of la
19                                              Hyperostosis frontalis interna is the term used to descr
20 ist revealed a significant relationship with hyperostosis; however, only 21% of women with high skull
21  mineralization (familial tumoral calcinosis/hyperostosis hyperphosphatemia syndrome, hypophosphatemi
22                  Diffuse Idiopathic Skeletal Hyperostosis (IDHS) is a multifactorial disease with a h
23 CMD) is a rare genetic disorder encompassing hyperostosis of craniofacial bones and metaphyseal widen
24 plasia (CMD) is a rare genetic disorder with hyperostosis of craniofacial bones and widened metaphyse
25  that the incisor phenotype is likely due to hyperostosis of mandibles, which distinguishes Ank (KI/K
26 s with renal osteodystrophy developed marked hyperostosis of the facial and cranial bones.
27  they had experienced an episode of cortical hyperostosis or not, had joint hyperlaxity, hyperextensi
28 SAPHO syndrome (synovitis, acne, pustulosis, hyperostosis, osteitis) is an inflammatory disorder of t
29 ents had SAPHO (synovitis, acne, pustulosis, hyperostosis, osteitis) syndrome; both had complete clea
30  Like axial PsA, diffuse idiopathic skeletal hyperostosis phenotypes, which can be indistinguishable
31 tics of thoracic diffuse idiopathic skeletal hyperostosis (T-DISH) in the Black patients using the co
32 in response was established and the onset of hyperostosis that can be suppressed with a chemical chap
33 ations coupled with maxillary and mandibular hyperostosis that progressively worsened with age.