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1 ed with endothelial dysfunction and vascular hyperpermeability.
2 ling increases inflammation-induced vascular hyperpermeability.
3 o deliver NO to subcellular targets to cause hyperpermeability.
4 angiogenesis and associated chronic vascular hyperpermeability.
5 naling in a mouse model for retinal vascular hyperpermeability.
6 esponsible for caveolae-mediated endothelial hyperpermeability.
7 s tumor growth and tumor-associated vascular hyperpermeability.
8 Rho signaling, monolayer disruption, and EC hyperpermeability.
9 lding domain (AP-Cav) reduces eNOS-regulated hyperpermeability.
10 rface VE-cadherin and inhibition of vascular hyperpermeability.
11 )-induced glomerular endothelial cell (GEnC) hyperpermeability.
12 akening of cell junctions and more robust EC hyperpermeability.
13 mbin, concomitant with increased endothelial hyperpermeability.
14 time course correlating with that of venular hyperpermeability.
15 at FAK contributes to VEGF-elicited vascular hyperpermeability.
16 n but significantly attenuated VEGF-elicited hyperpermeability.
17 itrite (ONOO(-)), we abrogated B box-induced hyperpermeability.
18 suffer both systemic and pulmonary vascular hyperpermeability.
19 tor (VEGF)-induced angiogenesis and vascular hyperpermeability.
20 lial growth factor (VEGF)-localized vascular hyperpermeability.
21 rs of inflammation, coagulation and vascular hyperpermeability.
22 of histologic evidence of mucosal damage and hyperpermeability.
23 nger) on the development of cytokine-induced hyperpermeability.
24 liorate the development of IFN-gamma-induced hyperpermeability.
25 mediator of cytokine-induced gut epithelial hyperpermeability.
26 ithin MYLK-L may limit SOCE-induced vascular hyperpermeability.
27 nhibition reduced SIN-1-but not SNAP-induced hyperpermeability.
28 e characterized by angiogenesis and vascular hyperpermeability.
29 e to several mediators that increase venular hyperpermeability.
30 have deleterious effects related to vascular hyperpermeability.
31 factor (VEGF)-induced cell proliferation and hyperpermeability.
32 e-induced endothelial barrier disruption and hyperpermeability.
33 endothelial glycocalyx layer and triggering hyperpermeability.
34 ced capillary network of VAT, and intestinal hyperpermeability.
35 and Erk1/2, as well as NOS1 expressions and hyperpermeability.
36 e iBRB and the promotion of retinal vascular hyperpermeability.
37 ponsible for TBI-induced BBB dysfunction and hyperpermeability.
38 en reported to inhibit VEGF-induced vascular hyperpermeability.
39 ility and mechanisms regulating UTMC-induced hyperpermeability.
40 oform VEGF165 stimulates vascular growth and hyperpermeability.
41 ed attenuation of IL-1beta-induced monolayer hyperpermeability.
42 s beneficial effects against TBI-induced BBB hyperpermeability.
43 dal exudation by reducing choroidal vascular hyperpermeability.
44 of a distinct CNV corresponding to the ICGA hyperpermeability.
45 I) is characterized by increased endothelial hyperpermeability.
46 during sepsis and, therefore, contributes to hyperpermeability.
47 endothelial junction disruption and vascular hyperpermeability.
48 n and p120 at the adherens junction and with hyperpermeability.
49 in the cytosol is necessary for PAF-induced hyperpermeability.
50 asma membrane to the cytosol is required for hyperpermeability.
51 the cytosol is necessary for agonist-induced hyperpermeability.
52 I and is not involved in endothelial barrier hyperpermeability.
54 ement (80.9%, vs.54.2% p = 0.007), choroidal hyperpermeability (50% vs 25.8%, p = 0.013) and lower ba
55 4.4% vs 28.7%; P < .001), choroidal vascular hyperpermeability (70.3% vs 17.2%; P < .001), and widesp
58 s and inducing blood-labyrinth barrier (BLB) hyperpermeability, along with changes in endothelial cel
59 ed 1) VEGF signaling pathway leading to GEnC hyperpermeability and 2) the modulatory effects of stati
61 preconditioned with ESCs could reverse cell hyperpermeability and apoptosis in vitro and protect aga
62 ents IFN-gamma-induced intestinal epithelial hyperpermeability and ATP depletion, possibly by fosteri
63 rowth factor antagonist, blocks the vascular hyperpermeability and blood flow increases induced by el
65 stration of ASP4058 also suppressed vascular hyperpermeability and CNV, and the effect was comparable
66 nees completely abrogated DENV-2 NS1-induced hyperpermeability and cross-inhibited hyperpermeability
67 reperfusion (I/R), losartan blocked vascular hyperpermeability and decreased infarct size, hemorrhage
68 y mediators of VEGF-induced endothelial cell hyperpermeability and demonstrates the modulatory effect
70 t mice were also refractory to lung vascular hyperpermeability and edema in a lipopolysaccharide-indu
71 ESL structure are believed to cause vascular hyperpermeability and entrap immune cells during sepsis,
73 rmined how procalcitonin induces endothelial hyperpermeability and how targeting procalcitonin protec
74 yte adhesion induced by endothelial junction hyperpermeability and ICAM-1 expression during inflammat
75 ndothelium-intrinsic dysregulation underlies hyperpermeability and implicate the cytoplasmic serine/t
78 sion and nitric oxide production to vascular hyperpermeability and intensified anaphylactic responses
79 Padi4KO mice were protected from intestinal hyperpermeability and metabolic impairment due to the tr
80 in angiogenesis, and JAM-C blockade reduced hyperpermeability and neovascularization in hypoxia-indu
82 ly ameliorates the severity of microvascular hyperpermeability and preserves multi-organ function in
83 se VEGF 121 genes induced localized vascular hyperpermeability and produced PE only after direct impl
84 monolayers were protected from microvascular hyperpermeability and retained tight junction integrity
85 chronic alcohol misuse can induce intestinal hyperpermeability and significantly shift microbial comm
87 otective mechanism of ANP against pathologic hyperpermeability and suggests a novel pharmacological i
88 nd-binding domain of RAGE, reversed vascular hyperpermeability and suppressed accelerated atheroscler
89 he microbiota-intestinal interface, inducing hyperpermeability and the systemic spillover of bacteria
90 RAs are thought to reduce choroidal vascular hyperpermeability and thickness by inhibiting the minera
91 of calpains in mediating BBB dysfunction and hyperpermeability and to test the effect of calpain inhi
92 al protein 1 (NS1) induces human endothelial hyperpermeability and vascular leak in mice, and NS1 vac
94 thermore, PGD(2) attenuated cytokine-induced hyperpermeability and zonula occludens-1 downregulation
96 ase-9 (MMP-9) is critical to TBI-induced BBB hyperpermeability, and doxycycline possesses anti-MMP-9
98 vessels serves as a barrier against vascular hyperpermeability, and its maintenance is critical to or
99 in promoting lymphatic enlargement, vascular hyperpermeability, and leukocyte recruitment, thereby le
100 Knockout of Sting ameliorated retinal NV, hyperpermeability, and leukostasis in Pparalpha(-/-) mic
102 ernalization is required for agonist-induced hyperpermeability, and suggests that a mechanism by whic
103 ay was used to examine VEGF-induced vascular hyperpermeability, and the mouse corneal model of angiog
104 mitogen that promotes angiogenesis, vascular hyperpermeability, and vasodilation by autocrine mechani
106 Increased neovascularization and vascular hyperpermeability are integral processes in tumors, and
107 ular mechanisms by which VEGF acts to induce hyperpermeability are poorly understood and in vivo mode
109 beta-ladder involved in inducing endothelial hyperpermeability as measured by transendothelial electr
110 de, cavtratin, was able to correct the tumor hyperpermeability as well as attenuate the increased tum
111 ulmonary alveolar epithelial and endothelial hyperpermeability as well as edema formation, all of whi
112 o attenuated Tat-induced endothelial and BBB hyperpermeability as well as transendothelial migration
113 dent biphasic blood flow stasis and vascular hyperpermeability, as determined by intravital microscop
115 n sulfate as a potential target for treating hyperpermeability associated with ischemic disease.
117 e were used to monitor dermal acute vascular hyperpermeability (AVH) and passive systemic anaphylaxis
119 re, as shown in Robo4(-/-) mice that develop hyperpermeability, but how Robo4 signals remained unclea
120 c injuries also promotes BBB dysfunction and hyperpermeability, but the underlying mechanisms are not
121 ith AICAR attenuated LPS-induced endothelial hyperpermeability by activating the Rac/Cdc42/PAK pathwa
122 drophobic statin, reversed VEGF-induced GEnC hyperpermeability by preventing MLC diphosphorylation, a
124 zes adheren junctions, resulting in vascular hyperpermeability, by converging with the VEGFA/VEGFR2/c
132 rotected EC monolayers from thrombin-induced hyperpermeability, disruption of intercellular junctions
134 Increased expression of PDE2 can mediate hyperpermeability effects of paracrine endothelial NP/GC
136 actor in the intrastrial fluid-blood barrier hyperpermeability exhibited in the mice is down-regulati
138 mice, this modest increase in HIF-dependent hyperpermeability factors was sufficient to promote vesi
139 ) breakdown and the associated microvascular hyperpermeability followed by brain edema are hallmark f
141 Sprague Dawley (SD) rats only showed retinal hyperpermeability from 3 to 10 days after the onset of d
142 hy (OIR), BN rats developed retinal vascular hyperpermeability from postnatal day 12 (P12) to P22 wit
144 te prevented IgE-mediated cutaneous vascular hyperpermeability, hypothermia, elevation in plasma hist
145 bitor, L-NMMA, blocked VEGF-induced vascular hyperpermeability in all ocular and nonocular tissues, p
146 aled dilated choroidal vessels and choroidal hyperpermeability in areas corresponding to altered AF o
150 yline as a therapeutic inhibitor of vascular hyperpermeability in human clinical conditions is promis
151 open-1-one (3PO) prevented histamine-induced hyperpermeability in human microvascular endothelial cel
152 d tumor necrosis factor (TNF)-alpha-mediated hyperpermeability in human umbilical vein endothelial ce
154 nctions with intercellular gap formation and hyperpermeability in microvascular endothelial cells.
156 intestinal mucosal injury, inflammation, and hyperpermeability in NASH + aleglitazar + JWH015 mice.
157 ensity/fibrosis, and intestinal inflammation/hyperpermeability in nonalcoholic steatohepatitis (NASH)
159 in the signal transduction of microvascular hyperpermeability in response to growth factors and infl
162 orway (BN) rats developed sustained vascular hyperpermeability in the retina during the entire experi
163 l protein Gc, potently inhibited endothelial hyperpermeability in vitro and vascular leak in vivo dur
164 ycosaminoglycans on the endothelium, causing hyperpermeability in vitro and vascular leakage in vivo
166 paran sulfate-protein interactions inhibited hyperpermeability in vivo, suggesting heparan sulfate as
168 side counteracted lipopolysaccharide-induced hyperpermeability in wild-type mice in vivo as well as i
169 that a large component of pulmonary vascular hyperpermeability induced by activation of PMNs adherent
170 paran sulfate biosynthesis in vivo decreases hyperpermeability induced by both VEGF(165) and VEGF(121
171 nduced hyperpermeability and cross-inhibited hyperpermeability induced by DENV-1, -3, and -4 NS1.
172 vitro and in vivo and attenuates endothelial hyperpermeability induced by inflammatory and edemagenic
173 taining caveolae to plasma membrane inhibits hyperpermeability induced by platelet-activating factor
174 trate a previously unrecognized mechanism of hyperpermeability induced by rises in cytosolic cAMP.
175 lin-1 in VVO structure in the acute vascular hyperpermeability induced by VEGF-A and in pathological
178 n strongly suppressed thrombin-induced HUVEC hyperpermeability, inhibiting ERK1/2 activation had no e
179 ling pathway that mediates VEGF-induced GEnC hyperpermeability involves RhoA activation leading to ac
180 cular dysfunction resulting from endothelial hyperpermeability is a common and important feature of c
182 A feature of thrombin-enhanced endothelial hyperpermeability is contraction of endothelial cells (E
183 ought to determine whether IFN-gamma-induced hyperpermeability is increased under acidic conditions.
184 ly, losartan-mediated inhibition of vascular hyperpermeability is mediated by the inhibition of phosp
186 induced elevation of PlGF is responsible for hyperpermeability mainly through increasing activation o
187 clusion, modulation of O&NS by R-107 reduced hyperpermeability markers and improved multi-organ funct
188 s correlated with reduction of microvascular hyperpermeability, neutrophil infiltration, and endothel
189 ines, alveolar neutrophil accumulation, lung hyperpermeability, NO production, and lipid peroxidation
191 y is controlled by CCR7 and that the chronic hyperpermeability of collecting vessels observed in Ccr7
198 retinal reattachment, reduction of choroidal hyperpermeability on ICGA and improvement of visual acui
199 th unremarkable results except for choroidal hyperpermeability on indocyanine green angiography (ICGA
200 atial correlation between choroidal vascular hyperpermeability on UWF ICGA images and areas of choroi
203 tor) successfully reversed the microvascular hyperpermeability phenotype of Cav-1 knock-out mice.
204 O in mice reduced histamine-induced vascular hyperpermeability, prevented vascular leakage in passive
205 arget for limiting oxidant-mediated vascular hyperpermeability, protein-rich edema formation, and acu
206 (KO) neonatal mice exhibit male-specific BBB-hyperpermeability, reduced neuronal excitability, and im
207 an isoform that inhibits blood-brain barrier hyperpermeability, reduced the number of stalled capilla
210 and vitronectin, respectively, prevented the hyperpermeability response to GRGDdSP and GPenGRGDSPCA.
212 ar endothelial monolayers, which displayed a hyperpermeability response to neutrophils and MLCK in a
213 l elements of the signalling pathway for the hyperpermeability response to PAF; (3) iNOS does not aff
214 a common role in mediating the microvascular hyperpermeability response to vascular endothelial growt
216 n endothelial monolayers displayed a similar hyperpermeability response to VEGF which was greatly att
221 line permeability; (b) eNOS is essential for hyperpermeability responses in inflammation; and (c) mol
223 s not affect either baseline permeability or hyperpermeability responses to PAF; and (4) caveolin-1 i
225 y prevented the VEGF-induced Ca2+ influx and hyperpermeability similar to the inhibitory effects seen
226 ence supports a role for an ischemia-induced hyperpermeability state regulated, in part, by VEGF.
227 lated with choroidal thickness and choroidal hyperpermeability, supporting that the pathogenesis of P
228 xhibited higher ability to cause endothelial hyperpermeability than exosomes from the non metastatic
229 eas of previous or ongoing SRF and choroidal hyperpermeability that can assist in the diagnosis of CS
230 are characterized by abnormal morphology and hyperpermeability that together cause inefficient delive
231 that mediate endothelial barrier leakiness (hyperpermeability) that are important in the pathogenesi
232 on of Epac also counteracts thrombin-induced hyperpermeability through down-regulation of Rho GTPase
234 s on the induction of iNOS expression and/or hyperpermeability to hydrophilic solutes in cultured ent
237 rst time that CCL2 induces brain endothelial hyperpermeability via Rho/PKCalpha signal pathway intera
238 explored the hypothesis that PAF stimulates hyperpermeability via S-nitrosation (SNO) of adherens ju
239 oxidative stress-induced pulmonary vascular hyperpermeability via transcellular and paracellular pat
248 ipopolysaccharide-induced pulmonary vascular hyperpermeability was significantly reduced in CD44 knoc
250 this study, the effect of doxycycline on BBB hyperpermeability was studied utilizing molecular modeli
252 etastasis and their vessels exhibit vascular hyperpermeability, we hypothesized that CysLT(2)R, via i
253 al models of acute lung injury with vascular hyperpermeability, we observed that HTJ-1 knockdown bloc
254 of acute lung injury with pulmonary vascular hyperpermeability, we observed that selective lung silen
256 resulted in increased basal permeability and hyperpermeability when stimulated by thrombin and TNF-a.
257 oxide synthase (NOS1) expressions as well as hyperpermeability, whereas inhibition of p38MAPK pathway
258 ction and of SNO blocked PAF-induced SNO and hyperpermeability, whereas inhibition of the cGMP pathwa
259 angiogenesis and associated chronic vascular hyperpermeability, whether induced by VEGF-A(164) or by
260 FU does not induce apoptosis rather vascular hyperpermeability, which can be alleviated by Resveratro
261 ndrome with diarrhea (IBS-D) have intestinal hyperpermeability, which contributes to their diarrhea a
262 Intestinal PARP activation resulted in gut hyperpermeability, which developed in PARP(+/+) but not
263 uced F-actin remodeling and Rho-dependent EC hyperpermeability, while expression of a phosphorylation
264 ICGA showed the characteristic choroidal hyperpermeability, while there was no evidence of choroi