ライフサイエンスコーパス: hyperplastic

1 ere non-neoplastic lesions (of which 62 were hyperplastic).

2 dividuals; most serrated polyps detected are hyperplastic.

3 the images of the test set as neoplastic or hyperplastic.

4 lveoli and the mesenchyme remained thick and hyperplastic.

5 A total of 273 colorectal glands (126 hyperplastic, 147 adenomatous) were isolated via laser c

6 On this basis, we predicted that hyperplastic aberrant crypt foci (ACF), a putative preca

7 een BRAF mutations and serrated histology in hyperplastic ACF supports the idea that these lesions ar

8 ver, that the lesions formerly classified as hyperplastic actually represent a heterogeneous group of

9 Furthermore, MUFA-HFD-fed mice displayed hyperplastic adipose tissue, with enhanced adipogenic po

10 bnormal production of corticotropin in these hyperplastic adrenal glands.

11 er RNA (mRNA) was detected in all samples of hyperplastic adrenal tissue.

12 h local production of the peptide within the hyperplastic adrenals.

13 subpopulation of steroidogenic cells in the hyperplastic adrenals.

14 and alpha-cells, with high expression in the hyperplastic alpha-cells of Gcgr(-/-) mice.

15 DCK was elevated in hyperplastic alveolar epithelial cells of patients with

16 l heterogeneity of IPF, we hypothesized that hyperplastic alveolar epithelial cells regulate the fibr

17 Interrogating the D2-HAN (hyperplastic alveolar nodule) model of breast cancer dor

18 helial cell proliferation and development of hyperplastic alveolar nodules and increased expression l

19 creased the percentage of mice with multiple hyperplastic alveolar nodules.

20 A test set of images (96 hyperplastic and 188 neoplastic polyps, smaller than 5 m

21 icant reduction in vascular surface areas in hyperplastic and angiogenic lesions in pancreatic islets

22 d that Stat3-deficient mice initially formed hyperplastic and early adenoma-like lesions that later c

23 epidermal keratinocytes become progressively hyperplastic and eventually produce tumors.

24 cardial cushions in FOG-2 deficient mice are hyperplastic and fail to remodel appropriately, suggesti

25 ly, mammary glands from transgenic mice were hyperplastic and highly vascularized.

26 e mechanistic insights into the link between hyperplastic and hypertrophic growth of cardiomyocytes.

27 In addition, the hyperplastic and inflammatory responses to topical phorb

28 transcriptional activity contributes to the hyperplastic and invasive phenotype of synoviocytes that

29 utation that elevates Dpp signaling was also hyperplastic and rescued.

30 whereas diameter decreased significantly in hyperplastic and SCC lesions.

31 -associated CS, especially in the context of hyperplastic and/or adenomatous polyps.

32 e terms risk or risk factor, and serrated or hyperplastic, and polyps or adenomas, and colorectal (or

33 st that LMO7 is upregulated in human intimal hyperplastic arteriovenous fistula and cardiac allograft

34 The isolation of additional loci that affect hyperplastic as well as neoplastic growth indicates the

35 fibrosis characterized by fibroblastic foci, hyperplastic AT2 cells, and increased susceptibility of

36 PA200 was upregulated in hyperplastic basal cells and myofibroblasts of fibrotic

37 orneal wound-healing defect characterized by hyperplastic basal progenitor cells (some of which under

38 ticularly tumor infiltration (P = .0044) and hyperplastic blood vessels (P = .0005).

39 r cells in the cardinal veins, together with hyperplastic, blood filled lymph sacs and hyperplastic d

40 Histologic features include hyperplastic bone consisting of mature cortical and trab

41 Cxcr4 and identify a molecular signature in hyperplastic breast lesions that signifies inappropriate

42 body mass and whole body energy expenditure, hyperplastic brown/white adipose tissues and larger hype

43 mTORC1 was also activated in the hyperplastic but not involuted follicles of HIV-associat

44 during goitrogenic stimulation abrogates the hyperplastic but not the hypertrophic thyrocyte response

45 mice, the mutant ICC stem cells were grossly hyperplastic but remained imatinib-resistant.

46 genetic hit is often required to push these hyperplastic cells to malignancy and confer mutation-spe

47 s syndrome mice showed that in some lesions, hyperplastic cells were predominantly GFP/Akt1E17K-posit

48 This is accompanied by hyperplastic changes in cells and desmoplastic alteratio

49 specific vivo-morpholino sequences inhibited hyperplastic cholangiocyte proliferation, liver damage,

50 sion of DDB2 and RNF43 was observed in human hyperplastic colonic foci.

51 tem called DNN-CAD to identify neoplastic or hyperplastic colorectal polyps less than 5 mm.

52 ormal terminal duct lobular units (TDLUs) by hyperplastic columnar epithelial cells is one of the mos

53 luate the significance of PKD1 in normal and hyperplastic conditions.

54 ced inflammation and dramatically attenuated hyperplastic cryptal response to infection in this group

55 The hyperplastic-deficient esophagus has a distinct expressi

56 th hyperplastic, blood filled lymph sacs and hyperplastic dermal lymphatic vessels.

57 s possible to differentiate adenomatous from hyperplastic diminutive (1-5 mm) polyps during endoscopy

58 yonic stem (ES) cells, we identify mammalian hyperplastic discs protein EDD, a known E3 ubiquitin lig

59 TTA indicated that the architecture of human hyperplastic discs protein is preserved.

60 t the DDMI protocol, we redesigned the human hyperplastic discs protein to bind to the kinase domain

61 yroidectomy, 100 to 300 mg of adenomatous or hyperplastic diseased parathyroid tissue was prepared an

62 s to determine which cells gave rise to both hyperplastic ductal cells and duct-associated insulin(+)

63 herefore, we tested the hypothesis that both hyperplastic ductal cells and their associated insulin(+

64 We found that hyperplastic ductal cells arose largely from acinar cell

65 tructures, corresponding to normal or mildly hyperplastic ductal epithelium.

66 t islet endocrine cells can intercalate into hyperplastic ducts as they develop.

67 del that develops insulin(+) cell-containing hyperplastic ducts in response to the growth factor TGFa

68 ct cells in the epithelium that makes up the hyperplastic ducts of both chronic pancreatitis and panc

69 ase in the frequency of medulloblastomas and hyperplastic EGL lesions, formed by preneoplastic GCPs.

70 It is not expressed in normal or hyperplastic endometrial.

71 These hyperplastic enlarged lobular units (HELUs) are importan

72 however, AERD was distinguished from chronic hyperplastic eosinophilic sinusitis by the prominent exp

73 y has a severe course accompanied by chronic hyperplastic eosinophilic sinusitis with nasal polyps, b

74 from control subjects, patients with chronic hyperplastic eosinophilic sinusitis, and patients with A

75 at NAFs can inhibit the growth of normal and hyperplastic epi-thelium but are less able to regulate t

76 ML3 is a useful marker for normal and benign hyperplastic epidermal development, whereas the loss of

77 lin D1, and MMP7 in normal (NT), tumour (T), hyperplastic epidermis and/or squamous papilloma (Hyp/Pa

78 of hair shafts with increased apoptosis, and hyperplastic epidermis during this time.

79 ated ATM and other markers of the DDR in the hyperplastic epidermis of transgenic mice expressing E2F

80 ion was localized to the upper region of the hyperplastic epidermis.

81 AB2IP(-/-) mice, the prostate gland exhibits hyperplastic epithelia, in which AR becomes more active.

82 d by abnormal tissue repair characterized by hyperplastic epithelial cell formation, is a pathogenic

83 alysis revealed a novel mediator, MMP-19, in hyperplastic epithelial cells adjacent to fibrotic regio

84 Bik reduces hyperplastic epithelial cells by releasing calcium from

85 rmore, after prolonged exposure to allergen, hyperplastic epithelial cells persist longer, and nuclea

86 in IPF lungs and colocalized with MMP-19 in hyperplastic epithelial cells.

87 We show that MMP3 overexpression promotes hyperplastic epithelial growth, surprisingly, in a nonpr

88 nasal epithelium when compared to normal or hyperplastic epithelium.

89 revents cardiovascular anomalies including a hyperplastic expansion within a large Ccm1-deficient vas

90 The hyperplastic field of normal-looking cells surrounding t

91 All carcinomas and hyperplastic foci on MR images were localized on the bas

92 Surprisingly, by 18 mo of age, the number of hyperplastic foci was reduced, the ducts of the VP and M

93 sion yielded enhanced mammary gland density, hyperplastic foci, cellular dysplasia, and mammary carci

94 ted a ductal thickening accompanied by small hyperplastic foci.

95 ed classification system for differentiating hyperplastic from adenomatous polyps.

96 aging expertise is required to differentiate hyperplastic from neoplastic polyps with high levels of

97 A total of 119 polyps (81 neoplastic, 38 hyperplastic) from 75 patients was assessed.

98 ed expression of 32,381 murine genes in nine hyperplastic ganglia harvested at three time points and

99 s with CVID+AIC displayed irregularly shaped hyperplastic GCs, whereas GCs were scarce and small in p

100 utonomous growth, leading to a homogeneously hyperplastic gland and to the development of follicular

101 nsive centrosome amplification in tumors and hyperplastic glands prior to tumor formation suggests th

102 of PTEN or activation of AKT in regenerated hyperplastic glands that were surrounded by wild-type st

103 t had four-gland hyperplasia, of which three hyperplastic glands were not localized.

104 lands (9 adenomas, 1 atypical adenoma, and 9 hyperplastic glands) were removed from 17 patients, and

105 on in parathyroid chief cells in adenoma and hyperplastic glands, and also in normal parathyroid by i

106 in SH, EGFR activation is the cause of both hyperplastic growth and VDR reduction and therefore infl

107 of the vertebrate embryonic heart occurs by hyperplastic growth as well as the incorporation of cell

108 age-independent growth and conferred benign, hyperplastic growth in a skin-like environment.

109 into new adipocytes during the formation and hyperplastic growth of white adipose tissue.

110 proliferation and plays an important role in hyperplastic growth, which has a positive effect on meat

111 Similar p53 activity occurred in hyperplastic growths that were triggered either by the R

112 genes, myocardial genes, and development of hyperplastic hearts.

113 astic brown/white adipose tissues and larger hyperplastic hearts.

114 ce endoscopic interpretations of adenoma and hyperplastic histology for polyps 5 mm and smaller in si

115 High-confidence predictions of adenoma and hyperplastic histology were correct for 91% and 95%, res

116 Endoscopic predictions of adenoma and hyperplastic histology were made with high confidence fo

117 pic features associated with adenomatous and hyperplastic histology.

118 ophthalmia, failed lens differentiation, and hyperplastic hyaloid vessels.

119 ogenitors are true stem cells for normal and hyperplastic ICC and give rise to GIST.

120 ivity and postjunctional neural responses in hyperplastic ICC tissues appeared normal but were up-reg

121 DNN-CAD classified polyps as neoplastic or hyperplastic in 0.45 +/- 0.07 seconds-shorter than the t

122 bing depth and plaque, papilla bleeding, and hyperplastic index scores were recorded.

123 beta-catenin, treatment shifted cells within hyperplastic intestinal crypts from a stem cell to a tra

124 VS28 mice showed a hyperplastic intestinal epithelium and, strikingly, deve

125 ovascular angiogenesis, reduced outgrowth of hyperplastic intima, and minimized CD4(+)CD103(+) tissue

126 course of RA, the synovium transforms into a hyperplastic invasive tissue that causes destruction of

127 It is a concentric vascular intimal hyperplastic lesion composed of smooth muscle-like cells

128 ute to chronic inflammation, and its role in hyperplastic lesion development requires further examina

129 oped renal injury with loss of podocytes and hyperplastic lesion formation; beta-arrestin-1 expressio

130 transformation of benign renal cysts into a hyperplastic lesion, suggesting that fumarate-induced se

131 of undifferentiated nestin(+) cells in both hyperplastic lesions and primary tumors that may serve a

132 ransgenic mice (WAP-HA-14-3-3zeta) developed hyperplastic lesions and showed increased susceptibility

133 d tissues correlates with the development of hyperplastic lesions and the presence of PthA4.

134 uitary, the p27CK- protein dominantly caused hyperplastic lesions and tumors in multiple organs, incl

135 We did not observe a detectable DDR in early hyperplastic lesions arising in transgenic mice expressi

136 These results suggest that glomerular hyperplastic lesions derive from the proliferation and m

137 a significant inhibition in the formation of hyperplastic lesions during tumor progression.

138 ession of IGF-1(des) was sufficient to cause hyperplastic lesions in all mice, however the well-diffe

139 astoma development in these mice begins with hyperplastic lesions in early postnatal sympathetic gang

140 drogenase (ALDH) as well as by generation of hyperplastic lesions in humanized fat pads of NOD (nucle

141 opmental defect of the prostate and prostate hyperplastic lesions in mouse.

142 Prostate hyperplastic lesions in Mp(+/-) mice also induced stroma

143 gammaH2AX expression was elevated in hyperplastic lesions in the mammary fat pads of BALB-neu

144 The cellular composition of the resulting hyperplastic lesions is controversial, although a popula

145 LA-exposed SHHF rats developed atypical hyperplastic lesions of bronchiolar epithelial cell orig

146 teristically develop severe, diffuse intimal hyperplastic lesions that eventually compromise luminal

147 -regulated during multistep progression from hyperplastic lesions to islet tumors.

148 When hyperplastic lesions were further classified into serrat

149 levels of IGF-1 facilitate the emergence of hyperplastic lesions while imposing a strong IGF-1-depen

150 an prevented podocyte loss, formation of the hyperplastic lesions, and normalized expression of glome

151 ut mice leads to the development of prostate hyperplastic lesions, and this effect was mediated throu

152 ogen receptor (AR) does not give rise to any hyperplastic lesions, but when combined with high levels

153 eolar type II (ATII) cells were able to form hyperplastic lesions, some of which progressed to adenom

154 as a reduction in the number and severity of hyperplastic lesions, the endometrial phenotype persiste

155 , and CD4(+) and gammadelta T cells in mouse hyperplastic lesions.

156 ll GCPs and, remarkably, reduces the area of hyperplastic lesions.

157 colonoscopy, and obtained tissue blocks for hyperplastic lesions.

158 Hyperplastic livers and tumors from YAP(S127A) mice had

159 pport a novel role for the CaR in preventing hyperplastic lung disease in utero.

160 We obtained specimens of hyperplastic macronodular adrenal tissue from 30 patient

161 However, hyperplastic mammary epithelium was a mixture of GFP/Akt

162 rrow reference cells) recapitulate the known hyperplastic mechanism of weight gain with pioglitazone

163 Bmp9-knockout (KO) pups presented hyperplastic mesenteric collecting vessels that maintain

164 Second, affected organs showed variable hyperplastic, metaplastic, and connective tissue changes

165 ent membrane (BM) in all neoplastic, but not hyperplastic, models examined.

166 ugh Kras/KRAS mutation promotes serrated and hyperplastic morphologic features in colon epithelium, i

167 Plxnd1 deficiency in zebrafish induced hyperplastic morphology in visceral adipose tissue (VAT)

168 evels in both human papilloma virus-positive hyperplastic murine skin and a subset of human squamous

169 ed expansion and oncogenic transformation of hyperplastic neuroblasts, thus promoting progression to

170 plastic) or reactive (nonmalignant, lymphoid hyperplastic) nodes.

171 d the percentage of mice exhibiting multiple hyperplastic nodules, invasive mammary cancers and cance

172 If HZ disruption is severe, hyperplastic OB-ISCs expand across the interorgan bounda

173 The hyperplastic ocular phenotypes were largely suppressed b

174 for patients whose colonoscopies identified hyperplastic (odds ratio [OR] = 3.1; 95% CI, 1.7-5.5) or

175 common and accurate endoscopic prediction of hyperplastic or adenomatous polyp histology could reduce

176 45%-52% after colonoscopies that identified hyperplastic or adenomatous polyps.

177 5 or more GI polyps, 1 or more of which was hyperplastic or hamartomatous (N = 397), were prospectiv

178 at seen in normal (nonmalignant, nonlymphoid hyperplastic) or reactive (nonmalignant, lymphoid hyperp

179 s lacking the tumor suppressor PTEN to cause hyperplastic overgrowth.

180 ge and reduced the incidence of diabetes and hyperplastic pancreatic islets at 6 months of age.

181 rmoid carcinoma cell line A431, which mimics hyperplastic parathyroid cells.

182 CAAT enhancer binding protein-beta, in human hyperplastic parathyroid glands and in the human epiderm

183 e parathyroid adenomas in 6 patients, and 10 hyperplastic parathyroid glands in 3 patients).

184 posite characteristics, the degenerative and hyperplastic pathologies of aging are at least partly li

185 tribute to both age-related degeneration and hyperplastic pathologies, including cancer.

186 ea that it also drives both degenerative and hyperplastic pathologies, most likely by promoting chron

187 ially among vertebrates, aging also promotes hyperplastic pathologies, the most deadly of which is ca

188 n in later life, leading to hypertrophic and hyperplastic pathologies.

189 entrations, and a reduction in the number of hyperplastic Peyer's patches.

190 Ovariectomized Mig-6(d/d) mice exhibit this hyperplastic phenotype in the presence of E2 and P4 but

191 We identified a hyperplastic phenotype of ISMC, uniquely adapted to an i

192 type genome, and upon stratification, a more hyperplastic phenotype, with an increase in the number o

193 100A8/A9 and 27 other genes and reversed the hyperplastic phenotype.

194 study were a total of 688 adenoma cases, 210 hyperplastic polyp cases, and 1306 polyp-free controls f

195 and the association between the report of a hyperplastic polyp in the baseline CC report and the pro

196 d be in part attributed to the presence of a hyperplastic polyp in the baseline CC.

197 idual crypts from six colonic adenomas and a hyperplastic polyp were microdissected and characterized

198 to estimate ORs and 95% CIs among cases (556 hyperplastic polyp, 1753 adenoma, and 208 SSL) and contr

199 he most advanced lesion present: no polyp, a hyperplastic polyp, a tubular adenoma less than 1 cm in

200 xclusive categories, including normal colon, hyperplastic polyp, nonadvanced adenoma, and advanced ad

201 l polyp were recognized: the adenoma and the hyperplastic polyp.

202 nd 1.30 in the 55 cases vs 235 controls with hyperplastic polyps (95% CI, 0.96-1.77).

203 Gastric hyperplastic polyps (GHPs) have a potential risk of neop

204 regions containing normal epithelium (NE) or hyperplastic polyps (HP) to be distinguished from region

205 ecursors to colorectal cancer (CRC), whereas hyperplastic polyps (HPPs) have low risk of progression

206 444 cases with adenomas only, 662 cases with hyperplastic polyps (HPPs) only, and 437 cases with sync

207 re phosphorylated in serrated areas of human hyperplastic polyps (HPPs), sessile serrated adenomas, a

208 6 of 372) of serrated lesions, respectively; hyperplastic polyps (HPs) accounted for 58.9% (219 of 37

209 Individuals with SPs were classified into hyperplastic polyps (n = 34; 32%), traditional serrated

210 a (odds ratio: 1.60; 95% CI: 1.12, 2.29) and hyperplastic polyps (odds ratio: 1.85; 95% CI: 1.09, 3.1

211 itivity 0.94; 95% CI 0.90-0.97) and 55 of 62 hyperplastic polyps (specificity 0.89; 0.78-0.95), with

212 HETE were significantly higher in those with hyperplastic polyps and adenomas compared to those with

213 tubular and sessile serrated adenomas versus hyperplastic polyps and normal mucosa.

214 d adenomas (TSAs) are now distinguished from hyperplastic polyps and recognized as precursors to colo

215 The family of serrated lesions includes hyperplastic polyps and sessile serrated adenomas withou

216 ns with no polyps at baseline and those with hyperplastic polyps at baseline (1.1% [12 of 1057] and 2

217 These serrated polyps include not only hyperplastic polyps but also traditional serrated adenom

218 eported that colon carcinomas, adenomas, and hyperplastic polyps exhibiting a serrated histology were

219 low risk adenoma in 158 cases (11.3 %), and hyperplastic polyps in 119 cases (8.5 %).

220 lying conditions were hemorrhoids in 7 (6%), hyperplastic polyps in 4 (3.5%), adenomatous polyps in 2

221 UFAs were not associated with adenomatous or hyperplastic polyps in either men or women.

222 cid was associated with an increased risk of hyperplastic polyps in men (P-trend = 0.03), which was n

223 tion between the outcome and the presence of hyperplastic polyps in the baseline CC, showed a statist

224 d be considered a mixed polyp syndrome, with hyperplastic polyps most prevalent, with a risk of early

225 ases with adenomatous polyps only (n = 639), hyperplastic polyps only (n = 294), and both types of po

226 for adenoma only, 0.2 (95% CI: 0.1, 0.3) for hyperplastic polyps only, and 0.2 (95% CI: 0.2, 0.4) for

227 tous polyps only, 5.0 (95% CI: 3.3, 7.3) for hyperplastic polyps only, and 6.9 (95% CI: 4.4, 11.1) fo

228 2% of patients were found to have incidental hyperplastic polyps or adenomas with low-grade dysplasia

229 vary among countries, but SSLs and proximal hyperplastic polyps require special attention in assignm

230 s of cigarette smoking and were stronger for hyperplastic polyps than for adenoma.

231 aging system in the identification of distal hyperplastic polyps that do not need resection, as well

232 the precursor to colorectal cancer, whereas hyperplastic polyps were viewed as innocuous lesions wit

233 st set, the DNN-CAD identified neoplastic or hyperplastic polyps with 96.3% sensitivity, 78.1% specif

234 However, failure to recognize adenomas (vs hyperplastic polyps), or discarding a polyp with advance

235 ly unimportant lesions (diminutive adenomas, hyperplastic polyps).

236 No SNP was significantly associated with hyperplastic polyps, and only rs6983267 was significantl

237 those with no polyps, those with one or more hyperplastic polyps, and those with one or more adenomas

238 31st 2005, without reported lesions or with hyperplastic polyps, based on secondary data extracted f

239 stology, they were wild type for BRAF; among hyperplastic polyps, KRAS mutations were found mainly in

240 mages of neoplastic polyps and 681 images of hyperplastic polyps, obtained from the picture archiving

241 The specificity in individuals with normal, hyperplastic polyps, or nonadvanced adenomas was 82.0% (

242 SPs comprise hyperplastic polyps, sessile serrated adenomas/polyps (S

243 e assigning serrated polyps to categories of hyperplastic polyps, traditional serrated adenomas, and

244 radic high-grade dysplastic adenomas, and 19 hyperplastic polyps--and tissue derived from patients wi

245 intake on risk of colorectal adenomatous and hyperplastic polyps.

246 Of the 64, half had hyperplastic polyps.

247 g intake in relation to both adenomatous and hyperplastic polyps.

248 ith an elevated risk of both adenomatous and hyperplastic polyps.

249 An inverse association trend was found for hyperplastic polyps.

250 er for sessile serrated adenomas/polyps than hyperplastic polyps.

251 ize and was not engendered by nonneoplastic (hyperplastic) polyps.

252 were found to accelerate the development of hyperplastic precancerous lesions.

253 urred in microphthalmic eyes with persistent hyperplastic primary vitreous (more recently termed pers

254 ting in eye disease mimicking the persistent hyperplastic primary vitreous (PHPV) found in Arf-null m

255 logical process resembling severe persistent hyperplastic primary vitreous (PHPV), a human eye diseas

256 ilar to that of human eye disease persistent hyperplastic primary vitreous (PHPV).

257 evelop eye disease reminiscent of persistent hyperplastic primary vitreous (PHPV).

258 mutants strongly resembles human persistent hyperplastic primary vitreous cases and may provide insi

259 suggest that autosomal recessive persistent hyperplastic primary vitreous is caused by N46H and is e

260 syndrome closely resembles human persistent hyperplastic primary vitreus (PHPV), attributed to faile

261 RP782 uptake tracks the hyperplastic process in vascular remodeling and provides

262 and loss of RB is proposed to facilitate the hyperplastic proliferation associated with tumorigenesis

263 wever, we have previously shown that SCs are hyperplastic, rather than neoplastic, and able to induce

264 n disease pathogenesis, become activated and hyperplastic, releasing proinflammatory factors and tiss

265 damaged blood-brain barrier and a subsequent hyperplastic response that results in a dense scar that

266 mount an appropriate compensatory beta-cell hyperplastic response to maintain normoglycemia.

267 exhibited a markedly exaggerated neointimal hyperplastic response to wire injury of the femoral arte

268 d with the inhibition of the UVR-induced (i) hyperplastic response, (ii) Hsp90beta-PKCvarepsilon inte

269 eby down-regulating Pim1 and suppressing the hyperplastic response.

270 We further demonstrate that hyperplastic SCs accumulate non-cell-autonomously via si

271 kin show follicles in prolonged telogen with hyperplastic sebaceous glands.

272 olyps, including at least 1 hamartomatous or hyperplastic/serrated polyp.

273 es blood vessels to coalesce into large flat hyperplastic sinuses with no distinctive hierarchical or

274 aser-capture microdissection of normal skin, hyperplastic skin and papillomas showed that amplificati

275 ids (UFs) are benign tumors characterized by hyperplastic smooth muscle cells and excessive depositio

276 -treated HNSCC lines, human HNSCC tumors and hyperplastic squamous epithelia of transgenic mice overe

277 hibition of NF-kappaB increases apoptosis in hyperplastic stages of tumor development and decreases p

278 This persistent hyperplastic state appeared to result from an equilibriu

279 umor cells and their transformation beyond a hyperplastic state.

280 elopmentally-timed apoptotic response in the hyperplastic sympathoadrenal cells.

281 s with MYCN to increase the proliferation of hyperplastic sympathoadrenal precursor cells, leading to

282 nced cell proliferation, consistent with the hyperplastic synovial tissue observed in MPS patients.

283 endent cell proliferation and development of hyperplastic three-dimensional acini without affecting a

284 Distinguishing hyperplastic thymus from tumors was better with SII sign

285 changes in the timing of the transition from hyperplastic to hypertrophic growth of cardiomyocytes co

286 Mammalian cardiomyocytes undergo a critical hyperplastic-to-hypertrophic growth transition at early

287 RNAs may have important functions in cardiac hyperplastic-to-hypertrophic growth transition.

288 ative activity and is likely associated with hyperplastic-to-hypertrophic growth transition.

289 rointestinal lymphoid hyperplasia, extensive hyperplastic tonsils, thymus hyperplasia, autoimmune lym

290 Mutations in the expanded gene act as hyperplastic tumor suppressors, interfere with cell comp

291 els, including loss-of-function mutations in hyperplastic tumour suppressors in the Hippo-Salvador-Wa

292 EoE), in which basal progenitor cells become hyperplastic upon proinflammatory stimulation.

293 f cytokeratin 14-positive basal cells in the hyperplastic urothelial mucosa in male Foxa1 knockout mi

294 ysis AV graft hyperplasia and possibly other hyperplastic vascular disorders.

295 we report that differentiated adipocytes are hyperplastic when SIRT1 is knocked down stably in mouse

296 r-gradient variation the majority seem to be hyperplastic whereby non-native individuals express phen

297 Klf5CN palpebral epidermis was hyperplastic with 7-9 layers of keratinocytes, compared

298 Nkx2-5 mutants died shortly after birth with hyperplastic working myocytes and conduction system incl

299 o not contain mucosal microbiota and exhibit hyperplastic yet inefficient GC responses that favor the

300 weak PSCA staining on tumor cells present in hyperplastic zones and in areas that contained focal and