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1 ately 25-50 are reduced, with 6-8 considered hyperreactive.
2 ased from endothelium is ultralarge (UL) and hyperreactive.
3 not conclude whether or not asthmatic ASM is hyperreactive.
4 hat natural ovarian steroids directly reduce hyperreactive 5-HT and thromboxane A2-stimulated Ca2+ an
5 y exposed workers and control subjects to be hyperreactive (95% confidence interval, 1.8-25.2; p = 0.
6 against syngeneic MHC-II(+) skin grafts, (2) hyperreactive against third-party MHC-II(+) skin grafts,
7 ant therapeutic targets for the treatment of hyperreactive airway disease.
8 tself is not required for the asthmatic-like hyperreactive airway response.
9  class had diminished lung function and more hyperreactive airways compared with all other classes.
10 ty and creating a NO deficiency that induces hyperreactive airways.
11 rs the TCR threshold and renders lymphocytes hyperreactive and capable of unwanted immune responses.
12    Platelets from patients with diabetes are hyperreactive and demonstrate increased adhesiveness, ag
13                        The sixth subject was hyperreactive and differed from the other subjects.
14 d mice demonstrated that their platelets are hyperreactive and form larger thrombi.
15 , CD44(high), but not CD44(low) T cells, are hyperreactive and hyperproliferative in vivo.
16                 Reticulated platelets (RPs), hyperreactive and RNA-rich, are associated with increase
17 ollapse, 23% of highly exposed subjects were hyperreactive as compared with only 11% of moderately ex
18 nd hyperreactivity persisted in 55% of those hyperreactive at 1 and/or 3 months.
19 ically silencing these neurons abolished the hyperreactive broncho-constrictions, even in the presenc
20 rlapping with those identified previously as hyperreactive by administration of exogenous reagents (t
21 e c oxidase-negative/succinate dehydrogenase-hyperreactive (COX-/SDH++) fibers from normal aging huma
22                   FA reacts with privileged, hyperreactive cysteine sites in the proteome, including
23 work is the first to directly identify seven hyperreactive cysteines: 1040, 1303, 2436, 2565, 2606, 2
24  primarily mediated through highly reactive (hyperreactive) cysteines.
25 of PTE assume a specific conformation with a hyperreactive guanylate (G*) in SHAPE structure probing,
26 ibody-drug conjugates, and identification of hyperreactive methionine residues in whole proteomes.
27 over ligandable proteins and sites harboring hyperreactive methionine within the human proteome.
28 ll transfer from long-term exposed mice into hyperreactive mice also restored normal airway responsiv
29 how that normal responsiveness in previously hyperreactive mice, achieved after long-term allergen ch
30 oxide nanoparticles (SPIONs) as recoverable, hyperreactive microwave absorbers for sludge prehydrolys
31 represents an important mechanism behind the hyperreactive nature of basophils that has long been obs
32         PRESS performs well in identifying a hyperreactive phenotype in patients with PAD (AUC [cross
33 yeloproliferative neoplasms (MPNs) exhibit a hyperreactive phenotype.
34 the rs956115 marker of the IRS-1 gene have a hyperreactive platelet phenotype and increased risk of M
35  marker were more commonly associated with a hyperreactive platelet phenotype.
36 et messenger RNA (mRNA) from subjects with a hyperreactive platelet phenotype.
37 d tools exist to identify individuals with a hyperreactive platelet phenotype.
38 eripheral artery disease (PAD), those with a hyperreactive platelet response (>60% aggregation) to 0.
39 anges in platelet gene expression creating a hyperreactive platelet, despite antiplatelet therapy.
40 tion in humans, but can cumulatively lead to hyperreactive platelets and increase risk for negative o
41                            Both children had hyperreactive platelets, as determined by whole blood pl
42  attributed to multiple mechanisms including hyperreactive platelets, hypercoagulable status, and end
43                                              Hyperreactive responses to the combination of 5-HT and U
44 t alterations in the allergen-induced airway hyperreactive responses.
45 et therapies, and it has been suggested that hyperreactive reticulated platelets underlie this altere
46                                              Hyperreactive RPs drive arterial thrombosis, whereas pro
47     Leveraging this chemistry, we discovered hyperreactive sites responsible for acrolein-induced mod
48 ted for reducing the rate of CPM labeling of hyperreactive SR thiols (IC50 = 0.3 and 1.8 microM, resp
49 oup is essential for modulating the state of hyperreactive SR thiols.
50                                              Hyperreactive sulfhydryl groups associated with the Ca(2
51 nd dose-dependently interact with a class of hyperreactive sulfhydryl groups localized on ryanodine-s
52 lcoumarin (CPM) to measure the reactivity of hyperreactive sulfhydryl moieties on sarcoplasmic reticu
53 itions previously shown to selectively label hyperreactive sulfhydryls and eliminate redox sensing.
54 ely and reversibly alters the redox state of hyperreactive sulfhydryls localized in the RyR/Ca2+ chan
55                                Alkylation of hyperreactive sulfhydryls on RYR1 with N-ethylmaleimide
56                                              Hyperreactive sulfhydryls previously shown to reside wit
57                                          The hyperreactive systemic inflammatory response seen in age
58                                          The hyperreactive T residue in the six sites detected as wel
59 lated to their ability to selectively modify hyperreactive thiols regulating normal functioning of mi
60            In summary, TRAF3 renders B cells hyperreactive to antigens and TLR agonists, promoting au
61 ound that pre-GT WASp-deficient B cells were hyperreactive to B cell receptor stimulation (BCR stimul
62 onsequence, TIPE2 knockout myeloid cells are hyperreactive to Poly (I:C) stimulation, and TIPE2 knock
63  is a feature of asthma in which airways are hyperreactive to stimuli causing extensive airway narrow
64       Here, we show that people with SCI are hyperreactive to uncertainty and that this might be a ke
65              Antigen-challenged animals were hyperreactive to vagal stimulation, and demonstrated los
66 ent antigen-presenting cells (APCs) but were hyperreactive to wild-type APCs.
67 ence of E3Q EcFpg, the Sp nucleotide (nt) is hyperreactive toward cleavage by MPE-Fe(II)-generated hy
68 together with a pathological accumulation of hyperreactive ultra-large VWF (UL-VWF) multimers.
69 age (IL-6), resulting in the accumulation of hyperreactive ULVWF in plasma and on the surface of endo
70 ease with thrombospondin motif) converts the hyperreactive unusually large (UL) forms of von Willebra
71          To test this hypothesis, we treated hyperreactive VMC from ovariectomized (ovx) monkeys in v
72              ADAMTS13 cleaves ultralarge and hyperreactive von Willebrand factor (ULVWF) freshly rele
73 These findings suggest an important role for hyperreactive VWF in SCD pathology and connect SCD to ot