ライフサイエンスコーパス: hypersensitive

1 ined tolerant; all Group B patients remained hypersensitive.

2 is insufficient to cause cells to be GM-CSF hypersensitive.

3 ACC2 knockout mutants, by contrast, are hypersensitive.

4 e of 10 patients with CRU (10%) were aspirin hypersensitive.

5 also cause health problems to people who are hypersensitive.

6 Here we describe an expanded search for hypersensitive accessions of Arabidopsis, evaluate wheth

7 nucleotide resolution, coincident with DNase hypersensitive and ATAC-seq sites at a low sequencing bu

8 Both hypersensitive and control glutamatergic terminals were

9 HCM mutants were hypersensitive and DCM mutants were hyposensitive to Ca(

10 surface poly-N-acetyl-lactosamine and led to hypersensitive and hyperresponsive immunocytes.

11 the DSB-induced G2-M checkpoint, inducing a hypersensitive and prolonged arrest.

12 T cells are equivalent to the ones formed in hypersensitive and tolerant patients, which indicates th

13 NCODE database showed the presence of DNaseI hypersensitive and transcription factors binding sites i

14 ase was designed and used as the basis for a hypersensitive assay.

15 An example is BC hyperacusis (hypersensitive BC hearing)-an unnerving symptom experien

16 a strong level of rescue to the Arabidopsis hypersensitive bzip19 bzip23 double mutant under Zn defi

17 Here, we experimentally evolved an IFN-beta-hypersensitive CA mutant which showed decreased binding

18 ed by receptor-like protein Cf-2, triggering hypersensitive cell death (HR) and disease resistance.

19 effector strongly suppresses both basal and hypersensitive cell death innate immune responses, and i

20 In Arabidopsis, NRG1 is required for the hypersensitive cell death response (HR) and full oomycet

21 oxygen species and enhancement of localized hypersensitive cell death.

22 inal 29 amino acids are sufficient to induce hypersensitive cell death.

23 ative genomic analysis was performed between hypersensitive cells and cells categorized as least sens

24 Hypersensitive cells underwent early S-phase arrest at d

25 putationally designed mutant, E181K, that is hypersensitive, confirming predictions derived from in s

26 ructs, corresponding to roughly 3500 DNase I hypersensitive (DHS) sites, into the mouse retina by ex

27 The KRAS nuclease-hypersensitive element (NHE) region contains a G-rich el

28 uman KRAS proto-oncogene contains a nuclease-hypersensitive element located upstream of the major tra

29 We also describe a set of 37 'hypersensitive' genes which were frequently targeted by

30 clade A of type 2C protein phosphatases: ABA-HYPERSENSITIVE GERMINATION 1 (AHG1) and AHG3.

31 ANT1-deficient animals are insulin-hypersensitive, glucose-tolerant, and resistant to high

32 Unexpectedly, the rapamycin hypersensitive growth arrest of vip1-1 cells was depende

33 e filtrates that have the capacity to elicit hypersensitive (HR) cell death and disease resistance in

34 The peanut proteins however cause hypersensitive immunogenic responses in certain individu

35 ergy, that is an overreaction to allergen in hypersensitive individuals, results from the production

36 Arabidopsis hypersensitive-induced reaction (AtHIR) proteins functio

37 gene encoding a putative protein similar to hypersensitive-induced response proteins (HIR) was ident

38 In Solomon, the hypersensitive inducible genes such as pathogenesis-rela

39 hat this ultrafine nanorod material exhibits hypersensitive intense red emission (610 nm) with good b

40 often render an addict's brain reward system hypersensitive, leaving the individual more susceptible

41 -based mechanism of resistance, six showed a hypersensitive-like response while three had elevated SA

42 We find that maize MNase-hypersensitive (MNase HS) regions localize around active

43 tified eed1Delta/Delta as the first farnesol hypersensitive mutant of C. albicans.

44 ngolipid-depleted human cells and identified hypersensitive mutants in genes of membrane trafficking

45 x networks, buildings, or vessels, where the hypersensitive nature of multiple interference paths cha

46 mice, in which immunostimulation resulted in hypersensitive neutrophils.

47 is sensorially mediated by a massive pair of hypersensitive, night-vision eyes [5-7].

48 opportunity of safer and more cost-effective hypersensitive NMR applications employing low-power lase

49 ll response induced in the blood and skin of hypersensitive patients and healthy volunteers.

50 Soybean beverages represent a risk for PR-10 hypersensitive patients and should be avoided.

51 y volunteers, and the mycobiome signature of hypersensitive patients differed from that of normally s

52 570, 84% CD4(+)) from blood of piperacillin-hypersensitive patients proliferated and secreted TH1/TH

53 Analysis of plasma from hypersensitive patients revealed the same pattern and le

54 o define the epitopes formed in tolerant and hypersensitive patients taking the beta-lactam antibioti

55 T-cell clones were generated from immediate hypersensitive patients' blood by serial dilution and re

56 period, 182 positive DPT (accounting for 171 hypersensitive patients) were analysed.

57 sitive DPT were analyzed (accounting for 285 hypersensitive patients).

58 B cells from hypersensitive patients, but not controls, were stimulat

59 T lymphocytes are activated in piperacillin-hypersensitive patients.

60 T-cell clones were generated from 4 hypersensitive patients.

61 OsHAC4 is the causal gene for the As(V)-hypersensitive phenotype.

62 nhibitor fluridone rescued the mybs1 glucose-hypersensitive phenotype.

63 e receptor Tim50 to promote the transport of hypersensitive precursors into the matrix.

64 Administration of fungicide to hypersensitive rats reduced their visceral hypersensitiv

65 , necrosis, nephrotoxicity and often develop hypersensitive reactions.

66 ing at enhancers, promoters, and other DNase hypersensitive regions not marked with canonical histone

67 ys, overlap with human cardiac tissue DNaseI hypersensitive regions, and predicted impact of sequence

68 , ORC binds nonspecifically to open (DNase I-hypersensitive) regions containing active chromatin mark

69 This hypersensitive regulatory switch is entirely dependent o

70 sitic weed Striga gesnerioides, developing a hypersensitive response (HR) at the site of parasite att

71 The hypersensitive response (HR) is a localized programmed c

72 , effector recognition by the host elicits a hypersensitive response (HR) that overcomes the inhibiti

73 ene, PAD4, and salicylate, are disabled, the hypersensitive response (HR) typical of ETI is abolished

74 ulation of reactive oxygen species (ROS) and hypersensitive response (HR), a rapid programmed death o

75 unction results in reduced growth and yield, hypersensitive response (HR)-like lesions, accumulation

76 cell death of the infected area through the hypersensitive response (HR).

77 taneous lesions that show characteristics of hypersensitive response (HR).

78 at the pathogen infection sites, termed the hypersensitive response (HR).

79 s a rapid and local cell death reaction, the hypersensitive response (HR).

80 SERK3 is required for the effector-triggered hypersensitive response and resistance of tomato against

81 overexpression of SINA3 interferes with the hypersensitive response cell death triggered by multiple

82 PM1) P. syringae strains, conserving typical hypersensitive response features.

83 The lipase/esterase also elicited a hypersensitive response in grapevine.

84 ivation of immune receptors that trigger the hypersensitive response in plants.

85 rapefruit (Citrus x paradisi) suppresses the hypersensitive response induced by Xanthomonas citri ssp

86 Furthermore, the csrA mutant did not induce hypersensitive response on tobacco or cause disease on i

87 t analysis method on a small set of bacteria hypersensitive response phenotypes and identified a sing

88 Defense was mediated by a hypersensitive response that included necrotic lesions,

89 A typical plant defense strategy is the hypersensitive response that results in host cell death

90 A significant hypersensitive response to IFNs was identified in lupus

91 SDE15 also suppresses the hypersensitive response triggered by the Xanthomonas ves

92 terminal Y/FxC motif, that induced a strong hypersensitive response when co-expressed with Pm1a in N

93 response generally encompasses a defensive 'hypersensitive response' (HR) that involves programmed c

94 ylation significantly relieved ER stress and hypersensitive response, and facilitated the folding/ass

95 he major dominant gene for simple resistance hypersensitive response, Cr1 We describe new markers and

96 sis revealed that oxidative phosphorylation, hypersensitive response, DNA repair, stomata closure, bi

97 bacterial mutant strains, and assays for the hypersensitive response, salicylic acid (SA) accumulatio

98 g, response to stimulus, regulation of plant hypersensitive response, sequence-specific DNA binding t

99 nt dead and dying tissues due to an aberrant hypersensitive response.

100 Plant genets varied widely for an induced 'hypersensitive' response in which meristem cells become

101 were aggravated by gene networks involved in hypersensitive responses (PAR1, RIN4, PBS1 etc.).

102 ABA-hypersensitive responses are suppressed in alix-1 plants

103 hormonal determinants, followed by intrinsic hypersensitive responses to counter infestation process

104 ades, as well as prominent genes involved in hypersensitive responses, cell wall fortification, and h

105 ly, anti-LRP5/6 VHHs block the growth of Wnt-hypersensitive Rnf43/Znrf3-mutant intestinal organoids t

106 died two mutations in Nav1.8 associated with hypersensitive sensory neurons: G1662S reported in painf

107 Here we present PlantDHS, a plant DNase I hypersensitive site (DHS) database that integrates histo

108 ce, which leverages cell-type specific DNAse Hypersensitive Site (DHS) information from the NIH Epige

109 d that allows us to generate maps of DNase I-hypersensitive site (DHS) of mouse preimplantation embry

110 uman silencers, we utilized H3K27me3-DNase I hypersensitive site (DHS) peaks with tissue specificity

111 in various cells using Encode Roadmap DNase-hypersensitive site data.

112 nucleosome positioning for MNase-seq, DNase hypersensitive site mapping, site annotation and motif i

113 ing (snDrop-seq) and single-cell transposome hypersensitive site sequencing (scTHS-seq).

114 n sequencing and microarray data and DNase I hypersensitive site sequencing data.

115 In this study, we applied DNase-seq (DNase I hypersensitive site sequencing) to study changes of chro

116 of the KRAS gene contains a GC-rich nuclease hypersensitive site with three potential DNA secondary s

117 The Lockd promoter contains a DNase-hypersensitive site, binds numerous transcription factor

118 d NucDHS, a nucleosome that covers the DNase hypersensitive site, in unintegrated viral DNA.

119 folds from 1.36 to 3.1) as well as the DNase hypersensitive sites (1.58-2.42 fold), H3K4Me1 (1.23-1.4

120 DNase I hypersensitive sites (DHSs) are a hallmark of chromatin

121 DNase I hypersensitive sites (DHSs) are generic markers of regul

122 tory elements; therefore, mapping of DNase I hypersensitive sites (DHSs) enables the detection of act

123 DNase I hypersensitive sites (DHSs) provide important informatio

124 Over half of embryonic DNase I hypersensitive sites (DHSs) were annotated as noncoding,

125 AP-1 which created thousands of new DNase I-hypersensitive sites (DHSs), enabling ETS-1 and RUNX1 re

126 bility and DNA methylation patterns at DNase hypersensitive sites (DHSs).

127 s in promoter regions that intersect DNase I hypersensitive sites (DHSs).

128 ory DNA elements are associated with DNase I hypersensitive sites (DHSs).

129 ry DNA elements can be identified as DNase I hypersensitive sites (DHSs).

130 isruptive mutations within fetal CNS DNase I hypersensitive sites (i.e., putative regulatory regions)

131 DNA fragment including only its four DNase I hypersensitive sites (lacking the large spacer regions)

132 ched with MH-seq reads are referred as MNase hypersensitive sites (MHSs).

133 a shared region of 39 kb that contains DNAse hypersensitive sites active at a restricted time window

134 Global analyses of DNase I-hypersensitive sites and 3D genome architecture, linking

135 e structure within about 1.6 million DNase I-hypersensitive sites and show that the overwhelming majo

136 in wild-type cells, suggesting that the four hypersensitive sites contain most of the CSR-promoting f

137 with, and maintain the intensity of DNase I hypersensitive sites genome wide, in resting but not in

138 s information from H3K27ac signal at DNase I hypersensitive sites identified from published human and

139 his technique enables genome-wide mapping of hypersensitive sites in a wide range of cell populations

140 ypermethylation signature enriched for DNase Hypersensitive Sites in acute myeloid leukemia.

141 erization of the most highly mutated DNase I hypersensitive sites in breast cancer (using in silico a

142 didate noncoding driver mutations in DNase I hypersensitive sites in breast cancer and experimentally

143 enoc7arcinoma cell line (LNCaP) or by DNaseI hypersensitive sites in cancer cell lines.

144 e observed significant enrichment in DNase I hypersensitive sites in fetal heart and lung.

145 we profile parental allele-specific DNase I hypersensitive sites in mouse zygotes and morula embryos

146 ld tend to contain more micrococcal nuclease hypersensitive sites in their promoters, a proxy for ope

147 me as accessible, with the majority of MNase hypersensitive sites marking proximal promoters, but als

148 In the vicinity of active genes and DNase I hypersensitive sites nucleosomes are organized into peri

149 s strong enhancer regions containing DNase I hypersensitive sites overlapping the rs874040 linkage di

150 precipitation sequencing (ChIP-seq), DNase I hypersensitive sites sequencing (DNase-seq), and whole-g

151 We present a transposome hypersensitive sites sequencing assay for highly sensiti

152 Analysis of DNase I hypersensitive sites sequencing data revealed an open ch

153 sequence alone, with the highest accuracy at hypersensitive sites shared across cell types.

154 single guide RNA libraries to target DNase I hypersensitive sites surrounding a gene of interest, we

155 genes with paternal allele-specific DNase I hypersensitive sites that are devoid of DNA methylation

156 We identify ten DNase I hypersensitive sites that are significantly mutated in b

157 By analyzing the DNase I hypersensitive sites under 349 experimental conditions,

158 Enrichment of SNPs associated with DNase I-hypersensitive sites was also found in many tissue types

159 However, correlations with DNase I hypersensitive sites were different for all vectors, ind

160 We found that 40% DHSs (DNaseI hypersensitive sites) were diminished under darkness.

161 for 17 TFs, 3 histone modifications, DNase I hypersensitive sites, and high-resolution promoter-enhan

162 d in chicken lung overlapped half of DNase-I hypersensitive sites, coincided with active histone modi

163 site methylation, CGIs, co-localized DNase I hypersensitive sites, transcription factor binding sites

164 sitions of 4 histone modifications and DNase hypersensitive sites, Wilson et al reveal many more of t

165 ma cells by inducing and maintaining DNase I hypersensitive sites.

166 and termination sites, enhancers and DNase I hypersensitive sites.

167 r chromatin binding and induction of DNase I hypersensitive sites.

168 n transcription-factor-binding-sites and DNA-hypersensitive-sites.

169 events per bombarded sample in spectinomycin-hypersensitive Slavice and Columbia acc2 knockout backgr

170 Cells lacking USP45 are hypersensitive specifically to UV irradiation and DNA in

171 sensory signals from visceral organs produce hypersensitive spots on the skin (neurogenic spots), cau

172 ferred multidrug resistance on KAM32, a drug hypersensitive strain of Escherichia coli.

173 In our study, 21 patients with hypersensitive teeth were tested using nonpainful and pa

174 aim was to determine whether these putative hypersensitive terminals could constitute a significant

175 ordered cortical microtubule arrays that are hypersensitive to a microtubule-depolymerizing drug.

176 t in the rgs1 mutant background makes plants hypersensitive to a subset of abscisic acid-mediated res

177 The era1 mutant is hypersensitive to ABA during seed germination and shows

178 tive in ABA-induced stomatal closure and are hypersensitive to ABA during seed germination.

179 In addition, the rgga knockout mutant was hypersensitive to ABA in root growth and survival tests

180 that fip1-2 is more tolerant to salt but is hypersensitive to ABA or Cd.

181 cell outward-rectifying K(+) -channel) were hypersensitive to abscisic acid, but insensitive to drou

182 hat the two PLCgamma2 mutants are strikingly hypersensitive to activation by Rac2 such that even wild

183 The authors found that these lesions are hypersensitive to Akt inhibitors which bind to the ATP b

184 are defective in ASCC foci formation and are hypersensitive to alkylating agents.

185 lls to IL-15, rendering naive CD8(+) T cells hypersensitive to antigen stimulation characterized by e

186 Mutants of gamma-ECS and PCS1 were hypersensitive to As and had higher root-to-shoot As tra

187 A rice mutant hypersensitive to As(V), but not to As(III), was isolate

188 identify genes whose loss makes tumor cells hypersensitive to ATR inhibition, we performed CRISPR/Ca

189 lants carrying the BRI1(Y898F) mutation were hypersensitive to BRs.

190 lines exhibit developmental defects and are hypersensitive to camptothecin (CPT) and cis-platin.

191 insensitive to salt and osmotic stresses and hypersensitive to cell wall stressors.

192 Patients with persistent PTH are hypersensitive to CGRP, which underscores its pathophysi

193 C) cell lines and report that nearly 20% are hypersensitive to CHKi monotherapy.

194 A (Csa(-/-)) and group B (Csb(-/-)) mice are hypersensitive to cisplatin, in contrast to global genom

195 R) branch of nucleotide excision repair, are hypersensitive to cisplatin-induced hearing loss and sen

196 -R273H-expressing cancer cells renders cells hypersensitive to cisplatin.

197 r the polarity gene scribble (scrib(KD)) are hypersensitive to compaction, that interaction with wild

198 Single ump1 mutants were hypersensitive to conditions that induce proteotoxic, sa

199 ts from Nuclear Magnetic Resonance (NMR) are hypersensitive to conformational changes and ensembles i

200 c overexpression of ARR10, Arabidopsis lines hypersensitive to cytokinin were generated and used to c

201 oxically, this inhibition renders stem cells hypersensitive to cytotoxic stress, as tumour regenerati

202 cyp86a2 and lacs2 mutants were hypersensitive to diphenyleneiodonium but could be rever

203 -deficient cells, devoid of most miRNAs, are hypersensitive to DISE, suggesting cellular miRNAs prote

204 sav6 mutants are hypersensitive to DNA damage induced by ultraviolet (UV)

205 maH2AX foci, we found that fbl17 mutants are hypersensitive to DNA double-strand break-induced genoto

206 omologous recombination and consequently are hypersensitive to DNA-damaging agents, including cisplat

207 nogenic and biallelic mutations in SPRTN are hypersensitive to DPC-inducing agents due to a defect in

208 ants of AtPTPN were hyposensitive to ABA but hypersensitive to drought stresses, whereas plants with

209 inally, METTL13 depletion renders PDX tumors hypersensitive to drugs that target growth-signaling pat

210 der homeostatic conditions and are similarly hypersensitive to DSS-induced colitis.

211 ture, the neo1-1(ts) mutant became extremely hypersensitive to duramycin, although the sensitivity to

212 tions in iron homeostasis-related genes were hypersensitive to E9591 and LMT.

213 also observed that KLF3-deficient mice were hypersensitive to endotoxin and exhibited elevated level

214 The silenced lines were hypersensitive to exogenous auxin, while levels of endog

215 The yuc1, yuc4, and ahp6 mutants are hypersensitive to exogenous cytokinin and 1-napthylphtha

216 ally normal under homeostatic conditions but hypersensitive to experimental colitis induced by dextra

217 mtp8 mutants were only hypersensitive to Fe deficiency when Mn was present in t

218 2-deficient ovarian cancer cell line that is hypersensitive to floxuridine, we show that GSK-3 phosph

219 fective mutants of Arabidopsis thaliana were hypersensitive to freezing before and after cold acclima

220 Moreover, CTCF-deficient cells are hypersensitive to genotoxic stress such as ionizing radi

221 Sam68 deleted cells are hypersensitive to genotoxicity caused by DNA damaging ag

222 ted that Sam68-deleted cells and animals are hypersensitive to genotoxicity caused by DNA-damaging ag

223 CDKAL1 mutant insulin+ cells were also hypersensitive to glucolipotoxicity.

224 ll populations in the BM and spleen that are hypersensitive to granulocyte macrophage-CSF due to hype

225 ic uptake regulator (Fur) renders mstA cells hypersensitive to H2O2 Conversely, induction of chromoso

226 th Ala substitutions of Cys276 or Cys303 are hypersensitive to high-light conditions during greening.

227 utant in heme biosynthesis, hem13-1, that is hypersensitive to HU.

228 uperoxide dismutase 2 and catalase, and were hypersensitive to hydrogen peroxide.

229 mir-274 mutant larvae are hypersensitive to hypoxia, which is suppressed by miR-27

230 Chicken cells lacking Rnf4 are hypersensitive to hyroxyurea, DNA alkylating drugs and D

231 L-15, and deletion of Cish rendered NK cells hypersensitive to IL-15, as evidenced by enhanced prolif

232 eukemic Tp53-/-Lnk-/- pro-B progenitors were hypersensitive to IL-7, exhibited marked self-renewal in

233 gation of Zeo1.b, and that Zeo1.b itself was hypersensitive to inhibition by MeJA but less responsive

234 s are viable under normal conditions but are hypersensitive to inhibition of purine nucleotide synthe

235 uding Tip60, BRG1 and NBS1, and renders mice hypersensitive to ionizing radiation (IR).

236 reases genomic instability, and renders mice hypersensitive to IR.

237 ction of reactive oxygen species (ROS), were hypersensitive to iron and pro-oxidants, and accumulated

238 presynaptic excitatory neurotransmission is hypersensitive to isoflurane in Ndufs4(KO) mice due to t

239 d JAK2 protein levels and signaling and were hypersensitive to JAKi.

240 lmonella strains with mutations of phoPQ are hypersensitive to killing by RNS generated in vitro.

241 Here, we identified and characterized the HYPERSENSITIVE TO LATRUNCULIN B1 (HLB1) protein isolated

242 l gene, Retinoic acid induced-1 (Rai1), were hypersensitive to light such that light eliminated alert

243 Parasites with low levels of EXP1 became hypersensitive to low nutrient conditions, indicating th

244 A Us3-deficient virus was hypersensitive to low-energy-induced stress as infected

245 3 kinase binding and Akt activation, and are hypersensitive to MEK inhibition.

246 e demonstrate that SIRT1-deficient mESCs are hypersensitive to methionine restriction/depletion-induc

247 The Arabidopsis fen1-1 mutant is hypersensitive to methyl methanesulfonate and shows redu

248 Their lymphoblasts were hypersensitive to MMC and MMC-induced monoubiquitination

249 nuclease that specifically repairs ICLs, are hypersensitive to most ABQ prodrugs, a phenotype exacerb

250 ry cell lines were structurally immature and hypersensitive to neutralization by human antibodies com

251 bellar Purkinje cells (PCs) are particularly hypersensitive to NPC1 deficiency and degenerate earlier

252 utants exhibited altered morphology and were hypersensitive to nutrient-limiting conditions.

253 XP-C patients are specifically hypersensitive to ocular damage, and XP-F and XP-G patie

254 In addition, the bon mutants are hypersensitive to osmotic stress in growth inhibition.

255 dopsis thaliana amy3 bam1 double mutants are hypersensitive to osmotic stress, showing impaired root

256 Cells lacking E2F1 are hypersensitive to oxidative stress due to the defects in

257 displayed reduced catalase activity and were hypersensitive to oxidative stress.

258 /-)RNF168(-/-) cells lack RAD51 foci and are hypersensitive to PARP inhibitor, whereas forced targeti

259 homologous recombination (HR) deficient, are hypersensitive to PARPi through the mechanism of synthet

260 damage, with cells from these patients being hypersensitive to particular genotoxic drugs, indicating

261 we identified an Arabidopsis mutant, hps10 (hypersensitive to Pi starvation 10), which is morphologi

262 calize in neurons and that Mrap2 KO mice are hypersensitive to PKR1 stimulation.

263 HR-deficient cancers are hypersensitive to Poly (ADP ribose)-polymerase (PARP) in

264 ologous recombination (HR) and renders cells hypersensitive to poly (ADP-ribose) polymerase (PARP) in

265 the suggestion that in intact cells they are hypersensitive to Rac family small GTPases or to the ups

266 ce have normal intestinal morphology but are hypersensitive to radiation injury in the intestine comp

267 We found that mutants in PHO84 are hypersensitive to rapamycin and in response to phosphate

268 in replication, FBXO31-knockdown cells were hypersensitive to replication stress-inducing agents and

269 Consequently, the first S phase cells are hypersensitive to replication stress.

270 1(D325A/D325A) and Ripk1(D325A/+) cells were hypersensitive to RIPK3-dependent TNF-induced apoptosis

271 Although telomeres are hypersensitive to ROS-mediated 8-oxoguanine (8-oxoG) for

272 te that expressing a variant of NPR4 that is hypersensitive to SA could enhance SA-mediated basal imm

273 In addition, attrappc11/rog2 mutants are hypersensitive to salinity, indicating an undescribed ro

274 We isolated a salt-sensitive mutant named hypersensitive to salt stress (hss) from an ethyl methan

275 An Arabidopsis thaliana tno1 mutant is hypersensitive to salt stress and partially mislocalizes

276 The Y1F strain was also hypersensitive to several different cellular stresses th

277 and SCAR20 disease patient-derived cells are hypersensitive to SFA-mediated lipotoxic cell death.

278 ce with reduced DAT expression (DAT-HT) were hypersensitive to short active (SA; 19:5 L:D) photoperio

279 [PSI+] variants hypersensitive to Ssb1/2p have distinguishable biologica

280 The PrimPol-knockdown strain was hypersensitive to tenofovir treatment, indicating that P

281 ulation of PV5 expression renders P. berghei hypersensitive to the antimalarial drugs artesunate, chl

282 ive to ABA in dose-response assays, but also hypersensitive to the GA synthesis inhibitor PAC.

283 DCDC-deficient mutants are hypersensitive to the genotoxic agent methyl methanesulf

284 ed that BRCA1-deficient tumors are initially hypersensitive to the inhibition of topoisomerase I/II a

285 ion of this regulon and notably are strongly hypersensitive to the proteasome inhibitors MG132 and bo

286 Cells depleted of Ube2w alone are not hypersensitive to the same DNA damaging agents.

287 Deletion mutants of ULS1 are hypersensitive to the Top2 poison acriflavine (ACF), act

288 NK-A20(-/-) cells were hypersensitive to TNF-induced cell death and could be re

289 Moreover, C. elegans gcna-1 mutants are hypersensitive to TOP2 poison.

290 Finally, UBR5 and BMI1 KO cells are hypersensitive to UV, which supports the notion that fau

291 f mitochondrial complex I and are strikingly hypersensitive to VAs yet resistant to the intravenous a

292 d Haspin inhibitor-treated HCT116 cells were hypersensitive to VX-680.

293 on of Blimp1 and that Fancc(-/-) B cells are hypersensitive to Wnt activation during ASC differentiat

294 inguishable biological properties from those hypersensitive to Zuo1p or Ssz1p.

295 s (PECTIN LYASE-LIKE, ASPARTYL PROTEASE, DWD-HYPERSENSITIVE-TO-ABA3, and YELLOW STRIPE-LIKE5) were fu

296 sequence, loss of diphthamide rendered cells hypersensitive toward TNF-mediated apoptosis.

297 Applications of this hypersensitive transport for optical and microwave limit

298 ation-inducing strategy for treatment of Wnt-hypersensitive tumors.

299 To investigate how an IFN-hypersensitive virus can evolve to overcome IFN-beta-med

300 ong-Evans rats separated from their mothers (hypersensitive) with non-handled (normally sensitive) ra