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1 h could be used to reduce both hypotonia and hypertonia.
2 for RepReOx improved prediction of postnatal hypertonia.
3 edicted which fetuses will develop postnatal hypertonia.
4 genation is a critical trigger for postnatal hypertonia.
5 etus to H-I is crucial to the development of hypertonia.
6  consensus definitions of different types of hypertonia.
7 ized by increased reflexes leading to muscle hypertonia.
8  wide-based gait or an inability to walk and hypertonia.
9 e phase and chronically persists with muscle hypertonia.
10 mities including congenital contractures and hypertonia.
11                 In hippocampal tissue slices hypertonia (+25, +50 and +100 mmol/l fructose) reversibl
12  with verbal ability (6/9), hypotonia (7/9), hypertonia (6/9), and involuntary movements (3/9).
13  with gait impairment (94%), limb spasticity/hypertonia (76%), oculomotor (71%) and behavioural abnor
14 iduals with mutations in SLC6A5 present with hypertonia, an exaggerated startle response to tactile o
15 ancy, impaired speech, stereotypic behavior, hypertonia and a specific facial gestalt.
16 xia-ischemia at preterm gestation results in hypertonia and abnormalities in motor control.
17   Hyperekplexia is characterised by neonatal hypertonia and an exaggerated startle reflex in response
18 d by tactile or auditory stimuli, leading to hypertonia and apnea episodes.
19 ed by tactile or auditory stimuli, producing hypertonia and apnea episodes.
20 ised by exaggerated startle reflexes, muscle hypertonia and apnoea.
21  reflex and non-reflex components of spastic hypertonia and intramuscular blood perfusion among indiv
22 there is no animal model that reproduces the hypertonia and motor disturbances of this disorder.
23 n significance was identified in a male with hypertonia and spasticity without syndromic features.
24 ongenital contractures; and (5) marked early hypertonia and symptoms of extrapyramidal involvement.
25 t mechanics, known clinically as spasticity, hypertonia, and paresis.
26 intractable seizures, acquired microcephaly, hypertonia, and psychomotor retardation and died at age
27 ewborns with the hallmark characteristics of hypertonia, apnea, and noise or touch-induced nonepilept
28 in the hypertonic (hyrt) gene exhibit severe hypertonia as their primary symptom.
29 model for studying the molecular etiology of hypertonia associated with human neurological diseases.
30 ave important implications for understanding hypertonia associated with neurological disorders.
31                                              Hypertonia depressed voltage-gated sodium, potassium and
32 BV had an acute effect on modulating spastic hypertonia dominated by hyperreflexia in people with chr
33 settings to facilitate precise evaluation of hypertonia, essential for tailoring individual treatment
34                                          The hypertonia group had significantly less of a nadir in ap
35 uscular hypotonia was reported in 13/25, and hypertonia in 10/25.
36 identified as the genetic defect that causes hypertonia in mice.
37 entral WM tracts in newborn rabbit kits with hypertonia in our model of cerebral palsy.
38  levodopa had significantly more dyskinesia, hypertonia, infection, headache, and nausea than those r
39 f in utero placental insufficiency, in which hypertonia is accompanied by marked abnormalities in mot
40 aluations of neuromuscular disorders such as hypertonia mostly rely on perception-based scales, the i
41                    We studied the effects of hypertonia on voltage-gated currents of freshly isolated
42 rogressive congenital motor dysfunction with hypertonia or dyskinesia.
43 cluding facial asymmetry, marfanoid habitus, hypertonia, osteoporosis and unsteady gait.
44 esponses, alongside episodic and generalized hypertonia, that presents within the first month of life
45 alformation, axial hypotonia with peripheral hypertonia, visual problems, and conductive hearing impa
46                                              Hypertonia was associated with microstructural WM injury
47 r deficits in newborn kits, including muscle hypertonia, were assessed by neurobehavioral testing.
48                                              Hypertonia, which is characterized by stiff gait, abnorm
49                                              Hypertonia, which results from motor pathway defects in
50  brachycephaly, optic atrophy, seizures, and hypertonia with hyperreflexia.