ライフサイエンスコーパス: hypertriglyceridemic

1 LpL knockout mice; however, these mice were hypertriglyceridemic.

2 , hyperleptinemic, hypercholesterolemic, and hypertriglyceridemic.

3 The morbidity rate of hypertriglyceridemic acute pancreatitis (HTG-AP) increas

4 We report a patient with DKA-induced hypertriglyceridemic acute pancreatitis treated successf

5 ue against diabetic progression via its anti-hypertriglyceridemic and anti-hyperglycemic effects.

6 cterized by hyperglycemic, hyperinsulinemic, hypertriglyceridemic, and enhanced noradrenergic indexes

7 Fasted HcB-19 mice are hypoglycemic, hypertriglyceridemic, and have higher than normal levels

8 ess mice became obese, hypercholesterolemic, hypertriglyceridemic, and hyperinsulinemic compared with

9 s after preincubation with either fasting or hypertriglyceridemic blood.

10 In addition, hypertriglyceridemic but not fasting blood inhibited the

11 injury in mice on a low-phosphate diet under hypertriglyceridemic conditions.

12 In addition, SCD1 deficiency prevented the hypertriglyceridemic effect and reduced hepatic triglyce

13 Moreover, hypertriglyceridemic events and abnormal international n

14 creased in both the hypercholesterolemic and hypertriglyceridemic groups (298 +/- 29 and 342 +/- 31 n

15 Mean values for PD and CAL in the two hypertriglyceridemic groups were significantly higher th

16 stance in a non-obese prediabetic hereditary hypertriglyceridemic (HHTg) rats.

17 The effect of normo (NTG)- and hypertriglyceridemic (HTG)-VLDL on cultured human umbili

18 [NL]) and those with moderately elevated TG (hypertriglyceridemic [HTG]) were studied on both a contr

19 ism by which statins lower VLDL (and LDL) in hypertriglyceridemic individuals is by stimulation of li

20 litate the development of atherosclerosis in hypertriglyceridemic individuals, we used in situ hybrid

21 Hypertriglyceridemic men aged 39-66 y (n = 34) participa

22 We found that hypertriglyceridemic mice exhibit mild pancreatic injury

23 Hypertriglyceridemic pancreatitis (HTGP) is featured by

24 gnosing and treating patients with suspected hypertriglyceridemic pancreatitis; or diagnosing hypertr

25 n of lipoproteins is increased when familial hypertriglyceridemic patients are treated with drugs tha

26 n of VLDL to an apoC-III-dominated system in hypertriglyceridemic patients characterized by reduced c

27 LDL from hypertriglyceridemic patients or prepared in vitro by th

28 01 on Cardiovascular Health and Mortality in Hypertriglyceridemic Patients With Cardiovascular Diseas

29 ring drugs, and lower triglyceride levels in hypertriglyceridemic patients.

30 altered, LDL levels may rise, especially in hypertriglyceridemic patients.

31 When LpL was added to hypertriglyceridemic plasma, dextran accumulation within

32 ce received an adenovirus encoding Pltp, the hypertriglyceridemic response to T0901317 was partially

33 Two hypertriglyceridemic siblings were homozygous for the sa

34 Results from 2 Phase 1 studies in hypertriglyceridemic subjects are reported here.

35 Lipid changes in hypertriglyceridemic subjects were similar to those obse

36 rated clinical and TNF-alpha response in the hypertriglyceridemic subjects.

37 supports further clinical investigations in hypertriglyceridemic subjects.

38 Fasting and postprandial (hypertriglyceridemic) subjects were injected with endoto

39 asted VLDLr knockout (VLDLr0) mice were more hypertriglyceridemic than controls (2-fold greater trigl

40 istics seem to be common to postprandial and hypertriglyceridemic very low density lipoproteins as we

41 vivo to examine the capacity of fasting vs. hypertriglyceridemic whole blood to attenuate the effect