1 ated the relation between VAS and underlying hypervitaminosis A assessed by retinol isotope dilution

2                           The odds of having hypervitaminosis A at baseline were higher in children c

3                     Vitamin A deficiency and hypervitaminosis A cause disruption of normal cellular h

4     At baseline, 63.6% of these children had hypervitaminosis A defined as total liver retinol reserv

5                               The finding of hypervitaminosis A in Zambian children was supported by

6 population and could avoid the potential for hypervitaminosis A that was observed with the use of pre

7 ders such as obesity and hypoparathyroidism, hypervitaminosis A, tetracycline use and thyroid replace

8 using negative consequences in children with hypervitaminosis A.

9 retinyl ester concentrations are elevated in hypervitaminosis A.

10 osis, closely resemble the manifestations of hypervitaminosis A.

11 ed tumor growth and that induced symptoms of hypervitaminosis A.

12  of total serum vitamin A indicate potential hypervitaminosis, but this cutoff was derived from small

13 uption of the Klotho gene in mice results in hypervitaminosis D and a syndrome resembling accelerated

14 n of Fgf-23 in mice (Fgf-23(-/-)) results in hypervitaminosis D, abnormal mineral metabolism, and red