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1 ation development: trophoblast, epiblast and hypoblast.
2  does not require paracrine support from the hypoblast.
3  formation but suppresses development of the hypoblast.
4 th a riboprobe for Crescent, a marker of the hypoblast.
5  only parietal endoderm, a derivative of the hypoblast.
6 the segmental plate, epiblast, mesoderm, and hypoblast.
7 yos and stem cell models of the epiblast and hypoblast.
8 ntiate into extraembryonic trophectoderm and hypoblast.
9  order of genes associated with emergence of hypoblast.
10 wth factor from the extraembryonic endoderm (hypoblast, a cell layer unique to amniotes) directs the
11                To reevaluate the role of the hypoblast/ADE (lower layer) in patterning the chick ecto
12 ith or without tissue replacement), that the hypoblast/ADE (lower layer) is required and sufficient f
13                         The spreading of the hypoblast also directs cell movements in the adjacent ep
14 e failed to demonstrate a clear role for the hypoblast and anterior definitive endoderm (ADE) in patt
15 in which they are expressed, the pregastrula hypoblast and anterior lateral endoderm, respectively.
16                                              Hypoblast and endoblast (a second lower layer formed und
17 esizing retinoids are expressed in the avian hypoblast and in tissues directly involved in head patte
18 e cardiac-inducing capacities of pregastrula hypoblast and stage 5 anterior lateral endoderm.
19        Finally, the relationship between the hypoblast and the definitive endoderm was defined by fol
20  embryo, has focused on Koller's sickle, the hypoblast and the posterior marginal zone.
21  cells bearing some resemblance to epiblast, hypoblast and trophectoderm lineages.
22  extra-embryonic membranes - trophoblast and hypoblast - and the formation of a flat embryonic disc,
23 ose induced by an extraembryonic tissue, the hypoblast, and are normally expressed in the pregastrula
24 alyses confirm segregation of trophectoderm, hypoblast, and epiblast with high fidelity to the human
25 d inhibition by Cerberus from the underlying hypoblast, and finally a late inhibition from Lefty emit
26  Epiblasts were separated from the mesoderm, hypoblast, and primitive streak, dissociated to produce
27 yngeal endodermal precursors of the anterior hypoblast, anticipating both temporally and spatially th
28 s, we show that the node, head mesoderm, and hypoblast are interchangeable to begin any of these indu
29 ocysts, implying segregation of epiblast and hypoblast, as in rodent embryos.
30 by SOX17, FOXA2 and GATA4 in sequence as the hypoblast becomes committed.
31                           We report that the hypoblast can induce a set of very early markers that ar
32 non-genetic approaches to generate authentic hypoblast cells (naive hPSC-derived hypoblast-like cells
33 of asymmetrically positioned extra-embryonic hypoblast cells expressing inhibitors of BMP, NODAL and
34  of embryo-fated epiblast and yolk-sac-fated hypoblast cells from uncommitted inner cell mass (ICM) c
35                            Lateral zebrafish hypoblast cells initiate dorsal convergence near mid-gas
36 calation at its interface with the displaced hypoblast cells.
37 udies suggest a signaling cascade in which a hypoblast-derived activin/TGFbeta signal is required pri
38 ndoderm/Node fate hinges on balanced WNT and hypoblast-derived NODAL, which is extinguished upon endo
39                Here, we hypothesise that the hypoblast-derived primary yolk sac serves as a source fo
40 s regulating cardiac myogenesis in avians, a hypoblast-derived signal acting on epiblast and mediated
41              These findings suggest that the hypoblast-derived signal likely acts upstream of propose
42  is underway by stage 3, indicating that the hypoblast-derived signal occurs shortly before specifica
43                       A goosecoid-expressing hypoblast did not form under the induced streak, indicat
44 lium, or in the presence of the mesoderm and hypoblast, did not undergo myogenesis.
45                         In the human embryo, hypoblast differentiation has not previously been charac
46 t human naive pluripotent stem cell (PSC) to hypoblast differentiation proceeds via reversion to a tr
47        Thus, stem cell models that reproduce hypoblast differentiation provide valuable alternatives.
48 enes, we present a basic blueprint for human hypoblast differentiation, lending support to the propos
49                                 However, the hypoblast does induce transient expression of the early
50                             We show that the hypoblast does not fit the criteria for a head organizer
51 sen's node as a cell monolayer that replaces hypoblast during chick gastrulation.
52 toderm before separation of the epiblast and hypoblast, either via differentiation of the hypoblast f
53 a transitional ICM-like state from which the hypoblast emerges in concordance with the trajectory in
54 cific transcription factors GATA6 and SOX17 (hypoblast factors) or GATA3 and TFAP2C (encoding AP2gamm
55 nhibition in the early blastocyst suppresses hypoblast formation.
56 se observations indicate that segregation of hypoblast from the bipotent ICM is dependent on FGF/Erk
57 hypoblast, either via differentiation of the hypoblast from the established epiblast, or production o
58                                  The primate hypoblast gives rise to a transient primary yolk sac, wh
59 ising influences of the organiser, the chick hypoblast has been suggested to be the homologue of the
60 rdiac mesodermal precursors in the zebrafish hypoblast immediately following gastrulation.
61 with enriched perforations near the anterior hypoblast in embryos, suggesting a conserved mechanism f
62 rm in early blastocysts, and of epiblast and hypoblast in late blastocysts.
63 n is strongly expressed in the yolk cell and hypoblast in the early gastrula, just preceding the appe
64 of Hoxa-1 message during gastrulation in the hypoblast in the head region.
65                    However, the formation of hypoblast in the human is apparently not dependent upon
66 l differences in epiblast responsiveness and hypoblast inductiveness restrict appearance of cardiac m
67                                          The hypoblast is an essential extraembryonic tissue set asid
68                 Here we argue that the chick hypoblast is equivalent to the mouse VE, based on fate,
69                        This dual role of the hypoblast is more consistent with the Nieuwkoop model th
70 al plate or a forebrain, suggesting that the hypoblast is not a head organizer and that other signals
71                   In vitro, the induction of hypoblast is synergistically enhanced by limiting trophe
72 ss, and subsequently identifying presumptive hypoblast, is PDGFRA, followed by SOX17, FOXA2 and GATA4
73 express ndr1, while ndr2 RNA is found in the hypoblast layer of the shield and later in notochord, pr
74      These cells involute to join the deeper hypoblast layer where they adopt a migratory, mesenchyma
75 uthentic hypoblast cells (naive hPSC-derived hypoblast-like cells (nHyCs))-known to give rise to one
76 e for SOX17 in the specification of anterior hypoblast-like cells(2).
77      Modulation of the subpopulations in the hypoblast-like compartment demonstrates that extraembryo
78 ed by genetically manipulating the DKK1/OTX2 hypoblast-like domain.
79  embryonic epiblast-like and extra-embryonic hypoblast-like lineages, establishes key signalling hubs
80 iation of cells toward the trophectoderm and hypoblast lineages compared with that for control embryo
81 ent, whereby segregation of the epiblast and hypoblast lineages occurs during maturation of the mamma
82                      After gastrulation, the hypoblast might protect prospective forebrain cells from
83 ating ovine embryos: trophoblast maturation, hypoblast migration, embryonic disc formation, disappear
84  exogenous FGF stimulation leads to expanded hypoblast molecular marker expression, at the expense of
85 s) that resemble the post-implantation human hypoblast molecularly and functionally.
86 cells of the gastrula that involute into the hypoblast, motility appears wild-type.
87                         However, neither the hypoblast nor any of these factors or combinations there
88 the absence of axial-expressing cells in the hypoblast of oep mutants.
89                           Our data support a hypoblast origin of the extra-embryonic mesoderm and pri
90 ies as well as the generation of an anterior hypoblast pole and posterior domain.
91                                          The hypoblast possessed broad capacity to induce heart muscl
92                             We find anterior hypoblast specification is NODAL dependent, as in the mo
93 rowth factor (FGF) signaling is critical for hypoblast specification.
94              Here we explore the role of the hypoblast (the chick equivalent of the AVE) in the early
95 oth embryonic (epiblast) and extraembryonic (hypoblast) tissues.
96                         One such tissue, the hypoblast (visceral endoderm in mouse), acquired a role
97 eriments demonstrated that a signal from the hypoblast was required to induce cardiac myogenesis in t
98 oderm, which generates the placenta; and the hypoblast, which forms both the anterior signalling cent
99 er lineages: the trophectoderm, epiblast and hypoblast, which together form the blastocyst.
100 ell stage blastomere to the epiblast (body), hypoblast (yolk sac), and trophectoderm (placenta).