ライフサイエンスコーパス: hypocotyl

1 pylar endosperm) and RAD (radicle plus lower hypocotyl).

2 ross the epidermis below the meristem in the hypocotyl.

3 gerated apical hook, and a thickening of the hypocotyl.

4 along the length of the Arabidopsis thaliana hypocotyl.

5 ed the site of light perception to the upper hypocotyl.

6 tic lesions were observed at the base of the hypocotyl.

7 ous and occurs more prominently in the basal hypocotyl.

8 m, phloem, and primary xylem in the stem and hypocotyl.

9 l controls of cell elongation in Arabidopsis hypocotyl.

10 steeper auxin signaling gradient across the hypocotyl.

11 tion occurs, but it differs between stem and hypocotyl.

12 stinct, permissive temperature sensor in the hypocotyl.

13 cell elongation in different regions of the hypocotyl.

14 h in rapidly elongating roots and dark-grown hypocotyls.

15 genes are defective in stomata formation in hypocotyls.

16 tability are aberrant in etiolated xxt1 xxt2 hypocotyls.

17 that promotes cell elongation in Arabidopsis hypocotyls.

18 veral candidate regulators in the elongating hypocotyls.

19 growth, and, in etiolated seedlings, shorter hypocotyls.

20 issect their trafficking routes in etiolated hypocotyls.

21 growth of Arabidopsis (Arabidopsis thaliana) hypocotyls.

22 resulting in low activity of PIF3 and short hypocotyls.

23 rlapping patterns of expression in etiolated hypocotyls.

24 transcript and late flowering and elongated hypocotyls.

25 psi1-1 seedlings have shorter roots and hypocotyls.

26 in the cotyledon tissue but not meristems or hypocotyls.

27 extent of axial cell expansion in dark-grown hypocotyls.

28 PHOTOPERIODIC CONTROL OF HYPOCOTYL 1 (PCH1) and PCH1-LIKE (PCHL) were shown to di

29 esent evidence that PHOTOPERIODIC CONTROL OF HYPOCOTYL 1 (PCH1) functions both as an essential struct

30 nd that an Arabidopsis LIGHT-DEPENDENT SHORT HYPOCOTYLS 1 and Oryza G1 (ALOG) family protein, named M

31 level of a poplar clock gene, LATE ELONGATED HYPOCOTYL 2 (LHY2), which controls FT2 expression.

32 ld), roots (4.3-fold), bolts (4.5-fold), and hypocotyls (2-fold).

33 ith the signalling component Non-Phototropic Hypocotyl 3 (NPH3).

34 otropic signalling component Non-Phototropic Hypocotyl 3 (NPH3).

35 The impact ethylene has on hypocotyl 3D cell anisotropy identified the preferential

36 n of the organ boundary gene LIGHT-SENSITIVE HYPOCOTYL 4 restored RZ function and stem growth in the

37 f5 (pifq) mutants; the dynamics of ELONGATED HYPOCOTYL 5 (HY5) and LONG HYPOCOTYL IN FAR-RED (HFR1) p

38 ion of the transcription activator ELONGATED HYPOCOTYL 5 (HY5) that is associated with chromatins of

39 ELONGATED HYPOCOTYL 5 (HY5), a basic domain/leucine zipper (bZIP)

40 nsitivity, albeit independently of elongated hypocotyl 5 (HY5).

41 (IAA) transport and its accumulation in the hypocotyl above the point of excision where adventitious

42 racterized by reduced growth of the root and hypocotyl, an exaggerated apical hook, and a thickening

43 opposite effects on phyB's functions in the hypocotyl and cotyledon despite inducing similar photobo

44 yB-FP) in the epidermal cells of Arabidopsis hypocotyl and cotyledon.

45 clarify cell-specific auxin function in the hypocotyl and highlight the complexity of cell type inte

46 rnode elongation, a mutant with an elongated hypocotyl and internodes but wild-type petioles was iden

47 hows that the mechanisms underlying rhythmic hypocotyl and leaf growth differ.

48 rphogenic development pattern, showing short hypocotyl and long roots.

49 med thermomorphogenesis, is characterized by hypocotyl and petiole elongation and hyponastic growth a

50 ght leads to enhanced auxin signaling in the hypocotyl and, upon phototropic stimulation, a steeper a

51 duced genome-wide gene expression changes in hypocotyls and cotyledons separately.

52 ABP1 on transcriptomic changes in dark-grown hypocotyls and investigated the consequences of gene exp

53 PGX1(AT) plants, PGX2(AT) plants have longer hypocotyls and larger rosette leaves, but they also uniq

54 angles and gravitropic behavior of seedling hypocotyls and primary roots.

55 detected in the epidermal layers of leaves, hypocotyls and roots; in the root, it was predominantly

56 play skotomorphogenic development, with long hypocotyls and short roots.

57 ar interactions in the plant embryonic stem (hypocotyl), and analyzing these using quantitative netwo

58 arly flowering and increased length of root, hypocotyl, and petiole when compared with Col-0 and jaz4

59 ter primary roots, longer root hairs, longer hypocotyls, and altered lateral root formation.

60 vascular cells of cotyledons, leaves, roots, hypocotyls, and anthers.

61 tosis to endoreplication was lower in abcb19 hypocotyls, and fluorescence microscopy showed the CCS52

62 Increased elongation growth of roots, hypocotyls, and petioles in warm temperatures are hallma

63 al targeting approach therefore excludes the hypocotyl apex as the site for light perception for phot

64 -GFP (P1-GFP) expression was targeted to the hypocotyl apex of the phot-deficient mutant using the pr

65 te in more detail the functional role of the hypocotyl apex, and the regions surrounding it, in estab

66 n of CUC3::P1-GFP was clearly visible at the hypocotyl apex, with weaker expression in the cotyledons

67 ocotyl is a prerequisite for phot1-dependent hypocotyl bending.

68 ds expression of Bn-FAE1.1 into the axis and hypocotyl but also acts negatively to repress expression

69 omain B-class GATA genes, most strikingly in hypocotyls but also in cotyledons.

70 t light leads to the transient elongation of hypocotyls by stabilizing the ACS5 protein during the da

71 to far-red shade by the cotyledons triggers hypocotyl cell elongation and auxin target gene expressi

72 Quantitative analysis of hypocotyl cell growth in the nek6-1 mutant demonstrated

73 ng growth by modulating DNA accessibility of hypocotyl cell size regulatory genes.

74 The two proteins were identified in hypocotyl cell wall extracts by proteomics.

75 fying the major array pattern classes in the hypocotyl cell.

76 his manipulation, a large population of host hypocotyl cells are delayed in cell cycle exit and maint

77 over, we demonstrate that Golgi transport in hypocotyl cells can be accurately predicted from the act

78 in microtubule dynamics in spr1 eb1b mutant hypocotyl cells correlated well with the severity of gro

79 n cytoskeleton in both growing and elongated hypocotyl cells has structural properties facilitating e

80 the hypocotyl where it induces elongation of hypocotyl cells.

81 of the cortical microtubule cytoskeleton in hypocotyl cells.

82 tensive transcriptome reconfiguration in the hypocotyls compared with the cotyledons.

83 protein to be lower in the nuclei of abcb19 hypocotyls compared with wild type.

84 n mtp8-2 mutant, Mn no longer accumulates in hypocotyl cortex cells and sub-epidermal cells of the em

85 ls, followed by the root apical meristem and hypocotyl, cotyledons, and shoot apical meristem.

86 The elongated cells of the hypocotyl create a variety of microtubule array patterns

87 or instance, secondary growth of Arabidopsis hypocotyls creates a radial pattern of highly specialize

88 We acquired hypocotyl cross-sections from tiled high-resolution imag

89 s into the known inhibitory role of light in hypocotyl development.

90 systemic, related to the disturbance of host hypocotyl developmental programs by preventing cell cycl

91 clustered stomata in the leaves, whereas the hypocotyls did not have any stomata.

92 High-temperature-mediated hypocotyl elongation additionally involves localized cha

93 wth medium greatly enhances the reduction in hypocotyl elongation and cellulose content of shv3svl1 T

94 ole of phyA-dependent CKI1 expression in the hypocotyl elongation and hook development during skotomo

95 me 2 (CRY2) mediate blue light inhibition of hypocotyl elongation and long-day (LD) promotion of flor

96 t receptor that mediates light inhibition of hypocotyl elongation and long-day promotion of floral in

97 ion, but both were active in AtD14-dependent hypocotyl elongation and secondary shoot growth.

98 Here, we study hypocotyl elongation as a proxy for shoot elongation and

99 of response from inhibition to promotion of hypocotyl elongation by light.

100 emonstrate that the magnitude of Suc-induced hypocotyl elongation depends on the day length and light

101 light/dark cycles, we found that Suc-induced hypocotyl elongation did not occur in tps1 mutants and o

102 Our study reveals that plants regulate hypocotyl elongation during seedling establishment by co

103 rmomorphogenesis but does not interfere with hypocotyl elongation during shade avoidance.

104 thylene, accentuates the effects of light on hypocotyl elongation during the dark-to-light transition

105 in-signaling machinery to regulate etiolated hypocotyl elongation growth in Arabidopsis.

106 iologically, Glc and BR interact to regulate hypocotyl elongation growth of etiolated Arabidopsis (Ar

107 -6 double mutant displayed severe defects in hypocotyl elongation growth similar to its bri1-6 parent

108 In seedlings, these proteins repress hypocotyl elongation in a daylength- and sucrose-depende

109 ibit folate biosynthesis in plants, restrict hypocotyl elongation in a sugar-dependent fashion.

110 at, when overexpressed, resulted in enhanced hypocotyl elongation in etiolated Arabidopsis thaliana s

111 ediating a signal that underlies Suc-induced hypocotyl elongation in light/dark cycles.

112 nt with ethylene or auxin inhibitors reduced hypocotyl elongation in PIF4 overexpressor (PIF4ox) and

113 gibberellins (GAs) antagonistically regulate hypocotyl elongation in plants.

114 e fluence rate response where suppression of hypocotyl elongation increases incrementally with light

115 Hypocotyl elongation is a highly coordinated physiologic

116 Under short-day (SD) photocycles, hypocotyl elongation is maximal at dawn, being promoted

117 s thaliana) seedlings are grown in the dark, hypocotyl elongation is promoted, whereas root growth is

118 elicits shade- and high temperature-induced hypocotyl elongation largely independently of 3-IPA-medi

119 As GA signalling directs hypocotyl elongation largely through promoting PIF activ

120 In contrast, the reduced hypocotyl elongation of ethylene biosynthesis and signal

121 Mutation in DET1 changed the sensitivity of hypocotyl elongation of mutant seedlings to GA and paclo

122 articipates positively in the control of the hypocotyl elongation response to plant proximity, a role

123 Plants lacking PGX1 display reduced hypocotyl elongation that is complemented by transgenic

124 ts in fertility, and enhanced sensitivity of hypocotyl elongation to red but not to far-red or blue l

125 We found that Suc-induced hypocotyl elongation under light/dark cycles does not in

126 ight- and phytochrome-mediated regulation of hypocotyl elongation under red (R) and FR illumination.

127 xin and gibberellin signaling in Suc-induced hypocotyl elongation under short photoperiods.

128 er transcription factor that participates in hypocotyl elongation under short-day conditions.

129 cient phyA-dependent pathway that suppresses hypocotyl elongation when challenged by shade from nearb

130 -regulated protein stability drives rhythmic hypocotyl elongation with peak growth at dawn.

131 reviously uncharacterized LHE (LIGHT-INDUCED HYPOCOTYL ELONGATION) gene, which we show impacts light-

132 ppressed chlorophyll synthesis, promotion of hypocotyl elongation, and formation of a closed apical h

133 active in KAI2-dependent seed germination or hypocotyl elongation, but both were active in AtD14-depe

134 rphogenesis, as illustrated by inhibition of hypocotyl elongation, cotyledon opening, and leaf greeni

135 e overexpressors has differential effects on hypocotyl elongation, leaf shape, and petiole length, as

136 e of these genes in the control of greening, hypocotyl elongation, phyllotaxy, floral organ initiatio

137 g deoxystrigolactones to inhibit Arabidopsis hypocotyl elongation, regulate seedling gene expression,

138 ure in distinct developmental traits such as hypocotyl elongation, root elongation, and flowering tim

139 lthough only SMAX1 regulates germination and hypocotyl elongation, SMAX1 and SMXL6,7,8 have complemen

140 er HY5 uses additional mechanisms to inhibit hypocotyl elongation.

141 ce by DELLAs correlates closely with reduced hypocotyl elongation.

142 ngs have opposite BR-response phenotypes for hypocotyl elongation.

143 HXK1)-mediated pathway to regulate etiolated hypocotyl elongation.

144 eviously shown to be important regulators of hypocotyl elongation.

145 ular anisotropy driving Arabidopsis thaliana hypocotyl elongation.

146 ding seed germination, stomatal closure, and hypocotyl elongation.

147 and genetically acts through BIN2 to inhibit hypocotyl elongation.

148 how that cotyledon-generated auxin regulates hypocotyl elongation.

149 athways, such as photoperiodic flowering and hypocotyl elongation.

150 r Phytochrome Interacting Factor 4 (PIF4) on hypocotyl elongation.

151 sor of brassinosteroid signaling, to repress hypocotyl elongation.

152 ging of a structure-function relationship in hypocotyl epidermal cell patterning through global topol

153 tation of Arabidopsis (Arabidopsis thaliana) hypocotyl epidermal cells, dynamic cortical microtubules

154 aliana with transcriptional profiling of the hypocotyl epidermis from Brassica rapa, we show that aux

155 he expense of IAA-Glu (IAA-glutamate) in the hypocotyl epidermis.

156 ted with growth symmetry breaking within the hypocotyl epidermis.

157 In the hypocotyl expansion zone, indaziflam caused an atypical

158 comparison, average speed in the A. thaliana hypocotyl expressing GFP-AtCESA6 was 184 +/- 86 nm min(-

159 Hypocotyl extension in the shade and outgrowth of new le

160 In hypocotyls, GA levels were reduced in a phytochrome inte

161 light responses, including seed germination, hypocotyl gravitropism, and chlorophyll biosynthesis, by

162 the circadian clock, and we review seedling hypocotyl growth as a paradigm of PIFs acting at the int

163 promotes PIF4/PIF5 protein accumulation and hypocotyl growth at both 22 degrees C and 17 degrees C,

164 integrates light and temperature control of hypocotyl growth by promoting PIF4 and PIF5 protein abun

165 a molecular understanding of the control of hypocotyl growth by these proteins.

166 Hypocotyl growth during seedling emergence is a crucial

167 Moreover, the stimulatory role of light on hypocotyl growth during the dark-to-light transition pro

168 peratively stimulate a transient increase in hypocotyl growth during the dark-to-light transition via

169 ATAF2 modulates hypocotyl growth in a light-dependent manner, with the p

170 night temperature difference [-DIF]) inhibit hypocotyl growth in Arabidopsis (Arabidopsis thaliana).

171 r basis for the phyB-mediated suppression of hypocotyl growth in Arabidopsis.

172 Ethylene and light antagonistically control hypocotyl growth in either continuous light or darkness.

173 Previous studies have shown that hypocotyl growth in low red to far-red shade is largely

174 g is required in many cell types for correct hypocotyl growth in shade, with a key role for the epide

175 xpression, coinciding with the initiation of hypocotyl growth in the early evening, is positively cor

176 rowth, light treatment transiently increases hypocotyl growth in wild-type etiolated seedlings.

177 brief heat shocks enhance the inhibition of hypocotyl growth induced by light perceived by phytochro

178 sensitive to MeJA- and COR-induced root and hypocotyl growth inhibition.

179 This suggests that hypocotyl growth is elicited by both local and distal au

180 signaling pathways and uncover differential hypocotyl growth of red light-grown seedlings in respons

181 me and root growth; control of cotyledon and hypocotyl growth requires simultaneous phyA activity in

182 ng; instead, it showed auxin activity in the hypocotyl growth test.

183 sential for plant cold acclimation, promotes hypocotyl growth under ambient temperatures in Arabidops

184 and the conditional use of GA-ATHB5-mediated hypocotyl growth under optimal conditions may be used to

185 HsfB2b is also involved in the regulation of hypocotyl growth under warm, short days.

186 ses of gene expression, cotyledon unfolding, hypocotyl growth, and greening observed in the phyA muta

187 e changes of single hypocotyl protoplasts or hypocotyl growth, both at high temporal resolution.

188 is both necessary and sufficient to initiate hypocotyl growth, but we also provide evidence for the f

189 with the negative regulatory role of HOS1 in hypocotyl growth, HOS1-defective mutants exhibited elong

190 However, how ethylene and light regulate hypocotyl growth, including seedling emergence, during t

191 ast to the known inhibitory role of light in hypocotyl growth, light treatment transiently increases

192 In contrast to vastly studied hypocotyl growth, little is known about diel regulation

193 ic diurnal variation in Arabidopsis thaliana hypocotyl growth, we found that cellulose synthesis and

194 ing phenotypes, including increased stem and hypocotyl growth, which increases the likelihood of outg

195 g converge to influence the transcription of hypocotyl growth-promoting SAUR19 subfamily members.

196 elayed seed germination in the dark and long hypocotyl growth.

197 ion of genes involved in cell elongation and hypocotyl growth.

198 within the molecular framework driving rapid hypocotyl growth.

199 (PIF3), a key transcription factor promoting hypocotyl growth.

200 specific expression of PIF4 has no effect on hypocotyl growth.

201 esponsible for a component of ploidy-related hypocotyl growth.

202 that HMR acts upstream of PIFs in regulating hypocotyl growth.

203 4), a key transcription factor that promotes hypocotyl growth.

204 lly understood how the phytochromes modulate hypocotyl growth.

205 duced an auxin-like swelling response but no hypocotyl growth.

206 In Arabidopsis, the seedling hypocotyl has emerged as an exemplar model system to stu

207 The middle and upper hypocotyl have a greater requirement for GA to promote c

208 UV-B also stabilizes the bHLH protein LONG HYPOCOTYL IN FAR RED (HFR1), which can bind to and inhib

209 date a previously unidentified role for long hypocotyl in far red 1, a negative regulator of the PIFs

210 levels of the transcriptional cofactor LONG HYPOCOTYL IN FAR RED1, which also binds to PIF1 and othe

211 This latter regulation requires LONG HYPOCOTYL IN FAR RED1/SLENDER IN CANOPY SHADE1 and phyto

212 mics of ELONGATED HYPOCOTYL 5 (HY5) and LONG HYPOCOTYL IN FAR-RED (HFR1) proteins; and the epistatic

213 In addition, the long hypocotyl in far-red light phenotype of the laf6 mutant

214 n studies and proposed to interact with LONG HYPOCOTYL IN FAR-RED1 (HFR1), a (b)HLH protein that inhi

215 Arabidopsis (Arabidopsis thaliana) Short Hypocotyl in White Light1 (SHW1) encodes a Ser-Arg-Asp-r

216 During secondary growth of hypocotyls in Arabidopsis thaliana, the xylem undergoes

217 are required for an efficient elongation of hypocotyls in response to auxin and for the correct expr

218 However, the elongation of hypocotyls in response to auxin was impaired in the muta

219 of free auxin in specialized organs such as hypocotyls in response to shade and high temperature.

220 e1 mutants, which produce strongly elongated hypocotyls in response to shade.

221 ugar-sensing mechanisms in the elongation of hypocotyls in response to Suc.

222 , HOS1-defective mutants exhibited elongated hypocotyls in the light.

223 similar to its Arabidopsis counterpart (long hypocotyls in white and blue light), but also several ad

224 ile constitutive expression of PCH1 shortens hypocotyls independent of day length.

225 in transport and that auxin transport in the hypocotyl is a prerequisite for phot1-dependent hypocoty

226 The Arabidopsis thaliana hypocotyl is a robust system for studying the interplay

227 IF- and light-regulated stomata formation in hypocotyls is critically dependent on LLM-domain B-GATA

228 ases, and morphogenetic capacity of cultured hypocotyls is reduced.

229 One of these, LATE ELONGATED HYPOCOTYL, is known in A. thaliana to regulate many stre

230 e with Sl-MMPs in the apoplast of the tomato hypocotyl, it exhibited increased stability in transgeni

231 ent and aboveground plant tissues, including hypocotyls, leaves, and stems.

232 Hypocotyl length determination is a widely used method t

233 ined, these results show that PIF3 regulates hypocotyl length downstream, whereas PIF4 and PIF5 regul

234 ntrast to pif4 and pif5 mutants, the reduced hypocotyl length in pif3 cannot be rescued by either ACC

235 d seed weight and internode length, enhanced hypocotyl length in red light, inhibited primary root gr

236 delineate Arabidopsis (Arabidopsis thaliana) hypocotyl length kinetics in response to ethylene and sh

237 h downstream, whereas PIF4 and PIF5 regulate hypocotyl length upstream of an auxin and ethylene casca

238 th AtHY5, which does not cause any change in hypocotyl length when overexpressed in Arabidopsis, the

239 BBX19 expression by RNA interference reduces hypocotyl length, and its constitutive expression promot

240 abidopsis plants, resulting in a decrease in hypocotyl length.

241 ted to leaf number and complexity as well as hypocotyl length.

242 onfucosylated xyloglucan, rescued dark-grown hypocotyl lengthening of ABP1 knockdown seedlings.

243 of the core clock regulators LATE ELONGATED HYPOCOTYL (LHY) and CIRCADIAN CLOCK ASSOCIATED1 (CCA1) i

244 sic mutant alleles accumulate LATE ELONGATED HYPOCOTYL (LHY) and CIRCADIAN CLOCK ASSOCIATED1 (CCA1) s

245 DIAN CLOCK ASSOCIATED1 (CCA1)/LATE ELONGATED HYPOCOTYL (LHY) and the evening gene TIMING OF CAB EXPRE

246 CLOCK ASSOCIATED1 (CCA1) and LATE ELONGATED HYPOCOTYL (LHY).

247 rcadian clock gene P. hybrida LATE ELONGATED HYPOCOTYL (LHY; PhLHY) regulates the daily expression pa

248 Finally, the decreased XyG abundance in hypocotyl longitudinal cell walls of germinating embryos

249 pstream regulators we identified a LONG PALE HYPOCOTYL (LPH) gene whose activity is indispensable for

250 early flowering plants with overly elongated hypocotyls mainly in short days.

251 is approach has been used exclusively on the hypocotyl of Arabidopsis thaliana.

252 The hypocotyls of Arabidopsis (Arabidopsis thaliana) also el

253 also disordered the cellular arrangement of hypocotyls of Arabidopsis plants, resulting in a decreas

254 organ (cotyledons) and in rapidly elongating hypocotyls of Arabidopsis thaliana PIFs initiate transcr

255 Hypocotyls of SOB3 mutant seedlings grown in white light

256 Etiolated hypocotyls of the quadruple atlazy1,2,3,4 mutant were es

257 Examination of hypocotyls of these plants revealed normal vasculature i

258 In Arabidopsis dark-grown hypocotyls, one PME (AtPME3) and one PMEI (AtPMEI7) were

259 ruption of BAS1 and SOB7 abolishes the short-hypocotyl phenotype of ATAF2 loss-of-function seedlings

260 background is likely causative for the long hypocotyl phenotype previously attributed to disrupted A

261 hyB background partially suppressed its long hypocotyl phenotype.

262 Specifically, SOB3 mutant hypocotyl phenotypes, which are readily apparent when th

263 During hypocotyl photomorphogenesis, light signals are sensed b

264 We measured either volume changes of single hypocotyl protoplasts or hypocotyl growth, both at high

265 Elongation of the Arabidopsis hypocotyl pushes the shoot-producing meristem out of the

266 lar level with reduced cell expansion in the hypocotyl relative to the wild type.

267 The induction of cell elongation in hypocotyls requires temperature sensing in cotyledons, f

268 ings expressing PIF3(K13R) show an elongated hypocotyl response, elevated photoprotection and higher

269 to reflect metabolism in the cotyledons and hypocotyl/root axis (HRA).

270 is defective in cell expansion in dark-grown hypocotyls, roots, and adult plants.

271 oy a custom image-based method for measuring hypocotyl segment elongation with high resolution and a

272 In contrast, hypocotyl segments overexpressing a PP2C.D phosphatase a

273 sing the mechanical extensibility of excised hypocotyl segments.

274 ed from upper (growing) regions of 3-day-old hypocotyls showed ploidy levels to be lower in abcb19 mu

275 pch1 seedlings have overly elongated hypocotyls specifically under short days while constitut

276 In hypocotyls, the combination of circadian expression of P

277 In secondarily thickened hypocotyls, these enzymes had positive effects on vessel

278 In hypocotyls, these plants failed to transition to true ra

279 c stress in roots and ABA transport from the hypocotyl to the shoot and root.

280 ulations available for Arabidopsis etiolated hypocotyls to clarify how auxin is perceived and the dow

281 ncrease the growth of specific organs (e.g., hypocotyls) to enhance access to sunlight.

282 downregulate symplasmic permeability during hypocotyl tropic response.

283 Cell elongation in the basal part of the hypocotyl under -DIF was restored by both 1-aminocyclopr

284 SHORT HYPOCOTYL UNDER BLUE1 (SHB1) is a key regulatory gene of

285 Overexpression of Arabidopsis SHORT HYPOCOTYL UNDER BLUE1::uidA (SHB1:uidA) in canola produc

286 ly suppressed excessive radial growth of the hypocotyl vasculature during secondary growth.

287 We focused on phenotypes of hypocotyl vasculatures caused by double mutation in EREC

288 This newly synthesized auxin moves to the hypocotyl where it induces elongation of hypocotyl cells

289 Subsequently, auxin travels to the hypocotyl, where it triggers local brassinosteroid-induc

290 l processes including cell elongation in the hypocotyl, whether or not it modulates cell proliferatio

291 ngs grow initially through elongation of the hypocotyl, which is regulated by signaling pathways that

292 ve morphological changes including elongated hypocotyls, which is predominantly regulated by a bHLH t

293 bidopsis thaliana line with longer etiolated hypocotyls, which overexpresses a gene encoding a polyga

294 t known, nor is their function understood in hypocotyls, which undergo considerable radial expansion.

295 expression of PIF4 induces constitutive long hypocotyls, while vasculature-specific expression of PIF

296 expansion; for example, epidermal cells from hypocotyls with reduced CP are longer than wild-type cel

297 -based reporter of mitosis throughout abcb19 hypocotyls without an equivalent effect on mitosis promp

298 Upon floral transition, the hypocotyl xylem gained a competency to respond to GA in

299 ARY WALL THICKENING PROMOTING FACTORs in the hypocotyl xylem.

300 iformly expressed throughout the Arabidopsis hypocotyl, yet decapitation experiments have localized t