ライフサイエンスコーパス: hypocotyl elongation

1 er HY5 uses additional mechanisms to inhibit hypocotyl elongation.

2 r Phytochrome Interacting Factor 4 (PIF4) on hypocotyl elongation.

3 rfi2-1 mutation in the expression of CO and hypocotyl elongation.

4 ive regulator of phyB-mediated inhibition of hypocotyl elongation.

5 lowering, circadian cotyledon movements, and hypocotyl elongation.

6 utamate and glycine synergism also regulates hypocotyl elongation.

7 oreceptor mediating blue light inhibition of hypocotyl elongation.

8 shoot initiation, and brassinolide-mediated hypocotyl elongation.

9 mental processes including de-etiolation and hypocotyl elongation.

10 diating the blue light-induced inhibition of hypocotyl elongation.

11 toreceptor, cryptochrome (CRY1), involved in hypocotyl elongation.

12 as observed in light-dependent inhibition of hypocotyl elongation.

13 ectable consequences for the photocontrol of hypocotyl elongation.

14 rease in auxin levels resulting in increased hypocotyl elongation.

15 dopsis thaliana which mediates inhibition of hypocotyl elongation.

16 although it has no significant effect on the hypocotyl elongation.

17 altered responses to NPA in root curling and hypocotyl elongation.

18 of vegetative photomorphogenesis, including hypocotyl elongation.

19 cts in miRNA biogenesis and thermoresponsive hypocotyl elongation.

20 is required for the full effects of auxin on hypocotyl elongation.

21 sor of brassinosteroid signaling, to repress hypocotyl elongation.

22 (PIFs) 4 and 5 to produce a daily rhythm of hypocotyl elongation.

23 timulates ABA biosynthesis to fully regulate hypocotyl elongation.

24 ing to promoting elongation, thereby driving hypocotyl elongation.

25 hypocotyl growth with minimal suppression of hypocotyl elongation.

26 ous defects, including for flowering time or hypocotyl elongation.

27 /Broad 1, 2 & 3) in blue-light regulation of hypocotyl elongation.

28 ES redundantly contribute to Heatin-mediated hypocotyl elongation.

29 ce by DELLAs correlates closely with reduced hypocotyl elongation.

30 and genetically acts through BIN2 to inhibit hypocotyl elongation.

31 ngs have opposite BR-response phenotypes for hypocotyl elongation.

32 HXK1)-mediated pathway to regulate etiolated hypocotyl elongation.

33 eviously shown to be important regulators of hypocotyl elongation.

34 ular anisotropy driving Arabidopsis thaliana hypocotyl elongation.

35 athways, such as photoperiodic flowering and hypocotyl elongation.

36 ding seed germination, stomatal closure, and hypocotyl elongation.

37 how that cotyledon-generated auxin regulates hypocotyl elongation.

38 curately describes wild-type root growth and hypocotyl elongation.

39 nced resistance to DELLA accumulation during hypocotyl elongation.

40 mote the known shade-induced acceleration of hypocotyl elongation.

41 NG FACTOR4 (PIF4) and PIF5 in the control of hypocotyl elongation.

42 h no reduction in postgermination radical or hypocotyl elongation.

43 ponents acting downstream of phyA to inhibit hypocotyl elongation.

44 oral initiation and blue light inhibition of hypocotyl elongation.

45 re and is required for temperature-dependent hypocotyl elongation.

46 fore abrogate PIF3-mediated light control of hypocotyl elongation.

47 acid [IAA], 2,4-D, 1-NAA) have no effect on hypocotyl elongation.

48 Unlike 2,4-D alone, this pro-2,4-D enhanced hypocotyl elongation.

49 gs and identified 100 compounds that promote hypocotyl elongation.

50 opmental defects, including reduced root and hypocotyl elongation.

51 iting a thermotolerance defect as assayed by hypocotyl elongation, 10-day-old hot1 seedlings were als

52 High-temperature-mediated hypocotyl elongation additionally involves localized cha

53 umpkin hypocotyls during the period of rapid hypocotyl elongation after which mRNA levels declined, w

54 with SPY acting in the GA pathway to inhibit hypocotyl elongation and also acting as a light-regulate

55 a hy5 mutant, with regards to inhibition of hypocotyl elongation and anthocyanin accumulation.

56 cryptochrome), which mediates inhibition of hypocotyl elongation and anthocyanin biosynthesis, has r

57 s concluded that CRY1-mediated inhibition of hypocotyl elongation and anthocyanin production requires

58 It requires rapid hypocotyl elongation and apical hook formation, both of

59 ns similar to cry1 and include inhibition of hypocotyl elongation and blue light-dependent anthocyani

60 wth medium greatly enhances the reduction in hypocotyl elongation and cellulose content of shv3svl1 T

61 s to specifically mediate SHB1 regulation of hypocotyl elongation and CHLOROPHYLL a/b BINDING PROTEIN

62 B and cry2 in the control of flowering time, hypocotyl elongation and circadian period by the clock.

63 s show only small defects in photocontrol of hypocotyl elongation and cotyledon opening under prolong

64 motion of cotyledon expansion, repression of hypocotyl elongation and flowering time in addition to o

65 endent circadian period effects, and altered hypocotyl elongation and flowering time.

66 ariety of physiological processes, including hypocotyl elongation and flowering time.

67 ole of phyA-dependent CKI1 expression in the hypocotyl elongation and hook development during skotomo

68 ings, BMAA caused a 2- to 3-fold increase in hypocotyl elongation and inhibited cotyledon opening dur

69 functionally implicated in the inhibition of hypocotyl elongation and known to be a direct target of

70 f auxin-regulated growth processes including hypocotyl elongation and lateral root formation.

71 hypersensitivity to red light inhibition of hypocotyl elongation and light-regulated gene expression

72 me 2 (CRY2) mediate blue light inhibition of hypocotyl elongation and long-day (LD) promotion of flor

73 t receptor that mediates light inhibition of hypocotyl elongation and long-day promotion of floral in

74 ving a unique role in regulating FR-mediated hypocotyl elongation and meristem- and/or leaf primordia

75 However, ethylene-induced inhibition of hypocotyl elongation and petiole epinasty are normal in

76 e 2 (CRY2) mediates blue light inhibition of hypocotyl elongation and photoperiodic control of floral

77 ight receptor regulating light inhibition of hypocotyl elongation and photoperiodic flowering in Arab

78 many aspects of plant development, including hypocotyl elongation and photoperiodic induction of flow

79 that CRY2 mediates blue light inhibition of hypocotyl elongation and photoperiodic promotion of flor

80 etween light and auxin signaling pathways in hypocotyl elongation and phototropism responses.

81 bri1, did not respond to brassinosteroids in hypocotyl elongation and primary root inhibition assays,

82 t photomorphogenesis, such as suppression of hypocotyl elongation and promotion of cotyledon expansio

83 ion, but both were active in AtD14-dependent hypocotyl elongation and secondary shoot growth.

84 assic glucose signaling responses, including hypocotyl elongation and seed germination, with exposure

85 lines, including FR-dependent inhibition of hypocotyl elongation and stimulation of anthocyanin accu

86 mediates blue light-dependent inhibition of hypocotyl elongation and stimulation of cotyledon openin

87 seedling development, such as inhibition of hypocotyl elongation and the promotion of greening, acti

88 vibrational stimulation, with an increase in hypocotyl elongation and up-regulation of TCH gene expre

89 t of an end-of-day pulse of far-red light on hypocotyl elongation, and a decrease in the number of ro

90 responses such as germination, inhibition of hypocotyl elongation, and anthocyanin accumulation in Ar

91 ppressed chlorophyll synthesis, promotion of hypocotyl elongation, and formation of a closed apical h

92 erellic acid (GA) promotes germination, stem/hypocotyl elongation, and leaf expansion during seedling

93 o key factors that antagonistically regulate hypocotyl elongation, and promote the abundance of both

94 Root growth and hypocotyl elongation are convenient downstream physiolog

95 Here, we study hypocotyl elongation as a proxy for shoot elongation and

96 However, hypocotyl elongation assays indicated that suppression o

97 on of root growth, lateral root development, hypocotyl elongation at high temperature, and apical dom

98 in enhanced accumulation of ELF3 and reduced hypocotyl elongation at warm temperature.

99 nd class of IAA precursor, displayed reduced hypocotyl elongation but normal cotyledon size and only

100 active in KAI2-dependent seed germination or hypocotyl elongation, but both were active in AtD14-depe

101 ty of the inhibitory effect of blue light on hypocotyl elongation, but phytochrome photoreceptors als

102 by increasing GA biosynthesis, but inhibits hypocotyl elongation by decreasing the responsiveness to

103 It is likely that the stimulation of hypocotyl elongation by Heatin might be independent of i

104 of response from inhibition to promotion of hypocotyl elongation by light.

105 The extent of inhibition of hypocotyl elongation by NPA increased in a fluence-rate-

106 ased upon induction of R-hypersensitivity of hypocotyl elongation by overexpression of the apoprotein

107 However, the promotion of hypocotyl elongation by SMAX1 and SMXL2 is, in contrast

108 Seedling root growth and hypocotyl elongation can be accurately predicted using a

109 adruple pifq mutant displays clearly reduced hypocotyl elongation compared to wild-type in response t

110 esC), Arabidopsis seedlings exhibit dramatic hypocotyl elongation compared with seedlings grown at 20

111 tomorphogenic responses in plants, including hypocotyl elongation, cotyledon expansion, and control o

112 rphogenesis, as illustrated by inhibition of hypocotyl elongation, cotyledon opening, and leaf greeni

113 We show that the hypocotyl elongation defect in the det3 mutant is condit

114 ry and coding sequences of RCN1, whereas the hypocotyl elongation defect of rcn1 plants can be comple

115 emonstrate that the magnitude of Suc-induced hypocotyl elongation depends on the day length and light

116 light/dark cycles, we found that Suc-induced hypocotyl elongation did not occur in tps1 mutants and o

117 tifies apical hook opening and inhibition of hypocotyl elongation during photomorphogenesis of Arabid

118 Our study reveals that plants regulate hypocotyl elongation during seedling establishment by co

119 rmomorphogenesis but does not interfere with hypocotyl elongation during shade avoidance.

120 thylene, accentuates the effects of light on hypocotyl elongation during the dark-to-light transition

121 16 functions to promote seed germination and hypocotyl elongation during the early stages of Arabidop

122 lue light (BL) rapidly and strongly inhibits hypocotyl elongation during the photomorphogenic respons

123 ly mediate light-induced inhibition of stem (hypocotyl) elongation during the development of photoaut

124 and differentiation, and the retardation in hypocotyl elongation enables growth and development in d

125 nsport, growth, and gravitropism, while rcn1 hypocotyl elongation exhibited enhanced ethylene respons

126 st few seconds of blue light and to suppress hypocotyl elongation for at least 120 min.

127 We developed a screen, based on hypocotyl elongation, for mutants of Arabidopsis thalian

128 reviously uncharacterized LHE (LIGHT-INDUCED HYPOCOTYL ELONGATION) gene, which we show impacts light-

129 in-signaling machinery to regulate etiolated hypocotyl elongation growth in Arabidopsis.

130 sensitivity of cry1 and cry2 in controlling hypocotyl elongation growth in Brassica.

131 iologically, Glc and BR interact to regulate hypocotyl elongation growth of etiolated Arabidopsis (Ar

132 -6 double mutant displayed severe defects in hypocotyl elongation growth similar to its bri1-6 parent

133 of four loci required for thermotolerance of hypocotyl elongation, hot1-1, hot2-1, hot3-1, and hot4-1

134 In seedlings, these proteins repress hypocotyl elongation in a daylength- and sucrose-depende

135 ibit folate biosynthesis in plants, restrict hypocotyl elongation in a sugar-dependent fashion.

136 The pattern of hypocotyl elongation in constant light includes a daily

137 Moreover, SlBBX28 knockdown reduced hypocotyl elongation in darkness-grown tomato.

138 ve, photoreceptor imposing the inhibition of hypocotyl elongation in deetiolating seedlings in respon

139 ecific, as the bim409 mutant exhibits normal hypocotyl elongation in etiolated (dark grown) plants (+

140 at, when overexpressed, resulted in enhanced hypocotyl elongation in etiolated Arabidopsis thaliana s

141 Mechanisms controlling hypocotyl elongation in etiolated seedlings reaching the

142 ke other BR-deficient mutants, the defect of hypocotyl elongation in fk-J79 cannot be corrected by ex

143 d mutants that exhibit reduced inhibition of hypocotyl elongation in FRc.

144 so uncover a new role for PCH1 in regulating hypocotyl elongation in LDs.

145 results lead us to the view that the rate of hypocotyl elongation in light is determined by at least

146 ediating a signal that underlies Suc-induced hypocotyl elongation in light/dark cycles.

147 showed significantly enhanced inhibition of hypocotyl elongation in low-white, red, far-red, blue, a

148 nt with ethylene or auxin inhibitors reduced hypocotyl elongation in PIF4 overexpressor (PIF4ox) and

149 gibberellins (GAs) antagonistically regulate hypocotyl elongation in plants.

150 essing phyC displayed enhanced inhibition of hypocotyl elongation in Rc, but were unchanged in respon

151 A similar additive effect on hypocotyl elongation in red and blue light is also obser

152 to modulate the phyB-mediated inhibition of hypocotyl elongation in red light and to function togeth

153 We measured a decrease in hypocotyl elongation in response to acidic pH in the tra

154 that nitrilases are involved in promotion of hypocotyl elongation in response to high temperature and

155 ACTING FACTOR 4 (PIF4), is also required for hypocotyl elongation in response to high temperature.

156 verexpression of AtRALF23 impairs BL-induced hypocotyl elongation in seedlings, and mature overexpres

157 In contrast, hypocotyl elongation in the dark was decreased independe

158 Arabidopsis thaliana developmental response, hypocotyl elongation in the dark, to detail the underpin

159 e oil and cannot utilize applied sucrose for hypocotyl elongation in the dark.

160 egulated genes in the dark, and HY5 inhibits hypocotyl elongation in the light.

161 tions of additional AHL genes in suppressing hypocotyl elongation in the light.

162 chemical library for compounds that restored hypocotyl elongation in the pif4-2-deficient mutant back

163 By contrast, inhibition of hypocotyl elongation in xct is hyposensitive to red ligh

164 e fluence rate response where suppression of hypocotyl elongation increases incrementally with light

165 induction of seed germination, inhibition of hypocotyl elongation, induction of cotyledon opening, ra

166 Hypocotyl elongation is a highly coordinated physiologic

167 ive to floral development is accelerated and hypocotyl elongation is accentuated in these mutants und

168 Hypocotyl elongation is controlled by several signals an

169 ansition from darkness to light, the rate of hypocotyl elongation is determined from the integration

170 Hypocotyl elongation is driven by increased auxin biosyn

171 The effect of BMAA on hypocotyl elongation is light specific.

172 Under short-day (SD) photocycles, hypocotyl elongation is maximal at dawn, being promoted

173 s thaliana) seedlings are grown in the dark, hypocotyl elongation is promoted, whereas root growth is

174 elicits shade- and high temperature-induced hypocotyl elongation largely independently of 3-IPA-medi

175 As GA signalling directs hypocotyl elongation largely through promoting PIF activ

176 s throughout the plant life cycle, including hypocotyl elongation, leaf expansion and apical dominanc

177 and signaling, ultimately leading to proper hypocotyl elongation, leaf expansion, and inflorescence

178 e overexpressors has differential effects on hypocotyl elongation, leaf shape, and petiole length, as

179 SlPIF4 promotes hypocotyl elongation mainly by activating the transcript

180 In contrast, the reduced hypocotyl elongation of ethylene biosynthesis and signal

181 Mutation in DET1 changed the sensitivity of hypocotyl elongation of mutant seedlings to GA and paclo

182 that causes a two- to three-fold increase in hypocotyl elongation on Arabidopsis seedlings grown in t

183 e of these genes in the control of greening, hypocotyl elongation, phyllotaxy, floral organ initiatio

184 e decreased response to red light (R) of the hypocotyl elongation rate in these mutants.

185 o far-red, red, and blue-light inhibition of hypocotyl elongation, reduced chlorophyll and anthocyani

186 g deoxystrigolactones to inhibit Arabidopsis hypocotyl elongation, regulate seedling gene expression,

187 prolonged continuous red light (Rc) promotes hypocotyl elongation relative to dark controls, the reci

188 onse to high temperature and Heatin-mediated hypocotyl elongation requires the NITRILASE1-subfamily m

189 The normal BMAA-induced hypocotyl elongation response observed on wild-type seed

190 ants are shown to lack an end-of-day-far-red hypocotyl elongation response that requires a stable Pfr

191 articipates positively in the control of the hypocotyl elongation response to plant proximity, a role

192 UV-A, and green light for the inhibition of hypocotyl elongation response.

193 h red and far-red light in the inhibition of hypocotyl elongation response; a classic phytochrome phe

194 ophore inactivation and associated disparate hypocotyl elongation responses under far-red (FR) light.

195 ure in distinct developmental traits such as hypocotyl elongation, root elongation, and flowering tim

196 thermotolerance defects in seed germination, hypocotyl elongation, root growth, and seedling survival

197 alterations of red light effects on seedling hypocotyl elongation, rosette leaf morphology, and chlor

198 lthough only SMAX1 regulates germination and hypocotyl elongation, SMAX1 and SMXL6,7,8 have complemen

199 ts exhibit hypersensitive photoinhibition of hypocotyl elongation, suggesting that MAC3A/3B positivel

200 nse was necessary for (a). the inhibition of hypocotyl elongation that develops within minutes of the

201 Plants lacking PGX1 display reduced hypocotyl elongation that is complemented by transgenic

202 cues with opposite effects on growth control hypocotyl elongation through intricate mechanisms.

203 ression in fhy1-3 caused an insensitivity of hypocotyl elongation to FR and blue light (B) indistingu

204 characteristics include hypersensitivity of hypocotyl elongation to inhibition by white, blue, red,

205 erring ethylene insensitivity restore normal hypocotyl elongation to rcn1.

206 ts in fertility, and enhanced sensitivity of hypocotyl elongation to red but not to far-red or blue l

207 HsfA1d is a positive regulator of hypocotyl elongation under chilling.

208 f-function of one ribosome gene also reduced hypocotyl elongation under chilling.

209 Here we use hypocotyl elongation under dark to investigate the molec

210 bidopsis, SnRK2.2/3/6, exert dual effects on hypocotyl elongation under far-red (FR) and red (R) ligh

211 me-mediated degradation of phytochrome A and hypocotyl elongation under far-red light.

212 f COP1 function, as indicated by exaggerated hypocotyl elongation under light conditions.

213 We found that Suc-induced hypocotyl elongation under light/dark cycles does not in

214 ight- and phytochrome-mediated regulation of hypocotyl elongation under red (R) and FR illumination.

215 ere, we unveil a role for TZP in fine-tuning hypocotyl elongation under red light and long-day condit

216 epistatic to these B subunits in regulating hypocotyl elongation under red light.

217 the physiological effects of IBA and IAA on hypocotyl elongation under several light conditions were

218 xin and gibberellin signaling in Suc-induced hypocotyl elongation under short photoperiods.

219 er transcription factor that participates in hypocotyl elongation under short-day conditions.

220 retain and exert a dual capacity to modulate hypocotyl elongation under these conditions, by concomit

221 nclude that the RCN1 protein affects overall hypocotyl elongation via negative regulation of ethylene

222 The rhythm of hypocotyl elongation was entrained by light-dark cycles

223 The fluence rate response of hypocotyl elongation was examined and showed a biphasic

224 When inhibition of hypocotyl elongation was measured, elf3 mutant seedlings

225 fferent blue-light receptors act to suppress hypocotyl elongation, we measured relative elemental gro

226 ediated by glutamate and glycine, as well as hypocotyl elongation, were inhibited by 6,7-dinitroquino

227 cient phyA-dependent pathway that suppresses hypocotyl elongation when challenged by shade from nearb

228 y to red and far-red light in the control of hypocotyl elongation, whereas increments in TOC1 gene do

229 display a phenotype of reduced inhibition of hypocotyl elongation, which is specific to far-red light

230 -regulated protein stability drives rhythmic hypocotyl elongation with peak growth at dawn.

231 strated that protein levels increased in the hypocotyl elongation zone when shifted from the dark to