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1 elayed seed germination in the dark and long hypocotyl growth.
2 interact with target proteins and to promote hypocotyl growth.
3 31 accelerates ELF3 degradation and promotes hypocotyl growth.
4  CCA1, and LHY in regulating light-inhibited hypocotyl growth.
5  its ubiquitination and turnover to modulate hypocotyl growth.
6 and a regulator of warm temperature-mediated hypocotyl growth.
7  suggesting that MAC3A/3B positively control hypocotyl growth.
8 4), a key transcription factor that promotes hypocotyl growth.
9 lly understood how the phytochromes modulate hypocotyl growth.
10 duced an auxin-like swelling response but no hypocotyl growth.
11 ion of genes involved in cell elongation and hypocotyl growth.
12 within the molecular framework driving rapid hypocotyl growth.
13 (PIF3), a key transcription factor promoting hypocotyl growth.
14 esponsible for a component of ploidy-related hypocotyl growth.
15 that HMR acts upstream of PIFs in regulating hypocotyl growth.
16 specific expression of PIF4 has no effect on hypocotyl growth.
17 apyrases at least as rapidly as it inhibited hypocotyl growth.
18 idopsis thaliana, high-resolution studies of hypocotyl growth accomplished by computer-assisted elect
19 strate that SLOMO is a negative regulator of hypocotyl growth, also under warm temperature conditions
20 to root elongation, but cytokinin effects on hypocotyl growth and ethylene synthesis in these seedlin
21 ga/spy/gal-3 is almost insensitive to GA for hypocotyl growth and its bolting stem is taller than the
22  epistatic to hrb1 under blue light for both hypocotyl growth and light-regulated gene expression res
23 nment, this dual action will strongly retard hypocotyl growth and promote cotyledon opening and expan
24 ions in phyA largely suppress the randomized-hypocotyl growth and the short-hypocotyl phenotype of th
25 ses of gene expression, cotyledon unfolding, hypocotyl growth, and greening observed in the phyA muta
26 tyledon unhooking, unfolding, and expansion, hypocotyl growth, and the accumulation of chlorophylls a
27  the circadian clock, and we review seedling hypocotyl growth as a paradigm of PIFs acting at the int
28 ype seed and mutant seedlings have decreased hypocotyl growth as compared to wildtype seedlings when
29                                              Hypocotyl growth assays in monochromatic light and micro
30                                Together with hypocotyl growth assays showing that the sensitivity and
31 r-null and phosphor-mimetic seedlings affect hypocotyl growth at 22 and 28 degrees C by modulating th
32  promotes PIF4/PIF5 protein accumulation and hypocotyl growth at both 22 degrees C and 17 degrees C,
33 red the effects of a reduced PMF on root and hypocotyl growth, ATP-induced skewed root growth, and ra
34 e changes of single hypocotyl protoplasts or hypocotyl growth, both at high temporal resolution.
35 is both necessary and sufficient to initiate hypocotyl growth, but we also provide evidence for the f
36 ensation of CRYs fine-tunes light-responsive hypocotyl growth by balancing the opposed effects of HY5
37 idative environment favored the promotion of hypocotyl growth by COP1 under shade.
38  integrates light and temperature control of hypocotyl growth by promoting PIF4 and PIF5 protein abun
39         We present a model for regulation of hypocotyl growth by specific molecules found in this pat
40  a molecular understanding of the control of hypocotyl growth by these proteins.
41 ool in 5ptase11 mutants, we correlated these hypocotyl growth changes with a small increase in the 5P
42                                      Maximal hypocotyl growth coincides with the phase during which t
43  The xct mutation also causes sugar-specific hypocotyl growth defects, in which mutants are short in
44 g elements and their relationship underlying hypocotyl growth direction.
45 ences the auxin transport process to control hypocotyl growth during de-etiolation.
46                                              Hypocotyl growth during seedling emergence is a crucial
47   Moreover, the stimulatory role of light on hypocotyl growth during the dark-to-light transition pro
48 peratively stimulate a transient increase in hypocotyl growth during the dark-to-light transition via
49 A depletion partly explains the resetting of hypocotyl growth dynamics during photomorphogenesis.
50 with the negative regulatory role of HOS1 in hypocotyl growth, HOS1-defective mutants exhibited elong
51  are tightly regulated in light to fine-tune hypocotyl growth; however, details of the mechanisms tha
52                              ATAF2 modulates hypocotyl growth in a light-dependent manner, with the p
53 night temperature difference [-DIF]) inhibit hypocotyl growth in Arabidopsis (Arabidopsis thaliana).
54                            Here we show that hypocotyl growth in Arabidopsis thaliana during the nigh
55 luence-rate blue light (BL) rapidly inhibits hypocotyl growth in Arabidopsis, as in other species, af
56 r basis for the phyB-mediated suppression of hypocotyl growth in Arabidopsis.
57 4 in the temperature-dependent plasticity of hypocotyl growth in Arabidopsis.
58 f red light and a hypersensitive response in hypocotyl growth in continuous red light of higher fluen
59  Ethylene and light antagonistically control hypocotyl growth in either continuous light or darkness.
60 , defects in cell-cell adhesion, and reduced hypocotyl growth in etiolated seedlings.
61             Previous studies have shown that hypocotyl growth in low red to far-red shade is largely
62  det1 did not show significant inhibition of hypocotyl growth in response to UV-B, while det2 was str
63 g is required in many cell types for correct hypocotyl growth in shade, with a key role for the epide
64 ologs in Arabidopsis resulted in compromised hypocotyl growth in the dark, suggesting a functional co
65 xpression, coinciding with the initiation of hypocotyl growth in the early evening, is positively cor
66  the molecular basis for circadian gating of hypocotyl growth in the early evening.
67 rowth, light treatment transiently increases hypocotyl growth in wild-type etiolated seedlings.
68     However, how ethylene and light regulate hypocotyl growth, including seedling emergence, during t
69 on also generated transversal asymmetries in hypocotyl growth, indicating poor coordination among dif
70  brief heat shocks enhance the inhibition of hypocotyl growth induced by light perceived by phytochro
71          In wild-type Arabidopsis seedlings, hypocotyl growth inhibition and cotyledon expansion were
72 uced AtPP7 expression levels exhibit loss of hypocotyl growth inhibition and display limited cotyledo
73                             Phototropism and hypocotyl growth inhibition are modulated by the coactio
74 nterference of phytochrome A (phyA)-mediated hypocotyl growth inhibition in far-red (FR) light.
75                          Kinetic analyses of hypocotyl growth inhibition in response to ethylene and
76 hat SOB3 and ESC act redundantly to modulate hypocotyl growth inhibition in response to light.
77 on the order of minutes, that phyA initiated hypocotyl growth inhibition upon the onset of continuous
78 esses the phytochrome-modulated responses of hypocotyl growth inhibition, sucrose-stimulated anthocya
79  sensitive to MeJA- and COR-induced root and hypocotyl growth inhibition.
80 onse to photoperiod and a blue light-induced hypocotyl growth inhibition.
81                           This suggests that hypocotyl growth is elicited by both local and distal au
82 etic pigments are promoted by light, whereas hypocotyl growth is inhibited.
83 onditions studied, from UV to far-red, early hypocotyl growth is rapidly and robustly suppressed with
84 ast to the known inhibitory role of light in hypocotyl growth, light treatment transiently increases
85                In contrast to vastly studied hypocotyl growth, little is known about diel regulation
86                                              Hypocotyl growth of B19OE seedlings in red light was ver
87  signaling pathways and uncover differential hypocotyl growth of red light-grown seedlings in respons
88                         We compared root and hypocotyl growth of the single, double, and triple hydro
89                                              Hypocotyl growth often depends on sensing in green tissu
90 cessary for light-dependent randomization of hypocotyl growth orientation.
91 B transgene complements the phyB-1 red light hypocotyl growth phenotype completely, the PB-phyD and P
92 Loss of ABCB19 partially suppressed the cry1 hypocotyl growth phenotype in blue light.
93 s phenotype is the opposite of the increased hypocotyl growth phenotype previously described for othe
94 ream modules participate in diurnal rhythmic hypocotyl growth: PIF4 and/or PIF5 modulation of auxin-r
95 g converge to influence the transcription of hypocotyl growth-promoting SAUR19 subfamily members.
96 es, we found that SPA1 caused an increase in hypocotyl growth rate after approximately 2 h of continu
97 ight resulted in automatic quantification of hypocotyl growth rate, apical hook opening, and phototro
98   Auxin signaling and ABCB19 protein levels, hypocotyl growth rates, and apical hook opening were mea
99  of SLOMO activity controls the abundance of hypocotyl growth regulators, such as DWF1, through ubiqu
100 me and root growth; control of cotyledon and hypocotyl growth requires simultaneous phyA activity in
101 with warm temperature produces a synergistic hypocotyl growth response that dependent on PHYTOCHROME-
102 lue-light photoreceptors to coordinate these hypocotyl growth responses is still unclear.
103 alpha3 triple mutants also displayed reduced hypocotyl growth, smaller cotyledon size and a reduced n
104  hypocotyl, which reduced the sensitivity of hypocotyl growth specifically to blue light in long-term
105                                              Hypocotyl growth suppression by blue light was assessed
106 ion in the regulation of gene expression and hypocotyl growth suppression in Arabidopsis.
107 ng; instead, it showed auxin activity in the hypocotyl growth test.
108 g establishment, blue and red light suppress hypocotyl growth through the cryptochrome 1 (cry1) and p
109 TUTIVELY PHOTOMORPHOGENIC 1 (COP1), promotes hypocotyl growth to facilitate access to light.
110 sential for plant cold acclimation, promotes hypocotyl growth under ambient temperatures in Arabidops
111 and the conditional use of GA-ATHB5-mediated hypocotyl growth under optimal conditions may be used to
112 HsfB2b is also involved in the regulation of hypocotyl growth under warm, short days.
113 ating the level of ELF4 and thermoresponsive hypocotyl growth under warm-temperature conditions.
114 LF4) in Arabidopsis, and confers accelerated hypocotyl growth under warm-temperature conditions.
115 erotrimeric G protein in R and FR control of hypocotyl growth using a loss-of-function approach.
116 ic diurnal variation in Arabidopsis thaliana hypocotyl growth, we found that cellulose synthesis and
117 ing phenotypes, including increased stem and hypocotyl growth, which increases the likelihood of outg
118 unfolding, seed germination and agravitropic hypocotyl growth with minimal suppression of hypocotyl e

 
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