ライフサイエンスコーパス: hypodermis

1 ghters lose competence after fusing with the hypodermis.

2 f hyp6 with hyp7, the major syncytium of the hypodermis.

3 al cells or the posterior half of the dorsal hypodermis.

4 large amounts of fat in their intestines and hypodermis.

5 ction is not strictly limited to the lateral hypodermis.

6 ng a terminally differentiated adult lateral hypodermis.

7 rminal dendritic branches between muscle and hypodermis.

8 germline, gonadal sheath, coelomocytes, and hypodermis.

9 lialized tunnels and fibrotic regions of the hypodermis.

10 s on the sex and developmental stages of the hypodermis.

11 idermal keratinocytes and fibroblasts in the hypodermis.

12 nstruct consisting of epidermis, dermis, and hypodermis.

13 ne expression between the seam cells and the hypodermis.

14 hogenesis or establishment of the dermis and hypodermis.

15 and the preadipocytes and adipocytes of the hypodermis.

16 pression is reduced in the intestine and the hypodermis.

17 , while 5B is predominantly expressed in the hypodermis.

18 on of the key target gene, lin-3/EGF, in the hypodermis.

19 le in timing terminal differentiation of the hypodermis.

20 in the proper morphogenesis of the anterior hypodermis.

21 or and retarded adult gene expression in the hypodermis.

22 ion of Clr (Soc) function is required in the hypodermis.

23 nt cell types, including neurons, muscle and hypodermis.

24 ot in body-wall muscle but most likely is in hypodermis.

25 , is a requisite for fungal clearance in the hypodermis.

26 ted signaling from the nervous system to the hypodermis.

27 UNC-52 must be synthesized primarily by the hypodermis.

28 fascicles, among muscle bundles, and in the hypodermis.

29 le MAB-3 is expressed throughout the lateral hypodermis.

30 onic gene pathway: LIN-29 accumulates in the hypodermis abnormally early, during the third larval sta

31 Consistent with the hypodermis acting as an exopher phagocyte, we found that

32 and effector SEC-10/exocyst activity in the hypodermis, along with the CED-1 phagocytic receptor, is

33 t-1 and elt-3 are expressed in the embryonic hypodermis (also called the epidermis).

34 e located extracellularly between muscle and hypodermis and are essential for muscle development in b

35 infiltrate in the epidermis, dermis, and/or hypodermis and are often associated with systemic inflam

36 ssed in a group of unidentified cells in the hypodermis and at low level in the muscle tissue, but th

37 he formation of an opening through the adult hypodermis and cuticle that is used for egg laying and m

38 ssembly and attachment to basement membrane, hypodermis and cuticle.

39 OSR-1 is expressed in the hypodermis and intestine, and expression of OSR-1 in hyp

40 d, 800-nucleotide noncoding RNA expressed in hypodermis and intestine.

41 osm-7 is expressed in the hypodermis and may be secreted.

42 Ac-GST-1 was immunolocalized to the parasite hypodermis and muscle tissue and weakly to the intestine

43 ired for postembryonic downregulation in the hypodermis and nervous system and contains multiple puta

44 me utilization in the Caenorhabditis elegans hypodermis and provide a mechanism for the regulation of

45 pilla (DP) alternates between the dermis and hypodermis and results in varying oxygen levels for the

46 7 to promote terminal differentiation of the hypodermis and the cessation of molting in C. elegans.

47 is expressed in the cytosol of cells of the hypodermis and the intestine.

48 TR-mediated regulation of hbl-1, in both the hypodermis and the ventral nerve cord.

49 n causes strong precocious phenotypes in the hypodermis and vulva when expressed from multicopy array

50 ssembly and attachment to basement membrane, hypodermis, and cuticle.

51 on of subcellular patterning between muscle, hypodermis, and dendrites.

52 ratosis, marked thickening of the dermis and hypodermis, and early hair follicle generation in newbor

53 Signaling from neurons, hypodermis, and intestine regulate the appearance and be

54 rogradely, branches and turns within the ray hypodermis, and is nonciliated.

55 eir medial tips and are required to pull the hypodermis around the equator of the embryo.

56 Smad is expressed in pharynx, intestine and hypodermis, as has been previously reported for the type

57 and morphogenetic movements of the posterior hypodermis, as well as posterior-to-anterior cell fate t

58 how that MEC-8 is a nuclear protein found in hypodermis at most stages of development and not in most

59 at span from the exoskeletal ECM through the hypodermis, basement membrane ECM, to the muscles, coupl

60 f body wall muscle and on the regions of the hypodermis between body wall muscle quadrants, indicatin

61 the main sites of MIF production are in the hypodermis, body wall muscles and in the nuclei of devel

62 rly cell positions are observed in posterior hypodermis, both in the C-lineage cells that express pal

63 required to degrade exopher contents in the hypodermis but not for exopher-phagosome resolution into

64 e also found that overexpression of MEC-8 in hypodermis but not muscle can suppress certain unc-52 mu

65 sential function of 5B when expressed in the hypodermis, but 5B is incapable of carrying out SM chemo

66 hat is expressed in the cuticle-synthesizing hypodermis coincident with each larval molt.

67 and neutrophils to the Coccidioides-infected hypodermis com pared to wild-type mice; however, neutrop

68 ts, in addition to the defects in the dorsal hypodermis, consistent with the DIE-1 expression pattern

69 lacking this region to express in posterior hypodermis correlates with the inability of this constru

70 presence of the native CPZ-1 protein in the hypodermis/cuticle of larval and adult stages and along

71 on by RNAi suppressed the aberrant vulva and hypodermis development phenotypes of let-7(n2853) mutant

72 ion involves cross-tissue signaling from the hypodermis (epidermis) to the intestine to promote repro

73 the AB lineage, which generates most of the hypodermis (epidermis), a tissue with which muscle inter

74 DRAG-1 is normally expressed in the ventral hypodermis excepting the vulval region, its ectopic expr

75 ogenesis, including localized rupture of the hypodermis, failure of the midbody to elongate properly,

76 The antigen, although expressed by hypodermis, first reflects the pattern of muscle element

77 formulation to the homeostatic state of the hypodermis following SC injection.

78 utonomously required in the posterior dorsal hypodermis for intercalation.

79 ssed in two head cells at all stages, in the hypodermis from mid-second larval stage (L2) to the four

80 Mutations in mua-3 cause a separation of the hypodermis from the cuticle, suggesting this gene is req

81 al cell types and outside the pharynx in the hypodermis, hindgut, and vulva.

82 hat express rde-4(+) in body-wall muscles or hypodermis, however, enable silencing selectively in the

83 opment, and identify a critical role for the hypodermis in supporting pigment cell development.

84 al distribution throughout the epidermis and hypodermis in the HS lesion.

85 protein folding catalysts in the nematode's hypodermis indicated this to be a functionally important

86 or rounded tail, retraction of the tail tip hypodermis involves a temporally ordered set of cell fus

87 The epiboly of the Caenorhabditis elegans hypodermis involves the bilateral spreading of a thin ep

88 In particular, daf-9::GFP expression in the hypodermis is absolutely dependent on daf-12, the nuclea

89 The time of LIN-29 appearance in the hypodermis is controlled by the heterochronic gene pathw

90 o boundaries of hypodermal cells, shows that hypodermis is disorganized in these embryos.

91 The initial migration of the hypodermis is led by a quartet of cells, which exhibit p

92 of SMA-3, we find that SMA-3 activity in the hypodermis is necessary and sufficient for normal body s

93 of the type I TGF-beta receptor SMA-6 in the hypodermis is needed during adulthood to generate olfact

94 er transcriptionally from the differentiated hypodermis is poorly understood.

95 ind that overexpression of fmo-4 in just the hypodermis is sufficient for these benefits, and that th

96 daf-9 expression exclusively in the hypodermis is sufficient to restore reproductive develop

97 Hypodermis is the predominant site of Staphylococcus aur

98 The skin, including the hypodermis, is the largest organ of the body.

99 t human CaGC accumulated in the cells of the hypodermis-lateral chord of adult and larval parasites.

100 ed that Bm-MIF was localized to cells of the hypodermis/lateral chord, the uterine wall, and larvae d

101 at adipocytes, usually located in the deeper hypodermis layer, were present in the upper dermis layer

102 The hypodermis likely senses the availability of amino acids

103 In the vulva, sex myoblasts, and hypodermis, lin-42 activity prevents stage-specific cell

104 remodeling and cellular infiltration of the hypodermis measured by a newly developed computer-aided

105 hst-2 expression is found in the hypodermis, muscle, distal tip cells (DTCs), and in neur

106 ion was equivalent for MCs in the dermis and hypodermis of all three strains, but only the WT mice sh

107 are not properly organized within the dorsal hypodermis of die-1(w34) embryos, consistent with interc

108 COL-1 protein is specifically located in the hypodermis of G. pallida adult females.

109 d lin-42 mutants, and fails to accumulate in hypodermis of lin-4 mutants.

110 ales showed specific accumulation within the hypodermis of the body regions exposed to the soil envir

111 epithelial cell rearrangement in the dorsal hypodermis of the Caenorhabditis elegans embryo, in whic

112 marked alterations in elastic fibers of the hypodermis of Tsk animals.

113 rearrangement (intercalation) in the dorsal hypodermis, or embryonic epidermis, of the C. elegans em

114 velopment, for secretion of cuticle from the hypodermis, or for the function of muscle, in contrast t

115 While expression of rde-4(+) in intestine, hypodermis, or neurons using a repetitive transgene can

116 protein (Ce-CPL-1::GFP) was expressed in the hypodermis, pharynx, and gonad.

117 zed and are not restricted to regions of the hypodermis previously contacted by muscle.

118 They are synthesized in the hypodermis prior to secretion and incorporation into the

119 propose that DRAG-1 expressed in the ventral hypodermis signals via the UNC-40 receptor expressed in

120 orphic, scleromorphic, or scleromorphic with hypodermis), similarly to the performance of four reflec

121 ytial blastoderm, the Caenorhabditis elegans hypodermis, skeletal muscle and filamentous fungi.

122 mportant cis-regulatory region directing the hypodermis-specific expression of this operon gene of C.

123 estricted to tissue types that attach to the hypodermis, specifically body wall muscles, vulval muscl

124 ntiation, and morphogenesis of the posterior hypodermis, spicules, and proctodeum.

125 ore, constitutive expression of daf-9 in the hypodermis suppresses dauer arrest of daf-7 mutant anima

126 ursor cell fate or fuse with the surrounding hypodermis (the F fate).

127 coding the secreted GpX is restricted to the hypodermis, the outermost cellular layer of the nematode

128 eficient mice there was increased persistent hypodermis thickening and an elevated leptin level, whic

129 generate ectopic PAL-1-dependent muscle and hypodermis, tissues normally made by the C blastomere.

130 actor beta (TGF-beta) Sma/Mab pathway in the hypodermis to control reproductive aging, and that it do

131 es via the IIS and Rag-TORC1 pathways in the hypodermis to coordinately control progenitor cell behav

132 pmental timing pathway that functions in the hypodermis to nonautonomously coordinate the mobilizatio

133 lts suggest that EGL-15 and CLR-1 act in the hypodermis to regulate fluid homeostasis in worms.

134 demonstrate that the IIS pathway acts in the hypodermis to regulate nutrition-responsive reactivation

135 Loss of lin-57 function causes the hypodermis to terminally differentiate and acquire adult

136 al link to transmit muscle forces across the hypodermis to the cuticle.

137 :mCherry fusion protein is released from the hypodermis to the surrounding matrices and fluids during

138 ing extracellular matrix and epithelium (the hypodermis) to the cuticle.

139 a an intervening basal lamina and epidermis (hypodermis), to the cuticle.

140 promote expression of the mlt-10 gene in the hypodermis whenever the exoskeleton is remade.

141 transcription is primarily activated in the hypodermis where it is also sufficient to drive sulfur a

142 and to induce vulval fates in the underlying hypodermis, whereas FBF, FOG-1, and FOG-3 control germ-l

143 2)S results in HIF-1 activity throughout the hypodermis, whereas hypoxia causes HIF-1 activity in the

144 ticed for hyp6, a syncytial component of the hypodermis, which indicated that the marker may serve as

145 the terminal differentiation of the lateral hypodermis, which occurs during the final (fourth) molt.

146 entiation in a stem cell-like lineage in the hypodermis, while human let-7 has been implicated in lun

147 ws a linear increase compared with muscle or hypodermis, with the lowest heme threshold in neurons.

148 l death from the middle of the dermis to the hypodermis within 2 h to 4 h after the initial injury, n

149 fusion construct is expressed throughout the hypodermis within the cells that abundantly produce the