1  cases had a consistent clinical picture and hypoglycorrhachia.

2                  The laboratory signature is hypoglycorrhachia.

3 velopmental delay, acquired microcephaly and hypoglycorrhachia.

4 quired microcephaly, spasticity, ataxia, and hypoglycorrhachia.

5 y more frequently than NMOSD (p < 0.05); CSF hypoglycorrhachia (11%, p = 0.25) and elevated angiotens

6                                              Hypoglycorrhachia and decreased erythrocyte 3-OMG uptake

7  disease or death were preterm birth, severe hypoglycorrhachia, CSF/blood glucose ratio <0.10, and mo

8  the glucose transporter GLUT1, resulting in hypoglycorrhachia, infantile seizures, and developmental

9 G), impaired motor activity, incoordination, hypoglycorrhachia, microencephaly, decreased brain gluco

10 vation were each seen in 87.5% of cases, and hypoglycorrhachia was present in 18.8% of cases.

11 vation were each seen in 87.5% of cases, and hypoglycorrhachia was present in 18.8% of cases.