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1 c nodules with subfoci of HCC, hyperintense, hypointense.
2 high-grade dysplastic nodules, hyperintense, hypointense.
3 c low-grade dysplastic nodules, hypointense, hypointense.
4  low-grade dysplastic nodules, hyperintense, hypointense.
5 version recovery hyperintense (18 of 18), T1-hypointense (17 of 18), and diffusion-hyperintense (15 o
6 /- 12, and 238 msec +/- 17, respectively) or hypointense (296 msec +/- 27, 163 msec +/- 12, and 199 m
7 isointense (8.5/s vs 9.5/s, p=0.02) and T(1) hypointense (7.7/s vs 9.5/s, p=0.003) compared with NAWM
8 66 from lesions on T2-weighted MR images (43 hypointense and 23 isointense on T1-weighted MR images),
9 lesions were classified in T1-isointense, T1-hypointense and black holes.
10 an enhance the separation between fibrous T2-hypointense and cellular T1-enhancing components.
11        Thermal ablation zones were uniformly hypointense and had a surrounding bright rim on T2-weigh
12                     On the other hand, it is hypointense and less evident in T1-weighted images.
13  inversion recovery (FLAIR) hyperintense, T1-hypointense, and appeared as perivascular demyelinated l
14                             In parallel, the hypointense areas on MR images at the injection sites de
15                              Within lesions, hypointense areas on phase images did not always represe
16                       We observed that these hypointense areas showed difference according to the gen
17 and at the external surface of the cortex, a hypointense band deeper in the cortex, and a hyperintens
18                                          OCT hypointense bands are a novel biomarker in RVOD indicati
19                              OCT angiography hypointense bands were detected in the superficial and d
20            When obtained on the same device, hypointense bands were thinner and more numerous at lowe
21  (eg, myometrial thinning, intraplacental T2-hypointense bands, uterine bulge, serosal hypervasculari
22 had less volume shrinkage and became more T1 hypointense between months 3 and 12.
23                   The UCCA and the number of hypointense brain lesions on T1-weighted images were the
24 r MR imaging parameters, including number of hypointense brain lesions on T1-weighted MR images, pres
25                              UCCA, number of hypointense brain lesions on T1-weighted MR images, pres
26 lacing surrounding structures, presenting as hypointense cable-like nerve bundles.
27 tion by effectively separating responding T2-hypointense-collagenized-mature components from potentia
28                         All lesions appeared hypointense compared to testicular tissue on T1W and T2W
29                                  Glands were hypointense compared with the liver on T1-weighted image
30  of 52 (73%) participants with NMOSDs had T1-hypointense cord lesions.
31 on of cord swelling, and decreased volume of hypointense core and edema at the last time points.
32  "Bagel Sign" pattern: a central lesion with hypointense core and hyperintense rim with or without co
33                               Over time this hypointense core reduced in size and in some animals was
34                  Also, at this time point, a hypointense core was identified on T1, PD, and T2 weight
35 ity, characteristic of edema, surrounded the hypointense core.
36                     The predictive values of hypointense cores (HIC) in balanced steady-state free pr
37                                              Hypointense cores within CMI on balanced steady-state fr
38 e lesions had lower ADCs compared with their hypointense counterparts.
39                                          The hypointense (dark) areas in T2-weighted images were not
40     MRI, owing to its ability to demonstrate hypointense endocyst, can act as a useful adjunct to cor
41  contrast than adjacent normal prostate, and hypointense features on T2-weighted imaging; these findi
42   Cerebral microbleeds (CMBs) are defined as hypointense foci visible on T2*-weighted and susceptible
43 e defined as discrete, well-defined markedly hypointense foci within the adnexal lesion on T2-weighte
44 mall HCC, hyperintense, hypointense (n = 7); hypointense, hyperintense (n = 2); hyperintense, hyperin
45 l 14 siderotic low-grade dysplastic nodules, hypointense, hypointense.
46 n T2-weighted images that showed significant hypointense in striatum region which close to the germin
47 in vascular tissue, and they are found to be hypointense in T1 sequences and hyperintense in T2 seque
48 asts agents behave as hyperintense in T2 and hypointense in T1.
49 ocardial hemorrhage was taken to represent a hypointense infarct core with a T2* value of <20 ms.
50 (n = 3); hyperintense, hyperintense (n = 1); hypointense, isointense (n = 1).
51  ganglion cell, and inner nuclear layer; the hypointense layer 2, the outer nuclear layer and the inn
52                        A BH was defined as a hypointense lesion on a T1 pre-contrast image that coinc
53 on volume (0.45% vs 13.00%, p<0.0001) and T1 hypointense lesion volume (36.68% vs 60.93%, p<0.0001).
54 tense lesion volume [T2LV]), the ratio of T1 hypointense lesion volume [T1LV] to T2LV [T1:T2]), brain
55 asures of chronic lesion activity such as T1 hypointense lesion volume accumulation and mean normaliz
56  lesion activity measured by longitudinal T1 hypointense lesion volume accumulation in new focal T2 l
57  lesion activity measured by longitudinal T1 hypointense lesion volume accumulation in slowly expandi
58 on that most of total brain non-enhancing T1 hypointense lesion volume accumulation was derived from
59 d tiw versus DT (nominal p<0.001); T2 and T1 hypointense lesion volume change was lower for sc IFN be
60 ional measures such as T2 hyperintense or T1 hypointense lesion volumes.
61 ively) lesions, except for the Cho value for hypointense lesion, which was significantly lower.
62 ional areas, T2 hyperintense lesions, and T1 hypointense lesions (38.1% +/- 2.6%, 45.0% +/- 2.6%, 51.
63  in T(1) isointense (44.6 +/- 7.2 mM) and T1 hypointense lesions (46.8 +/- 8.3 mM) compared with norm
64                            Mean NAA level in hypointense lesions (5.30 mmol/L +/- 2.27 [standard devi
65  (66.0%, p<0.001), annualised rate of new T1 hypointense lesions (62.8%, p<0.001) and CUA lesions per
66  of lateral ventricles, Dawson's fingers, T1 hypointense lesions (multiple sclerosis), fluffy lesions
67  lesions (all P<0.001) and new T(1)-weighted hypointense lesions (P<0.001, P<0.001, and P=0.002, resp
68 ns (both BG-12 doses), and new T(1)-weighted hypointense lesions (thrice-daily BG-12) (nominal P<0.05
69 adolinium-enhancing (Gd+) T1 lesions, new T1 hypointense lesions and combined unique active (CUA) les
70                           Binary masks of T1-hypointense lesions and lesion probability maps were pro
71 er sodium concentration was observed in T(1) hypointense lesions in secondary-progressive (49.0 +/- 7
72  characterize the spatial distribution of T1-hypointense lesions in the spinal cord at MRI, its assoc
73 ection of blood-brain barrier break down and hypointense lesions on T1-weighted images, magnetization
74 1 mL, p=0.024) and a higher number of new T1-hypointense lesions over 0-12 months (p=0.005) as well a
75 mbers of new T2, gadolinium-enhancing and T1 hypointense lesions were lower with sc IFN beta-1a qw (n
76  imaging with ultra-highfield MRI that phase hypointense lesions were significantly more prevalent in
77 radient-recalled-echo MR images demonstrated hypointense lesions with variable contrast material enha
78            Brain white matter T2 lesions, T1-hypointense lesions, cortical and cord lesions were iden
79 olved on repeat imaging, and often showed T1 hypointense lesions.
80  microbleeds, which appear as small dot-like hypointense lesions.
81             Each ligament was well seen as a hypointense linear structure with MR arthrography.
82 h the number of alternating hyperintense and hypointense lines depicted.
83 rast-to-noise ratio between hyperintense and hypointense liver regions, coefficient of variation, and
84 ic analysis; at MR imaging, PTLD appeared as hypointense masses on T1-and T2-weighted images with min
85 For both diseases, T1-weighted images showed hypointense masses with progressive enhancement (differe
86 pecific pattern of hyperintensity within the hypointense medial globus pallidus.
87 intense rim or detached internal T2-weighted hypointense membrane, a correct diagnosis of hydatid cys
88 ted images, respectively: HCC, hyperintense, hypointense (n = 3); hyperintense, hyperintense (n = 1);
89                     Small HCC, hyperintense, hypointense (n = 7); hypointense, hyperintense (n = 2);
90 nosis of acute RVOD and alternating bands of hypointense OCTA flow signal on en face projections.
91 hyperintense areas on T1-weighted images and hypointense on fat-suppressed T1-weighted images, compat
92 ntrifugal DCE lesions appeared isointense or hypointense on phase images, whereas centripetal DCE les
93                                     None was hypointense on proton-density- or T2-weighted images.
94  intrathoracic and subcutan masses as mainly hypointense on T1-weighted images and hyperintense on T2
95 te depiction of IRE ablation zones that were hypointense on T1-weighted images and hyperintense on T2
96 eatures were the following: all lesions were hypointense on T2- and hyperintense (n=12) and isointens
97               Infiltrative lesions that were hypointense on T2-weighted images were better characteri
98 lesions that shrink least and become more T1 hypointense over time suggests that the rim might mark f
99                        In acute lesions, the hypointense phase rim reflects the expanding inflammator
100 whereas centripetal DCE lesions showed thin, hypointense phase rims that clearly colocalized with the
101 ic lesions also selected for the presence of hypointense phase rims, the findings were stable over ti
102 , in cerebral white matter and brain stem, a hypointense region on T1-weighted images.
103                            MRIs demonstrated hypointense regions at the periphery of the tumors where
104 BBBD evident in contrast-enhanced MRI and to hypointense regions in T2*-weighted MRI.
105  oxide nanoparticles are detected in vivo as hypointense regions in the liver up to two weeks post in
106  ADC threshold, (ii) visual determination of hypointense regions on ADC maps, and (iii) visual determ
107 al microbleeds, observed as small, spherical hypointense regions on gradient echo (GRE) or susceptibi
108 ice that received labeled cells demonstrated hypointense regions within the tumor that evolved over t
109  Fibroids were classified as hyperintense or hypointense relative to skeletal muscle on pretreatment
110    APAs (mean size, 20 x 16 mm) were iso- or hypointense relative to the liver on T1-weighted images
111 r lesions located throughout the brain and a hypointense rim around some WM lesions.
112 nse pulmonary cystic lesion with T2-weighted hypointense rim or detached internal T2-weighted hypoint
113 nal, well-defined margin, lobulation, and/or hypointense rim, together with restricted diffusion and
114 signal, well-defined margin, lobulation, and hypointense rim, were detected in a higher proportion of
115 ed over time and developed a continuous dark hypointense ring at a consistent time point.
116 , fluid-fluid or air-fluid level, incomplete hypointense ring due to hemosiderin deposition, pseudotu
117               High-field MRI defined 2 novel hypointense signal abnormalities on T(2) -weighted image
118 elected for treatment had no hyperintense or hypointense signal intensity changes on the DW images or
119 sence of fluid collection with isointense or hypointense signal on T1-weighted images, fluid-equivale
120 ed by subcortical linear or arc lesions with hypointense signal on T1-weighted imaging and hyperinten
121                        Benign lesions showed hypointense signal on T2W and DWI.
122             The hypothesis that thin tubular hypointense signals in the myocardium of rat hearts at S
123 categorized into four subgrades: subgrade A, hypointense; subgrade B, inhomogeneous; subgrade C, hype
124 antified on a three-point ordinal scale (0 = hypointense to brain parenchyma, 1 = isointense to brain
125 ioma (hyperintense) from radiation necrosis (hypointense to isointense) by APT MRI.
126 ced more than metastases, they also remained hypointense to liver on T1-weighted images (from -4.87 +
127           In comparison, metastases remained hypointense to liver on T1-weighted images (from -5.77 +
128                   At MR imaging, tumors were hypointense to liver on T1-weighted images (n = 11) and
129 R images, but a central scar was depicted as hypointense to surrounding tumor in nine cases.
130                           All first HGs were hypointense to the MTG and were either iso- or hypointen
131                                  The STG was hypointense to the MTG in 54 (90%) hemispheres and in th
132 pointense to the MTG and were either iso- or hypointense to the STG.
133 eighted images ranged from 0.136 (moderately hypointense) to 0.529 (encapsulation).
134                                           T1 hypointense volume and signal intensity on T1-weighted M
135 sity and greater relative accumulation of T1 hypointense volume in slowly expanding/evolving lesions
136 -hyperintense) and rarefied or cystic (FLAIR-hypointense) white matter, and ventricular and extracere
137                     Thermal lesions appeared hypointense with hyperintense surrounding rims with all

 
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