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1 eridol-treated controls, which were markedly hypokinetic.
2  was similar to that of segments that became hypokinetic (1.06 +/- 0.72 versus 1.02 +/- 0.77 mL.g-1.m
3 ricular segment at peak stress, with normal, hypokinetic, akinetic, dyskinetic, and aneurysmal segmen
4                                         Most hypokinetic and 40% of akinetic/dyskinetic myocardial se
5 nce of surgical therapy for the treatment of hypokinetic and hyperkinetic movement disorders has, how
6 surgical treatment for medication-refractory hypokinetic and hyperkinetic movement disorders, and it
7 nternus (GPi) to treat medically intractable hypokinetic and hyperkinetic movement disorders, the cou
8  marrow transplantation partially attenuated hypokinetic and motor deficits in HD mice.
9          Accumulating evidence suggests that hypokinetic aspects of Parkinson's disease and their imp
10  activity in premotor areas in patients with hypokinetic catatonia.
11 of viable segments that recover to normal or hypokinetic contractility.
12 vel therapeutic strategies for patients with hypokinetic disorders, such as Parkinson's disease.
13  their role in movement "vigor," leading to "hypokinetic dysarthria" or "hypophonia." This is an addi
14 ular and circuit mechanisms that explain the hypokinetic features of Parkinson's disease.
15 signs that are specific for hyperkinetic and hypokinetic functional motor symptoms.
16  The aim of the study was to distinguish the hypokinetic gait disorder in idiopathic normal pressure
17                           In conclusion, the hypokinetic gait disorder in NPH can be quantitatively d
18 oregistered with contrast enhancement in the hypokinetic infarct on MRI.
19 mutant is sufficient to elicit age-dependent hypokinetic motor deficits and DA neuron loss in vivo, a
20 which is significantly correlated with their hypokinetic motor deficits.
21  exhibit multiple late-onset and progressive hypokinetic motor deficits.
22 d indirect pathways, have been linked to the hypokinetic motor output associated with the disease.
23                                          The hypokinetic motor symptoms of Parkinson's disease (PD) a
24 uency band, is believed to be related to the hypokinetic motor symptoms of the disorder.
25 kinsonism (n=4), or a mixed hyperkinetic and hypokinetic movement disorder (n=2).
26 rkinson's disease (PD) is the most prevalent hypokinetic movement disorder, and symptomatic PD pathog
27 ne with the emergence of hyperkinetic and/or hypokinetic movement disorder, cognitive deterioration a
28         Parkinson's disease, the most common hypokinetic movement disorder, has received much attenti
29 uthors describe rare, late Parkinsonism-like hypokinetic movement disorders (also known as movement a
30        The presence of both hyperkinetic and hypokinetic movement disorders such as ataxia, tremor, a
31 erebellar ataxia with concomitant hyper- and hypokinetic movement disorders, severe early-onset cereb
32  injected with autologous MSCs into akinetic/hypokinetic myocardial territories not receiving bypass
33 netic susceptibility changes associated with hypokinetic myocardial wall motion and microvascular obs
34  most common early DCM features, followed by hypokinetic nondilated cardiomyopathy (4%).
35  disease, isolated ventricular dilation, and hypokinetic nondilated cardiomyopathy; or phenotype-nega
36 n with established dilated cardiomyopathy or hypokinetic nondilated phenotype, which in the advanced
37                                     Severely hypokinetic or akinetic segments showed a 2.4-fold incre
38 triatal brain regions and show a paradoxical hypokinetic response to D1 agonist.
39 left ventricular ischemia, but a dilated and hypokinetic right ventricle suggested pulmonary patholog
40 re, combining dystonia and chorea, and (2) a hypokinetic score, combining bradykinesia and rigidity.
41  graded each LV segment as normal (score=1), hypokinetic (score=2), or akinetic (score=3).
42 tabolism was negatively correlated only with hypokinetic scores in the cuneus (z score = 3.95, P < .0
43 as only limited by false positive results in hypokinetic segments and not by the test interval or cli
44 obal longitudinal strain and the presence of hypokinetic segments in placebo-treated GRMD dogs.
45 criteria for predicting recovery of severely hypokinetic segments on preoperative imaging were tracer
46 .001) due to false positive results (16%) in hypokinetic segments.
47 A) receptor antagonists could ameliorate the hypokinetic symptoms of advanced Parkinson's disease pat
48 e in alleviating Parkinson's disease-related hypokinetic symptoms, and sensing-enabled neurostimulato
49 efect conceivably has a role in parkinsonian hypokinetic symptoms.
50 oped atypical parkinsonism, and two a severe hypokinetic syndrome with a tendency to eye closure and
51 ta- and gamma1-synucleins were more severely hypokinetic than animals lacking one or the other synucl