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1 ere normometabolic, and 21% of subjects were hypometabolic.
2 tabolic rate and body temperature (Tb), is a hypometabolic adaptation to stressful environmental cond
3 ic, normometabolic, or at the border between hypometabolic and normal cortex (metabolic "border zones
4 lization of epileptogenic brain regions, but hypometabolic areas are often larger than or can miss ep
5 s performed to evaluate the role of severely hypometabolic areas.
6                   In contrast, m-/+ mice are hypometabolic at ambient temperature and eumetabolic at
7                  We conclude that there is a hypometabolic component of the rat brain adaptation to c
8 pamine nerve terminal function under extreme hypometabolic conditions, but loss of this dopamine-deri
9 tive adaptation that may indicate a cerebral hypometabolic conservation of energy at altitude and/or
10 to 0.967 (metaROI), and from 0.801 to 0.983 (hypometabolic convergence index).
11  Alzheimer discrimination analysis tool, the hypometabolic convergence index, and a set of meta-analy
12                              This feature of hypometabolic cortex is remarkably similar to that of st
13 e more heterogeneous: there were significant hypometabolic effects in posterior cingulate, precuneus,
14 urviving anoxia (zero oxygen) for hours in a hypometabolic, energy-conserving state.
15                    Our data suggest that the hypometabolic epileptogenic focus seen in [18F]FDG-PET s
16 s of interest (ROIs), which included (1) the hypometabolic epileptogenic regions and (2) the homologo
17               The findings differ from prior hypometabolic findings in ME/CFS plasma.
18    On the autonomic side, Trpv1 KO mice were hypometabolic (had a lower oxygen consumption) and hyper
19             Torpor is a naturally occurring, hypometabolic, hypothermic state engaged by a wide range
20  lasts for 5-7 months and comprises weeks of hypometabolic, hypothermic torpor interspersed with 24-4
21              Physiological trade-offs led to hypometabolic-hypothermic responses due to LPS, but not
22  renal cell tumor embolism that hemorrhaged (hypometabolic lesion).
23 ities in 19 of 22 intracranial metastases, 2 hypometabolic lesions, and 1 renal cell tumor embolism t
24 ied with their metabolic phenotype: The most hypometabolic line [exhibiting low levels of oxidative p
25 entia, although our findings imply posterior hypometabolic mismatch is related more to S than vascula
26                         Following up on this hypometabolic mismatch with CSF metabolite and proteome
27 s over cortical areas, which were defined as hypometabolic, normometabolic, or at the border between
28  patients, the temporal lobe (which appeared hypometabolic on PET) was determined to be the site of t
29 ones significantly more frequently than over hypometabolic or normometabolic regions.
30 l background, G93A SOD1 mice become markedly hypometabolic, overweight and less active.
31          Hypermetabolic, normometabolic, and hypometabolic patients were identified with the use of B
32 ive decline, but did not affect the regional hypometabolic pattern or cognitive profile.
33  Compound B positron emission tomography, AD hypometabolic pattern shown on fludeoxyglucose F 18 posi
34        In this analysis, the only persisting hypometabolic region was that centred over the left ante
35                     Cortical areas bordering hypometabolic regions can be highly epileptogenic and sh
36                                          The hypometabolic regions in each patient were identified wi
37 equently overlay metabolic border zones than hypometabolic regions.
38 e the threshold to multiple depolarizing and hypometabolic spreading depolarization triggers.
39 the thought that humans can undergo a severe hypometabolic state analogous to hibernation borders on
40                                         This hypometabolic state is manifested by lowered neuronal gl
41 body temperature and enter a hypothermic and hypometabolic state known as torpor.
42 res and food shortage by actively entering a hypometabolic state known as torpor.
43                                  Despite the hypometabolic state of these cells, DNP-MRSI of 1-(13) C
44                                          The hypometabolic state of torpor is a widely utilized and w
45 restriction produces a transient hypothyroid-hypometabolic state that normalizes on return to energy-
46                     In addition to a general hypometabolic state, the dorsoventral difference disappe
47 s treatment with H2S can induce a reversible hypometabolic state, which can protect organs from ische
48 nsform an organ or a cellular network into a hypometabolic state, without loss of functional integrit
49 nerate adenosine triphosphate, and a general hypometabolic state.
50 l cell bodies and axons leading to a chronic hypometabolic state.
51 ntry and recovery from the energy-conserving hypometabolic state.
52 colysis was also low, thereby establishing a hypometabolic state.
53 flammation, energy metabolic deficits, and a hypometabolic state.
54 cal and mechanistic aspects into how various hypometabolic states are initiated in mammals are poorly
55 cuitry that regulate extreme hypothermic and hypometabolic states, and enables genetic access to moni
56  our findings suggest that energy-conserving hypometabolic states, such as dormancy, might have evolv
57                 During bacterial infections, hypometabolic states, which could be elicited by competi
58  in ion leakage across cell membranes during hypometabolic states-has highlighted the energetic benef
59 etabolites were decreased, consistent with a hypometabolic syndrome.
60 hmia of the thalamocortical loop, leading to hypometabolic theta activity in the MGB.
61 ) on the evolution of the volume of severely hypometabolic tissue in anaesthetized baboons and compar
62                                 The severely hypometabolic tissue volume, as defined by a threshold o
63 role in the induction of the hypothermic and hypometabolic torpid state in hibernating mammals, we in
64 ke (n = 189) and 85% of lesions with iso- or hypometabolic uptake (n = 33) were correctly identified
65                          Moreover, the final hypometabolic volume where cerebral metabolic rate of ox
66 elationship between histological and chronic hypometabolic volumes (r = 0.84; p < 0.001).
67                               However, these hypometabolic volumes regressed in the chronic stage (p
68                    E2m-/+ mice are obese and hypometabolic, whereas E2+/p- mice are lean and hypermet
69 mentia did not extend caudal to the atrophic/hypometabolic zone.