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1 Asthmatics are known to have esophageal hypomotility.
2 disorders, spastic disorders, and esophageal hypomotility.
3 lvement, and less frequent distal esophageal hypomotility.
4 ra-esophageal acidification cause esophageal hypomotility.
6 an important role in the pathogenesis of gut hypomotility and are critical for the production of the
8 ss of Capa/CapaR signaling causes intestinal hypomotility and impaired nutrient absorption, which gra
9 the role of slow wave disruption in inducing hypomotility and the prostaglandin dependence of dysrhyt
10 estimates for ileus (severe gastrointestinal hypomotility) and pneumonia were 5.3% and 29.5%, respect
12 d by a single endocannabinoid pathway (e.g., hypomotility by the 2-AG/MAGL pathway) and, interestingl
13 nduced acute liver failure, gastrointestinal hypomotility, constipation, diarrhea, gastrointestinal b
14 nduced acute liver failure, gastrointestinal hypomotility, constipation, diarrhea, gastrointestinal b
16 ues 1-205) develop profound gastrointestinal hypomotility, dilated pupils with impaired light respons
17 well as Raynaud's phenomenon and esophageal hypomotility diminished, while pulmonary hypertension, p
19 ic fibrosis, dry mouth, and gastrointestinal hypomotility disorders, and for pharmacological dissecti
21 e of intestinal smooth muscle cells produced hypomotility from signal-transduction decoupling of chol
27 ty (corpus and fundus dysmotility and antral hypomotility), pyloric resistance to gastric outflow (py
28 to five disease categories: gastrointestinal hypomotility, seizures, pneumonia, other acute respirato