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1 nide, when simultaneously added with EtOH to hyponatremic buffer, failed to reverse the EtOH-induced
2               Both groups were studied under hyponatremic conditions and/or with the addition of p38(
3 otonic media (112 mM NaCl, mimicking in vivo hyponatremic conditions associated with EtOH withdrawal)
4                  One patient had unsuspected hyponatremic encephalopathy and died of cardiopulmonary
5                        Prompt recognition of hyponatremic encephalopathy and early treatment with hyp
6 eview of articles involving risk factors for hyponatremic encephalopathy and treatment of hyponatremi
7 emia with bolus hypertonic saline to reverse hyponatremic encephalopathy by increasing the serum sodi
8                             In patients with hyponatremic encephalopathy exhibiting neurologic manife
9 hyponatremic encephalopathy and treatment of hyponatremic encephalopathy in critical illness.
10                                              Hyponatremic encephalopathy is a frequently encountered
11                                              Hyponatremic encephalopathy, symptomatic cerebral edema
12 sure and can be the initial manifestation of hyponatremic encephalopathy.
13 genic pulmonary edema can be associated with hyponatremic encephalopathy.
14              Here, we describe a 3-month-old hyponatremic infant found to have a phenylalanine 229 to
15 iuretic hormone levels increased more in the hyponatremic patients (P < .02), despite their consisten
16                                           As hyponatremic patients have elevated circulating arginine
17                        Optimal management of hyponatremic patients involves weighing the risk for ill
18 and edema and normalized serum sodium in the hyponatremic patients.
19  actions of Avpr inhibitors used commonly in hyponatremic patients.
20                   The water-loaded rats were hyponatremic (plasma Na+, 98 to 122 mmol/L) and manifest
21 , chronically hypernatremic, and chronically hyponatremic rats received pituitary stalk compression (
22                                  However, in hyponatremic rats SC caused degeneration of 97% of the A
23                    In both normonatremic and hyponatremic rats, infusion of hypertonic saline without
24                                           In hyponatremic rats, oxytocin and vasopressin cells also r
25                                 In contrast, hyponatremic SC rats exhibited peak water intakes of 600