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1 ecific function in addition to their role in hypoosmotic adaptation.
2 igher growth rates and were more tolerant of hypoosmotic and high-temperature stress than the wild ty
3  as safety valves preventing cell lysis upon hypoosmotic cell swelling: the channels open under membr
4                                        After hypoosmotic challenge, OHCs shortened and their diameter
5 discontinuity, exhibits tolerance to extreme hypoosmotic challenge, whereas populations native to bra
6 ng the cell swelling that occurs following a hypoosmotic challenge.
7 hand, a small decrease in TonEBP level under hypoosmotic condition was attenuated by Ift88 or Kif3a k
8 Osm/kg) induce chromatin condensation, while hypoosmotic conditions (100 mOsm/kg) cause decondensatio
9                                              Hypoosmotic conditions activate volume-regulated anion c
10 ine released from pituicytes under basal and hypoosmotic conditions may act to suppress axon terminal
11 ndings are as follows: brain edema mimicking hypoosmotic conditions stimulates the formation of new O
12 ts, including poor growth and motility under hypoosmotic conditions, and the inability to invade plan
13 ncreased in the pituitary of juveniles under hypoosmotic conditions, and treatment with PRL-L blocked
14                                           In hypoosmotic conditions, which elicit cell edema, OAP for
15 ory volume decrease in astrocytes exposed to hypoosmotic conditions.
16 f different structural classes as well as by hypoosmotic conditions.
17 es through the channel opening under extreme hypoosmotic conditions.
18           The adaptation of rhizobacteria to hypoosmotic environments is also examined in the present
19 ->3),beta-(1-->6)-D-glucans during growth in hypoosmotic environments, and evidence is growing that t
20 erence toxicants was tested in isosmotic and hypoosmotic exposure media in RTgill-W1 cells.
21                                        Thus, hypoosmotic exposure medium should be considered for aqu
22 s in high-water-permeability membranes under hypoosmotic gradients of different magnitude, as well as
23 mutant strain was only slightly sensitive to hypoosmotic growth conditions compared with the ndvB mut
24  radiotracer assays in cells challenged with hypoosmotic medium (30% reduction in osmolarity).
25                                 Exposures in hypoosmotic medium significantly increased sensitivity o
26  These data suggest that volume expansion by hypoosmotic medium stimulates movement of skAE1 from an
27 ions of taurine significantly decreased with hypoosmotic perfusion, while glutamate, glutamine, and G
28             Conversely, induced expansion by hypoosmotic pressure accelerates osteogenesis.
29 rent chondrocyte membrane tension, including hypoosmotic prestrain, high compression magnitudes, and
30 ated with acute and acclimatory responses to hypoosmotic shock and posits unique mechanisms that enab
31 annels prevent cells from lysing upon sudden hypoosmotic shock by opening and releasing solutes and w
32 s environment in the presence and absence of hypoosmotic shock by reacting a charged sulfhydryl reage
33 ortic and bovine retinal ECs were exposed to hypoosmotic shock for 2 minutes, were allowed to recover
34 n channel required for pollen to survive the hypoosmotic shock of rehydration and for full male ferti
35   When Escherichia coli cells are subject to hypoosmotic shock they are subject to substantial flows
36 hat limits periplasmic volume increase under hypoosmotic shock, allowing osmotic pressure build-up in
37 "cell integrity" MAPK pathway by heat shock, hypoosmotic shock, and actin perturbation, and we report
38       Furthermore, in response to hyper- and hypoosmotic shock, E. coli cells resumed their preshock
39 owed to react, in the presence or absence of hypoosmotic shock, with cells expressing mechanosensitiv
40 this hypopeak increased with the size of the hypoosmotic shock, with increased water flow.
41                          As a consequence of hypoosmotic shock, yeast cells swell rapidly and increas
42  protect cells from structural damage during hypoosmotic shock.
43 in the protection of bacterial cells against hypoosmotic shock.
44 in the protection of bacterial cells against hypoosmotic shock.
45 ol uptake and glycerol efflux in response to hypoosmotic shock.
46 r role in releasing the pressure built up by hypoosmotic shock.
47 bulin-disrupting nocodazole, and cells under hypoosmotic shock.
48 conductance (MscS) protects bacteria against hypoosmotic shock.
49  response, tension and volume recovered from hypoosmotic shocks but not from hyperosmotic shocks.
50 uctural mutations in the envelope respond to hypoosmotic shocks.
51 ic properties of OHCs, we exposed cells to a hypoosmotic solution for varying durations and then punc
52 are sensitive to prolonged exposure to hyper/hypoosmotic solutions, temperature changes, mechanical m
53 tography analyses demonstrated that in vitro hypoosmotic stimulation (reduction of 40 mOsm/kg) of iso
54  increase in maximal cell volume elicited by hypoosmotic stimulation was significantly smaller in AQP
55 f the TonEBP target genes were responsive to hypoosmotic stimulus in control and Ift88 or Kif3a knock
56 ing in vivo two-photon imaging, we show that hypoosmotic stress (20% reduction in osmolarity) initiat
57 ective in normoxic slices (400 microm) after hypoosmotic stress altered the ECS to mimic ischemia.
58                          Channel gating upon hypoosmotic stress and the role of lipids in this proces
59 vis Wilson clone, cells responded quickly to hypoosmotic stress by increasing their brevetoxin cell q
60          We conclude that plastids are under hypoosmotic stress during normal plant growth and dynami
61 se two alpha1 variants were less tolerant of hypoosmotic stress than the wild type and produced CPs w
62  (<1 pg per cell), was unable to balance the hypoosmotic stress, and although brevetoxin production r
63 e of urea conduction in UT-B is increased by hypoosmotic stress, and that the site of osmoregulation
64  of the cell were significantly decreased by hypoosmotic stress, but were unchanged by hyperosmotic s
65 oxicity were similar to those induced by the hypoosmotic stress, but, in contrast to the latter, they
66 d MSL3 contain leaf epidermal plastids under hypoosmotic stress, even during normal growth and develo
67 -regulate mitochondrial genes in response to hypoosmotic stress.
68 1 could regulate their volume in response to hypoosmotic stress.
69 se valve" that protects bacterial cells from hypoosmotic stress.
70 elief valve", protecting bacteria from acute hypoosmotic stress.
71 e valve," allowing bacteria to survive acute hypoosmotic stress.
72  remodeling of cortical actin in response to hypoosmotic stress.
73                                              Hypoosmotic swelling markedly reduced microvilli number
74 nal genomic basis of population variation in hypoosmotic tolerance.
75                                      After a hypoosmotic transfer, there was an additional effect: a
76                                        After hypoosmotic treatment of isolated mitochondria, mitochon
77  the Arp2/3-depleted cells can be rescued by hypoosmotic treatment.
78                 Cellular volume expansion by hypoosmotic volume expansion but not volume expansion by
79                                         Upon hypoosmotic volume expansion nearly half of the skAE1 is